ライフサイエンスコーパス: penicillin

1 inhibitory concentration (MIC) >2 mug/mL to penicillin.

2 curred with ciprofloxacin, erythromycin, and penicillin.

3 l between those treated and not treated with penicillin.

4 was the least susceptible species overall to penicillin.

5 estigate the impact on the susceptibility to penicillin.

6 ed drug allergies in this population were to penicillins (12.8%), sulfonamide antibiotics (7.4%), opi

7 lin-sulbactam, 9 [69.2%]; and institution C: penicillin, 32 [72.7%], P < .001).

8 erythromycin (256 mug/mL from 4 mug/mL), and penicillin (8 mug/mL from 4 mug/mL), indicating higher l

9 es (9), amphenicols (2), cephalosporins (7), penicillins (8), macrolides (8), benzimidazoles (20), co

10 visiae to produce and secrete the antibiotic penicillin, a beta-lactam nonribosomal peptide, by takin

11 on of other secondary metabolites, including penicillin, affecting the expression of PN genes.

12 ability of both aztreonam and carbapenems in penicillin-allergic subjects.

13 e aimed to determine the optimal approach to penicillin allergies among medical inpatients.

14 Failure to address inpatient penicillin allergies results in more broad-spectrum anti

15 oximately 90-99% of patients with a label of penicillin allergy (PenA) are not allergic when comprehe

16 clude identification of HLA associations for penicillin allergy and a microRNA biomarker/mechanism fo

17 the diagnosis of patients with a history of penicillin allergy has also been included.

18 uated internal medicine inpatients reporting penicillin allergy in 3 periods: (1) standard of care (S

19 A documented penicillin allergy is associated with increased morbidit

20 Reported penicillin allergy rarely reflects penicillin intoleranc

21 tic review was to identify whether inpatient penicillin allergy testing affected clinical outcomes du

22 Inpatient penicillin allergy testing is safe and effective in ruli

23 Inpatient penicillin allergy testing led to a change in antibiotic

24 Inpatient penicillin allergy testing was associated with decreased

25 documented penicillin allergy that underwent penicillin allergy testing were included.

26 Inpatients having a documented penicillin allergy that underwent penicillin allergy tes

27 Patients with a documented penicillin allergy who require penicillin should be test

28 porin antibiotics among inpatients reporting penicillin allergy.

29 used, even in individuals with a history of penicillin allergy.

30 testing is safe and effective in ruling out penicillin allergy.

31 erococcus (VRE) in patients with and without penicillin "allergy" at hospital admission.

32 Cases with penicillin "allergy" averaged 0.59 (9.9%; 95% CI, 0.47-0

33 Subjects with a penicillin "allergy" history are exposed to significantl

34 Subjects with a penicillin "allergy" history spend significantly more ti

35 A penicillin "allergy" history, although often inaccurate,

36 ble cases) unique hospitalized subjects with penicillin "allergy" to 2 unique discharge diagnosis cat

37 bjects who had 307 nonimmediate reactions to penicillins (almost exclusively aminopenicillins) and ha

38 cts with T cell-mediated hypersensitivity to penicillins, almost exclusively aminopenicillins.

39 ive bacterial pathogens showed resistance to penicillin, ampicillin and amoxicillin.

40 l second- or third-generation cephalosporin, penicillin, ampicillin, ampicillin-sulbactam, amoxicilli

41 The rise in PSSA makes penicillin an increasingly viable treatment option.

42 ontaining, bicyclic compounds, considered as penicillin analogs with an additional free thiol.

43 517/993 (52.1%) isolates were susceptible to penicillin and 946/993 (95.3%) were susceptible to oxaci

44 sceptibility across species, particularly to penicillin and ceftriaxone, and across geographical regi

45 P and ST-when completed-increased the use of penicillin and cephalosporin antibiotics among inpatient

46 Resistance of VGS to penicillin and erythromycin was determined by the epsilo

47 MICs for penicillin and erythromycin were correlated (P <0.05).

48 NCTC1 was resistant to penicillin and erythromycin, and contained a complement

49 218), and they were generally susceptible to penicillin and fluoroquinolones but not to erythromycin.

