ライフサイエンスコーパス: perfusate

1 challenges with or without treatments to the perfusate.

2 ximately 20 mM when NAD+ was included in the perfusate.

3 ycle intermediates in tissue versus effluent perfusate.

4 f other transporters were independent of the perfusate.

5 A levels, NSD-1015 20 microM was included in perfusate.

6 rin, and concentration of fibronectin in the perfusate.

7 cence over time after dyes were removed from perfusate.

8 (2.8 +/- 1.0 microM) were identified in the perfusate.

9 1 to 5 micromol/L) was added to the coronary perfusate.

10 essed when SR11302 (100 nM) was added in the perfusate.

11 moval of HCO3-/CO2 from the pulmonary artery perfusate.

12 with reference to biochemical changes in the perfusate.

13 rate of the respective isotope added to the perfusate.

14 nium, an inhibitor of SACs, was added to the perfusate.

15 motic solutions or a reduced level of normal perfusate.

16 g/dL bovine serum albumin or a protein-free perfusate.

17 han twice as fast if HDL was included in the perfusate.

18 by lowering the calcium concentration in the perfusate.

19 sine or 5.0 mM cycloleucine was added to the perfusate.

20 ere studied with and without acivicin in the perfusate.

21 elective beta-adrenergic stimulation) to the perfusate.

22 x-line radical adduct signal was detected in perfusate.

23 ed by addition of atropine (1 microM) to the perfusate.

24 omol/L) or propafenone (1 micromol/L) to the perfusate.

25 ion was performed using an erythrocyte-based perfusate.

26 perfusion with a plasma-free red cell-based perfusate.

27 1 mg/mL) or MnTBAP (0.3 mg/mL), added to the perfusate.

28 < 0.001), and E-selectin (P < 0.001) in the perfusate.

29 educed the ATP concentration detected in the perfusate.

30 were 10-fold higher than that in the kidney perfusates.

31 were increased in the nonstreptokinase group perfusates.

32 total of 51 analytes, 34 were measurable in perfusates.

33 after transplantation and the NMR data from perfusates.

34 opionic acid from [5,6,7-(13)C(3)]heptanoate perfusates.

35 ration of BK stabilised at 378 +/- 48 pg (ml perfusate)(-1), that of trypsin-activated BK precursor w

36 e groups (n = 4 each) were differentiated by perfusate: (1) isolated red blood cells (RBCs) (current

37 Addition of enalaprilat to the perfusate (10 mM) prevented the conversion of exogenousl

38 ction in lactate dehydrogenase levels in the perfusate (333+/-22 vs.103+/-8 U/L) and an increase in b

39 With [U-13C16] palmitate in the perfusate, agmatine significantly increased the output o

40 minutes of ex situ perfusion, at which point perfusate alanine transaminase level was 1152 IU/L and u

41 The use of an autologous whole donor blood perfusate allowed 24H of preservation without functional

42 In whole liver, H/R significantly increased perfusate ALT.

43 Perfusate analysis demonstrated reduced fibrin generatio

44 ficial cerebrospinal fluid and the collected perfusate analyzed for ATP and SP content using the fire

45 on permits pharmacologic manipulation of the perfusate and aids in the pretransplant assessment of th

46 s in the interstitial fluid diffuse into the perfusate and are collected.

47 and PCO2 electrodes simultaneously measured perfusate and effluent pH and PCO2.

48 Net flux should equal zero when perfusate and interstitial concentrations are equal.

49 ed increases in proinflammatory cytokines in perfusate and lung lavage fluid, compared to control.

50 rfused rat kidney model using a protein-free perfusate and perfusates containing bovine serum albumin

51 on of indomethacin (30 micromol l-1) to both perfusate and superfusate reduced the positive correlati

52 Perfusate and urine samples were obtained at baseline an

53 Cl-free perfusate and/or bumetanide (10(-5) M) was used to inhib

54 resence of extracellular histones in machine perfusates and deceased donor kidney viability.

55 tion strategies, and the impact of different perfusates and leukocyte filters.

