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1 n young, nulliparous women with abdominal or pelvic pain.
2 one-year history of dysmenorrhea and cyclic pelvic pain.
3 have an adnexal mass with acute or subacute pelvic pain.
4 % of women and is associated with persistent pelvic pain.
5 and (3) an operation to address the groin or pelvic pain.
6 eeding, primary amenorrhea, pelvic mass, and pelvic pain.
7 s mutation led to his chronic infections and pelvic pain.
8 diated adaptive immune mechanisms in chronic pelvic pain.
9 i isolate (CP-1) from a patient with chronic pelvic pain.
10 t important for the care of men with chronic pelvic pain.
11 ed NOD mice was sufficient to induce chronic pelvic pain.
12 onic fungal rash of the scrotum, and chronic pelvic pain.
13 n and erosion and increased dyspareunia, and pelvic pain.
14 ndent condition that causes dysmenorrhea and pelvic pain.
15 infertility, ectopic pregnancy, and chronic pelvic pain.
16 a treatment option for patients with chronic pelvic pain.
17 as therapeutic targets for treating chronic pelvic pain.
18 inary urgency; and 2.2 (95% CI, 1.2-3.9) for pelvic pain.
19 ymptoms include reduced fertility and severe pelvic pain.
20 oing laparotomy, 20 of whom reported chronic pelvic pain.
21 s review of surgical patients with groin and pelvic pain, 2008-2013, was conducted in a single-surgeo
23 (26 of 31 patients) in the context of acute pelvic pain and 92.6% (25 of 27 patients), 88.9% (24 of
26 n of reproductive age and is associated with pelvic pain and infertility, and its pathogenesis is not
34 necological disorder, associated with severe pelvic pain and reduced fertility; however, its molecula
35 oms of endometriosis are chronic intolerable pelvic pain and subfertility or infertility, which profo
36 lammatory condition in women that results in pelvic pain and subfertility, and has been associated wi
37 characterized by histologic endometritis or pelvic pain and tenderness plus one of the following: or
38 defined as 1) histologic endometritis or 2) pelvic pain and tenderness plus oral temperature >38.8 d
39 ladder pain syndrome, a clinical syndrome of pelvic pain and urinary urgency/frequency in the absence
40 logue scales (VAS) for dysmenorrhea, chronic pelvic pain, and deep dyspareunia and answered questions
42 syndromes irritable bowel syndrome, chronic pelvic pain, and fibromyalgia were assessed by questionn
44 ing is warranted in patients presenting with pelvic pain, and more aggressive pain management strateg
45 g-term outcomes such as dyspareunia, chronic pelvic pain, and vaginal distortion, which may occur eve
48 approach in diagnosing and treating chronic pelvic pain, because it is common knowledge that differe
51 ion of abdominal or pelvic injury that cause pelvic pain, bowel obstruction, and infertility in women
52 our patients presented with abdominal and/or pelvic pain, but only one of these four had cyclic pain
53 me, interstitial cystitis, and other chronic pelvic pain (CPP) disorders often occur concomitantly.
55 adolescents with dysmenorrhea and/or chronic pelvic pain (CPP) who are suspected to have endometriosi
57 vis may contribute to etiology of functional pelvic pain disorders such as interstitial cystitis/blad
58 e in improving dysmenorrhea and nonmenstrual pelvic pain during a 6-month period in women with endome
65 howed that CP1 induced and sustained chronic pelvic pain in NOD mice, an attribute not exhibited by a
66 We developed an infection model of chronic pelvic pain in NOD/ShiLtJ (NOD) mice with a clinical Esc
73 e of dorsal root ganglia neurons to decrease pelvic pain; (iv) decreases proinflammatory, estrogen-do
74 d controlled trial of 487 women with chronic pelvic pain lasting longer than 6 months without or with
78 plays a central role in the pathogenesis of pelvic pain, one of the core symptoms of endometriosis.
79 tension-type headache was 4% (2-9); chronic pelvic pain or prostatitis was 11% (8-17); and fibromyal
80 .12-4.69, I(2) = 0%; 3 studies), and chronic pelvic pain (OR, 2.73; 95% CI, 1.73-4.30, I(2) = 40%; 10
81 algia (OR, 3.35; 95% CI, 1.51-7.46), chronic pelvic pain (OR, 3.27; 95% CI, 1.02-10.53), and function
82 is a common cause of both cyclic and chronic pelvic pain, reduced fertility, and reduced quality-of-l
85 isorders(MHD) in chronic prostatitis/chronic pelvic pain syndrome (CP/CPPS) have been widely studied.
88 Prostatitis [chronic prostatitis/chronic pelvic pain syndrome (CP/CPPS)] is a common condition in
90 categorize male chronic prostatitis/chronic pelvic pain syndrome and interstitial cystitis/bladder p
91 zation, and treatment of prostatitis/chronic pelvic pain syndrome based on the National Institutes of
92 The etiology of chronic prostatitis/chronic pelvic pain syndrome in men is unknown but may involve m
93 Treatment of chronic prostatitis/chronic pelvic pain syndrome is often empirical because clinical
96 nditions such as chronic prostatitis/chronic pelvic pain syndrome, interstitial cystitis/bladder pain
100 thus serve as initiating agents for chronic pelvic pain through mechanisms that are dependent on bot
102 e complications cause lifelong, irreversible pelvic pain, vaginal shortening, vaginal narrowing, seve
103 inical response with respect to nonmenstrual pelvic pain was 50.4% in the lower-dose elagolix group a
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