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1 lly significant (P < 0.05) benefits favoring pegaptanib.
2             At 2 years, the VA trend favored pegaptanib.
3 izumab and 200-fold more potent than that of pegaptanib.
4 ed safety risk resulting from treatment with pegaptanib 0.3 mg in individuals with neovascular age-re
5                      More patients receiving pegaptanib (0.3 mg), as compared with sham injection, ma
6 9), followed by ranibizumab (44.1%, n = 41), pegaptanib (7.5%, n = 7), and aflibercept (5.4%, n = 5).
7                                              Pegaptanib, an anti-vascular endothelial growth factor t
8  (bevacizumab, ranibizumab, aflibercept, and pegaptanib) and intraocular steroids (triamcinolone and
9                     One hundred thirty-three pegaptanib- and 127 sham-treated subjects were in the ye
10 k 54, >/= 10 letter gains seen in 49 (36.8%) pegaptanib- and 25 (19.7%) sham-treated subjects (odds r
11                                              Pegaptanib appears to be an effective therapy for neovas
12                           In the group given pegaptanib at 0.3 mg, 70 percent of patients lost fewer
13 treous injection into one eye per patient of pegaptanib (at a dose of 0.3 mg, 1.0 mg, or 3.0 mg) or s
14                        Subjects who received pegaptanib by randomization or change in dose assignment
15                          Unlike bevacizumab, pegaptanib did not interact directly with VEGF-A(165)b.
16    Studies for bevacizumab, aflibercept, and pegaptanib in DME were limited but also in favor of anti
17 ith the terms "bevacizumab OR ranibizumab OR pegaptanib OR aflibercept" and the limitations "humans"
18 amer therapeutic approved for use in humans (Pegaptanib or Macugen).
19                                   In year 1, pegaptanib or sham was administered every 6 weeks with f
20 esponse relationship, for all three doses of pegaptanib (P<0.001 for the comparison of 0.3 mg with sh
21  10 percent in the group receiving 0.3 mg of pegaptanib (P<0.001).
22                                 Intravitreal pegaptanib sodium (0.3 mg) improved vision and reduced c
23                    To evaluate the safety of pegaptanib sodium 0.3 mg intravitreal injection in the t
24 icantly greater effect of the combination of pegaptanib sodium and PDT on lesion size in choroidal ne
25                           The combination of pegaptanib sodium and PDT resulted in the regression of
26                        Two anti-VEGF agents, pegaptanib sodium and ranibizumab, are currently approve
27                            Pretreatment with pegaptanib sodium appeared to decrease the efficacy of P
28 t provides level I evidence for intravitreal pegaptanib sodium for DME.
29 ion of pegaptanib sodium nor the efficacy of pegaptanib sodium in the inhibition of VEGF165 binding t
30 nt of ocular neovascularization with PDT and pegaptanib sodium may provide a more effective approach
31                                 In contrast, pegaptanib sodium monotherapy yielded little regression
32 T did not affect the chemical composition of pegaptanib sodium nor the efficacy of pegaptanib sodium
33 herapy's (PDT's) effects on the integrity of pegaptanib sodium were analyzed by HPLC, a VEGF165-bindi
34 evacizumab, 935 triamcinolone acetonide, 121 pegaptanib sodium) were reviewed for 2465 patients betwe
35       Early studies of the anti-VEGF agents, pegaptanib sodium, ranibizumab, bevacizumab, VEGF trap,
36 al acuity among patients receiving 0.3 mg of pegaptanib was better than in those receiving sham injec
37  trial first showed that an anti-VEGF agent (pegaptanib) was able to prevent vision loss in neovascul

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