56 increased ATP concentrations in the bladder perfusate, and also increased voiding frequency; these e

57 eristics, reduced the release of [Ca++] into perfusate, and ameliorated mitochondrial ischemic injury

58 tors VII and X were evaluated in pre-bypass, perfusate, and pericardial wound blood before and during

59 Protein binding was measured in each of the perfusates, and the venous outflow was collected to dete

60 nts of acetic acid and propionic acid in the perfusate are comparable to the labeling pattern of acet

61 ents by removal of bicarbonate ions from the perfusate at this point, which resulted in further swell

62 Furthermore, we measured perfusate ATP concentration with a luciferin-luciferase

63 Urocortin 10(-8)M was added to the perfusate before I, before I and during R, and during R

64 ion of a specific caspase-9 inhibitor to the perfusate before ischemia prevented endothelial apoptosi

65 When added to the perfusate, benzamil almost completely inhibited the elev

66 n and conjugates were measured in intestinal perfusates, bile, plasma, and urine.

67 These perfusate biomarkers can be potentially used for more pr

68 We analyzed the diagnostic accuracy of the perfusate biomarkers glutathione S-transferase, lactate

69 The diagnostic accuracy of the perfusate biomarkers glutathione S-transferase, LDH, hea

70 viable; however, current tools and urine and perfusate biomarkers to identify these viable kidneys ar

71 Although TTX (1 microm) or a calcium-free perfusate both caused reductions in the power amplitude

72 PPi levels in the perfusates both in the liver and kidney of Abcc6(-/-) ra

73 to allow the use of biologically appropriate perfusate buffers containing high salt content.

74 s attenuated if calcium was removed from the perfusate but not by external vein stenting.

75 dent on the immediate presence of SIT in the perfusate, but independent of the amount of SIT that had

76 eticulum Ca(2+)-ATPase inhibition or reduced perfusate [Ca(2+)], which indicates a Ca(2+)-mediated me

77 ificantly higher systolic performance at low perfusate [Ca2+] compared with R278C-Tg hearts, which we

78 At baseline perfusate calcium of 1.2 mmol/L, myocyte fractional shor

79 ractional shortening in the presence of high-perfusate calcium of 3.5 mmol/L.

80 In the presence of high-perfusate calcium, both myocyte fractional shortening an

81 similarly increased in the presence of high-perfusate calcium.

82 The NMR spectroscopy of the perfusate can identify differences in the metabolomic pr

83 lin-dibutyryl cAMP (db-cAMP) cocktail to the perfusate caused no significant change in Jv in either w

84 ne, and the Ca(2+) ionophore) to the myocyte perfusate caused tachycardia, contracture, and fibrillat

85 With acivicin in perfusate, cellular concentrations were reduced but ther

86 nd hypertonic shock, and rapid withdrawal of perfusate chloride.

87 cretion but replacing bath Na+ with NMDG+ or perfusate Cl- with gluconate- had no effect.

88 he maximum prednisolone concentration in the perfusate (Cmax) by 3.0 and 2.2 fold, respectively.

89 Impermeant CA inhibitors abolished net perfusate CO2 loss and increased net HCO3- gain, whereas

90 quilibrated under flow for 30 min, using the perfusate collected during the final 10 min of the equil

91 jected to push-pull perfusion of the MPA and perfusates collected at 30 min intervals were analyzed f

92 Perfusates collected from a separate group of rats were

93 50% (NH4)2SO4 saturation of perfusates collected from Langendorff rat heart preparat

94 s of the putative algogen endothelin (ET) in perfusates collected from the tumor sites of hyperalgesi

95 a) perfusion increased net CO2 loss from the perfusate compared with controls (pH 6.4 saline, PCO2 ap

96 The minimum perfusate concentration of NMDA needed to elicit feeding

97 d not be discarded because of high biomarker perfusate concentration.

98 yclohexyladenosine (10 microM) in the tubule perfusate confirmed the ability of the afferent arteriol

99 ed decorticate rat preparation, hyperosmotic perfusate consisted of either 135 mm NaCl, or a non-NaCl

100 The perfusate contained isolated syngeneic red blood cells a

101 minutes with unmodified perfusate (control), perfusate containing 20 nM dopamine, dopamine+2,3-butane

102 ased when 1 micro mol/l GLY was added to the perfusate containing 5 mmol/l glucose.

103 Perfusate containing either 4 microM oxyHb or SNO-Hb inc

104 Addition of L-glutamate or sucralose to a perfusate containing low glucose (20 mM) each activated

105 Electroosmosis was used to pull perfusate containing secreted insulin into 4-cm-long rea

106 Excess glutathione (GSH) added to perfusate containing SNO-Hb resulted in a 20 to 40% fall

107 Then 7.5 microM VIP was added to the perfusate containing VIP(10-28) at the three concentrati

108 ney model using a protein-free perfusate and perfusates containing bovine serum albumin.

