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1 The highest ash content was 3.9% (melon and peach).
2 tent varied from 3% (Surinam cherry) to 39% (peach).
3 ulatory networks for fruit quality traits in peach.
4 ved clinical association between cypress and peach.
5 .3% of GB positive patients were positive to peach.
6 scale post-harvest physiological disorder in peach.
7 to environmental stresses in flower buds of peach.
8 and sorbitol accumulation in flower buds of peach.
9 tool to assist fruit quality improvement in peach.
10 hrimp, peanut, hazelnut, celeriac, apple and peach.
11 ergic subjects that tolerate both peanut and peach.
12 us Prunus, including cherries, apricots, and peaches.
13 o a dwarf phenotype similar to that of dw/dw peaches.
15 nut allergy and tolerance to peach; Group 3, peach allergy and tolerance to peanut; Group 4, nonaller
17 Mediterranean patients: Group 1, peanut and peach allergy; Group 2, peanut allergy and tolerance to
21 the DORMANCY ASSOCIATED MADS-BOX genes from peach and hypothesize that it may play a direct role in
23 yphenolic extracts and fractions of selected peach and plum genotypes were evaluated for cell viabili
24 created from cold-acclimated bark tissue of peach and selectively probed using an antibody directed
26 y of enhancing phenolic compound contents in peaches and nectarines by post-harvest irradiation with
28 sibling deciduous and evergreen genotypes of peach, and also inducible by water deficit in cv. Rio Os
29 strongly correlated with IgE against walnut, peach, and apple and against Chenopodium, plane tree, an
30 commercialized in Brazil (strawberry, mango, peach, and orange) were analyzed and indicated the absen
35 persicae Sulzer, commonly known as the green peach aphid (GPA), which is an important phloem sap-cons
37 Arabidopsis (Arabidopsis thaliana) and green peach aphid (GPA; Myzus persicae Sulzer), we found that
38 PAD4) is essential for defense against green peach aphid (GPA; Myzus persicae) and the pathogens Pseu
40 ana shoots by the phloem sap-consuming green peach aphid (GPA; Myzus persicae), an agronomically impo
41 globally important economic pest - the green peach aphid (Myzus persicae) - growing on 34 plant taxa,
42 8 also show enhanced resistance to the green peach aphid (Myzus persicae) compared with wild-type con
43 se pathways was investigated following green peach aphid (Myzus persicae) feeding on Arabidopsis.
44 ays with the generalist phloem-feeding green peach aphid (Myzus persicae), and in weight-gain assays
45 se to feeding by its aphid vector, the green peach aphid (Myzus persicae), and increases aphid fecund
46 w (Hyaloperonospora arabidopsidis) and green peach aphid (Myzus persicae), but retained susceptibilit
47 oEL displayed reduced fecundity of the green peach aphid (Myzus persicae), indicating enhanced resist
48 is gap we exposed four naive clones of green peach aphid (Myzus persicae), maintained on the model cr
55 ial diet both decrease Myzus persicae (green peach aphid) reproduction, suggesting a direct toxic or
57 eding herbivore-host plant system: the green peach aphid, Myzus persicae, feeding on multiple brassic
63 sis (cotton mealybug), Myzus persicae (green peach aphids) and Bemisia tabaci (silver leaf whitefly).
