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1 dead cells was saturated by the addition of pentosan polysulfate.
2 g, subcutaneously, in olive oil)/5% glucose, pentosan polysulfate (10 mg/kg, subcutaneously in 5% glu
3 scrapie protease-resistant PrP accumulation, pentosan polysulfate and a porphyrin compound, indium (I
4 ytic digestion of protamine, and polyanions (pentosan polysulfate and heparin) based on the strong bi
6 , unlabeled heparan sulfate, and heparin and pentosan polysulfate, but not in the presence of chondro
12 lfate = dermatan sulfate = dextran sulfate > pentosan polysulfate, implicating the importance of the
13 spectropolarimetry measurements showed that pentosan polysulfate induced a conformational change in
14 vCJD) have been treated with intraventicular pentosan polysulfate (iPPS) and extended survival has be
15 In this study, we examined the effect of pentosan polysulfate on the development of CsA nephropat
16 r U-73122, by blocking the IP3 receptor with pentosan polysulfate or heparin, and by buffering intrac
17 taneously in 5% glucose) plus CsA, olive oil/pentosan polysulfate, or olive oil/5% glucose for 30 day
18 om normal mice was used to determine whether pentosan polysulfate (PPS) inhibited proliferation and a
20 of the glycosaminoglycan (GAG)-like molecule pentosan polysulfate (PPS) to alleviate virus-induced ar
25 Stepwise dosing (5 to 60 mug/m(2)/day) plus pentosan polysulfate SP54 (n = 3) resulted in no treatme
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