戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 sugar enones (dihydropyranones) derived from pentoses.
2 d for seven out of eight possible isomers of pentoses.
3 f a variety of C-nucleosides and fluorinated pentoses.
4 ation of the C(5) osone, D-xylosone (D-threo-pentose-2-ulose), showing that this transposition may be
5                                 However, the pentose analogue fructose can also enter glycolysis thro
6 tic oligomannose structures, the presence of pentose and deoxyhexose residues on their N-glycans.
7  protein is more closely related to archaeal pentose and glucose phosphomutases than to bacterial glu
8  and aldarate metabolism (P=9.0x10(-6)), and pentose and glucuronate interconversions (P=3.0x10(-6))
9  phenylpropanoid and flavonoid biosynthesis, pentose and glucuronate interconversions and starch and
10  of microorganisms to efficiently co-ferment pentose and hexose sugars, especially glucose and xylose
11  site and prevents release of the UDP-4-keto-pentose and NADH intermediates.
12 etric amounts of C2 metabolites from hexose, pentose and triose phosphates without carbon loss.
13 toses), SnHypSys II (20 amino acids with six pentoses), and SnHypSys III (20 amino acids with either
14 cellular membranes to water, five of the six pentoses, and selected aldohexoses, ketohexoses, and thr
15 and purification of nine SIL-IS for hexose-, pentose-, and triose-phosphates, UDP-glucose, and adenos
16 due to the relative stability of diglycoside pentose anthocyanins.
17                         Specifically, the 12 pentoses are much more structurally similar to one anoth
18 atalyze retro-aldol reactions of hexoses and pentoses at these moderate temperatures, are shown to be
19  III (20 amino acids with either six or nine pentoses) by their sequential appearance in SnpreproHypS
20 C(4)H(8)O(4)), heptoses (C(7)H(14)O(7)), and pentoses (C(5)H(10)O(5)), including the ribose found in
21 re and smaller energetic differences between pentose conformations than between hexose conformations.
22 inor reaction products such as furfural from pentose dehydration.
23 fficient route towards biologically relevant pentose derivatives is described.
24 ange of aldose sugars, encompassing hexoses, pentoses, disaccharides, and trisaccharides, and is able
25 de range of oligosaccharidic combinations of pentose, hexose, deoxyhexose and hexuronic acid were acc
26                           Many components of pentoses, hexoses and disaccharides were identified usin
27 cid of lipid components.) Many components of pentoses, hexoses and disaccharides were identified usin
28  member of the LacS subfamily of galactoside-pentose hexuronide translocators was identified for upta
29 olyol derived from xylose, the most abundant pentose in lignocellulosic biomass.
30 ides and arabinose, the second most abundant pentose in nature.
31           Here, we show that non-fermentable pentoses inhibit growth and end-product formation during
32  UDP-4-keto-D-glucuronic acid and UDP-4-keto-pentose intermediates.
33 outes to convert biomass-derived hexoses and pentoses into valuable C2, C3, and C4 products such as g
34 hexose isomers, the chiral separation of the pentose isomers was significantly more challenging.
35 solute configuration determination of all 12 pentose isomers, including the d and l enantiomers for a
36 ously described xylans, this xylan carries a pentose linked 1-2 to the alpha-1,2-d-glucuronic acid (G
37 ified by the addition of essential genes for pentose metabolism.
38 pplied to the identification of any isolated pentose monosaccharide using only microgram quantities a
39 ished inhibitors of NAPDH production via the pentose pathway (dehydroandrostenedione or norepinephrin
40 nstrated a metabolic interconnection between pentose pathway and 11 beta-HSD1 oxo-reductase activitie
41 ids, which together sustain the nonoxidative pentose pathway for purine synthesis.
42 stem and its main reducing power source, the pentose pathway, as demonstrated by a 50 and 70% drop in
43 H) activity, the key enzyme of the oxidative pentose pathway.
44 ed by one or two CQAs covalently linked with pentose (Pent) residues (1-12) were identified, along wi
45 s a cyclic route that includes the oxidative pentose phosphate (OPP) pathway and the glucose-6-phosph
46 rhof-Parnas (EMP) pathway] and the oxidative pentose phosphate (OPP) pathway.
