ライフサイエンスコーパス: periaqueductal

1 rve nuclei, inferior and superior colliculi, periaqueductal and pontine gray matter, and the red nucl

2 and in the lateral and dorsal aspects of the periaqueductal central gray.

3 periaqueductal gray (54%); and ventrolateral periaqueductal gray (52%) when compared with basal bindi

4 ); dorsal raphe nucleus (53%); dorsal medial periaqueductal gray (54%); and ventrolateral periaqueduc

5 her OT receptor activity in the ventrocaudal periaqueductal gray (cPAGv) contributes to mothers' redu

6 ductal gray (vl PAG) versus the dorsolateral periaqueductal gray (dl PAG), in the rabbit, elicits two

7 lating neuronal activity of the dorsolateral periaqueductal gray (dl-PAG) through excitatory and inhi

8 mate receptors (mGlu(5)) in the dorsolateral periaqueductal gray (dlPAG) and mobilizing 2-AG.

9 that VMHdm/c projection to the dorsolateral periaqueductal gray (dlPAG) induces inflexible immobilit

10 The dorsal and ventral periaqueductal gray (dPAG and vPAG, respectively) are em

11 al gray (VPAG), or sympathoexcitatory dorsal periaqueductal gray (DPAG), differentially modulates CBF

12 We demonstrate in rats that the periaqueductal gray (PAG) affects motor systems at the f

13 The periaqueductal gray (PAG) and amygdala are known to be i

14 The ventrolateral periaqueductal gray (PAG) and pontine reticular formatio

15 ceptors, acts by an unknown mechanism in the periaqueductal gray (PAG) and raphe magnus (RM) to stimu

16 could be mediated in part by actions in the periaqueductal gray (PAG) and the dorsal raphe nucleus (

17 CeL neurons directly project to the midbrain periaqueductal gray (PAG) and the paraventricular nucleu

18 se VMH efferents travel caudally through the periaqueductal gray (PAG) and then ventrally through the

19 The midbrain periaqueductal gray (PAG) and ventromedial medulla (VMM)

20 The different subdivisions of the midbrain periaqueductal gray (PAG) are intricately (and different

21 attention or expectancy have identified the periaqueductal gray (PAG) as a key brainstem structure i

22 he generation of DRRs and stimulation of the periaqueductal gray (PAG) can induce the release of GABA

23 binds to mu-opioid receptors (MORs), and the periaqueductal gray (PAG) contains a dense population of

24 The periaqueductal gray (PAG) coordinates behaviors essentia

25 r 4 (TLR4)-mediated neuroinflammation in the periaqueductal gray (PAG) drives tolerance.

26 onnectivity fluctuations between the DMN and periaqueductal gray (PAG) dynamically tracked spontaneou

27 Activation of the dorsal periaqueductal gray (PAG) evokes defense-like behavior i

28 pothalamic nucleus (VMH) that project to the periaqueductal gray (PAG) form a crucial segment of the

29 NK(1)-Substance P receptors in the midbrain periaqueductal gray (PAG) in defensive rage behavior in

30 imulation in different parts of the midbrain periaqueductal gray (PAG) in the cat generates four diff

31 The periaqueductal gray (PAG) is a brain region involved in

32 The periaqueductal gray (PAG) is an important center that co

33 The ventrolateral periaqueductal gray (PAG) is an important neuronal netwo

34 The midbrain periaqueductal gray (PAG) is involved in many basic surv

35 Evidence suggests the periaqueductal gray (PAG) is involved in the integration

36 The periaqueductal gray (PAG) is known to be essential for v

37 t.SIGNIFICANCE STATEMENT We demonstrate that periaqueductal gray (PAG) microglia contribute to the se

38 expression is increased in the ventrolateral periaqueductal gray (PAG) neurons following precipitated

39 and spontaneous firings of rat ventrolateral periaqueductal gray (PAG) neurons, either mechanically d

40 tracellular Ca(2+) levels in dissociated rat periaqueductal gray (PAG) neurons, which express GPR55 m

41 ral nucleus, deep mesencephalic nucleus, and periaqueductal gray (PAG) of both sexes.

