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1 ygenase metabolites as has been shown in the periaqueductal gray ().
2 6) parabrachial/Kolliker-Fuse nuclei; and 7) periaqueductal gray.
3 found in the hypothalamus, dorsal raphe, and periaqueductal gray.
4 e in 9 regions, including lateral septum and periaqueductal gray.
5 om the ventromedial prefrontal cortex to the periaqueductal gray.
6 s of two general areas: the hypothalamus and periaqueductal gray.
7 r limitans nucleus, superior colliculus, and periaqueductal gray.
8 aining, as well as the cuneiform nucleus and periaqueductal gray.
9 he ventral secondary cortex targets midbrain periaqueductal gray.
10 l prefrontal cortex, insula, and dorsal pons/periaqueductal gray.
11 pontine reticular formation, and the lateral periaqueductal gray.
12 ing neurons of the substantia nigra, and the periaqueductal gray.
13 , pontine and caudal dorsal raphe nuclei and periaqueductal gray.
14 f the hypothalamus; and to the ventrolateral periaqueductal gray.
15 the stria terminals, subfornical organ, and periaqueductal gray.
16 ne analgesia elicited from the ventrolateral periaqueductal gray.
17 circuit comprising the nucleus accumbens and periaqueductal gray.
18 e midcingulate cortex, insula, amygdala, and periaqueductal gray.
19 it presynaptic GABA neurotransmission in the periaqueductal gray.
20 amygdala and globus pallidus, and bilateral periaqueductal gray.
21 this effect correlated with activity in the periaqueductal gray.
22 s known to have extensive projections to the periaqueductal gray.
23 matosensory cortex, anterior insula, and the periaqueductal gray.
24 periaqueductal gray (54%); and ventrolateral periaqueductal gray (52%) when compared with basal bindi
25 ); dorsal raphe nucleus (53%); dorsal medial periaqueductal gray (54%); and ventrolateral periaqueduc
26 m the medial prefrontal cortex to the dorsal periaqueductal gray, a brainstem area vital for defensiv
30 in structures involved in vocal production (periaqueductal gray and anterior hypothalamus) was signi
32 ially to the posterior hypothalamic area and periaqueductal gray and caudally along the brachium of t
33 Substantial labeling of the locus coeruleus, periaqueductal gray and forebrain regions occurred at la
35 y in the ventrolateral portion of the caudal periaqueductal gray and in the lateral septum in aggress
37 central roles in pain and affect, including periaqueductal gray and nearby dorsal raphe and nucleus
39 anterior hypothalamus) and of the midbrain (periaqueductal gray and paralemniscal tegmentum) reveal
40 ncreased functional connectivity between the periaqueductal gray and rostral anterior cingulate, as h
42 m and also expressed at much lower levels in periaqueductal gray and spinal cord, structures known to
43 system (CNS) pain modulatory regions (i.e., periaqueductal gray and subarachnoid lumbar spinal cord)
44 rtions of the lateral septum), midbrain (the periaqueductal gray and the intermediate layers of super
47 tal cortex, ventral striatum, temporal lobe, periaqueductal gray, and cerebellum in eight inbred stra
48 cells, neurons in the lateral parabrachial, periaqueductal gray, and dorsal raphe containing fluorog
51 rway motor control (i.e., the Kolliker-Fuse, periaqueductal gray, and intermediate reticular nuclei).
52 nd medial and ventrolateral divisions of the periaqueductal gray, and it sends a light input to the a
53 luding the lateral parabrachial nucleus, the periaqueductal gray, and laminae I-II of the spinal trig
54 lis, perifornical and anterior hypothalamus, periaqueductal gray, and lateral parabrachial nucleus.
56 f the lamina terminalis, supraoptic nucleus, periaqueductal gray, and medial nucleus of the solitary
57 ions, including the ventral tuberal nucleus, periaqueductal gray, and paraventricular regions of the
60 descending pain modulatory system (amygdala, periaqueductal gray, and rostral-ventromedial medulla),
61 BGluM increases in the ipsilateral striatum, periaqueductal gray, and somatosensory cortex, and in co
62 and/or downregulation in cerebellum, caudal periaqueductal gray, and spinal cord and attenuated tole
63 r nucleus, the medial and lateral lemniscus, periaqueductal gray, and the interpeduncular nucleus.