47 -Benson-Bassham (CBB) cycle, glycolysis, the pentose phosphate (PP) pathway and the tricarboxylic aci
48 e Entner-Doudoroff pathway and the oxidative pentose phosphate (PP) pathway.
49 ose catabolic pathways including glycolysis, pentose phosphate (PP), glycogenolysis, and polyols to t
50 irects the glycolytic intermediates into the pentose phosphate (PPP) and serine pathways.
51  glycolysis, in association with a decreased pentose phosphate activity and an increased ATP producti
52 in diabetic CPCs showed dysregulation of the pentose phosphate and glycero(phospho)lipid synthesis pa
53 ndance of enzymes in the glycolytic pathway (pentose phosphate and pyruvate metabolism) was further v
54  of compounds, multiple metabolites from the pentose phosphate and tryptophan metabolic pathways were
55                                The oxidative pentose phosphate cycle (OPPC) is necessary to maintain
56 after PH, and glucose use was shifted to the pentose phosphate cycle and oxidative metabolism.
57 ogenesis and reduced glucose use through the pentose phosphate cycle and oxidative metabolism.
58 egulation of glycolysis and the nonoxidative pentose phosphate cycle.
59 tradiol led to a significant upregulation in pentose phosphate flux and glycolytic intermediates in H
60 lycolysis by increasing glutamine uptake and pentose phosphate flux to generate energy and biomass.
61 s with HKCs showing consistently lower basal pentose phosphate flux.
62  showing that HEXOKINASE1-mediated oxidative pentose phosphate pathway (OPPP) metabolism is required
63                                The oxidative pentose phosphate pathway (OPPP) provides plants with im
64 e in the flux of the oxidative branch of the pentose phosphate pathway (ox-PPP) in response to the PD
65  that inhibition of the oxidative arm of the pentose phosphate pathway (oxPPP) is required for antima
66  a dependence on the oxidative branch of the pentose phosphate pathway (oxPPP), and oxPPP inhibition
67 genation elevates glycolytic flux and lowers pentose phosphate pathway (PPP) activity in mammalian er
68                         A sudden increase in pentose phosphate pathway (PPP) activity, the fastest kn
69  dehydrogenase (G6PD) is a key enzyme in the pentose phosphate pathway (PPP) and plays an essential r
70 R-ABL-ERK signaling in H929 cells drives the pentose phosphate pathway (PPP) and RNA biosynthesis, wh
71  two cytosolic NADPH-producing pathways, the pentose phosphate pathway (PPP) and the NADP-dependent m
72 ADP(+)/NADPH ratio controls flux through the pentose phosphate pathway (PPP) and the polyol pathway,
73 and miR-206 to direct carbon flux toward the pentose phosphate pathway (PPP) and the tricarboxylic ac
74 d and carbohydrate metabolites, particularly pentose phosphate pathway (PPP) and tricarboxylic acid (
75 cent case reports suggest a link between the pentose phosphate pathway (PPP) enzyme transaldolase (TA
76 y mediating fluxes through glycolysis or the pentose phosphate pathway (PPP) in an oxidative stress-d
77 increased glucose flux through the oxidative pentose phosphate pathway (PPP) in erythrocytes.
78                                The oxidative pentose phosphate pathway (PPP) is crucial for cancer ce
79 emonstrate that a metabolic shift toward the pentose phosphate pathway (PPP) is necessary for NET rel
80                              Here we add the pentose phosphate pathway (PPP) of T. brucei to the glyc
81                                          The pentose phosphate pathway (PPP) plays a critical role in
82 on increases the flux of glucose through the pentose phosphate pathway (PPP) to increase nucleotide p
83 -phosphogluconate dehydrogenase (PGD) in the pentose phosphate pathway (PPP) were found to be the NAD
84 6PD) is the first, rate-limiting step in the pentose phosphate pathway (PPP), a key metabolic pathway
85                                          The pentose phosphate pathway (PPP), metabolism in the citri
86 revealed that metabolites in the glycolysis, pentose phosphate pathway (PPP), pyrimidine biosynthesis
87  potential by shunting nectar glucose to the pentose phosphate pathway (PPP), resulting in a reductio
88 sphogluconate dehydrogenase in the oxidative pentose phosphate pathway (PPP), while 2-PG activates 3-
89 e metabolism and increased NADPH levels in a pentose phosphate pathway (PPP)-dependent manner.