42 ala by recording neurons in the amygdala and periaqueductal gray (PAG) of rats during fear conditioni

43 norecipient nuclei as well as in the DRN and periaqueductal gray (PAG) of the mesencephalon.

44 te nucleus (ARC) of the hypothalamus and the periaqueductal gray (PAG) of the midbrain.

45 M), but not following co-injections into the periaqueductal gray (PAG) or into the spinal subarachnoi

46 by lesions of brainstem regions such as the periaqueductal gray (PAG) or the rostral ventromedial me

47 The periaqueductal gray (PAG) orchestrates survival behavior

48 The midbrain periaqueductal gray (PAG) plays a central role in the de

49 nal motor system, in which the mesencephalic periaqueductal gray (PAG) plays a central role, as demon

50 The periaqueductal gray (PAG) plays an important role in mor

51 The midbrain periaqueductal gray (PAG) region is organized into disti

52 beta-endorphin in the rVLM as well as in the periaqueductal gray (PAG) that are involved in EA-mediat

53 ng emotions and projects to the amygdala and periaqueductal gray (PAG) to modulate emotional response

54 ng pain modulatory system that runs from the periaqueductal gray (PAG) to the spinal cord.

55 ult of increased microglia activation in the periaqueductal gray (PAG), a central locus mediating the

56 state functional connectivity (rs-fc) of the periaqueductal gray (PAG), a key region in the descendin

57 We found that pain PEs were encoded in the periaqueductal gray (PAG), a structure important for pai

58 havior in the cat elicited from the midbrain periaqueductal gray (PAG), and that such effects are blo

59 halamus, the amygdala, the hypothalamus, the periaqueductal gray (PAG), and the brainstem reticular f

60 ciation between the brainstem areas, such as periaqueductal gray (PAG), and the headache phase of mig

61 reas, the dorsal raphe nucleus (DRN) and the periaqueductal gray (PAG), in addition to EW.

62 d 81, 96, 106, and 82 tolerance genes in the periaqueductal gray (PAG), prefrontal cortex, temporal l

63 ministered systemically or directly into the periaqueductal gray (PAG), produces a significantly grea

64 stent with the location of areas such as the periaqueductal gray (PAG), rostral ventral medulla, and

65 We show that BDNF-containing neurons in the periaqueductal gray (PAG), the central structure for pai

66 ctions to the rostral midbrain including the periaqueductal gray (PAG), the deep layers of the superi

67 lowing administration into the ventrolateral periaqueductal gray (PAG), the dorsal PAG, and the rostr

68 or-expressing neurons (LepRb neurons) in the periaqueductal gray (PAG), the largest population of Lep

69 D), the lateral hypothalamic area (LHA), the periaqueductal gray (PAG), the parabrachial nucleus (Pb)

70 lateral and the upper lateral portion of the periaqueductal gray (PAG), the Su3 and PV2 nuclei of the

71 ds and to determine the role of the midbrain periaqueductal gray (PAG), which mediates stress-induced

72 procedures to affect c-Fos expression in the periaqueductal gray (PAG).

73 ted to be present in regions of the midbrain periaqueductal gray (PAG).

74 ic neurons and CRF receptors is the midbrain periaqueductal gray (PAG).

75 hypothalamus and dorsolateral aspect of the periaqueductal gray (PAG).

76 baroreflex attenuation) evoked by the dorsal periaqueductal gray (PAG).

77 t exert its effect on these functions is the periaqueductal gray (PAG).

78 ulates nociception and blood pressure in the periaqueductal gray (PAG).

79 ation of the mPFC subdivisions, amygdala and periaqueductal gray (PAG).

80 tex, insula, nucleus accumbens, amygdala and periaqueductal gray (PAG).

81 nucleus of the stria terminalis (BSTm), and periaqueductal gray (PAG).

82 r a network of forebrain structures plus the periaqueductal gray (PAG).

83 ral striatum, amygdala, midline thalamus and periaqueductal gray (PAG).

84 tic (MPO) produced dense labeling within the periaqueductal gray (PAG); anterogradely labeled fibers

85 tral tegmental area (VTA), and ventrolateral periaqueductal gray (PAGvl).

