ライフサイエンスコーパス: periapical

1 ance in the microbiological findings between periapical abscess and the maxillary sinus flora was fou

2 ial clinical diagnosis and the presence of a periapical abscess at surgery and at pathologic examinat

3 Clinical examination ruled out periapical abscess, periodontal abscess, and lateral per

4 patient underwent incision and drainage of a periapical abscess.

5 firm the importance of anaerobic bacteria in periapical abscesses and demonstrate their predominance

6 nificant association between the presence of periapical abscesses and oral viridans streptococci DNA-

7 Four patients had periapical abscesses at pathologic analysis, and the fif

8 Aspirate of pus from 5 periapical abscesses of the upper jaw and their correspo

9 ery, one of five calibrated examiners viewed periapical and bitewing radiographs of the surgical site

10 Periapical and panogram radiographs were taken.

11 raditionally performed using two-dimensional periapical and panoramic radiographs.

12 es successful, stimulating bleeding from the periapical area of the tooth can be challenging and in t

13 designed to enhance detection of crestal or periapical bone density changes and to help evaluate car

14 cient C3H/HeJ mice had significantly reduced periapical bone destruction compared to wild-type C3H/He

15 Periapical bone destruction occurs as a consequence of p

16 In the present study, we compared periapical bone destruction, sepsis, and inflammatory cy

17 or laborious histologic analyses to quantify periapical bone destruction.

18 the rapid and non-invasive quantification of periapical bone destruction.

19 results in the development of gingivitis and periapical bone loss, which apparently are associated wi

20 RC7 gene expression to simultaneously target periapical bone resorption and periapical inflammation.

21 Periapical bone resorption occurs following infection of

22 ted mice (40%; P < 0.05) exhibited increased periapical bone resorption, compared to wild-type contro

23 infected wild-type mice, which also had more periapical bone resorption.

24 dental pulp result in tissue destruction and periapical bone resorption.

25 ith inflammatory periapical lesions and with periapical cyst formation represents an interesting but

26 fore, the development of a new class of anti-periapical disease therapies is necessary and critical f

27 Periapical disease, an inflammatory disease mainly cause

28 ty to treat this disease in a mouse model of periapical disease.

29 ar nerve and mental foramen on panoramic and periapical films prior to implant placement; use of CT s

30 ts of canal anastomosis on the generation of periapical fluid pressure at different fluid flow rates

31 normal periodontal ligament (PDL) and of 12 periapical granulomas or cysts.

32 Normal PDL and periapical granulomas with scant inflammatory infiltrati

33 Periapical images obtained with a storage phosphor plate

34 port, a 54-year-old patient presented with a periapical infection involving the mesial root of the ma

35 e canal roof is discovered, enucleation of a periapical infection or subsequent implant placement can

36 s to be protective against infection-induced periapical inflammation and bone destruction via suppres

37 esign effective therapies that could prevent periapical inflammation and revolutionize current treatm

38 Periapical inflammation may also exacerbate inflammation

39 e root canal system of the tooth, leading to periapical inflammation, bone erosion, severe pain, and

40 terial infection-stimulated bone erosion and periapical inflammation, which confirms the potential th

41 eously target periapical bone resorption and periapical inflammation.

42 arkedly elevated IL-1alpha production within periapical inflammatory tissues (>10-fold) compared with

43 Radiographic evaluation revealed that the periapical lesion extended from the apex of the tooth to

44 progressive and significant increase in the periapical lesion size in both strains was observed.

45 o and decreased the number of T cells in the periapical lesion.

46 lt in bone-resorptive cytokine production in periapical lesion.

47 To investigate this question we compared periapical-lesion pathogenesis in RAG-2 severe combined

48 those of exposed immunocompetent controls in periapical-lesion size.

49 kening (odds ratio: 3.02, P <0.001), whereas periapical lesions and root canal fillings were not.

50 orptive cytokines IL-1alpha and IL-1beta, in periapical lesions and with decreased expression of the

51 l proliferation associated with inflammatory periapical lesions and with periapical cyst formation re

52 PCR) showed KGF expression in 4 specimens of periapical lesions but low or undetectable levels in nor

53 There was heavy cytokine staining in periapical lesions from both strains, especially in area

54 The development of periapical lesions in IL-17RA KO mice was significantly

55 crosis and the histomorphometric features of periapical lesions in scid vs. normal mice.

