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1 f WT-L1 adhesion was between 1-butanol and 1-pentanol.
2 esis of the 6-carbon alcohol, (S)-3-methyl-1-pentanol.
3 pentanol was 7.5 times higher than for (R)-2-pentanol.
4 from the boron complex upon treatment with 1-pentanol.
5 tane, n-hexane, n-heptane, 1 -butanol, and 1-pentanol.
6 2-hexanol, benzoic acid, benzaldehyde, and 1-pentanol.
8 -phenylethanol (PEA), 2-butanol (BUT), and 2-pentanol (2-PEN) with the highest ee up to 82.4% but als
11 ng natural E. coli metabolites into 4-methyl-pentanol (4MP) via coenzyme A (CoA)-dependent chemistry
12 evels of Oscillospira and higher levels of 1-pentanol and 2-butanone in NAFL patients compared to CTR
14 At optimized conditions (FLMs consisted of 1-pentanol and extraction solutions of deionized water and
17 ane gave, after protonolysis, (R)-3-methyl-1-pentanol as well as (R)- and (S)-3-methyl-1-hexanols in
18 for the alkane solutes), the saturation by 1-pentanol causes a dramatic increase of the alcohol parti
19 ropionate, trans-2-pentenoate, valerate, and pentanol, compounds with applications that include biofu
20 , 2,3 pinanediol, 3-oxo-ionol and 3-methyl-1-pentanol, coumaran and damascenone; in Rondinella, hexan
21 ids, namely cetyl trimethylammonium chloride/pentanol/D(2)O and sodium lauryl sulfate/octanol/brine l
22 ants (pentanoic acid, methyl pentanoate, and pentanol) did not recruit new areas of glomerular activa
23 ry were 2-amylfuran, (E/Z)-2,6-nonadienal, 1-pentanol, epoxylinalool, (Z)-jasmone, 2-acetylpyrrole, f
24 on cell-cell adhesion were antagonized by 1-pentanol (IC(50) = 715 microM) and 1-octanol (IC(50) = 3
25 ary phase is very small, both n-hexane and 1-pentanol partition strongly into and thereby swell the s
29 alkane solutes for both the n-hexane- and 1-pentanol-swollen stationary phases, the effects on the a
31 ohols (ethanol, 1-propanol, 1-butanol, and 1-pentanol) to the corresponding carboxylates was carried
32 for (R)-2-butanol, while the K(m) for (S)-2-pentanol was 7.5 times higher than for (R)-2-pentanol.
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