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1 and the putative neuroprotective effects of pergolide.
3 ptor agonists cabergoline (a D2 agonist) and pergolide (a D1/D2 agonist) administered in separate ses
4 d (a Bruton's tyrosine kinase inhibitor) and pergolide (a dopamine and serotonin receptor agonist) ro
6 irole, 7-hydroxy-dipropylamin-otetralin, and pergolide also induced sensitization, whereas the high a
7 e selective for D2-like receptors, with only pergolide and apomorphine potentially interacting with D
8 e and dexfenfluramine, the dopamine agonists pergolide and cabergoline, and more recently, the recrea
11 Like low dopamine, the D3 receptor agonists pergolide and PD 128907 reduced MSR amplitude in WT but
13 In this instance both the bromocriptine- and pergolide-based CoMFA models were similar to one another
19 eriments (e.g., during 10 microM dopamine or pergolide in WT), polysynaptic reflexes were facilitated
22 tive dopamine receptor agonists SKF38393 and pergolide into the medial prefrontal cortex (mPfc) on lo
24 -associated medications (e.g., fenfluramine, pergolide, methysergide, and cabergoline) have revealed
25 ted with pergolide use, we hypothesized that pergolide might have similar effects on hypoxic pulmonar
28 Although PAH has not been associated with pergolide use, we hypothesized that pergolide might have
29 eart valve disease (CLHVD) has been noted in pergolide users, reminiscent of that induced by certain
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