50 randomized to receive amoxicillin or benzyl penicillin and followed up for the primary outcome of tr

51 Antipseudomonal penicillin and fourth-generation cephalosporin monothera

52 Treatment for patients who cannot receive penicillin and management of patients who do not serolog

53 Evidence regarding penicillin and nonpenicillin regimens was reviewed from

54 ty to these agents can be predicted from the penicillin and oxacillin or cefoxitin results.

55 Routine testing of penicillin and oxacillin or cefoxitin should be used to

56 llin-binding proteins (PBPs), the targets of penicillin and related antibiotics, polymerize the glyca

57 e implies, these proteins are the targets of penicillin and related antibiotics.

58 Penicillin and related beta-lactams comprise one of our

59 cin with L. acidophilus being susceptible to penicillin and vancomycin, whereas L. rhamnosus and L. c

60 istory of IgE-mediated hypersensitivity to a penicillin and/or cephalosporin who were subsequently gi

61 ere significantly associated with allergy to penicillins and amoxicillin (P = 6.0 x 10(-4) and P = 4.

62 acyclines, fluoroquinolones, cephalosporins, penicillins and amphenicols) in the bovine urine is impo

63 -reactivity rate of approximately 1% between penicillins and both imipenem and meropenem, whereas a s

64 The cross-reactivity between penicillins and carbapenems for IgE-mediated reactions i

65 l as the absence of cross-reactivity between penicillins and cefuroxime, ceftriaxone, and aztreonam i

66 rns of allergologic-test positivity: to both penicillins and cephalosporins (group A), or only to cep

67 sting and laboratory investigations for both penicillins and cephalosporins are included.

68 Penicillins and cephalosporins are synthesized from a cl

69 The most common causes of SCARs were penicillins and cephalosporins for SJS/TEN and AGEP; gly

70 Cross-reactivity between penicillins and cephalosporins is discussed in detail.

71 acteriaceae generally cannot be treated with penicillins and cephalosporins.

72 that efficiently catalyzes the hydrolysis of penicillins and early cephalosporins but not oxyimino-ce

73 Penicillins and macrolides were the most common antibiot

74 iate and non-immediate allergic reactions to penicillins and other beta-lactams.

75 ecutive subjects with immediate reactions to penicillins and positive results on skin tests to at lea

76 clined most rapidly for fluoroquinolones and penicillins and reached baseline in 2-3 months.

77 crobials were trimethoprim-sulfamethoxazole, penicillin, and amoxicillin (22%, 8/37 each).

78 We examined the effects of erythromycin, penicillin, and virginiamycin at low concentrations refl

79 Carbapenems, penicillins, and cephalosporins were studied.

80 the most common antibiotics (cephalosporins, penicillins, and macrolides) used between age 3 months a

81 f moxifloxacin, carbapenems, antipseudomonal penicillins, and vancomycin were compared by using inter

82 nd rapid strategy for the extraction of four penicillin antibiotic residues (benzylpenicillin, cloxac

83 rgies" in general, but most those notably to penicillin, are associated with increased hospital use a

84 %) of patients in the amoxicillin and benzyl penicillin arms, respectively (risk difference, -0.3% [9

85 women is limited, but available data support penicillin as first-line therapy.

86 this is caused by RSV G glycoprotein binding penicillin binding protein 1a.

87 Isolates harbouring the mosaic penicillin binding protein 2 (PBP2)-considered a key mec

88 that controls activity of the bi-functional penicillin binding protein PBP A1, we discovered that Gp

89 precise temporal and spatial organization of penicillin binding proteins (PBPs) and associated protei

90 he surrounding medium, a process mediated by penicillin binding proteins (PBPs).

