ライフサイエンスコーパス: percussion

1 of these fragments also preserve evidence of percussion.

2 ury in hippocampal slices subjected to fluid percussion.

3 d rainbow trout killed by asphyxia in air or percussion.

4 d models induce head injury by lateral fluid percussion, a controlled cortical impact or impact accel

5 f traumatic brain injury, parasagittal fluid percussion, analysis of the metal load of metallothionei

6 ts underwent normothermic parasagittal fluid-percussion brain injury (1.8-2.1 atmospheres) followed b

7 transplanted at 24 h following lateral fluid percussion brain injury (FP) in rats.

8 Fluid percussion brain injury (FPI) elevates the CSF concentra

9 sensitive K (K(ca)) channel following fluid percussion brain injury (FPI) in newborn pigs equipped w

10 sitive K(+) (K(ca)) channels following fluid percussion brain injury (FPI) in newborn pigs equipped w

11 After fluid percussion brain injury (FPI) in the newborn pig, pial a

12 were then subjected to moderate/severe fluid percussion brain injury (FPI).

13 lation during hypotension after piglet fluid percussion brain injury (FPI).

14 on of superoxide anion (O(-)(2)) after fluid percussion brain injury (FPI).

15 artery dilation was impaired following fluid percussion brain injury (FPI).

16 cats were subjected to moderate/severe fluid percussion brain injury after intrathecal administration

17 Rats (n=45) were subjected to lateral fluid percussion brain injury and euthanized at 3 h to 28 days

18 l artery vasodilation was unchanged by fluid percussion brain injury and phenylephrine.

19 ented DNA at 12 or 24 hr after lateral fluid percussion brain injury in anesthetized rats.

20 hypotensive pial artery dilation after fluid percussion brain injury in females, but paradoxically ca

21 reased markedly during hypotension and fluid percussion brain injury in males but less in females.

22 oxically caused vasoconstriction after fluid percussion brain injury in males.

23 hanges were investigated using lateral fluid percussion brain injury in mice.

24 and reduces cerebral blood flow after fluid percussion brain injury in piglets.

25 n kinase C were measured after lateral fluid percussion brain injury in rats.

26 e and motor deficits following lateral fluid percussion brain injury in rats.

27 etrograde memory dysfunction following fluid percussion brain injury in rats.

28 index were determined before and after fluid percussion brain injury in untreated, preinjury, and pos

29 severity was induced using the lateral fluid percussion brain injury model in anesthetized rats (n =

30 24 hours to 2 months following lateral fluid percussion brain injury of moderate severity (2.4-2.6 at

31 The effect of fluid percussion brain injury on hippocampal long-term potenti

32 At 2 weeks after moderate fluid-percussion brain injury or sham surgery, adult male Spra

33 utoregulation during hypotension after fluid percussion brain injury through modulation of extracellu

34 utoregulation during hypotension after fluid percussion brain injury through modulation of extracellu

35 d elevated intracranial pressure after fluid percussion brain injury were greater in males, which wer

36 During hypotension and fluid percussion brain injury, pial artery dilation was impair

37 experiment 1, 15 min prior to central fluid percussion brain injury, rats (n=8 per group) were injec

38 ein kinase upregulation in males after fluid percussion brain injury.

39 eased more in males than females after fluid percussion brain injury.

40 e blunted by phenylephrine pre- or postfluid percussion brain injury.

41 sham surgery or moderate parasagittal fluid-percussion brain injury.

42 cerbates histopathologic outcome after fluid-percussion brain injury.

43 t, male rats were subjected to midline fluid percussion brain or sham injury and evaluated between 1d

44 a closed cranial window were connected to a percussion device consisting of a saline-filled cylindri

45 A parasagittal fluid percussion (FP) brain injury (1.8-2.1 atm) was induced i

46 c protein were measured after lateral fluid percussion (FP) brain injury in rats.

47 free fatty acids (FFAs) after lateral fluid percussion (FP) brain injury in the rat.

48 free fatty acids (FFAs) after lateral fluid percussion (FP) brain injury in the rat.

49 ation following lateral (parasagittal) fluid-percussion (FP) brain injury in the rat.

50 s in the adult rat brain after lateral fluid-percussion (FP) brain injury was characterized using ter

51 (1.8-2.1 atm), or severe (2.2-2.7 atm) fluid percussion (FP) injury (or sham surgery) and processed f

52 Because the lateral fluid-percussion (FP) model of experimental brain injury produ

53 hort- and long-term effects of lateral fluid percussion head injury on the perisomatic inhibitory con

54 Mild to moderate (1.7-2.1 atm) lateral fluid percussion head injury or sham operation was produced in

55 lity in the dentate gyrus 1 week after fluid percussion head trauma.

56 brain injury (TBI) induced by lateral fluid percussion in adult rats, using 2 new ligands for PET: (

57 derate injury was induced with midline fluid percussion in some of the rats.

58 sorder characterized by electrically silent, percussion-induced muscular contractions.

