ライフサイエンスコーパス: penicillin G

1 -lactam antibiotics (amoxicillin, oxacillin, penicillin G).

2 MIPs against Z-L-Phe, Z-L-glutamic acid, and penicillin G.

3 D)Ala-thioacetic acid [Bz-(D)Ala-(S)Gly] and penicillin G.

4 e does have a significant role in binding of penicillin G.

5 llin and, with the exception of two strains, penicillin G.

6 ctances were reduced by picrotoxin, zinc, or penicillin-G.

7 PBP2a acylation reaction, the value of K(m) (penicillin G) = 0.5 +/- 0.1 mM and kcat = 1 x 10(-3) s-1

8 significant matrix effects, quantitation of penicillin G, a common antimicrobial, is possible in pla

9 Penicillin G, a transpeptidation inhibitor, had no effec

10 The crystal structure of penicillin G acylase from Escherichia coli has been dete

11 y processing precursor mutant (Thr263Gly) of penicillin G acylase from Escherichia coli, which reveal

12 n G to 6-aminopenicillanic acid catalyzed by Penicillin G acylase in miniaturized stirred batch react

13 Penicillin G acylase is a periplasmic protein, cytoplasm

14 in control conditions and following systemic penicillin-G administration to adult cats.

15 or all stages of syphilis remains parenteral penicillin G, although the preparation, dose, route of a

16 pigrum isolates were in-vitro susceptible to penicillin G, amoxicillin, doxycycline, rifampicin and g

17 ventional beta-lactam antibiotics, including penicillin-G, amoxicillin, ampicillin, and cefazolin, ar

18 uction of beta-lactamase was correlated with penicillin G, ampicillin, and ampicillin-sulbactam MICs

19 nst seven different beta-lactam antibiotics (penicillin G, ampicillin, cephalothin, cefaclor, cefurox

20 as the W82A mutant reduced ring-expansion of penicillin G (an "unnatural" substrate).

21 re approximately the same as K(m) values for penicillin G and ampicillin found in the literature (~30

22 production correlated with susceptibility to penicillin G and ampicillin.

23 ctamase-negative strains were susceptible to penicillin G and ampicillin; all beta-lactamase-positive

24 ) pH dependencies for acylation of PBP 2x by penicillin G and Bz-(D)Ala-(S)Gly are identical, suggest

25 tant selectively catalyzed ring-expansion of penicillin G and had improved kinetic parameters (K(m) =

26 mined in its native form and in complex with penicillin G and penicillin G sulphoxide.

27 eractions of beta-lactam antibiotics such as penicillin-G and cefotaxime with normal, penicillin-susc

28 rate constants for the antibiotics imipenem, penicillin G, and ceftriaxone.

29 e, flutamide, flufenamic acid, the K salt of penicillin G, and form 4 of the drug 4-[4-(2-adamantylca

30 se in resistance to methicillin, cefotaxime, penicillin G, and nafcillin.

31 six antibiotics (clindamycin, ciprofloxacin, penicillin-G, and trimethoprim).

32 red-emitting dye with properly produced anti-penicillin G antibodies.

33 cure rates for those treated with benzathine penicillin G at a dosage of 2.4 million units administer

34 nts for early syphilis: 2.4 million units of penicillin G benzathine and that therapy enhanced with a

35 tly recommended, single-dose alternatives to penicillin G benzathine are available for treatment of i

36 doubts about the adequacy of the recommended penicillin G benzathine therapy for early syphilis.

37 avuligerus NP1, we have been able to convert penicillin G (benzylpenicillin) to deacetoxycephalospori

38 mmend the use of a single dose of benzathine penicillin G (BPG) for treating early syphilis in human

39 .4 million units of intramuscular benzathine penicillin G (BPG) is recommended for the treatment of e

40 Benzathine penicillin G (BPG) is the only recommended treatment to

41 ies; however, few receive optimal benzathine penicillin G (BPG) therapy to prevent disease progressio

42 (Delta310 mutant) enhanced ring-expansion of penicillin G by approximately 2-fold.

