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   1 RF 36, weakly sensitized cells to killing by penciclovir.                                            
     2 been confirmed as resistant to acyclovir and penciclovir.                                            
  
  
     5 the radioactively labeled substrates [8-(3)H]penciclovir ([8-(3)H]PCV), and 8-[(18)F]fluoropenciclovi
  
  
     8 -deoxyarabinofuranosyl-5-ethyluracil (FEAU), penciclovir, and 9-[4-fluoro-3-(hydroxymethyl)butyl]guan
     9 curred both among patients who first applied penciclovir cream in the prodrome and erythema stages an
  
  
  
  
    14 lovir, an oral form of the purine nucleoside penciclovir, has been shown to inhibit HBV replication. 
  
  
    17 mivudine]), and famciclovir (oral prodrug of penciclovir) induced depressions in viremia and intrahep
  
    19 e or lamivudine-resistant HBV to lamivudine, penciclovir, or adefovir but instead enhanced viral repl
  
    21 is patient's HSV-2 isolates to acyclovir and penciclovir sensitivity, although resistant virus reappe
    22 1.64; P=.003) also resolved more quickly for penciclovir-treated patients compared with patients who 
    23 lcers, and/or crusts) was 0.7 day faster for penciclovir-treated patients compared with those who rec
    24  compared the inhibition constants (K(i)) of penciclovir triphosphate (PCVTP, the active metabolite o
    25  triphosphate (FTCTP), adefovir diphosphate, penciclovir triphosphate, and lobucavir triphosphate was
  
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