86 sis that neurons in the rostral ventromedial periaqueductal gray (rvmPAG) are a source of inhibitory

87 electrical stimulation of the ventrolateral periaqueductal gray (vl PAG) versus the dorsolateral per

88 resynaptic GABA release in the ventrolateral periaqueductal gray (vlPAG) activates the descending ant

89 ulatory regions, including the ventrolateral periaqueductal gray (vlPAG) and locus ceruleus (LC).

90 een shown that the ventrolateral part of the periaqueductal gray (VLPAG) and the adjacent dorsal deep

91 The ventrolateral periaqueductal gray (vlPAG) is a critical structure in t

92 The ventrolateral periaqueductal gray (vlPAG) is a key structure in the de

93 The ventrolateral periaqueductal gray (vlPAG) is an integral locus for mor

94 een central amygdala (CeA) and ventrolateral periaqueductal gray (vlPAG) is implicated in several dis

95 The midbrain ventrolateral periaqueductal gray (vlPAG) is known to be a crucial sup

96 n (PG) E2 signaling within the ventrolateral periaqueductal gray (vlPAG) is pronociceptive in naive a

97 antagonist pretreatment in the ventrolateral periaqueductal gray (vlPAG) of rats.

98 Given that the ventrolateral periaqueductal gray (vlPAG) plays a major role in morphi

99 hat the ventrolateral column of the midbrain periaqueductal gray (vlPAG) region mediates the hypotens

100 ed IS induces DeltaFosB in the ventrolateral periaqueductal gray (vlPAG), and levels of the protein a

101 the arcuate nucleus (ARC) and ventrolateral periaqueductal gray (vlPAG).

102 arily to axon terminals in the ventrolateral periaqueductal gray (vlPAG).

103 responses, from the brainstem ventrolateral periaqueductal gray (vlPAG).

104 la (17%), midline raphe (40%), ventrolateral periaqueductal gray (VLPAG, 15%), lateral hypothalamic a

105 croinjecting morphine into the ventrolateral periaqueductal gray (vPAG) develops with repeated admini

106 istration of morphine into the ventrolateral periaqueductal gray (vPAG), a key structure contributing

107 of neurons of the sympathoinhibitory ventral periaqueductal gray (VPAG), or sympathoexcitatory dorsal

108 the antinociceptive action of ventrolateral periaqueductal gray (vPAG)-administered morphine.

109 l forebrain (SLEA) and the ventral tegmentum/periaqueductal gray (VT/PAG), while foci of increased si

110 Moreover, imminence-driven periaqueductal gray activity correlated with increased s

111 in structures involved in vocal production (periaqueductal gray and anterior hypothalamus) was signi

112 inhibition blocks ERK MAPK activation in the periaqueductal gray and caudal brain stem.

113 ially to the posterior hypothalamic area and periaqueductal gray and caudally along the brachium of t

114 as well as in discrete cells in the lateral periaqueductal gray and in the central gray nucleus.

115 y in the ventrolateral portion of the caudal periaqueductal gray and in the lateral septum in aggress

116 central roles in pain and affect, including periaqueductal gray and nearby dorsal raphe and nucleus

117 reticular formation, midbrain raphe nuclei, periaqueductal gray and parabrachial nucleus.

118 anterior hypothalamus) and of the midbrain (periaqueductal gray and paralemniscal tegmentum) reveal

119 ncreased functional connectivity between the periaqueductal gray and rostral anterior cingulate, as h

120 CB1R desensitization in the periaqueductal gray and spinal cord following 7 d of tre

121 m and also expressed at much lower levels in periaqueductal gray and spinal cord, structures known to

122 system (CNS) pain modulatory regions (i.e., periaqueductal gray and subarachnoid lumbar spinal cord)

123 rtions of the lateral septum), midbrain (the periaqueductal gray and the intermediate layers of super

124 ior paraventricular nucleus of the thalamus, periaqueductal gray and ventrolateral medulla.

125 and kappa opioid receptor agonists into the periaqueductal gray area (PAG).

126 ucleus, Kolliker-Fuse nucleus, ventrolateral periaqueductal gray area, central nucleus of the amygdal

127 caudal A10 cell group (A10dc) located in the periaqueductal gray area.

128 signaling is modulated in the ventrolateral periaqueductal gray by persistent inflammation different

129 tween the ventromedial prefrontal cortex and periaqueductal gray correlated with somatosensory decrea

130 nervation of the ventrolateral column of the periaqueductal gray distinguished the midbrain.

