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1 RF 36, weakly sensitized cells to killing by penciclovir.
2 been confirmed as resistant to acyclovir and penciclovir.
3     Additionally, a radiolabeled analogue of penciclovir, [(18)F]FHBG, which is similar in structure
4  by thymidine kinase (TK) assay using 8-(3)H-penciclovir (8-(3)H-PCV) as substrate.
5 the radioactively labeled substrates [8-(3)H]penciclovir ([8-(3)H]PCV), and 8-[(18)F]fluoropenciclovi
6                      Ex vivo substrate (8-3H Penciclovir) accumulation assays in 293T cells expressin
7 nce of adverse events was comparable between penciclovir and placebo groups.
8 -deoxyarabinofuranosyl-5-ethyluracil (FEAU), penciclovir, and 9-[4-fluoro-3-(hydroxymethyl)butyl]guan
9 curred both among patients who first applied penciclovir cream in the prodrome and erythema stages an
10                                              Penciclovir cream is the first treatment to clearly demo
11                                   Topical 1% penciclovir cream or vehicle control cream.
12                              The efficacy of penciclovir cream was apparent when therapy was initiate
13                    Subjects received topical penciclovir for 4 days during successive episodes of rec
14 lovir, an oral form of the purine nucleoside penciclovir, has been shown to inhibit HBV replication.
15 of these mutants to adefovir, lobucavir, and penciclovir in vitro and adefovir in vivo.
16 from these reactivations were susceptible to penciclovir in vitro.
17 mivudine]), and famciclovir (oral prodrug of penciclovir) induced depressions in viremia and intrahep
18             Famciclovir, the oral prodrug of penciclovir, inhibits HBV DNA replication.
19 e or lamivudine-resistant HBV to lamivudine, penciclovir, or adefovir but instead enhanced viral repl
20                                              Penciclovir (PCV), an antiherpesvirus agent in the same
21 is patient's HSV-2 isolates to acyclovir and penciclovir sensitivity, although resistant virus reappe
22 1.64; P=.003) also resolved more quickly for penciclovir-treated patients compared with patients who
23 lcers, and/or crusts) was 0.7 day faster for penciclovir-treated patients compared with those who rec
24  compared the inhibition constants (K(i)) of penciclovir triphosphate (PCVTP, the active metabolite o
25  triphosphate (FTCTP), adefovir diphosphate, penciclovir triphosphate, and lobucavir triphosphate was
26               Isolates remained sensitive to penciclovir when tested by a plaque reduction assay, and

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