91 f the bacterial cell wall, is synthesised by penicillin binding proteins (PBPs).

92 glycan synthesis and degradation mediated by penicillin binding proteins in the forespore and a cell

93 al sideromimic conjugated compounds bound to penicillin binding proteins PBP3 and PBP1a from Pseudomo

94 cholerae high-molecular-weight bifunctional penicillin binding proteins, PBP1a and PBP1b, in the fit

95 rmined that expression of the well-conserved penicillin-binding protein (PBP) 1A, prevented LOS-defic

96 erved heterogeneous localization dynamics of penicillin-binding protein (PBP) 1A, the synthase predom

97 otein LpoB is required for the activation of penicillin-binding protein (PBP) 1B, which is a major, b

98 y functional assays, showing binding of 2 to penicillin-binding protein (PBP) 2a.

99 of Streptococcus pneumoniae contain altered penicillin-binding protein (PBP) genes and occasionally

100 e acquisition of a nonnative gene encoding a penicillin-binding protein (PBP2a), with significantly l

101 onferring beta-lactam antibiotic resistance, penicillin-binding protein 2A (encoded by the mecA gene)

102 hylococcus aureus (MRSA), is able to inhibit penicillin-binding protein 2a (PBP2a) by triggering an a

103 The performance of a rapid penicillin-binding protein 2a (PBP2a) detection assay, t

104 antibiotics, the oxadiazoles, which inhibit penicillin-binding protein 2a (PBP2a) of MRSA.

105 (eg, oxacillin) depends on the production of penicillin-binding protein 2a (PBP2a), encoded by mecA M

106 An enzyme, called penicillin-binding protein 2a (PBP2a), is brought into t

107 The Alere penicillin-binding protein 2a test accurately detected a

108 Penicillin-binding protein 4 (PBP4) is a nonessential tr

109 evate AmpC expression is loss of function of penicillin-binding protein 4 (PBP4).

110 (ECD) consists of four repeats homologous to penicillin-binding protein and serine/threonine kinase a

111 ng of 1.2 million compounds for binding to a penicillin-binding protein and the subsequent demonstrat

112 MRSA strains have acquired a non-native penicillin-binding protein called PBP2a that cross-links

113 erived genomes possess mutations targeting a penicillin-binding protein coding gene (mrcA) that had n

114 s for the direct detection of IgE and of the penicillin-binding protein from Staphylococcus aureus (P

115 on is penicillin sensitive and assigned to a penicillin-binding protein motif.

116 Penicillin-binding protein PBP 2B is a key cell division

117 ates also potently inhibit the non-essential penicillin-binding protein PBP 5 by the same mechanism o

118 ls and is catalyzed by the essential class B penicillin-binding protein PBP2b transpeptidase (TP).

119 tro and in vivo with the major bi-functional penicillin-binding protein.

120 d scoring against the crystal structure of a penicillin-binding protein.

121 wall assembly mechanism assume that class A penicillin-binding proteins (aPBPs), the targets of peni

122 The biological roles of low molecular weight penicillin-binding proteins (LMW PBP) have been difficul

123 Penicillin-binding proteins (PBPs) are enzymes involved

124 High-molecular-weight penicillin-binding proteins (PBPs) are essential integra

125 Penicillin-binding proteins (PBPs) are involved in the s

126 Penicillin-binding proteins (PBPs) catalyze the crosslin

127 iotics is the D,D-transpeptidase activity of penicillin-binding proteins (PBPs) for synthesis of 4-->

128 erence affects the cross-linking activity of penicillin-binding proteins (PBPs) that assemble peptido

129 ave long been known to target enzymes called penicillin-binding proteins (PBPs) that build the bacter

130 jor synthases of this exoskeleton are called penicillin-binding proteins (PBPs)(1,2).