131 rojections from the ARC to the ventrolateral periaqueductal gray during EA at P5-P6 contribute to inh

132 (vmPFC), insula, amygdala, hypothalamus, and periaqueductal gray emerge as central brain structures u

133 wedge of labeled neurons in the dorsolateral periaqueductal gray extending into the deep layers of th

134 fralimbic prefrontal cortex, hippocampus and periaqueductal gray in extinction learning, while mainta

135 dies in the midbrain reticular formation and periaqueductal gray in four clinically documented and ge

136 ic motor nucleus, in a band just beneath the periaqueductal gray in the midbrain, in ventricular regi

137 rect evidence supporting the notion that the periaqueductal gray is a site for higher cortical contro

138 f the projections from the ICx to the dorsal periaqueductal gray is sufficient for provoking flight b

139 ned the role of eNOS within the dorsolateral periaqueductal gray mater (dlPAG) on cardiovascular resp

140 bnormal in hippocampus ( approximately 10%), periaqueductal gray matter ( approximately 13%), fimbria

141 the olfactory bulb ( approximately 10%) and periaqueductal gray matter ( approximately 16%).

142 Nitric oxide (NO) within the dorsal periaqueductal gray matter (dPAG) attenuated cardiovascu

143 al cortex, the lateral hypothalamus, and the periaqueductal gray matter (PAG) are involved in these c

144 ase in the total phosphatase activity in the periaqueductal gray matter (PAG) from morphine-pelleted

145 roinjection of dipyrone (metamizol) into the periaqueductal gray matter (PAG) in rats causes antinoci

146 The periaqueductal gray matter (PAG) is a major neuroanatomi

147 orsal horn (DH) by brain regions such as the periaqueductal gray matter (PAG) plays a critical role i

148 The periaqueductal gray matter (PAG) projections to the intr

149 The periaqueductal gray matter (PAG), a known modulator of s

150 nervate various targets, including thalamus, periaqueductal gray matter (PAG), and lateral parabrachi

151 nd the dorsolateral quadrant of the midbrain periaqueductal gray matter (PAG).

152 r ibotenic acid lesions of the ventrolateral periaqueductal gray matter (vlPAG) attenuated antinocice

153 -immunoreactive (TH-ir) cells in the ventral periaqueductal gray matter (vPAG) expressed Fos protein

154 reased gray matter in the midbrain including periaqueductal gray matter and nucleus cuneiformis, wher

155 e right amygdala and decreased activation in periaqueductal gray matter and the rostral anterior cing

156 rodents, that deep brain stimulation in the periaqueductal gray matter can rapidly and reversibly ma

157 Because chemokine administration into the periaqueductal gray matter inhibits opioid-induced analg

158 , followed by opioid administration into the periaqueductal gray matter of the brain results in an in

159 mygdala, prefrontal cortex, hippocampus, and periaqueductal gray matter to the control of conditioned

160 y cortex, the anterior cingulate cortex, the periaqueductal gray matter, and other regions.

161 cingulate cortex, caudate nuclei, brain stem periaqueductal gray matter, cerebellum, and occipital co

162 sterior hypothalamic nuclei), and brainstem (periaqueductal gray matter, dorsal and central superior

163 more modest, but labeling was strong in the periaqueductal gray matter, dorsal raphe nucleus, and la

164 tions to lateral hypothalamus, dorsal raphe, periaqueductal gray matter, pericerulear region, rostrov

165 rea, Edinger-Westphal nucleus, ventrolateral periaqueductal gray matter, reticular formation, peduncu

166 r cortices, nucleus accumbens, amygdala, and periaqueductal gray matter.

167 was mediated by projections to the midbrain periaqueductal gray matter.

168 nuclei, central nucleus of the amygdala, and periaqueductal gray matter.

169 from identified spino-parabrachial and spino-periaqueductal gray neurons indicated the presence of pa

170 eritonally) or neurotensin directly into the periaqueductal gray region of the brain.

171 crease in the levels of mu receptor in their periaqueductal gray region, while AS-MOR MM rats showed

172 the hand of nine normal subjects within the periaqueductal gray region.

