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1 lly significant (P < 0.05) benefits favoring pegaptanib.
2 At 2 years, the VA trend favored pegaptanib.
3 izumab and 200-fold more potent than that of pegaptanib.
4 ed safety risk resulting from treatment with pegaptanib 0.3 mg in individuals with neovascular age-re
6 9), followed by ranibizumab (44.1%, n = 41), pegaptanib (7.5%, n = 7), and aflibercept (5.4%, n = 5).
8 (bevacizumab, ranibizumab, aflibercept, and pegaptanib) and intraocular steroids (triamcinolone and
10 k 54, >/= 10 letter gains seen in 49 (36.8%) pegaptanib- and 25 (19.7%) sham-treated subjects (odds r
13 treous injection into one eye per patient of pegaptanib (at a dose of 0.3 mg, 1.0 mg, or 3.0 mg) or s
16 Studies for bevacizumab, aflibercept, and pegaptanib in DME were limited but also in favor of anti
17 ith the terms "bevacizumab OR ranibizumab OR pegaptanib OR aflibercept" and the limitations "humans"
20 esponse relationship, for all three doses of pegaptanib (P<0.001 for the comparison of 0.3 mg with sh
24 icantly greater effect of the combination of pegaptanib sodium and PDT on lesion size in choroidal ne
29 ion of pegaptanib sodium nor the efficacy of pegaptanib sodium in the inhibition of VEGF165 binding t
30 nt of ocular neovascularization with PDT and pegaptanib sodium may provide a more effective approach
32 T did not affect the chemical composition of pegaptanib sodium nor the efficacy of pegaptanib sodium
33 herapy's (PDT's) effects on the integrity of pegaptanib sodium were analyzed by HPLC, a VEGF165-bindi
34 evacizumab, 935 triamcinolone acetonide, 121 pegaptanib sodium) were reviewed for 2465 patients betwe
36 al acuity among patients receiving 0.3 mg of pegaptanib was better than in those receiving sham injec
37 trial first showed that an anti-VEGF agent (pegaptanib) was able to prevent vision loss in neovascul
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