43 a significant role in the ring expansion of penicillin G by resting cells or cell-free extracts.

44 AmpH bound strongly to penicillin G, cefoxitin, and cephalosporin C; was temper

45 The proposed method is based penicillin G conjugate labeled with red-emitting dye wit

46 lin), sodium dodecyl sulfate (+control), and penicillin-G (-control).

47 The deacylation rate constant for the PBP2a-penicillin G covalent complex was found to be 5.7 +/- 1.

48 mainstay of syphilis treatment is parenteral penicillin G despite the relatively modest clinical tria

49 posure to 20 microg/ml (27 times the MIC) of penicillin G for 6 h and 24 h, respectively.

50 orally was equivalent to that of benzathine penicillin G for the treatment of early syphilis in pers

51 vidence for a two-step acylation of PBP2x by penicillin-G has been demonstrated, and the dissociation

52 icillin determinants, all being positive for penicillin G in ICT.

53 rent analytical methods used to quantify the penicillin G in milk are based on HPLC, mass spectrometr

54 Detection of penicillin G in milk is of interest because of the wide

55 nsing approach for detecting the presence of penicillin G in milk.

56 ri is not eliminated by therapeutic doses of penicillin G; in contrast, doxycycline is effective.

57 Penicillin G, included in the endothelial cell growth me

58 industrially important metabolites urea and penicillin G is demonstrated.

59 ptomycin/ml of drinking water and 1,500 U of penicillin G/ml for 4 days and then ingested 10(7) CFU o

60 agents ceftiofur, enrofloxacin, florfenicol, penicillin G-novobiocin, pirlimycin, premafloxacin, and

61 Increased resistance to BACs and penicillin G of the two BACs-exposed communities is pred

62 bolite of ceftiofur), ampicillin, cefazolin, penicillin G, oxacillin, cloxacillin, naficillin, and di

63 of streptomycin (SM), tetracycline (TC), and penicillin G (PC-G) in milk.

64 uscular injection with 66,000 IU of procaine penicillin G per kg of body weight on days 8 to 10 (grou

65 Ampicillin (AMP), penicillin G (PG), penicillin V (PV), oxacillin (OXA), c

66 lactams, such as cephalothin, meropenem, and penicillin G, proceed through an electronically similar

67 of clinical and soil-derived strains reveal penicillin G resistance in 2 to 16% of isolates tested.

68 Following administration of penicillin-G, RF stimulation during nonREM sleep evoked

69 e of the amide bond in the benzylpenicillin (penicillin G) side-chain to produce phenylacetic acid an

70 All compounds but penicillin-G significantly slowed contraction in a dose-

71 ith respect to the microbial inhibition of a penicillin G standard.

72 llular response of Escherichia coli cells to penicillin G-streptomycin and cefazolin.

73 ve form and in complex with penicillin G and penicillin G sulphoxide.

74 complex with the slowly processed substrate penicillin G sulphoxide.

75 ll as three clinically relevant antibiotics (penicillin G, tetracycline, ciprofloxacin).

76 gests that during the acylation of PBP 2x by penicillin G the inherent chemical stability of penicill

77 anges during the enzymatic transformation of Penicillin G to 6-aminopenicillanic acid catalyzed by Pe

78 Procaine penicillin G was given as 12 h to one-half of the animal

79 p-49 and Phe-142, mimic interactions made by penicillin G when bound in the active site of TEM-1.

80 49) and Phe(142), mimic interactions made by penicillin G when bound in the beta-lactamase active sit

81 develop chromosomally mediated resistance to penicillin G, which for over 40 years was used to treat

82 markedly diminished the deacylation rate of penicillin G with a minimal impact on acylation, and abo

83 lar injection of 2.4 million U of benzathine penicillin G, with studies reporting 90% to 100% treatme