173 al cortex, lateral orbitofrontal cortex, and periaqueductal gray relative to controls but showed grea

174 The periaqueductal gray represents the final common pathway

175 und no evidence that neurons of the midbrain periaqueductal gray that project to the RVM are postsyna

176 ual anterior cingulate cortex (sgACC) to the periaqueductal gray to the rostral ventromedial medulla

177 rgic locus ceruleus and dopaminergic ventral periaqueductal gray wake neurons.

178 Activation in the periaqueductal gray was significantly increased during t

179 The PVH and ventrolateral periaqueductal gray were recipients of GABAergic outputs

180 ircuitry implicated in retaliation (amygdala/periaqueductal gray) in youths with DBD and low levels o

181 ephalic tegmentum, parabrachial nucleus, and periaqueductal gray), hypothalamus, limbic and paralimbi

182 measured in the dorsal central gray matter (periaqueductal gray), the locus coeruleus, the ventromed

183 g paraventricular nucleus, and ventrolateral periaqueductal gray); hypothalamic visceromotor pattern

184 m the medial prefrontal cortex to the dorsal periaqueductal gray, a brainstem area vital for defensiv

185 differential cardiac timing responses within periaqueductal gray, amygdala and insula.

186 The midbrain periaqueductal gray, an essential link between forebrain

187 s, anterior and posterior PVH, ventrolateral periaqueductal gray, and Barrington's nucleus.

188 tal cortex, ventral striatum, temporal lobe, periaqueductal gray, and cerebellum in eight inbred stra

189 cells, neurons in the lateral parabrachial, periaqueductal gray, and dorsal raphe containing fluorog

190 mRNA is also detected in mammillary nuclei, periaqueductal gray, and dorsal raphe.

191 medial parabrachial, solitary, ventrolateral periaqueductal gray, and interfascicular nuclei.

192 rway motor control (i.e., the Kolliker-Fuse, periaqueductal gray, and intermediate reticular nuclei).

193 nd medial and ventrolateral divisions of the periaqueductal gray, and it sends a light input to the a

194 luding the tuberomammillary nucleus, ventral periaqueductal gray, and locus coeruleus.

195 f the lamina terminalis, supraoptic nucleus, periaqueductal gray, and medial nucleus of the solitary

196 ions, including the ventral tuberal nucleus, periaqueductal gray, and paraventricular regions of the

197 rorubral area, ventrolateral division of the periaqueductal gray, and pontine central gray.

198 stria terminalis, paraventricular thalamus, periaqueductal gray, and precoeruleus.

199 descending pain modulatory system (amygdala, periaqueductal gray, and rostral-ventromedial medulla),

200 BGluM increases in the ipsilateral striatum, periaqueductal gray, and somatosensory cortex, and in co

201 and/or downregulation in cerebellum, caudal periaqueductal gray, and spinal cord and attenuated tole

202 r nucleus, the medial and lateral lemniscus, periaqueductal gray, and the interpeduncular nucleus.

203 minalis, along with their projections to the periaqueductal gray, are strongly implicated in freezing

204 nucleus, median eminence, infundibular stem, periaqueductal gray, area postrema, pontine raphe nucleu

205 nucleus, retrochiasmatic area, ventrolateral periaqueductal gray, Barrington's nucleus), hypothalamic

206 tex, anterior cingulate, the cerebellum, and periaqueductal gray, brain areas that mediate task perfo

207 vidence for substantial dual labeling in the periaqueductal gray, caudal portions of the zona incerta

208 the ventral tegmental area (VTA), habenula, periaqueductal gray, cerebellum, hypothalamus, and hippo

209 to predict arm and leg stimulation from the periaqueductal gray, control regions (e.g., white matter

210 l motor nucleus of the vagus), ventrolateral periaqueductal gray, dorsal parabrachial nucleus, perive

211 amus (LH), mediolateral septum, dorsolateral periaqueductal gray, dorsal raphe, ventral tegmental are

212 hese subdivisions included the ventrolateral periaqueductal gray, dorsomedial hypothalamus, dorsolate

213 ygdala, bed nucleus of the stria terminalis, periaqueductal gray, hippocampus, and dorsal anterior ci

214 idus nucleus, nucleus of the solitary tract, periaqueductal gray, hypothalamic paraventricular nucleu

215 ervation of the parabrachial nucleus and the periaqueductal gray, important nociceptive structures.

