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1 all measurement sites (except cerebral) and perfusion index.
2 and thenar) and finger photoplethysmographic perfusion index.
3 tically generated and were used to calculate perfusion indexes.
4 s 166.83 +/- 204.44; P = .002) rate, wash-in perfusion index (-42.29 +/- 59.21 vs 50.96 +/- 71.13; P
8 ies, there was linear correlation between MR perfusion indexes and the microsphere flow measurements
9 esence of pain, tissue oxygen saturation and perfusion index are further reduced by hypovolemia (lowe
11 when evaluating tissue oxygen saturation and perfusion index as markers of hypovolemia in trauma pati
12 perfusion was monitored with the peripheral perfusion index, capillary refill time, tissue oxygen sa
14 ssue oxygenation (all measurement sites) and perfusion index during hypovolemia and pain than during
15 ral capillary refill time and the peripheral perfusion index, followed by a significant increase at T
17 ficantly improved (p = 0.002); however, mean perfusion index, postductal saturation, and mean renal t
19 nductive plethysmography, oxygen saturation, perfusion index, regional cerebral and perirenal tissue
20 nal explanation for elevation in the Doppler perfusion index that occurs prior to tumor formation.
21 ssue oxygen saturation (except cerebral) and perfusion index were reduced by pain during normovolemia
22 genation in all measurement sites and finger perfusion index were reduced during hypovolemia/sham com
23 capillary segment density and the capillary perfusion index, which was prevented by both starches.
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