ライフサイエンスコーパス: peak systolic velocity

1 or equal to 3:1, and intrastent doubling of peak-systolic velocity.

2 Peak systolic velocity after administration of topical a

3 processed to reconstruct mitral annulus (MA) peak systolic velocity and displacement.

4 During stress, the peak systolic velocity and early diastolic velocity incr

5 beta values of the relationship between MA peak systolic velocity and LV end-diastolic volume and m

6 ional left ventricular function, assessed by peak systolic velocity and strain rate in 12 paired, non

7 The best-performing parameters were peak systolic velocity and V(MCA)/V(ICA) ratio.

8 Average volume of flow, peak systolic velocity, and diameter of residual lumen w

9 en kidneys and between regions was found for peak systolic velocity, but the magnitude of this variat

10 Increasing age decreased peak systolic velocity by 0.34 cm/sec/y.

11 2 months, defined as >/=2.5-fold increase in peak systolic velocity by duplex ultrasonography.

12 Internal carotid artery peak systolic velocities decrease with advancing age and

13 r or if flow approached 1,300 mL/min without peak systolic velocity greater than 400 cm/sec.

14 ive diagnostic US criteria for stenosis were peak-systolic velocity greater than 1.25 m/sec, internal

15 ive criteria for stenosis were also applied: peak-systolic velocity greater than 1.7 m/sec, ICA end-d

16 patients with isolated, asymptomatic AS and peak systolic velocity > or =4 m/s by Doppler echocardio

17 ort-/long-axis ratio <0.6, tricuspid annulus peak systolic velocity >/= 8 cm/s, and peak systolic lon

18 eroseptal-posterior delay, and SD in time to peak systolic velocity in the 12 left ventricular segmen

19 The SD in time to peak systolic velocity in the 12 left ventricular segmen

20 -/long-axis ratio >/= 0.6, tricuspid annulus peak systolic velocity <8 cm/s, and peak systolic longit

21 icuspid annular plane systolic excursion and peak systolic velocity, myocardial performance (expresse

22 ses in TDI indices occurred with dobutamine: peak systolic velocity of 4.41 +/- 1.07 to 6.67 +/- 1.07

23 significant decreases occurred with esmolol: peak systolic velocity of 4.46 +/- 0.94 to 2.31 +/- 0.81

24 arget artery of at least 70%, diagnosed by a peak systolic velocity of at least 3.0 m/s.

25 Duplex ultrasound peak systolic velocity of the cavernosal arteries increa

26 - 0.47 mm; p < 0.05) and correlated with the peak systolic velocity of the second palmar digital arte

27 Doppler-defined pre-procedural peak systolic velocities (PSV) and end-diastolic velocit

28 (c) ICA peak systolic velocity (PSV) and presence of plaque on g

29 Point, non-angle-corrected peak systolic velocity (PSV) and resistive index (RI) va

30 elocity (MFV), end diastolic velocity (EDV), peak systolic velocity (PSV) and resistive index (RI).

31 ICA peak systolic velocity (PSV) and the ratio of the PSV in

32 s was a focal twofold or higher elevation of peak systolic velocity (PSV) compared with the PSV immed

33 The range of peak systolic velocity (PSV) measurement (maximum minus

34 ic parvus tardus waveforms) with and without peak systolic velocity (PSV) thresholds (determined with

35 stically significant changes observed in the peak systolic velocity (PSV), end diastolic velocity (ED

36 Early systolic acceleration (ESA), peak systolic velocity (PSV), end diastolic velocity (ED

37 The range of CCA peak systolic velocities (PSVs) and end diastolic veloci

38 as evidenced by a higher mean graft/femoral peak systolic velocity ratio (1.6 vs. 0.90, P=0.006).

39 inal narrowing was assessed by graft/femoral peak systolic velocity ratio.

40 lic velocity greater than 0.4 m/sec, ICA/CCA peak-systolic velocity ratio greater than 2.0, and ICA/C

41 artery (ICA) to common carotid artery (CCA) peak-systolic velocity ratio of greater than or equal to

42 g-axis/length-area ratios, tricuspid annulus peak systolic velocity, RV peak longitudinal global syst

43 assessed by measuring differences in time-to-peak systolic velocity (T(SV)) between the RV free wall,

44 g-axis myocardial function was quantified by peak systolic velocity (Vs) and strain rate (SR) respons

45 ARAS was severe (60% to 99% stenosis) if peak systolic velocity was >200 cm/s.

46 Peak systolic velocity was additionally found to be inve

47 Peak systolic velocity was influenced by age (P =.008),

48 Peak systolic velocity was measured by Doppler velocimet

49 Preoperative portal peak systolic velocity was uniformly around 10 cm/sec.

50 rity, pulsatility index, resistive index, or peak-systolic velocity, was associated with malignancy.

51 Changes in TDI peak systolic velocity were correlated with changes in f

52 Resistive and pulsatility indexes and peak systolic velocity were documented.