216 ar and posterior hypothalamus, zona incerta, periaqueductal gray, intermediate layers of the contrala

217 sal lateral hypothalamic area, ventrolateral periaqueductal gray, lateral parabrachial nucleus and ca

218 terminalis, central nucleus of the amygdala, periaqueductal gray, lateral parabrachial nucleus, nucle

219 scle tone such as those in the ventrolateral periaqueductal gray, lateral pontine tegmentum, locus ce

220 ted [(35)S]GTPgammaS binding was observed in periaqueductal gray, locus coeruleus, lateral parabrachi

221 ulomotor nucleus, red nucleus, raphe nuclei, periaqueductal gray, locus coeruleus, trigeminal nucleus

222 he hypothalamus; and ventral tegmental area, periaqueductal gray, medial and posterior pretectal nucl

223 the wFMNs: superior colliculus, red nucleus, periaqueductal gray, mesencephalon, pons, and several nu

224 ial longitudinal fasciculus, supraoculomotor periaqueductal gray, nucleus of the optic tract, the inf

225 oidance) dependent on the extended amygdala, periaqueductal gray, or septum, all regions that project

226 he substantia nigra, ventral tegmental area, periaqueductal gray, parabrachial nucleus, and dorsal va

227 sal lateral hypothalamic area, ventrolateral periaqueductal gray, parabrachial nucleus, and nucleus o

228 ei, hippocampus, amygdala, substantia nigra, periaqueductal gray, paratrochlear nucleus, paralemnisca

229 the two fiber types was also observed in the periaqueductal gray, particularly in the vicinity of the

230 ypothalamic areas, Edinger-Westphal nucleus, periaqueductal gray, pedunculopontine tegmental nucleus,

231 d nucleus of the stria terminalis, amygdala, periaqueductal gray, raphe and parabrachial nuclei) and

232 gdala, hypothalamus, zona incerta, thalamus, periaqueductal gray, raphe nuclei, lateral parabrachial

233 olaminergic and cholinergic cell groups, the periaqueductal gray, several brainstem reticular nuclei,

234 formation, cerebellum, parabrachial nucleus, periaqueductal gray, thalamus, hypothalamus, amygdala, b

235 inalis, the medial and central amygdala, the periaqueductal gray, the dorsal raphe, and the locus coe

236 amic nuclei, the medial geniculate body, the periaqueductal gray, the ventral tegmental area, the sup

237 and lateral nuclei of the hypothalamus; and periaqueductal gray, ventral tegmental area, substantia

238 In lateral periaqueductal gray, virgin mice showed a significant Eg

239 etabolic decreases in insular cortex and the periaqueductal gray, were noted.

240 cerebellum, hippocampus, olfactory bulb, and periaqueductal gray, with in situ hybridization.

241 CG and OFC, mPFC, LHA, VMN, hippocampus, and periaqueductal gray, with largest effect sizes in mPFC a

242 mine the organization of the medial preoptic-periaqueductal gray-nucleus paragigantocellularis pathwa

243 s known to have extensive projections to the periaqueductal gray.

244 matosensory cortex, anterior insula, and the periaqueductal gray.

245 6) parabrachial/Kolliker-Fuse nuclei; and 7) periaqueductal gray.

246 found in the hypothalamus, dorsal raphe, and periaqueductal gray.

247 e in 9 regions, including lateral septum and periaqueductal gray.

248 om the ventromedial prefrontal cortex to the periaqueductal gray.

249 s of two general areas: the hypothalamus and periaqueductal gray.

250 r limitans nucleus, superior colliculus, and periaqueductal gray.

251 aining, as well as the cuneiform nucleus and periaqueductal gray.

252 he ventral secondary cortex targets midbrain periaqueductal gray.

253 l prefrontal cortex, insula, and dorsal pons/periaqueductal gray.

254 pontine reticular formation, and the lateral periaqueductal gray.

255 ing neurons of the substantia nigra, and the periaqueductal gray.

256 , pontine and caudal dorsal raphe nuclei and periaqueductal gray.