131 In Escherichia coli , the bifunctional penicillin-binding proteins (PBPs), PBP1A and PBP1B, pla

132 ne of very few compound classes that inhibit penicillin-binding proteins (PBPs), SBLs and, as recentl

133 Synthases called penicillin-binding proteins (PBPs), the targets of penic

134 pressing variants of its target enzymes, the penicillin-binding proteins (PBPs), with many amino acid

135 doglycan (PG) exoskeleton synthesized by the penicillin-binding proteins (PBPs).

136 ll wall biosynthesis through inactivation of penicillin-binding proteins (PBPs).

137 tagged mimics of the endogenous substrate of penicillin-binding proteins (PBPs).

138 Fluorescein-meropenem binds both penicillin-binding proteins and beta-lactam sensors and

139 The transglycosylase domain of penicillin-binding proteins is especially important, as

140 he relative localization patterns of class B penicillin-binding proteins Pbp2x and Pbp2b were used as

141 ght to work in concert with the PG synthases penicillin-binding proteins PBP3 and PBP1b.

142 Penicillin-binding proteins represent well-established,

143 can cell wall is synthesized by bifunctional penicillin-binding proteins such as PBP1b that have both

144 For many years, the penicillin-binding proteins were thought to be the key e

145 is of the bacterial cell wall (also known as penicillin-binding proteins, PBPs) have evolved to bind

146 elevant class A, C and D beta-lactamases and penicillin-binding proteins, resulting in intrinsic anti

147 biosynthesis and one encodes a homologue of penicillin-binding proteins.

148 d to monoclonal antibodies (MAb) specific to penicillin-binding-protein 2a of methicillin resistant (

149 ansmembrane proteins that have extracellular penicillin-binding-protein and serine/threonine kinase-a

150 The family of bacterial Penicillin-binding-protein And Serine/Threonine kinase-A

151 kin test positivity to amoxicillin and other penicillins but not to cephalosporins.

152 on were significant predictors of allergy to penicillins but not to cephalosporins.

153 demonstrating that skin-test sensitivity to penicillins can decrease over time and that allergic pat

154 yphilis is an acceptable alternate option if penicillin cannot be used.

155 All isolates were susceptible to penicillin, cefotaxime, and levofloxacin.

156 negative pathogens that threatens the use of penicillin, cephalosporin and carbapenem antibiotics to

157 Models were repeated separately for penicillins, cephalosporins and macrolides.

158 n, daptomycin and the beta-lactam-containing penicillins, cephalosporins and nocardicins.

159 ventional beta-lactam antibiotics, including penicillins, cephalosporins, and carbapenems, through va

160 f antibacterial beta-lactam drugs, including penicillins, cephalosporins, and carbapenems.

161 three other beta-lactam antibiotic families: penicillin/cephalosporins, clavams and carbapenems.

162 lawian first-line antibiotics amoxicillin or penicillin, chloramphenicol, and co-trimoxazole; 68.3% o

163 ored over time in the presence or absence of penicillin, ciprofloxacin, or doxycycline.

164 .91/patient), with 53.6% involving 1 or more penicillin class drug.

165 2-selective (SC-236) NSAIDs +/- antibiotics (penicillin, clindamycin) were given to mice challenged i

166 uding the cefoxitin-induced nitrocefin test, penicillin cloverleaf assay, and penicillin disk zone ed

167 ased chain length and resistance to lysis by penicillin compared to the Spn(-) strain, indicating tha

168 who received cefazolin or antistaphylococcal penicillins compared with vancomycin (HR, 0.57; 95% CI,

169 he beta-lactam and thiazolidine rings of the penicillin core into the linear tripeptide l-delta-amino

170 ning that patients with CLV allergy can take penicillin derivatives safely.