109 e reperfused for 120 minutes with unmodified perfusate (control), perfusate containing 20 nM dopamine

110 sulated clodronate, significantly attenuated perfusate cytokine levels, especially tumor necrosis fac

111 In contrast, atelectasis had no effect on perfusate cytokines compared to control but did induce s

112 In human eyes, hypo-osmotic perfusate decreased C 12%, whereas hyper-osmotic perfusa

113 Liver graft perfusate-derived KCs and in vitro-generated monocyte-de

114 r zinc (Zn2+ as 7 micromol l-1 ZnSO4) to the perfusate did not affect reabsorption of water, Na+ or K

115 Removing magnesium from the perfusate did not affect spontaneous subthreshold oscill

116 Interestingly, IL-6 levels in the perfusate did not increase after the lungs were removed

117 al of NO from Hb via transnitrosation to the perfusate did not reduce augmentation of HPV by SNO-Hb o

118 t rate (HR), coronary perfusion pressure and perfusate distribution to the myocardium.

119 After 24 hr of preservation, perfusate distribution was assessed, and oxygen consumpt

120 ol or isoprenaline had no effect on coronary perfusate distribution.

121 ive, at least for PO2 levels of carotid body perfusate down to approximately 40 mmHg.

122 (NMR) to predict graft outcome by analyzing perfusates during machine perfusion time.

123 six percent of the dose was recovered in the perfusate either as unchanged (-)-epicatechin (22 mg) or

124 perfusion characteristics (flow, resistance, perfusate electrolytes, and pH) were serially measured.

125 Responses to increases in perfusate flow (from 0 to 25 microL/min) and to the calc

126 nal allografts has been limited to assessing perfusate flow (PF) during hypothermic perfusion preserv

127 milk-like secretion, which was dependent on perfusate flow and contained a concentration of BK which

128 PP but not NPP significantly improved renal perfusate flow and urine production and significantly in

129 creases in diameter elicited by increases in perfusate flow from 0 to 10 microq/min were similar in a

130 cutoff of 30 kDa in a concentric probe and a perfusate flow rate of 2.0 muL min(-1), microdialysis re

131 After 10 mins, the perfusate flow was resumed at 20% of baseline flow and m

132 After 20 mins of VF, the perfusate flow was returned to baseline and a sinus rhyt

133 to the right ventricular endocardium and the perfusate flow was stopped.

134 The perfusate flow was then stopped for a 10-min interval an

135 n pressure for 30 minutes (in the absence of perfusate flow).

136 e perfusion pressure was linearly related to perfusate flux between 60 and 210 ml min(-1) and the flo

137 s factor-alpha (300 mug) was injected in the perfusate, followed 5 minutes later by melphalan at 1.5

138 icropipette electrodes and the collection of perfusate for analysis are other possibilities.

139 30 hour period with hourly sampling from the perfusate for measurement of general metabolism and clin

140 e able to (1) perfuse tissue and collect the perfusate for quantitative analysis of the solutes intro

141 High K+ (80 mM) was included in perfusate for two 30 min periods, 2.5 h apart.

142 s in smooth muscle cells downstream from the perfusate from an endothelium-intact arteriole was exami

143 py to examine the metabolomic profile of HMP perfusate from human cadaveric kidneys awaiting transpla

144 ng electrophoresis-based immunoassays of the perfusate from islets.

145 so decreased basal ATP concentrations in the perfusate from non-distended bladders and inhibited incr

146 acid, acetic acid, and propionic acid in the perfusate from rat liver exposed to Krebs-Ringer bicarbo

147 Perfusates from HMP kidneys were sampled at 45 min and 4

148 oncentrations in aortic and pulmonary artery perfusates from the working mouse heart before and after

149 Although perfusate G1 was the most effective solution for HBD kid

150 solution for HBD kidneys, the TFP additive (perfusate G2) more effectively reversed the vasospastic

151 dynamic variables of circulation, as well as perfusate gases and electrolytes (pH, pCO2, pO2, O2 satu

152 Furthermore, we demonstrated that liver perfusates, generated by isolated liver perfusion system

153 The kidneys were randomly assigned to a perfusate group (G): G1=MPS, G2=MPS+TFP, G3=MPS+AL, and

154 r between or through endothelial cells where perfusate had direct access to the basement membrane.

