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1  The highest ash content was 3.9% (melon and peach).
2 tent varied from 3% (Surinam cherry) to 39% (peach).
3 ulatory networks for fruit quality traits in peach.
4 ved clinical association between cypress and peach.
5 .3% of GB positive patients were positive to peach.
6 scale post-harvest physiological disorder in peach.
7  to environmental stresses in flower buds of peach.
8  and sorbitol accumulation in flower buds of peach.
9  tool to assist fruit quality improvement in peach.
10 hrimp, peanut, hazelnut, celeriac, apple and peach.
11 ergic subjects that tolerate both peanut and peach.
12 us Prunus, including cherries, apricots, and peaches.
13 o a dwarf phenotype similar to that of dw/dw peaches.
14 and the most common were to hazelnut (9.3%), peach (7.9%) and apple (6.5%).
15 nut allergy and tolerance to peach; Group 3, peach allergy and tolerance to peanut; Group 4, nonaller
16                                              Peach allergy is common too in Southern Europe.
17  Mediterranean patients: Group 1, peanut and peach allergy; Group 2, peanut allergy and tolerance to
18        Fruits of 'Suncrest' and 'Babygold 7' peach and 'Big Top' nectarine cultivars were irradiated
19 r, pepper, spinach, zucchini, grape, cherry, peach and apricot.
20 eta-cryptoxanthin and beta-carotene in kaki, peach and apricot.
21  the DORMANCY ASSOCIATED MADS-BOX genes from peach and hypothesize that it may play a direct role in
22 d carrying the nonautonomous mariner element peach and mRNA encoding the transposase.
23 yphenolic extracts and fractions of selected peach and plum genotypes were evaluated for cell viabili
24  created from cold-acclimated bark tissue of peach and selectively probed using an antibody directed
25             Whole genome sequences of apple, peach and strawberry are available to browse or download
26 y of enhancing phenolic compound contents in peaches and nectarines by post-harvest irradiation with
27 cal Exercise and Appetite in CHildren Study (PEACHES) and repeated by 113 mothers 3 y later.
28 sibling deciduous and evergreen genotypes of peach, and also inducible by water deficit in cv. Rio Os
29 strongly correlated with IgE against walnut, peach, and apple and against Chenopodium, plane tree, an
30 commercialized in Brazil (strawberry, mango, peach, and orange) were analyzed and indicated the absen
31                                       Apple, peach, and pear as non-citrus fruit fibres were examined
32  (eg, birch and alder) and foods (eg, apple, peach, and soy).
33                                    The green peach aphid (GPA) (Myzus persicae Sulzer) is an importan
34                                        Green peach aphid (GPA) Myzus persicae (Sulzer) is a phloem-fe
35 persicae Sulzer, commonly known as the green peach aphid (GPA), which is an important phloem sap-cons
36 rsicae (Sulzer), commonly known as the green peach aphid (GPA).
37 Arabidopsis (Arabidopsis thaliana) and green peach aphid (GPA; Myzus persicae Sulzer), we found that
38 PAD4) is essential for defense against green peach aphid (GPA; Myzus persicae) and the pathogens Pseu
39                         Extract of the green peach aphid (GPA; Myzus persicae) triggers responses cha
40 ana shoots by the phloem sap-consuming green peach aphid (GPA; Myzus persicae), an agronomically impo
41 globally important economic pest - the green peach aphid (Myzus persicae) - growing on 34 plant taxa,
42 8 also show enhanced resistance to the green peach aphid (Myzus persicae) compared with wild-type con
43 se pathways was investigated following green peach aphid (Myzus persicae) feeding on Arabidopsis.
44 ays with the generalist phloem-feeding green peach aphid (Myzus persicae), and in weight-gain assays
45 se to feeding by its aphid vector, the green peach aphid (Myzus persicae), and increases aphid fecund
46 w (Hyaloperonospora arabidopsidis) and green peach aphid (Myzus persicae), but retained susceptibilit
47 oEL displayed reduced fecundity of the green peach aphid (Myzus persicae), indicating enhanced resist
48 is gap we exposed four naive clones of green peach aphid (Myzus persicae), maintained on the model cr
49 e against a phloem-feeding insect, the green peach aphid (Myzus persicae).