90  of ribose, leading us to hypothesize that a pentose phosphate pathway (PPP)-responsive regulator med
91 lase (TKT), a key enzyme of the nonoxidative pentose phosphate pathway (PPP).
92 with H2O2 rapidly induces glycolysis and the pentose phosphate pathway (PPP).
93 cts carbon flux from the Calvin cycle to the pentose phosphate pathway (PPP).
94 s an anti-oxidant response by regulating the pentose phosphate pathway (PPP).
95 ylic acid cycle and the oxidative arm of the pentose phosphate pathway (PPP).
96 cose flux towards glycolysis relative to the pentose phosphate pathway (PPP).
97 C-null strains show indications of oxidative pentose phosphate pathway activation as well as increase
98                           Fasting stimulated pentose phosphate pathway activity and metabolism of [U-
99 that CP12 is essential to separate oxidative pentose phosphate pathway activity from Calvin-Benson cy
100              NADPH, a substrate for NOX, and pentose phosphate pathway activity increased with glucos
101 ose uptake, glucose metabolism and oxidative pentose phosphate pathway activity were similarly repres
102 tabolites provides an important biomarker of pentose phosphate pathway activity, triacylglycerol synt
103 sponse in the clinic and of monitoring tumor pentose phosphate pathway activity.
104 nd most abundant sugar in nature, is via the pentose phosphate pathway after a two-step or three-step
105 rovided the first link between the bacterial pentose phosphate pathway and activation of host IFN-bet
106 cemia by increasing glucose flux through the pentose phosphate pathway and enhancing fatty acid synth
107                                fluxes in the pentose phosphate pathway and gluconeogenesis were stabl
108 th a shift in glucose metabolism between the pentose phosphate pathway and glycolysis, (2) interactio
109 between the oxidative steps of the oxidative pentose phosphate pathway and glycolysis.
110 ection against hydrogen peroxide insult in a pentose phosphate pathway and GSH-dependent manner.
111 a fructose-2,6-bisphosphatase, promoting the pentose phosphate pathway and helping to lower intracell
112 yrophosphate production by the non-oxidative pentose phosphate pathway and late steps by modulating a
113 nsated by activation of anabolic metabolism (pentose phosphate pathway and lipogenesis) allowing live
114 glutathione biosynthesis or in the oxidative pentose phosphate pathway and other NADPH-producing enzy
115 of genes associated with photosynthesis, the pentose phosphate pathway and primary metabolism, but lo
116 ls further showed enhanced activation of the pentose phosphate pathway and the glutathione system, wh
117 se (G6PD) is the rate-limiting enzyme of the pentose phosphate pathway and the principal source of NA
118  is required to divert glucose flux into the pentose phosphate pathway and thereby generate sufficien
119 biosynthetic genes and discrete steps in the pentose phosphate pathway and tricarboxylic acid cycle t
120 ase step, promoting carbon overflow into the pentose phosphate pathway as evidenced by the increased
121 s of NADPH reflecting a higher activation of pentose phosphate pathway as this is accompanied with hi
122 nclude up-regulated NADPH production via the pentose phosphate pathway as well as activation of the N
123 TORC1 directs increased glucose flux via the pentose phosphate pathway back into glycolysis, thereby
124                           Glycolysis and the pentose phosphate pathway both play a central role in th
125 er occurs through diverting glucose into the pentose phosphate pathway by ADPr inhibition of glyceral
