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1 clinical outcomes (i.e., gastric cancer and peptic ulceration).
2 Total pancreatectomy was associated with peptic ulceration.
3 et of diabetes, body weight maintenance, and peptic ulceration.
4 ific vacA genotypes, cytotoxin activity, and peptic ulceration.
5 strains possessing cagA are associated with peptic ulceration.
6 helial injury (assessed histologically), and peptic ulceration.
7 te to the pathogenesis of gastric cancer and peptic ulceration.
8 to be a major cause of gastric carcinoma and peptic ulceration.
9 ons for the divergent trends in incidence of peptic ulceration and apparent trends in diagnosis of up
10 ylori is the strongest known risk factor for peptic ulceration and distal gastric cancer, and adheren
13 egative bacterium associated with gastritis, peptic ulceration, and gastric adenocarcinoma in humans,
14 Helicobacter pylori is the main cause of peptic ulceration, distal gastric adenocarcinoma, and ga
15 ication in patients with current or previous peptic ulceration, dyspepsia, or both who continued to u
16 the host environment, increasing the risk of peptic ulceration, gastric adenocarcinoma, and possibly
17 osa, which is associated with development of peptic ulceration, gastric atrophy, and gastric adenocar
18 pylori, an etiologic agent of gastritis and peptic ulceration in humans, synthesizes urease, a nicke
19 lly underlies the perceived relative lack of peptic ulceration in patients affected by cystic fibrosi
20 H. pylori infection is not a risk factor for peptic ulceration in patients with Zollinger-Ellison syn
21 ers an increased risk for the development of peptic ulceration, noncardia gastric adenocarcinoma, and
22 ith Helicobacter pylori is a risk factor for peptic ulceration, noncardia gastric adenocarcinoma, and
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