155 Restoring bicarbonate to the perfusate halted this swelling, and the corneas then thi

156 rospinal fluid was used as the microdialysis perfusate, Hcrt-1 no longer produced an increase in glut

157 Our data suggest no superiority of novel perfusates HTK-N and TiProtec in terms of tissue preserv

158 inistration of acriflavine directly into the perfusate immediately before IPC.

159 The addition of PGE1 to perfusate improved MP characteristics, reduced the relea

160 Urea was added to the perfusate in concentrations of 0.83, 2.5, 5.0, and 13.33

161 soluble complement receptor 1 (sCR1) to the perfusate in one further group (hDAF(+/-)/AbAbs/sCR1).

162 usate decreased C 12%, whereas hyper-osmotic perfusate increased C 44%.

163 or Bis-Mal-PEGHb (100 micromol/L) to buffer perfusate increased normoxic PAP and augmented HPV in si

164 ihydroguaiaretic acid-containing whole blood perfusate increased the rate of [3H]thymidine incorporat

165 ow-inflow) and the urea concentration in the perfusate (inflow).

166 showed decreasing lactate production in the perfusate (initial: 0.031 +/- 0.004 vs final: 0.007 +/-

167 Without fatty acids in the perfusate, insulin output in the Lard group (135 +/- 22

168 e (the sink, outer pipet) a buffer solution (perfusate) into each of the two pools.

169 During hypoxia the flow of blood (or perfusate) is maintained and, while there is a substanti

170 synaptic blockade, to identify EBs active at perfusate K(+) concentrations ([K(+)](o)) of 3, 6, and 9

171 ect out contribution of fatty acid uptake, a perfusate-lacking fatty acids was used.

172 ygen efficiency (P = 0.01), with lower blood perfusate lactate (P = 0.007).

173 The vascular flow parameters and the perfusate lactate clearance were similar in both groups.

174 Perfusate lactate concentration also decreased (0 hour,

175 Perfusate lactate concentration decreased from baseline

176 Perfusate lactate level fell from 7.2 to 0.3 mmol/L with

177 The evaluation protocol consisted of perfusate lactate, bile production, vascular flows, and

178 livers displaying good function during NMP, perfusate levels of ALT and D-dimers were low (</=3500 n

179 and significantly increased the reduction of perfusate levels of creatinine and urea during reperfusi

180 Perfusate levels of fatty acid binding protein, a marker

181 de synthase, along with significantly higher perfusate levels of the endogenous vasodilator nitric ox

182 air control, exposure to 70% N(2)O increased perfusate levels of the NO metabolites nitrite and nitra

183 ven when DMI (20 microM) was administered in perfusate, LNAA- still lowered DMI-induced DA and NE lev

184 ine in an organ bath, allowing access to the perfusate (luminal) and the bath (serosal) solutions.

185 ter-investigator variability using different perfusates makes comparisons difficult.

186 ggested that the removal of albumin from the perfusate may reduce EC-ECM attachment because hypertoni

187 as present in human aqueous humor and in the perfusate medium of perfusion-cultured human eyes.

188 sothiol], in association with an increase in perfusate [metHb].

189 When FFAs in the perfusate mimicked the quantity and composition of plasm

190 fusion (MPanox) or active oxygenation of the perfusate (MPox).

191 However, perindoprilat (10 mM in the perfusate, n = 7) significantly decreased RIF AngII conc

192 Interstitial infusion of AngI (100 nM in the perfusate, n = 7) significantly increased the RIF AngII

193 h the microdialysis probe (1 or 10 mM in the perfusate; n = 5 and 8, respectively) significantly incr

194 Replacing perfusate Na+ with NMDG+ reversibly inhibited net urea s

195 A significant (P < 0.05) difference in the perfusate nitrate concentration was observed in each loc

196 Perfusate of 26 transplanted cadaveric kidneys was analy

197 Addition to the perfusate of a medium-chain fatty acid (caprylic acid) t

198 Inclusion in the vein perfusate of drugs that reduce calcium entry (including

199 n to increase glutamate concentration in the perfusate of hippocampal slices and in purified rat hipp

200 rotein was markedly increased in serum or in perfusate of isolated heart following ischemia/reperfusi

201 ong decrease of APAP-GLUC secretion into the perfusate of Mrp3-/- livers.