171 to AX appears consistent, and a response to penicillin determinants only develops in a minority of c

172 se appears not to be modified by exposure to penicillin determinants, meaning that patients with CLV

173 known interactions (beta-lactamase-resistant penicillins [dicloxacillin] and carboxamide derivatives)

174 the accuracy of penicillin MIC testing, the penicillin disk diffusion test, and three beta-lactamase

175 % categorical agreement with blaZ PCR, while penicillin disk diffusion yielded one major error.

176 ompared to the blaZ PCR results, whereas the penicillin disk zone edge and cloverleaf tests showed se

177 cefin test, penicillin cloverleaf assay, and penicillin disk zone edge test.

178 en combined with AgNPs, while ampicillin and penicillin do not.

179 m complexes with AgNPs, while ampicillin and penicillin do not.

180 without tetracycline, while the presence of penicillin does not enhance the binding of Ag or Ag(+) r

181 erior to vancomycin or an antistaphylococcal penicillin, each in combination with low-dose, short-cou

182 s were implemented for the evaluation of the penicillin effect.

183 acquisition of pneumococci nonsusceptible to penicillin, erythromycin, clindamycin, penicillin plus e

184 lities of each of the colonizing isolates to penicillin, erythromycin, clindamycin, tetracycline, and

185 lin-susceptible isolates were treated with a penicillin family antimicrobial.

186 noninferiority of oral amoxicillin to benzyl penicillin for severe pneumonia may not be generalizable

187 methicillin-resistant Staphylococcus aureus, penicillin for Streptococcus pneumoniae, and an update o

188 mmend the use of a single dose of benzathine penicillin G (BPG) for treating early syphilis in human

189 .4 million units of intramuscular benzathine penicillin G (BPG) is recommended for the treatment of e

190 Benzathine penicillin G (BPG) is the only recommended treatment to

191 ies; however, few receive optimal benzathine penicillin G (BPG) therapy to prevent disease progressio

192 of streptomycin (SM), tetracycline (TC), and penicillin G (PC-G) in milk.

193 n G to 6-aminopenicillanic acid catalyzed by Penicillin G acylase in miniaturized stirred batch react

194 red-emitting dye with properly produced anti-penicillin G antibodies.

195 The proposed method is based penicillin G conjugate labeled with red-emitting dye wit

196 mainstay of syphilis treatment is parenteral penicillin G despite the relatively modest clinical tria

197 rent analytical methods used to quantify the penicillin G in milk are based on HPLC, mass spectrometr

198 Detection of penicillin G in milk is of interest because of the wide

199 nsing approach for detecting the presence of penicillin G in milk.

200 anges during the enzymatic transformation of Penicillin G to 6-aminopenicillanic acid catalyzed by Pe

201 -lactam antibiotics (amoxicillin, oxacillin, penicillin G).

202 significant matrix effects, quantitation of penicillin G, a common antimicrobial, is possible in pla

203 e, flutamide, flufenamic acid, the K salt of penicillin G, and form 4 of the drug 4-[4-(2-adamantylca

204 lar injection of 2.4 million U of benzathine penicillin G, with studies reporting 90% to 100% treatme

205 lin), sodium dodecyl sulfate (+control), and penicillin-G (-control).

206 All compounds but penicillin-G significantly slowed contraction in a dose-

207 ventional beta-lactam antibiotics, including penicillin-G, amoxicillin, ampicillin, and cefazolin, ar

208 tudy, 3 of whom were allocated to the benzyl penicillin group.

209 sk difference between amoxicillin and benzyl penicillin groups was prespecified at 7%.

210 16.8% by day 14 in the amoxicillin vs benzyl penicillin groups, respectively.

