ライフサイエンスコーパス: peptidase inhibitor

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1 indicates that it represents a new family of peptidase inhibitors.

2 behavioral and neurochemical effects of the peptidase inhibitors.

3 a subtractive strategy using class-specific peptidase inhibitors.

4 a mechanism that was partially prevented by peptidase inhibitors.

5 omain related to the Kunitz family of serine peptidase inhibitors.

6 We found that four splice variants of the peptidase inhibitor 16 (PI16) mRNA are among the most hi

7 CCL20, S100A7/A8/A9, DEFB4, lipocalin 2, and peptidase inhibitor 3 (p < 0.05), indicating a positive

8 kines (IL-12p40, IL-13, IL-22, CCL17, CCL18, peptidase inhibitor 3 [PI3]/elafin, and S100As) showed c

9 two negative control genes (cytochrome b and peptidase inhibitor 3) show no significant association.

10 (ie, IL-13, IL-22, CCL17, S100As, and elafin/peptidase inhibitor 3), and modulation of epidermal hype

11 imicrobials (IL-17A, IL-19, CCL20, LL37, and peptidase inhibitor 3/elafin), TH9/IL-9, IL-33, and inna

12 These findings indicate that NAAG peptidase inhibitors administered post-injury can signif

13 heters that were used to inject a mixture of peptidase inhibitors (amastatin, captopril and phosphora

14 ion in addition to 19 putative peptidases, 9 peptidase inhibitors and 7 C-type lectins that may funct

15 Serpins compose the largest superfamily of peptidase inhibitors and are well known as regulators of

16 epE is not inhibited by any of the classical peptidase inhibitors, and its amino acid sequence does n

17 such as beta-lactam antibiotics, antivirals, peptidase inhibitors, and others as substrates.

18 ses and identified IL-1 cytokines and serine peptidase inhibitors as the most dysregulated esophagus-

19 (Nqo1), peroxiredoxin-4 (Prdx4), and serine peptidase inhibitor b1b (Serpinb1b) and represses the ex

20 ced MOR-1 internalization in the presence of peptidase inhibitors, but little or no internalization i

21 ngs demonstrate that the application of NAAG peptidase inhibitors can reduce the deleterious motor an

22 tentially interdependent induction of serpin peptidase inhibitor, clade A member 3 (SERPINA3) and int

23 Cela1 was covalently bound to serpin peptidase inhibitor, clade A, member 1, resulting in a h

24 mily member 1 (CLCA1), periostin, and serine peptidase inhibitor, clade B (ovalbumin), member 2 (serp

25 ride channel regulator 1 (CLCA1), and serpin peptidase inhibitor, clade B (SERPINB2).

26 mutations were found in the SERPINF1 (Serpin Peptidase Inhibitor, Clade F) gene which encodes PEDF (p

27 8 (MTRNRL8), interleukin-8 (IL8), and serpin peptidase inhibitor, clade H (SERPINH1) and chemokine li

28 rally different HsPDF inhibitors and control peptidase inhibitors confirmed that inhibition of HsPDF

29 Attempts to base dd-peptidase inhibitor design on peptidoglycan structure, h

30 red on apolipoprotein E, jun, leptin, serpin peptidase inhibitor E type 1 and peroxisome proliferator

31 Used in combination with the cysteine peptidase inhibitor E-64, these substrates were able to

32 also differs from human in resisting tryptic peptidase inhibitors (e.g., aprotinin), while favoring a

33 ntimicrobial peptide and secretory leukocyte peptidase inhibitor genes.

34 ue secretory signal peptide and a 90-residue peptidase inhibitor I9 domain in addition to the 283-res

35 es to show the involvement of peptidases and peptidase inhibitors in biological pathways have been cr

36 f small-molecule inhibitors in the tables of peptidase-inhibitor interactions, a table of known cleav

37 along with the known substrate cleavages and peptidase-inhibitor interactions, and a link to the KEGG

38 tion of known substrate cleavages; tables of peptidase-inhibitor interactions; and dynamically genera

39 o synthesize both enantiomers of an aspartic peptidase inhibitor intermediate, 3-hydroxy-4-phenylpipe

40 Each page shows the peptidases and peptidase inhibitors involved in the pathway, along with

41 or-knockout lines for the ecotin-like serine peptidase inhibitor (ISP2; Deltaisp2/isp3), an inhibitor

42 Missense mutations in the serine peptidase inhibitor Kazal type 5 (SPINK5) skin barrier g

43 Serine peptidase inhibitor, Kazal type 1 (SPINK1) mRNA was very

44 In this study, we identified SPINT2 (serine peptidase inhibitor Kunitz type 2), a proteolytic inhibi

45 owing study, rats were administered the NAAG peptidase inhibitor PGI-02776 (10mg/kg) 30 min following

46 In the model, the zinc peptidase inhibitor, phosphoramidon, was docked in the a

47 on- and migration-related proteins including peptidase inhibitors, proteases, receptors, and integrin

48 ction/antimicrobial activity/detoxification, peptidase inhibitors, protein digestion (cysteine-, aspa

49 These peptidase inhibitors reduced the motor activation effect

50 Inhibition of the GCP II enzyme with NAAG peptidase inhibitors reduces the concentration of glutam

51 previously reported that secretory leukocyte peptidase inhibitor (SLPI) is regulated by the AR in a l

52 to elicit internalization were unaffected by peptidase inhibitors, the opioids released by veratridin

53 inal cord slices while superfusing them with peptidase inhibitors to prevent opioid degradation.

54 oral effects and support the conclusion NAAG peptidase inhibitors warrant further study as a novel an

55 ence of cysteine and aspartic peptidases and peptidase inhibitors, whereas other peptidase families w

56 ly 180-kDa multidomain membrane-anchored pan-peptidase inhibitor, which is cleaved by host endopeptid

57 hat will help in the design of additional DD-peptidase inhibitors with the potential to serve as lead

58 gned di-ester prodrug of the urea-based NAAG peptidase inhibitor ZJ-43, was tested for neuroprotectiv

59 pothesis that two structurally distinct NAAG peptidase inhibitors, ZJ43 and 2-(phosphonomethyl)pentan

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