59 he temporal profile of apoptosis after fluid percussion-induced traumatic brain injury (TBI) in rats

60 The late EPSCs in granule cells from fluid percussion-injured rats were not blocked by the NMDA rec

61 Animals were subjected to fluid percussion injury (2.2-2.4 atm; 1 atm = 101.3 kPa) and,

62 sham injury, lateral (LFPI) or central fluid percussion injury (CFPI) only, or to combined LFPI or CF

63 isoflurane, prepared for parasagittal fluid percussion injury (FPI) and randomly assigned to receive

64 Lateral fluid percussion injury (FPI) and sham-operated (Sham) rats we

65 to dilator opioids were blunted after fluid percussion injury (FPI) in newborn pigs.

66 y taken up by the injured brain, after fluid percussion injury (FPI) in the rat.

67 Using a fluid percussion injury (FPI) model and patch-clamp recordings

68 ontribute to TBI, we studied a lateral fluid percussion injury (FPI) model in mice.

69 This study used the rodent fluid percussion injury (FPI) model, in combination with elect

70 estigated the early effects of in vivo fluid percussion injury (FPI) on hippocampal synaptic potentia

71 o weeks after they had received a mild fluid percussion injury (FPI) or sham surgery.

72 rats (n=19) were performed mild brain fluid percussion injury (FPI) prior to exercise.

73 behavioral function following lateral fluid percussion injury (FPI) to the brain in male rats.

74 (ECoG) of rats weeks and months after fluid percussion injury (FPI), a model of traumatic brain inju

75 se (GAD) positive neurons between sham fluid percussion injury (FPI), FPI with sham Vagus Nerve Simul

76 del of closed head injury, the lateral fluid percussion injury (FPI), in the rat.

77 of cognitive recovery after a lateral fluid percussion injury (FPI).

78 s appear following a single episode of fluid percussion injury (FPI).

79 Lateral fluid percussion injury (LFP), a model of mild-moderate concus

80 ogy over 28 days following rat midline fluid percussion injury (mFPI) as a first step in exploiting m

81 Rats were given a sham or mild fluid percussion injury (mFPI), and behavioral testing, MRI, a

82 ce received a sham (n = 52) or midline fluid percussion injury (TBI; n = 57).

83 nduced in rats by rostral parasagittal fluid percussion injury and epilepsy patients evaluated with i

84 We used fluid percussion injury and in situ hybridisation to evaluate

85 -H mice were subjected to mild central fluid percussion injury and killed at various times between 15

86 ats were subjected to moderate central fluid percussion injury and killed between 30 min and 7 d afte

87 mild (33 degrees C) hypothermia after fluid-percussion injury combined with secondary hypoxia.

88 After lateral fluid percussion injury in adult rats, oral treatment with EVT

89 Rostral parasagittal fluid percussion injury in rats reliably induces a perilesiona

90 d previously that rostral parasagittal fluid percussion injury induces different types of chronic rec

91 rkable feature of rostral parasagittal fluid percussion injury is the occurrence, in the early months

92 oorly characterized by comparison with fluid percussion injury models.

93 cts of mild (1.4-1.5 atm) parasagittal fluid-percussion injury on the electrophysiology of this circu

94 The effect of moderate, central fluid percussion injury on the subcellular distribution of thi

95 The data support the conclusion that fluid percussion injury results in redistribution of the enzym

96 t the assessment of the time course of fluid percussion injury-induced epileptogenesis is dramaticall

97 ransgenic rodent model of parasagittal fluid percussion injury.

98 lcineurin activity increased following fluid percussion injury.

99 travenously at 30 min prior to midline fluid percussion injury.

100 significant benefits for sound quality of a percussion instrument (xylophone).

101 Using direct percussion, language-competent bonobo-chimpanzees Kanzi

102 The fluid-percussion model (FP) is one of the oldest and most comm

103 eviously characterized for the lateral fluid-percussion model of brain injury.

104 Rats were injured using lateral fluid percussion TBI (2.8 +/- .10 atm).

105 cted to moderate (1.8-2.2 atmospheres) fluid-percussion TBI and naive controls.

106 he hippocampus at the onset of central fluid percussion TBI and that the enhanced phospholipase C-cat

107 rus at 2 and 15 days following lateral fluid percussion TBI in adult rats.

108 selective hippocampal cell death after fluid-percussion TBI in rats, consistent with the reported red

109 hippocampal injury after experimental fluid percussion TBI in rats.

110 intraperitoneally 30 min after lateral fluid percussion TBI in the rat.

111 l deficits, in a moderate parasagittal fluid percussion TBI model.

112 ubjected to injury (INJ) consisting of fluid percussion TBI of 3 atm with concurrent 30 ml/kg graded

113 ithin a broader pattern of Palaeolithic bone percussion technology in Africa, Eurasia and North Ameri

114 He had no percussion tenderness of his spine, and a neurologic exa

115 nd lactate were measured after central fluid percussion traumatic brain injury (TBI) in rats.

116 nd 24 h after mild, moderate or severe fluid percussion traumatic brain injury (TBI).

117 cused injury than is seen with lateral fluid percussion which may have implications for the behaviora