126                            Inhibition of the pentose phosphate pathway by glucose-6-phosphate dehydro
127   Adiponectin also reversed induction of the pentose phosphate pathway by HFD.
128 ne phosphate and fructose-1,6-bisphosphate), pentose phosphate pathway components and Kreb's cycle in
129 non-photosynthetic conditions, the oxidative pentose phosphate pathway contributes to basic metabolis
130                                     Further, pentose phosphate pathway deregulation and impaired fatt
131 uiescent mutant 1 (NQM1), a paralogue to the pentose phosphate pathway enzyme transaldolase (TAL1), a
132  3.6% of glycolysis, and three non-oxidative pentose phosphate pathway exchange fluxes were calculate
133                                The oxidative pentose phosphate pathway flux was 3.6% of glycolysis, a
134  associated with a corresponding increase in pentose phosphate pathway flux, assessed using (13)C-lab
135 owever, LPS also induced a small decrease in pentose phosphate pathway fluxes and an increase in glut
136 e identified increased citric acid cycle and pentose phosphate pathway fluxes as consistent markers o
137 ol g(-1)h(-1) that was partly caused by high pentose phosphate pathway fluxes.
138 Embden-Meyerhof-Parnas, Entner-Doudoroff, or pentose phosphate pathway for glycolytic carbon metaboli
139                                  Loss of the pentose phosphate pathway had no effect on plaque format
140 rains cell proliferation, glycolysis and the pentose phosphate pathway in a catalytic-activity-indepe
141 x from glucose-6-phosphate (G6P) through the pentose phosphate pathway in egg extracts maintains NADP
142 te) was associated with perturbations in the pentose phosphate pathway induced initially by colistin
143 ogenase (H6PD) is the initial component of a pentose phosphate pathway inside the endoplasmic reticul
144  the two-step conversion of glucose into the pentose phosphate pathway intermediate 6-phosphogluconat
145 sphate cyclases are enzymes that utilize the pentose phosphate pathway intermediate, sedoheptulose 7-
146 chment of Embden-Meyerhof-Parnas pathway and pentose phosphate pathway intermediates indicated high a
147 ted glucose uptake and metabolism, increased pentose phosphate pathway intermediates, with a complime
148 rangements in the nonoxidative branch of the pentose phosphate pathway involving transaldolase that p
149                                          The pentose phosphate pathway is a key route of glucose meta
150                                          The pentose phosphate pathway is a major site of NADPH produ
151 er these conditions, NADPH generation by the pentose phosphate pathway is impaired, but AMPK induces
152                       Although the oxidative pentose phosphate pathway is important for tumor growth,
153  establish that the carbon overflow into the pentose phosphate pathway is mainly through its non-oxid
154 ial amounts of NADPH and do so even when the pentose phosphate pathway is operational.
155 in carbohydrate or hydroxyl acid metabolism, pentose phosphate pathway metabolites, or free fatty aci
156 ion and may enhance glucose turnover via the pentose phosphate pathway rather than through glycolysis
157 rnative route for directing glucose into the pentose phosphate pathway that bypasses hexokinase and t
158 otransposition, apoptosis, and the oxidative pentose phosphate pathway that these genes are involved
159 tes shunting of glucose-6-phosphate into the pentose phosphate pathway to generate reduced glutathion
160 hosphate is then converted by enzymes of the pentose phosphate pathway to glyceraldehyde 3-phosphate
161 ydrogenase, the TCA cycle, and the oxidative pentose phosphate pathway to obtain NADPH.
162 ,6-bisphosphatase, potentially promoting the pentose phosphate pathway to produce NADPH for antioxida
163 nverted by known enzymes of the nonoxidative pentose phosphate pathway to ribose-5-phosphate.
164 ferentially metabolized via the nonoxidative pentose phosphate pathway to synthesize nucleic acids an
165 pression of lipogenesis, glycolysis, and the pentose phosphate pathway triggered a strong growth rest
166  showed that: (i) flux through the oxidative pentose phosphate pathway varied independently of the re
167                            Activation of the pentose phosphate pathway was identified from the list o
168 and ribulose 5-phosphate 3-epimerase) in the pentose phosphate pathway were overexpressed, and a gera
169 ncluding glycerol oxidation, glycolysis, the pentose phosphate pathway, and carbon sources of stored
170 cluding glycolysis, the oxidative arm of the pentose phosphate pathway, and de novo lipid biosynthesi
171 ses including fatty acid esterification, the pentose phosphate pathway, and gluconeogenesis through t
172 lism, reactive oxygen species clearance, the pentose phosphate pathway, and glutathione homeostasis.