64 with this hypothesis, Myzus persicae (green peach aphids) prefer to settle on Nicotiana benthamiana
65 context, the syntenic regions identified in peach, apple and strawberry might be useful to interpret
67 characterised by floral and fruity flavours (peach/apricot, Muscat, melon, banana and strawberry) whi
68 hod to small RNA sequence data from soybean, peach, Arabidopsis and rice and provide experimental val
69 gene for the br mutation in Prunus persica (peach) associated with vertically oriented growth of bra
72 It is demonstrated that LTPs from wheat and peach bind a range of lipids in a variety of conditions,
75 enbuconazole, propiconazole, or pyridaben in peaches; carbendazim, imazalil, terbutryn, and thiabenda
76 Since matrix effects were observed in the peach commodity, organic acids were quantified by the st
78 mediate sorbitol synthesis in flower buds of peach concomitantly with specific chromatin modification
81 tionship between susceptibility of different peach cultivars (cvs) to the Mediterranean fruit fly (me
83 esults indicate that the phenolic extract of peach cultivars inhibits Abeta and alphaS fibril formati
87 al dehydrins (V. riparia YSK2, 60 kilodalton peach dehydrin [PCA60], barley dehydrin5 [Dhn5], Thellun
92 ow that China has been a critical region for peach evolution since long before human presence, much l
95 ble (except alpha-carotene and zeaxanthin in peach) for 13, 9.7, 5.7, 2.5 and 7.5months, respectively
98 o analyse 238 kaki, cashew apple, guava, and peach fruit and pulp samples, which were also analysed f
101 roperties, such as sweetness and acidity, in peach fruit by mid infrared spectroscopy is of interest
102 ic content and the antioxidant capacities of peach fruit extracts was found, indicating that phenolic
105 o increase the health-promoting potential of peach fruits and indirectly to ameliorate the aesthetic
107 -FTIR) was tested here on two populations of peach fruits issued from contrasting genitors providing
109 82, annotated as hypothetical protein in the peach genome sequence, was identified as a candidate gen
110 no acid sequence of LTP was identical in all peach genotypes but, for the first time, peel LTP was fo
111 gy; Group 2, peanut allergy and tolerance to peach; Group 3, peach allergy and tolerance to peanut; G
113 p 3 in vivo, a mouse model of anaphylaxis to peach has been produced and changes in the humoral and b
114 Allergic cross-reactions between cypress and peach have been reported, including an oral allergy synd
116 on is very bright but shows subtle yellow to peach hues which probably arise from the production of c
124 nation of As, Cd, Hg and Pb in NIST SRM 1547 peach leaves and SRM 1573a tomato leaves reference mater
131 onditions do not disrupt the 3D structure of peach LTP, explaining why LTPs retain their ability to b
133 cross-reactive allergen between cypress and peach might be responsible for the observed clinical ass
134 our physical and eight sensory properties of peach nectar were explored using the best-fit multiple l
138 h oranges (OR = 0.18; 95% CI: 0.06-0.51) and peaches (OR = 0.30; 95% CI: 0.13-0.67) had a decreased o
140 (P = .023), fresh oranges (P = .002), fresh peaches (P = .002), and collard greens/kale (P = .014).
144 able Rosaceae ESTs, the genetically anchored peach physical map, Rosaceae genetic maps and comprehens
151 se data define potential for improvements to peach production efficiency and fruit quality, particula
152 luate the allergenic properties of LTPs from peach (Pru p 3) and pellitory (Par j 1/Par j 2), major f
153 caused by lipid transfer protein (LTP) from peach (Pru p 3) is frequently associated with sensitizat
155 ivated in Tunisia: kaki (Diospyros kaki L.), peach (Prunus persica L.) and apricot (Prunus armeniaca
158 uence-based genotyping, and the high-quality peach (Prunus persica) genome reference sequence for sin
159 a recessive brachytic dwarfism trait (dw) in peach (Prunus persica) that has little or no effect on f
160 tein (LTP, Pru p 3) is the major allergen of peach (Prunus persica), and is in a greater abundance in
163 esentative crops such as apple (Malus spp.), peach (Prunus spp.), and strawberry (Fragaria spp.).
164 ADS-box transcription factors (DAM genes) in peach [Prunus persica (L.) Batsch] as potential candidat
166 d ripening stages in three climacteric (i.e. peach [Prunus persica] and two tomato [Solanum lycopersi
167 , blueberries, sweet cherries, table grapes, peaches, raspberries, and strawberries) in a postharvest
168 n blot analysis, indicating transposition of peach rather than random integration of the plasmid DNA.
169 wing two patterns: patients also allergic to peach, responding to Ara h 2 and Pru p 3, and patients a
171 of this region, BES were mapped against the peach scaffold_3 and BACs were anchored to the apricot m
174 h event in the southwestern USA, the 18.8 Ma Peach Spring Tuff, were formed by pyroclastic flows that
178 upational respiratory diseases in workers of peach tree crops have been reported punctually and have
183 rification of LTP from peel and pulp of four peach varieties [Gladys (white flesh), California (necta
185 nonspecific lipid transfer protein (LTP) of peach were compared with the homologous LTP1 of barley a
187 rst shown to directly impact endodormancy in peach where a deletion of a series of DAM resulted in lo
188 r two highly homologous genes are present in peach, whereas an additional member was detected under l
189 Robustness was demonstrated using fresh peaches, which provided recovery values within acceptabl
190 rases encoded by mycobacteriophages Bxz2 and Peaches with unusual and unpredictable specificities.
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