ライフサイエンスコーパス: perforant pathway

1 structures, the entorhinal verrucae and the perforant pathway.

2 m the layer II and III neurons that form the perforant pathway.

3 uency electrical stimulation of the afferent perforant pathway.

4 (ERC) to the dentate gyrus via axons of the perforant pathway.

5 communication and synaptic plasticity in the perforant pathway.

6 3, 7 and 14 days following lesioning of the perforant pathway.

7 tively scant immunoreactivity in the nascent perforant pathway.

8 rtical projection to the hippocampus via the perforant pathway.

9 feed-forward inhibition of granule cells via perforant pathway activation.

10 processing of de novo synthesized APP in the perforant pathway and at presynaptic sites in the hippoc

11 in excitability of fibre volleys in both the perforant pathway and granule cell mossy fibre projectio

12 and, to a lesser extent, stratum oriens; (3) perforant pathway-associated interneurons (n = 6) innerv

13 lumns of the fornix (P = 0.012), area of the perforant pathways bilaterally (right side: P = 0.028, l

14 ives inputs from the mossy-fiber and lateral perforant pathways, both of which contain and release op

15 erm potentiation initiated in the EC via the perforant pathway, but not the temporoammonic pathway.

16 e demonstrated previously that lesion of the perforant pathway causes the death of approximately 30%

17 abeled cells in the entorhinal cortex of the perforant pathway expressed predominantly m1 and m3, wit

18 Perforant pathway fibers monosynaptically activate granu

19 These data suggest that the loss of the perforant pathway fibers results in a compensatory incre

20 ractions between the Schaffer collateral and perforant pathways have been hypothesized.

21 uniformly occupies the terminal zone of the perforant pathway in tau-expressing mice.

22 o explored the role of FGFR-2 in response to perforant pathway lesion and observed enhanced FGFR-2 ex

23 3, 7, 14, 30, and 90 days after a unilateral perforant pathway lesion in the rat brain.

24 virus, we demonstrated that molecular layer perforant pathway (MOPP) cells innervated newborn granul

25 ss information is available about the medial perforant pathway (MPP), one of the major inputs to the

26 , we created unilateral knife lesions of the perforant pathway of transgenic mice that exhibit hippoc

27 To test this notion, we transected the perforant pathway (PP) of transgenic mice harboring eith

28 In contrast, the perforant pathway projection from EC to the dentate gyru

29 cortex, thereby disrupting the origin of the perforant pathway projection to the hippocampus, and amy

30 Stimulating the perforant pathway resulted in the activation of MOPP cel

31 ed stereology at 9 weeks after lesion of the perforant pathway revealed that the only ECL2 neuronal p

32 t that AMPA receptor subunits play a role in perforant pathway signal transduction.

33 In perforant pathway-stimulated animals, a population of gr

34 Perforant pathway stimulation for 24 hours, which evoked

35 Bilateral perforant pathway stimulation for 3 hours evoked granule

36 These results demonstrate that perforant pathway stimulation in mice reliably reproduce

37 Two daily 30-minute episodes of perforant pathway stimulation in Sprague-Dawley rats inc

38 During perforant pathway stimulation in urethane-anesthetized r

39 acid-induced status epilepticus or prolonged perforant pathway stimulation, translamellar inhibition

40 Bilateral perforant pathway stimulation-induced granule cell disch

41 rus, assessed with paired-pulse responses to perforant-pathway stimulation, revealed enhanced, and no

42 pathology, hippocampal sclerosis, TDP-43 and perforant pathway synaptic loss are the major contributo

43 eposits in the hippocampus were localized to perforant pathway terminal fields.

44 sits in this area leads to disruption of the perforant pathway terminal zone and apparent aberrant di

45 fter prolonged electrical stimulation of the perforant pathway that induced the expression of other n

46 the reorganization of the temporoammonic and perforant pathways that accompany hippocampal sclerosis,

47 Layer II of the EC gives rise to the perforant pathway, the major source of the excitatory in

48 he entorhinal cortex, is transported via the perforant pathway to presynaptic terminals in the dentat

49 ided neighboring cells that give rise to the perforant pathway to the dentate gyrus.

50 Paired-pulse stimulation of the perforant pathway was used to study the relation between

51 al stages of AD, the cells of origin for the perforant pathway within the entorhinal cortex are among