173 ical pathways, such as glycolysis, oxidative pentose phosphate pathway, and glycogen metabolism, were
174 ycolysis, through the oxidative/nonoxidative pentose phosphate pathway, and into the general phenylpr
175 way, nucleotide sugars, intermediates of the pentose phosphate pathway, and lipogenesis, including pr
176 through up-regulation of glucose influx, the pentose phosphate pathway, and NAD salvaging pathways.
177  for the complete non-oxidative phase of the pentose phosphate pathway, and others predicted to media
178 rom erythrose 4-phosphate, generated via the pentose phosphate pathway, and phosphoenolpyruvate.
179 tion of glucose flux between glycolysis, the pentose phosphate pathway, and serine biosynthesis seems
180 ed reduced glucose metabolism in glycolysis, pentose phosphate pathway, and sorbitol pathway, which m
181 r phosphates that constitute glycolysis, the pentose phosphate pathway, and the RNA and DNA backbone,
182 ent decrease in flux through glycolysis, the pentose phosphate pathway, and the tricarboxylic acid (T
183 h glycolysis and the oxidative branch of the pentose phosphate pathway, as well as to stimulate de no
184     Its major source is considered to be the pentose phosphate pathway, but cytosolic NADP(+)-depende
185 te of the cells with regard to the oxidative pentose phosphate pathway, Calvin cycle, tricarboxylic a
186 tration of metabolites of glycolysis and the pentose phosphate pathway, central metabolic players in
187  Idn1 act together to shunt glucose into the pentose phosphate pathway, creating an alternative route
188 lycolysis, the tricarboxylic acid cycle, the pentose phosphate pathway, fatty acid and nucleotide bio
189 lytic RA T cells diverted glucose toward the pentose phosphate pathway, generated more NADPH, and con
190 d zwf1, which encode the first enzyme in the pentose phosphate pathway, have a more severe growth phe
191 Fru-2,6-BPase), and through promotion of the pentose phosphate pathway, increases NADPH production to
192 ase (Tps1) is responsible for regulating the pentose phosphate pathway, intracellular levels of NADPH
193 metabolism of glucose from glycolysis to the pentose phosphate pathway, it was hypothesized to have a
194 products of glycolysis, the Krebs cycle, the pentose phosphate pathway, nucleobases, UDP-sugars, glyc
195 luding glucose and glutamine metabolism, the pentose phosphate pathway, nucleotide and fatty acid bio
196 abolites revealed a reduction in glycolysis, pentose phosphate pathway, polyamines and nucleotides, b
197 nase (G6PD), the key enzyme of the oxidative pentose phosphate pathway, provides reducing equivalents
198 rate metabolism (glycolysis/gluconeogenesis, pentose phosphate pathway, pyruvate metabolism), proteas
199 but instead of increasing the glycolysis and pentose phosphate pathway, the glucose is shunted throug
200    While NQM1 appears not to function in the pentose phosphate pathway, the interplay of NQM1 with VH
201 ently in clinical trials, on glycolysis, the pentose phosphate pathway, the tricarboxylic acid (TCA)
202 vity and redirected glucose flux through the pentose phosphate pathway, thereby conferring a selectiv
203 ase in TIGAR expression, which regulates the pentose phosphate pathway, treatment with the MUC1-C inh
204 ologs, RpiRc is a potential regulator of the pentose phosphate pathway, which also regulates RNAIII l
205 amental routes, glycolysis and the oxidative pentose phosphate pathway, which produces NADPH and the
206 th was potentiated by its suppression of the pentose phosphate pathway, which resulted in inhibition
207 xylic acid (TCA) cycle intermediates and the pentose phosphate pathway, which results in increased gl
208 hat a concerted preemptive activation of the pentose phosphate pathway, which targets both mRNA trans
209  produce NADPH from glucose is the oxidative pentose phosphate pathway, with malic enzyme sometimes a
210 nerated by metabolism of glucose through the pentose phosphate pathway, would have an anticonvulsant
211 sotope effect is only found in the reductive pentose phosphate pathway.