50 gated in Arabidopsis infested with the green peach aphid (Myzus persicae).
51  system, also reduced potato aphid and green peach aphid fecundity, respectively.
52 in host plant physiology and increased green peach aphid reproduction.
53 the alarm pheromone in Myzus persicae (green peach aphid) and many other aphid species.
54                   When Myzus persicae (green peach aphid) feeds on Arabidopsis aliphatic glucosinolat
55 ial diet both decrease Myzus persicae (green peach aphid) reproduction, suggesting a direct toxic or
56                        Remarkably, the green peach aphid, Myzus persicae, colonises plant species acr
57 eding herbivore-host plant system: the green peach aphid, Myzus persicae, feeding on multiple brassic
58 ea aphid, Acyrthosiphon pisum, and the green peach aphid, Myzus persicae.
59 ; parasitoid Aphidius colemani) of the green peach aphid, Myzus persicae.
60 ssion of Arabidopsis resistance to the green peach aphid.
61                               Although green peach aphids (Myzus persicae) are able to avoid most con
62      However, settling and survival of green peach aphids (Myzus persicae) were not affected.
63 sis (cotton mealybug), Myzus persicae (green peach aphids) and Bemisia tabaci (silver leaf whitefly).
64  with this hypothesis, Myzus persicae (green peach aphids) prefer to settle on Nicotiana benthamiana
65  context, the syntenic regions identified in peach, apple and strawberry might be useful to interpret
66               Carotenoids in orange, cherry, peach, apple, and kale were stable (except alpha-caroten
67 characterised by floral and fruity flavours (peach/apricot, Muscat, melon, banana and strawberry) whi
68 hod to small RNA sequence data from soybean, peach, Arabidopsis and rice and provide experimental val
69  gene for the br mutation in Prunus persica (peach) associated with vertically oriented growth of bra
70                 In the extrafloral nectar of peach, B was present as a mixture of sorbitol-B-sorbitol
71                                              Peach-based intensification was pronounced (300-400%) an
72  It is demonstrated that LTPs from wheat and peach bind a range of lipids in a variety of conditions,
73           Fruit quality traits are essential peach breeding program objectives since they determine c
74                             Fractionation of peach BY00P6653 extracts gave 4 fractions, with fraction
75 enbuconazole, propiconazole, or pyridaben in peaches; carbendazim, imazalil, terbutryn, and thiabenda
76    Since matrix effects were observed in the peach commodity, organic acids were quantified by the st
77  chains were more flexible in simulations of peach compared with barley LTP1.
78 mediate sorbitol synthesis in flower buds of peach concomitantly with specific chromatin modification
79                                    Pulp from peaches contained polygalacturonic acid and arabinogalac
80 677 and weaker Penta rootstock on 'Rich May' peach cultivar.
81 tionship between susceptibility of different peach cultivars (cvs) to the Mediterranean fruit fly (me
82               The volatile compounds of four peach cultivars (Prunus persica L.) were studied: Sudane
83 esults indicate that the phenolic extract of peach cultivars inhibits Abeta and alphaS fibril formati
84 ce' (850 h CR) and 'Springprince' (650 h CR) peach cultivars through winter 2008-2009.
85                                      We used peach cultivars with contrasting chilling requirements (
86 it quality, particularly for southern Europe peach cultivation conditions.
87 al dehydrins (V. riparia YSK2, 60 kilodalton peach dehydrin [PCA60], barley dehydrin5 [Dhn5], Thellun
88                                          The peach dehydrin gene encodes 472 amino acids with a predi
89                              The capacity of peach DMSO extracts to inhibit Candida albicans growth w
90                 Here, we report eight fossil peach endocarps from late Pliocene strata of Kunming Cit
91          The fossils are identical to modern peach endocarps, including size comparable to smaller mo
92 ow that China has been a critical region for peach evolution since long before human presence, much l
93                                              Peaches evolved their modern morphology under natural se
94 he enzymatic browning in minimally processed peaches for 8 days of storage.
95 ble (except alpha-carotene and zeaxanthin in peach) for 13, 9.7, 5.7, 2.5 and 7.5months, respectively
96 sfully applied to the analysis of samples of peach from two cultivars.