202 orbic acid (0.2 mM) was administrated in the perfusate of the ascorbic acid + electrical shock and as

203 Machine perfusates of 390 donations after circulatory death kidn

204 concentrations were significantly higher in perfusates of kidneys with posttransplant graft dysfunct

205 Dynorphin B was collected from spinal perfusates of rats pretreated with Delta(9)-THC, CP55,94

206 tly reduced, but the PPi levels in the liver perfusates of wild-type rats were 10-fold higher than th

207 omized to receive 3.0 mM supplemental GSH to perfusate or no supplementation (control) and were prese

208 Stimulus was then added to either the perfusate or the bath and the perfusate was collected fo

209 erfusate), to inhibit (hyperoxic, hypocapnic perfusate) or to stimulate (hypoxic, normocapnic perfusa

210 The first 6 livers were perfused at high perfusate oxygen tensions, and the subsequent 6 at near-

211 The effect of HOPE was dependent on perfusate oxygenation in the cold.

212 At 50% perfusate oxygenation, APD and LVDP were significantly h

213 n metabolism at hypothermia to the design of perfusates, perfusion machine technology, and drug thera

214 o influence MP characteristics when added to perfusate: PGE1, trifluoperazine, verapamil, and papaver

215 Increasing perfusate pH buffers decreased surface pH toward perfusa

216 en consumption and significantly neutralized perfusate pH.

217 activated in wound, but not in pre-bypass or perfusate plasma (monocyte chemotactic protein-1 = 29.5

218 Theophylline also improved the increase in perfusate PO(2) on transit through the lung after storag

219 nfusion time: 80.8+/-18.2 min; mean coronary perfusate pO(2): 631+/-235 mm Hg).

220 lungs with anoxic gas for 5 minutes reduced perfusate PO2 to 11+/-1.0 Torr.

221 on enhanced the outflow facility and lowered perfusate pressure in biomimetic TM scaffolds populated

222 fter 4 h of perfusion was independent of the perfusate, probably because normal kidneys can maintain

223 th tBHQ (10 micro M), both outflow rates and perfusate proMMP-3 level increased significantly within

224 ddition of 13-HODE to the LA-deficient blood perfusate promoted tumor 13-HODE uptake and a dose-depen

225 Perfusate protein expression during EVLP can differentia

226 The protein binding of MAG3 in the albumin perfusates ranged from 87% to 95%, significantly higher

227 An appreciable increase in perfusate recovery due to a shift in the directionality

228 ory clearance, so an appreciable increase in perfusate recovery of these metabolites was not observed

229 /-); La3+ (1 micromol/L) addition to wt lung perfusate reduced the agonist effect to that observed in

230 ing superoxide dismutase and catalase in the perfusate reduced the ESR signals.

231 ion of dmLSB (10 micromol/L) to the coronary perfusate restored the epicardial AP dome, reduced EDR a

232 ine-methyl ester (L-NAME, 5 mM) to the probe perfusate resulted in an inhibition of the histamine-ind

233 The addition of GSH supplementation to perfusate resulted in no significant differences in graf

234 NO-cyanometHb) on HPV, expired NO (eNO), and perfusate S-nitrosothiol (SNO) concentration in isolated

235 esulted in an approximately 50% reduction in perfusate [S-nitrosothiol], in association with an incre

236 r the collection of a pretreatment blood and perfusate sample, rats were injected (i.p.) with the veh

237 c bead array assay, we evaluated analytes in perfusate samples collected at 1 hour and 4 hours of EVL

238 Perfusate samples from the MPA were collected at the rat

239 The perfusate samples were analyzed for NE and dopamine (DA)

240 Blood and perfusate samples were collected at 30-min intervals for

241 Perfusate samples were collected every 30 min and analyz

242 o cortical cups 35 min prior to ischemia and perfusate samples were obtained prior to, during and fol

243 an NMDA receptor antagonist was added to the perfusate shortly before and during LTP induction.

244 g SNO-Hb resulted in a 20 to 40% fall in the perfusate SNO concentration, with a concomitant increase

245 in UW or machine perfused (MP) in UW-machine perfusate solution (MPS).

246 se (80 U/mL), but not catalase alone, in the perfusate solution prevented the reduction in dilation o

247 abbit TNF-alpha antibody to cardioplegia and perfusate solutions restored postischemic function.