212 o glycolysis and to the oxidative arm of the pentose phosphate pathway.
213 te dehydrogenase (G6PDH), a regulator of the pentose phosphate pathway.
214  increase in glucose metabolic flux into the pentose phosphate pathway.
215  by high levels of nutrient flux through the pentose phosphate pathway.
216 tributor to cytosolic NADPH is the oxidative pentose phosphate pathway.
217 onance assigned to 6-phosphogluconate in the pentose phosphate pathway.
218 o alterations in glucose metabolized via the pentose phosphate pathway.
219 by the Calvin-Benson-Bassham (CBB) reductive pentose phosphate pathway.
220 metabolism, including several mRNAs from the pentose phosphate pathway.
221 d failed to predict the use of the oxidative pentose phosphate pathway.
222 trate for NADPH generation via the oxidative pentose phosphate pathway.
223 ydrogenase (6-PGDH), the third enzyme of the pentose phosphate pathway.
224 nvolved in starch turnover and the oxidative pentose phosphate pathway.
225 mark of transformed cells, is to support the pentose phosphate pathway.
226 ated flux through the antioxidant-generating pentose phosphate pathway.
227 pgl), which encodes the second enzyme in the pentose phosphate pathway.
228  transport, a glucokinase and enzymes of the pentose phosphate pathway.
229 y between the TGF-beta signaling pathway and pentose phosphate pathway.
230 ed for 4-carbon saccharides arising from the pentose phosphate pathway.
231 -phosphate and d-xylulose 5-phosphate in the pentose phosphate pathway.
232  carbon dioxide assimilation via a reductive pentose phosphate pathway.
233 cycle and its upstream contributors, and the pentose phosphate pathway.
234 e groups comprising either glycolysis or the pentose phosphate pathway.
235 fts from primarily glycolytic to include the pentose phosphate pathway.
236 arboxylic acid (TCA) cycle and an incomplete pentose phosphate pathway.
237 ion of the reduced form of NADPH through the pentose phosphate pathway.
238 e ATP generating roles of glycolysis and the pentose phosphate pathway.
239 ne metabolism, glutamate metabolism, and the pentose phosphate pathway.
240  metabolism in the non-oxidative part of the pentose phosphate pathway.
241  glucose-6-phosphate dehydrogenase or in the pentose phosphate pathway.
242  focused on the importance of glycolysis and pentose phosphate pathway.
243 asing dependence upon glutaminolysis and the pentose phosphate pathway.
244 iphosphatase 4 (PFKFB4), drives flux through pentose phosphate pathway.
245 y by glycolysis and to a minor extent by the pentose phosphate pathway.
246  disorders, we focused on the glycolytic and pentose phosphate pathways (GPPPs).
247 ediates into the hexosamine biosynthesis and pentose phosphate pathways (PPP).
248               The oxidative and nonoxidative pentose phosphate pathways and the ratio between them al
249 etabolic intermediates in the glycolytic and pentose phosphate pathways as well as abnormal mitochond
250 s to the less efficient Entner-Doudoroff and pentose phosphate pathways on algal-dominated reefs.
251 erating in a gluconeogenic fashion), and the pentose phosphate pathways to form an unforeseen metabol
252 ioning of glucose carbons into C1/folate and pentose phosphate pathways, and increased tricarboxylic
253 nas, the Entner-Doudoroff, and the reductive pentose phosphate pathways.
254 : the Embden-Meyerhof, Entner-Doudoroff, and pentose phosphate pathways.
255 s in both the oxidative and the nonoxidative pentose phosphate pathways.