211 jectable agents (ampicillin, gentamicin, and penicillin) had low variable availability in first-level

212 Although resistance to penicillin has not yet been identified, an increasing nu

213 documented IgE-mediated hypersensitivity to penicillins have demonstrated a cross-reactivity rate of

214 lts with T cell-mediated hypersensitivity to penicillins have found a rate of cross-reactivity with c

215 al importance, including antibiotics such as penicillin, immunosuppressants such as cyclosporine, and

216 If a patient misses a dose of penicillin in a course of weekly therapy for late syphil

217 use of first generation beta-lactams such as penicillin in the years prior to the introduction of met

218 s, triazole derivatives, and combinations of penicillins, including beta-lactamase inhibitors) and tw

219 High-dose penicillin-induced seizures were characterized by a comb

220 and 10 Hz for its effects on a rat model of penicillin-induced seizures.

221 oglycan-like structures in Chlamydiaceae and penicillin inhibits cytokinesis, a phenomenon known as t

222 Reported penicillin allergy rarely reflects penicillin intolerance.

223 Penicillin is the drug of choice to treat syphilis.

224 Penicillin is the most common drug "allergy" noted at ho

225 Only one drug, penicillin, is recommended for syphilis treatment and re

226 We now show that low-dose penicillin (LDP), delivered from birth, induces metaboli

227 lin-binding proteins (aPBPs), the targets of penicillin-like drugs, function as the primary cell wall

228 characteristics, such as cephalosporins and penicillins, may be given safely to patients with a cert

229 g for 12 commonly used antimicrobial agents (penicillin, methicillin, erythromycin, clindamycin, tetr

230 (17%) were infected with VGS strains with a penicillin MIC >/= 2 microg/mL.

231 orts, 98% of patients infected by VGS with a penicillin MIC of >/= 2 microg/mL had at least 1 of the

232 The penicillin MIC test had 100% categorical agreement with

233 ates were selected to assess the accuracy of penicillin MIC testing, the penicillin disk diffusion te

234 The proportion of isolates with a penicillin minimal inhibitory concentration >2 microg/mL

235 ia was observed only for VGS isolates with a penicillin minimum inhibitory concentration (MIC) of >/=

236 taining combination therapy; antipseudomonal penicillin monotherapy versus fourth-generation cephalos

237 Penicillin non-susceptibility occurred in isolates from

238 ccine serotype associated with high rates of penicillin nonsusceptibility.

239 Overall, 91% (71/78) of isolates were penicillin nonsusceptible and 16.7% (13/78) were MDR.

240 events, or nasopharyngeal colonization with penicillin-nonsusceptible pathogens.

241 th 24 patients treated with intravenous (IV) penicillin only.

242 riod patients did not have increased odds of penicillin or cephalosporin use overall (adjusted odds r

243 ), we observed significant increased odds of penicillin or cephalosporin use overall in the APP perio

244 mputerized guideline significantly increased penicillin or cephalosporin use overall nearly 2-fold an

245 The primary outcome was use of a penicillin or cephalosporin, comparing interventions to

246 itive pathogens remain susceptible to either penicillin or chloramphenicol.

247 By exposing bacteria to nutrient broth and penicillin or ciprofloxacin, the authors were able to di

248 affect the resistance pattern of the VGS to penicillin or erythromycin.

249 ive therapy, specifically antistaphylococcal penicillins or cefazolin.

250 Cross-reactivity between penicillins or cephalosporins and carbapenems is anticip

251 The majority of patients with penicillin- or cephalosporin-related SCARs were able to

252 olides: OR, 0.98; 95% CI, .96-.99; P = .005; penicillins: OR [log(days)], 0.62; 95% CI, .44-.89; P =

253 r antistaphylococcal beta-lactams except for penicillin, oxacillin or cefoxitin, and ceftaroline.

254 sociated with increased nonsusceptibility to penicillin (P < 0.001).

255 lly avoided in the 10% of patients reporting penicillin (PCN) allergy, but most of these patients are

256 le to penicillin, erythromycin, clindamycin, penicillin plus erythromycin, and multiple drugs (>/=3 a

257 olates of NVT pneumococci not susceptible to penicillin (PNSP) in 2009 and compared them with the gen

258 eded to definitively determine the impact of penicillin prophylaxis on the trajectory of latent RHD.