248 red from donor urine at procurement and from perfusate soon after pump perfusion) were not different

249 PO2, PCO2 and pH in the venous effluent perfusate stabilised at 157 +/- 10 mmHg, 50.1 +/- 2.4 mm

250 responds to changes in the properties of the perfusate, such as the ionic strength ( ), pH, and catio

251 dition of copper or manganese to the luminal perfusate suggesting that these ions may compete with ir

252 depending on the Ca(2+) concentration of the perfusate; systolic function was significantly increased

253 etabolic perturbations, including changes in perfusate temperature, electromechanical uncoupling, and

254 usate) or to stimulate (hypoxic, normocapnic perfusate) the CB chemoreflex, while the systemic circul

255 In the 2.5 g/dL albumin perfusate, the EF of MAG3 was 44%, significantly less th

256 However, in the protein-free perfusate, the EF of MAG3 was 64%, equal to or higher th

257 F of the three EC complexes; in the 7.5 g/dL perfusate, the MAG3 EF fell to 18% versus 39%-45% for th

258 Recirculation of the perfusate through the support rat diminishes the need to

259 l of both dissolved gas and pH levels in the perfusate thus demonstrating applicability for a wide ra

260 ls, lowering the temperature of the coronary perfusate to induce mild hypothermia (32 degrees C-34 de

261 parative responses to cellular and acellular perfusates to identify these benefits.

262 normal CB blood gases (normoxic, normocapnic perfusate), to inhibit (hyperoxic, hypocapnic perfusate)

263 Addition of acivicin to the perfusate, to inhibit activity of the y-glutamyltransfer

264 significant increases in alveolar lavage and perfusate tumor necrosis factor-alpha, inflammatory cell

265 1)) or two (U(m+2)) (15)N was generated from perfusate [U-(15)N(4)]arginine.

266 d aspartate, respectively, were derived from perfusate [U-(15)N(4)]arginine.

267 ring release of acetylcholinesterase in cell perfusates using the Ellman reagent.

268 usate pH buffers decreased surface pH toward perfusate values.

269 Using fluorescence recovery, we determined perfusate velocity to calculate diameter changes under d

270 Increasing the perfusate viscosity improves speed by slowing dissipatio

271 nd loading conditions (varying extracellular perfusate viscosity).

272 The perfusate was collected and scleral permeability calcula

273 to either the perfusate or the bath and the perfusate was collected for another 30 min to measure th

274 The perfusate was continuously sampled by electroosmotic flo

275 Hearts were paced to increase workload and perfusate was deoxygenated to study the effects of myoca

276 )I-labelled albumin efflux into the vascular perfusate was determined.

277 Although the magnitude of the radical in perfusate was increased by ethanol, it was not derived d

278 NO(x) (NO(-)(2) + NO(-)(2) production in the perfusate was measured by chemiluminescence.

279 SAAP using an oxygen-carrying perfusate was more effective in this study than non-oxyg

280 Perfusate was plasma-based with a hemoglobin concentrati

281 The complement activity of the perfusate was reduced by CAB-2, as was the generation of

282 With the use of overlapping injections, the perfusate was sampled every approximately 10 s.

283 The WB perfusate was superior (vs RBC) for maintaining stabilit

284 Mucin in perfusates was quantified by periodic acid-Schiff's base

285 nality of metabolite excretion, from bile to perfusate, was noted in knockout mice only for conjugate

286 H+ and CO2 loss from the perfusate were accompanied by increases of PV H+ and tra

287 Enzyme biomarkers present in the kidney perfusate were all significantly reduced by the use of s

288 in (IL)-6 levels in the lung grafts and EVLP perfusate were also significantly lower after EVLP with

289 with isotonic solution and hourly samples of perfusate were collected and analyzed.

290 Ten-minute fractions of aCSF perfusate were collected from separate groups of room ai

291 Fractions of choroidal perfusate were collected, and fluorescence was measured

292 zure-like events (SLEs) in low-Mg2+/ high-K+ perfusate were measured in the CA3 region of the intact

293 and outflow facility and cAMP levels in the perfusate were measured simultaneously.

294 NE concentrations in the perfusate were measured using HPLC-EC and corticosterone

295 r pressure (IOP), and proMMP-3 levels in the perfusate were monitored.

296 ables flow and resistance, and the [Ca++] in perfusate, were significantly associated with delayed gr

297 or by isotonic replacement of sodium in the perfusate with choline or tetramethylammonium greatly at

298 Pike Laboratories Inc, Eagle, PA) is a novel perfusate with enhanced vasodilatory and antioxidant cap

299 rther reduced on combination of calcium-free perfusate with octanol (1 mM) and was abolished using a

300 Perfusates with modified osmolarity were used to cause a