256 ion (glycolysis) pathways, as well as to the pentose phosphate shunt and the hexosamine biosynthetic
257 ke for aerobic glycolysis, coupling with the pentose phosphate shunt to facilitate biosynthesis of nu
258 ding in the liver, including glycolysis, the pentose phosphate shunt, and lipogenic and sterol synthe
259 ed in glucose utilization by glycolysis, the pentose phosphate shunt, lipogenesis, and sterol synthes
260 is, and several glycolysis-related pathways (pentose phosphate, carbon fixation, aminoacyl-tRNA biosy
261                    Successful integration of pentose phosphate, nicotinamide adenine dinucleotide bio
262 ynthesized endogenously from glucose via the pentose-phosphate pathway (PPP) in stable isotope-assist
263  demonstrates an essential role of oxidative pentose-phosphate pathway reactions in peroxisomes, like
264  UDP-glucose; an alternative to the textbook pentose-phosphate pathway therefore predominates in plan
265 stressed cells protected Rpe and enabled the pentose-phosphate pathway to retain function.
266 nitrate assimilation, energy metabolism, and pentose-phosphate pathway were most affected.
267    Physiological analysis indicated that the pentose-phosphate pathway, in particular, was poisoned b
268 idation via the tricarboxylic acid cycle and pentose-phosphate pathway.
269 nd is linked through its product Xu5P to the pentose-phosphate pathway.
270 iting enzyme in the nonoxidative part of the pentose-phosphate pathway.
271 ctate even in the presence of oxygen via the pentose-phosphate pathway.
272 drolytic enzymes and also enzymes key to the pentose-phosphate pathway.
273 metabolites through the oxidative arm of the pentose-phosphate pathway.
274 idation via the TCA cycle to the glycolytic, pentose-phosphate, and glutaminolytic pathways.
275                                 We show that pentose-phosphate-pathway generation of NADPH is critica
276 perature are effective for the hydrolysis of pentose polymers in hemicellulose and also increase the
277 teen hexose, one fucose, one methyl, and two pentose residues; however, in this and most other strain
278 t in the hydrated pyrimidine ring but in the pentose ring, which is epimerized to arabinose in the mi
279 e called SnHypSys I (19 amino acids with six pentoses), SnHypSys II (20 amino acids with six pentoses
280 s demonstrated, including selectivity toward pentoses such as arabinose, ribose, and xylose to the ex
281  yeasts capable of efficient fermentation of pentoses such as xylose remains a key challenge in the p
282 DP-glucuronic acid to generate a UDP-4'-keto-pentose sugar and also catalyzes transfer of a formyl gr
283 ons with the radical at the chiral C4 of the pentose sugar and the intramolecularly C1-tethered olefi
284 stry, is characterized by high levels of the pentose sugar L-xylulose in blood and urine and deficien
285 inability of many microbes to metabolize the pentose sugars abundant within hemicellulose creates spe
286 dily ferments sugars derived from cellulose, pentose sugars from xylan are not metabolized.
287 somes, ability to rapidly ferment hexose and pentose sugars to ethanol, and ability to ferment synthe
288 ine and pyrimidine nucleotides, nucleosides, pentose sugars, and inorganic phosphate demonstrates tha
289 t competitively inhibited by ribose or other pentose sugars.
290 rment plant oligosaccharides and constituent pentose sugars.
291 mentation, operates with multiple hexose and pentose sugars.
292 f F. succinogenes to utilize non-cellulosic (pentose) sugars for growth.
293 on has been recognized for its importance in pentose synthesis and stabilization in prebiotic conditi
294 ybdate-catalyzed rearrangement of a branched pentose that is central to borate-moderated cycles that
295 l up-regulated genes potentially involved in pentose transport and metabolism, which were targeted fo
296 minimal energetic differences between the 12 pentoses, two unique fixed ligand kinetic method combina
297 gs build toward the engineering of efficient pentose utilization in yeast and provide a blueprint for
298 of other metabolic pathways, including sugar pentoses utilization and biogenesis of phosphoribosyl py
299 nal insights needed for engineering improved pentose utilizing strains of C. thermocellum and reveal
300 ains of Saccharomyces cerevisiae can use the pentose xylose, the fermentative capacity pales in compa

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。
 
Page Top