259 e rate of RHD progression and the ability of penicillin prophylaxis to improve outcome.

260 Penicillin prophylaxis was prescribed in 49.3% with over

261 firm noninferiority of amoxicillin to benzyl penicillin, provide estimates of risk of treatment failu

262 of antibiotics, beta-lactam (ampicillin and penicillin), quinolone (enoxacin), aminoglycoside (kanam

263 An increased prescription of penicillin (range 9.9%-49%) and cephalosporin (range 10.

264 proven, suspected, or possible IgE-mediated penicillin reactions (N = 838), the incidence of any typ

265 positive results on skin tests to at least 1 penicillin reagent underwent skin tests with aztreonam a

266 ed-reading skin test responses to at least 1 penicillin reagent.

267 d the percentage of isolates with high-level penicillin resistance from cultures taken from children

268 nually, and rates of methicillin-susceptible penicillin-resistant S. aureus (MSSA) did not change.

269 , including 31 penicillin-susceptible and 31 penicillin-resistant strains, were retrospectively revie

270 am-positive bacteria (e.g., MRSA, VRE, PRSP (penicillin-resistant Streptococcus pneumoniae)); however

271 tered either in T1 or T2, on the carriage of penicillin-resistant VGS.

272 In addition, the clonal spread of penicillin-resistant/intermediate phenotypes and a novel

273 ceptible to ciprofloxacin, erythromycin, and penicillin, respectively.

274 antifungal susceptibility testing of FLC and penicillin revealed their resistance pathways are merged

275 Use of vancomycin, penicillin, rifampin, and linezolid was associated with

276 triaxone, doxycycline, linezolid, meropenem, penicillin, rifampin, tetracycline, trimethoprim-sulfame

277 The latter function is penicillin sensitive and assigned to a penicillin-bindin

278 a documented penicillin allergy who require penicillin should be tested during hospitalization given

279 Penicillin skin testing (PST) with or without oral amoxi

280 n 3 periods: (1) standard of care (SOC), (2) penicillin skin testing (ST), and (3) computerized guide

281 Of the subset of patients with positive penicillin skin tests (n = 295), only 1 had a hypersensi

282 e orally with vancomycin or a combination of penicillin, streptomycin, and gentamicin (PSG) and then

283 vastly more complex antibiotics from nature: penicillin, streptomycin, tetracycline, and erythromycin

284 studies, cultured in media supplemented with penicillin-streptomycin (PenStrep) or vehicle control.

285 rates but maintain catalytic competency with penicillin substrates.

286 S. aureus isolates from 31 U.S. centers were penicillin susceptible.

287 om S. lugdunensis was isolated, including 31 penicillin-susceptible and 31 penicillin-resistant strai

288 Only 3/31 patients with penicillin-susceptible isolates were treated with a peni

289 Penicillin-susceptible S. aureus (PSSA) increased by 6.1

290 eta-lactamase positive and were resistant to penicillin, tetracycline, and ciprofloxacin.

291 ture review, suggests that extended-spectrum penicillins, tetracycline, and trimethoprim-sulfamethoxa

292 trial producer of the beta-lactam antibiotic penicillin, the most commonly used drug in the treatment

293 known strains of GAS are still sensitive to penicillin, there have been reports of antibiotic treatm

294 For penicillin, three of five E. faecium strains but none of

295 ive services include daily oral prophylactic penicillin up to the age of 5 years, annual transcranial

296 Post-AAT, prescribing of narrow-spectrum penicillins was more likely (adjusted odds ratio [aOR],

297 the best example, for which a single dose of penicillin (which literally costs pennies and that we ha

298 imum extraction sensitivity for the selected penicillins, which were analysed using an RP-HPLC method

299 with documented delayed hypersensitivity to penicillins who especially require them.

300 e "gold standard," the sensitivities of CLSI penicillin zone edge and nitrocefin-based tests for beta