56 on of KGF expression in the stromal cells of periapical lesions may play an important role in stimula

57 scessed RAG-2 teeth had significantly larger periapical lesions than did nonabscessed RAG-2 teeth (P

58 The cross-sectional area of periapical lesions was determined by image analysis of c

59 Periapical lesions were induced in 24 canine teeth of 6

60 Periapical lesions were induced in the lower first molar

61 y has been implicated in the pathogenesis of periapical lesions, although the extent to which these m

62 mine the influence of periodontal bone loss, periapical lesions, and root canal fillings on these sin

63 rior teeth, including periodontal bone loss, periapical lesions, and root canal fillings, were assess

64 nly within vasculature structures located in periapical lesions.

65 n infection caused by periodontal disease or periapical lesions.

66 Periapical lucency and sinus opacification were seen in

67 CT findings, including periapical lucency suggesting abscess, sinus opacificati

68 Abnormal periapical lucency, widening of the periodontal ligament

69 re needed to focus on histologic data around periapical microbiota to establish specific etiology and

70 Visible periapical or periodontal changes in dentition were anal

71 Asymptomatic periapical osteolysis, periodontal disease or dead teeth

72 cone beam computed tomography (CBCT) versus periapical (PA) radiographs in detecting PA changes at b

73 of abrogation of T- and B-cell mechanisms on periapical pathogenesis were then assessed.

74 the use of this model system for studies of periapical pathogenesis.

75 1 and 98.2 for implants placed in sites with periapical pathology and implants placed in sites withou

76 e implants placed in the sites demonstrating periapical pathology were followed in function for </=11

77 ts immediately placed in sites demonstrating periapical pathology yielded results comparable to those

78 te implant placement in a site demonstrating periapical pathology, and immediate implant placement in

79 thology and implants placed in sites without periapical pathology, respectively, according to publish

80 erapy present with hopeless teeth exhibiting periapical pathology.

81 Therefore, pulpal and periapical pathosis were independent of the presence of

82 For both models, periapical pressure increased with increasing irrigant f

83 D-speed vertical bitewing and periapical radiographs incorporating aluminum stepwedges

84 ated from digitalized standardized intraoral periapical radiographs obtained from natural teeth and d

85 Periapical radiographs of 18 African Americans with sick

86 Periapical radiographs of mandibular incisors from subje

87 ly assessed in 450 adults using standardized periapical radiographs of maxillary central incisors.

88 Panoramic and periapical radiographs showed a circumscribed 0.8x0.9-cm

89 andibles were measured from non-standardized periapical radiographs taken at abutment connection and

90 of apical portion of implant, and subsequent periapical radiographs taken demonstrated a radiolucent

91 same reference just after loading by digital periapical radiographs to determine the marginal bone lo

92 ralized bone matrix were included, and 1,536 periapical radiographs were analyzed.

93 Periapical radiographs were evaluated before surgery, po

94 Periapical radiographs were evaluated immediately after

95 Periapical radiographs were obtained, as well as two inc

96 Clinical parameters and periapical radiographs were registered on the day of imp

97 Digital photographs and periapical radiographs were taken after restoration.

98 and bleeding on probing) were measured, and periapical radiographs were taken at the time of implant

99 Periapical radiographs were taken using the long-cone te

100 evaluation was performed, and panoramic and periapical radiographs were taken.

101 eolar bone loss (ABL), measured on intraoral periapical radiographs with a modified Schei ruler metho

102 The periapical radiographs yielded stable peri-implant bone

103 fety was evaluated by clinical examinations, periapical radiographs, and occurrence of adverse experi

104 The periapical radiographs, obtained in a standardized manne

105 ant contact (distance bone-implant [DIB]) by periapical radiographs.

106 pulp test for tooth #15, and complete mouth periapical radiographs.

107 CBCT scanning, periapical radiography (PA), and direct measurements usi

108 sociation between the presence of persistent periapical radiolucency and root wall thickness ( P = 0.

109 As there was a periapical radiolucency, an endodontic consultation was

110 volve manipulation of gingival tissue or the periapical region of teeth or perforation of the oral mu

111 We combined data of widened periapical spaces (WPSs) and apical rarefactions to a sc

112 Periapical surgeries, implants and maxillary sinus lift

113 region is an important area with respect to periapical surgery, implant placement, and sinus lifts.

114 e that AAV-mediated Atp6i/TIRC7 knockdown in periapical tissues can inhibit endodontic disease develo

115 to evaluate whether evoked bleeding from the periapical tissues elicits the influx of MSCs into the r

116 IL-17 was strongly induced in periapical tissues in wild-type (WT) mice by 7 d after t

117 al debridement, intracanal bleeding from the periapical tissues was achieved, and intracanal blood sa

118 nguish between hypoxic and normoxic pulp and periapical tissues.