257 f the hypothalamus; and to the ventrolateral periaqueductal gray.

258 circuit comprising the nucleus accumbens and periaqueductal gray.

259 e midcingulate cortex, insula, amygdala, and periaqueductal gray.

260 it presynaptic GABA neurotransmission in the periaqueductal gray.

261 amygdala and globus pallidus, and bilateral periaqueductal gray.

262 this effect correlated with activity in the periaqueductal gray.

263 al midbrain/pontine tegmentum, including the periaqueductal gray/nucleus cuneiformis.

264 portion); ventromedial hypothalamus; lateral periaqueductal gray; and medial, central, and basolatera

265 pramammillary nuclei; ventrolateral midbrain periaqueductal gray; rostral and midlevel ventrolateral

266 ation of neurons in the lateral/dorsolateral periaqueductal grey (l/dlPAG) produces increases in hear

267 tial activation in individual columns of the periaqueductal grey (PAG) during breathlessness and its

268 The midbrain periaqueductal grey (PAG) lies at the heart of the defen

269 elta or kappa opioid receptors occurs in the periaqueductal grey (PAG) of adult male S-D rats.

270 ed from the rostral ACC (rACC), thalamus and periaqueductal grey (PAG) of CCI and sham-operated mice.

271 mol (250 pmol) into the caudal ventrolateral periaqueductal grey (PAG), but not at other sites in the

272 , deep layers of superior colliculus (DLSC), periaqueductal grey (PAG), or caudal pontine reticular f

273 or region (subthalamic nucleus, STN) and the periaqueductal grey (PAG), which have now been recorded

274 s originating in the brainstem ventrolateral periaqueductal grey (VL-PAG), which control the spinal p

275 The ventrolateral periaqueductal grey (vlPAG) has a well-established role

276 with overactive projections to the amygdala, periaqueductal grey and striatum, and an underactive med

277 he greatest neural changes were found in the periaqueductal grey area (PAG) where anticipation of exe

278 natomical tracing methods to define midbrain periaqueductal grey circuits for specific defensive beha

279 s freezing by disinhibition of ventrolateral periaqueductal grey excitatory outputs to pre-motor targ

280 f activity within dorsal anterior cingulate, periaqueductal grey matter (PAG) and superior temporal g

281 to increasing bladder volume was seen in the periaqueductal grey matter (PAG), in the midline pons, i

282 t to various brain areas including thalamus, periaqueductal grey matter (PAG), lateral parabrachial a

283 1 nucleus was found to project mainly to the periaqueductal grey matter (PAG), predominantly ipsilate

284 EM-off neurons (located in the ventrolateral periaqueductal grey matter (vlPAG) and lateral pontine t

285 have identified increased activity with the periaqueductal grey matter associated with stimulation o

286 inated release of 2-AG and anandamide in the periaqueductal grey matter might mediate opioid-independ

287 ockade of cannabinoid CB(1) receptors in the periaqueductal grey matter of the midbrain prevents non-

288 G concentrations and, when injected into the periaqueductal grey matter, enhances stress-induced anal

289 The dorsal raphe nucleus/ periaqueductal grey region of the midbrain and hippocamp

290 nucleus of the amygdala to the ventrolateral periaqueductal grey that produces freezing by disinhibit

291 eactivity of the amygdala, hypothalamus, and periaqueductal grey to angry facial expressions.

292 n secondary within-group analyses, increased periaqueductal grey volume was associated with role limi

293 s of the anterior cingulate cortex (ACC) and periaqueductal grey, areas involved in pain processing,

294 teral hypothalamus, midbrain tegmental area, periaqueductal grey, dorsal pons and various cortical ar

295 ased in the bilateral substantia nigra, left periaqueductal grey, right posterior cingulate cortex an

296 rtex, dentate gyrus, thalamus, hypothalamus, periaqueductal grey, superior colliculus, locus coeruleu

297 Dense SPL-IR areas included the periaqueductal grey, trigeminal nuclei, dorsal raphe, an

298 No c-Fos-ir was apparent in the periaqueductal grey.

299 ssing GABAergic neurons in the ventrolateral periaqueductal grey.

300 , retrosplenial cortex, medial thalamus, and periaqueductal/periventricular gray.