97                         Several cultivars of peach fruit (Prunus persica L.) were investigated.
98 o analyse 238 kaki, cashew apple, guava, and peach fruit and pulp samples, which were also analysed f
99        MPG1 and MPG2 were closely related to peach fruit and tomato abscission zone PGs, and MPG3 was
100                        Here we described the peach fruit as a system to link the phenotype of a slow
101 roperties, such as sweetness and acidity, in peach fruit by mid infrared spectroscopy is of interest
102 ic content and the antioxidant capacities of peach fruit extracts was found, indicating that phenolic
103 od for the determination of organic acids in peach fruit has been developed.
104 me that gluconic acid has been determined in peach fruit.
105 o increase the health-promoting potential of peach fruits and indirectly to ameliorate the aesthetic
106               Here, chemical biodiversity of peach fruits from fifteen varieties, at harvest and afte
107 -FTIR) was tested here on two populations of peach fruits issued from contrasting genitors providing
108                                              Peach fruits subjected to prolonged cold storage (CS) to
109 82, annotated as hypothetical protein in the peach genome sequence, was identified as a candidate gen
110 no acid sequence of LTP was identical in all peach genotypes but, for the first time, peel LTP was fo
111 gy; Group 2, peanut allergy and tolerance to peach; Group 3, peach allergy and tolerance to peanut; G
112                  The oldest evidence for the peach has been Chinese archaeological records dating to
113 p 3 in vivo, a mouse model of anaphylaxis to peach has been produced and changes in the humoral and b
114 Allergic cross-reactions between cypress and peach have been reported, including an oral allergy synd
115                             Among these, the peach HEC3-like gene FLESHY showed a strongly altered ex
116 on is very bright but shows subtle yellow to peach hues which probably arise from the production of c
117                                              Peach is widely thought to have origins in China, but it
118                                              Peach juices of distinct varieties, namely yellow- and r
119 sed analysis (PCA) proved useful to classify peach juices on the basis of variety.
120                      IgE-immunoblotting with peach leaf extract revealed in six patient sera a pair o
121                                The SBCT with peach leaf extract was positive in the asthmatic sensiti
122 pecific bronchial challenge test (SBCT) with peach leaf extract.
123         Those bands could be two isoforms of peach leaf lipid transfer proteins( LTP), so the recogni
124 nation of As, Cd, Hg and Pb in NIST SRM 1547 peach leaves and SRM 1573a tomato leaves reference mater
125 n NIST standard reference materials SRM 1547 Peach Leaves and SRM 1573a Tomato Leaves.
126                             Sensitization to peach leaves was the cause of occupational respiratory s
127 dard reference materials for polluted water, peach leaves, and tomato leaves.
128                      The allergen Pru p 3, a peach lipid transfer protein, has been well studied.
129                                       There, peach LTP (Pru p 3) seems to be the primary sensitizer,
130                                              Peach LTP was initially cleaved at Tyr79-Lys80 and then
131 onditions do not disrupt the 3D structure of peach LTP, explaining why LTPs retain their ability to b
132                      KEY MESSAGE: Pru p 3, a peach LTP, is located in pollinated flower styles and se
133  cross-reactive allergen between cypress and peach might be responsible for the observed clinical ass
134 our physical and eight sensory properties of peach nectar were explored using the best-fit multiple l
135 d with aroma active compounds of PEF-treated peach nectar.
136 itized to peach were mainly positive for the peach-nonspecific lipid-transfer protein.
137 immunoblot inhibition using sera specific to peach or pellitory pollen.
138 h oranges (OR = 0.18; 95% CI: 0.06-0.51) and peaches (OR = 0.30; 95% CI: 0.13-0.67) had a decreased o
139 TP being more resistant to cleavage than its peach ortholog.
140  (P = .023), fresh oranges (P = .002), fresh peaches (P = .002), and collard greens/kale (P = .014).
141 rotenoid extracts from two Amazonian fruits, peach palm (7.83+/-0.21) and mamey (6.90+/-0.44).
142 portant family which includes apple, cherry, peach, pear, raspberry, rose and strawberry.
143                                          The peach physical map can be viewed using WebFPC/WebChrom,
144 able Rosaceae ESTs, the genetically anchored peach physical map, Rosaceae genetic maps and comprehens
145 mes, the children were offered fruit (apple, peach, pineapple, or all 3 types).
146  This suggests a new biological activity for peach polysaccharides.
147                                          The peach potato aphid, Myzus persicae, is one of the most i
148                       Behavioural studies on peach-potato aphids showed that a reduced response to al
149 pression of the two GID1-like genes found in peach, PpeGID1c and PpeGID1b, was analyzed.
150                           We have shown that peach PpeS6PDH gene is down-regulated in flower buds aft
151 se data define potential for improvements to peach production efficiency and fruit quality, particula
152 luate the allergenic properties of LTPs from peach (Pru p 3) and pellitory (Par j 1/Par j 2), major f
153  caused by lipid transfer protein (LTP) from peach (Pru p 3) is frequently associated with sensitizat
154 al of 1.8 cM corresponding to ~364 Kb in the peach (Prunus persica L. Batsch) genome.
155 ivated in Tunisia: kaki (Diospyros kaki L.), peach (Prunus persica L.) and apricot (Prunus armeniaca
156 graveolens L.) and the extrafloral nectar of peach (Prunus persica L.).
157                                              Peach (Prunus persica) fruits from different varieties d
158 uence-based genotyping, and the high-quality peach (Prunus persica) genome reference sequence for sin
159 a recessive brachytic dwarfism trait (dw) in peach (Prunus persica) that has little or no effect on f
160 tein (LTP, Pru p 3) is the major allergen of peach (Prunus persica), and is in a greater abundance in
161 30% similar to an endopolygalacturonase from peach (Prunus persica).
162                                              Peach (Prunus persica, Rosaceae) is an extremely popular
163 esentative crops such as apple (Malus spp.), peach (Prunus spp.), and strawberry (Fragaria spp.).
164 ADS-box transcription factors (DAM genes) in peach [Prunus persica (L.) Batsch] as potential candidat
165 and nutritional homogeneity and quality with peach [Prunus persica (L.) Batsch].
166 d ripening stages in three climacteric (i.e. peach [Prunus persica] and two tomato [Solanum lycopersi
167 , blueberries, sweet cherries, table grapes, peaches, raspberries, and strawberries) in a postharvest
168 n blot analysis, indicating transposition of peach rather than random integration of the plasmid DNA.
169 wing two patterns: patients also allergic to peach, responding to Ara h 2 and Pru p 3, and patients a
170  in the extraction of pesticides from canned peach samples.
171  of this region, BES were mapped against the peach scaffold_3 and BACs were anchored to the apricot m
172 evelopment, DRO1 homologs in Arabidopsis and peach showed root-specific expression.
173                                  Much later, peach size and variety increased through domestication a
174 h event in the southwestern USA, the 18.8 Ma Peach Spring Tuff, were formed by pyroclastic flows that
175 ers of the dehydrin gene family may exist in peach that vary in their relation to ppdhn1.
176 reared to adulthood, and the transmission of peach to the F1 generation was tested by PCR.
177                          Specific IgE to GB, peach, tomato and nut-mix was measured.
178 upational respiratory diseases in workers of peach tree crops have been reported punctually and have
179 ed with extracts from leaves and branches of peach tree.
180         Most patients suffered symptoms when peach trees had leaves, specifically during thinning and
181 symptoms related to occupational exposure to peach trees.
182  these SNPs on downstream products from the 'peach v1.0' genome sequence was carried out.
183 rification of LTP from peel and pulp of four peach varieties [Gladys (white flesh), California (necta
184                                              Peach varieties clustered into four groups: two groups o
185  nonspecific lipid transfer protein (LTP) of peach were compared with the homologous LTP1 of barley a
186                       Patients sensitized to peach were mainly positive for the peach-nonspecific lip
187 rst shown to directly impact endodormancy in peach where a deletion of a series of DAM resulted in lo
188 r two highly homologous genes are present in peach, whereas an additional member was detected under l
189      Robustness was demonstrated using fresh peaches, which provided recovery values within acceptabl
190 rases encoded by mycobacteriophages Bxz2 and Peaches with unusual and unpredictable specificities.

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