64 rom neurons in the lateral parabrachial, the periaqueductal gray, and/or the dorsal raphe nuclei.
65 portion); ventromedial hypothalamus; lateral periaqueductal gray; and medial, central, and basolatera
66 minalis, along with their projections to the periaqueductal gray, are strongly implicated in freezing
68 ucleus, Kolliker-Fuse nucleus, ventrolateral periaqueductal gray area, central nucleus of the amygdal
70 nucleus, median eminence, infundibular stem, periaqueductal gray, area postrema, pontine raphe nucleu
71 g) analgesia elicited from the ventrolateral periaqueductal gray as measured by the tail-flick and ju
72 nucleus, retrochiasmatic area, ventrolateral periaqueductal gray, Barrington's nucleus), hypothalamic
73 tex, anterior cingulate, the cerebellum, and periaqueductal gray, brain areas that mediate task perfo
74 signaling is modulated in the ventrolateral periaqueductal gray by persistent inflammation different
75 vidence for substantial dual labeling in the periaqueductal gray, caudal portions of the zona incerta
76 the ventral tegmental area (VTA), habenula, periaqueductal gray, cerebellum, hypothalamus, and hippo
77 to predict arm and leg stimulation from the periaqueductal gray, control regions (e.g., white matter
78 tween the ventromedial prefrontal cortex and periaqueductal gray correlated with somatosensory decrea
79 partum lesions of the caudal intercollicular periaqueductal gray (cPAG-x) was confirmed and was exten
80 her OT receptor activity in the ventrocaudal periaqueductal gray (cPAGv) contributes to mothers' redu
82 ductal gray (vl PAG) versus the dorsolateral periaqueductal gray (dl PAG), in the rabbit, elicits two
83 lating neuronal activity of the dorsolateral periaqueductal gray (dl-PAG) through excitatory and inhi
85 that VMHdm/c projection to the dorsolateral periaqueductal gray (dlPAG) induces inflexible immobilit
87 l motor nucleus of the vagus), ventrolateral periaqueductal gray, dorsal parabrachial nucleus, perive
88 amus (LH), mediolateral septum, dorsolateral periaqueductal gray, dorsal raphe, ventral tegmental are
89 hese subdivisions included the ventrolateral periaqueductal gray, dorsomedial hypothalamus, dorsolate
91 al gray (VPAG), or sympathoexcitatory dorsal periaqueductal gray (DPAG), differentially modulates CBF
92 rojections from the ARC to the ventrolateral periaqueductal gray during EA at P5-P6 contribute to inh
93 (vmPFC), insula, amygdala, hypothalamus, and periaqueductal gray emerge as central brain structures u
94 wedge of labeled neurons in the dorsolateral periaqueductal gray extending into the deep layers of th
95 ygdala, bed nucleus of the stria terminalis, periaqueductal gray, hippocampus, and dorsal anterior ci
96 g paraventricular nucleus, and ventrolateral periaqueductal gray); hypothalamic visceromotor pattern
97 idus nucleus, nucleus of the solitary tract, periaqueductal gray, hypothalamic paraventricular nucleu
98 ephalic tegmentum, parabrachial nucleus, and periaqueductal gray), hypothalamus, limbic and paralimbi
99 ervation of the parabrachial nucleus and the periaqueductal gray, important nociceptive structures.
100 fralimbic prefrontal cortex, hippocampus and periaqueductal gray in extinction learning, while mainta
101 dies in the midbrain reticular formation and periaqueductal gray in four clinically documented and ge
102 ic motor nucleus, in a band just beneath the periaqueductal gray in the midbrain, in ventricular regi
103 ircuitry implicated in retaliation (amygdala/periaqueductal gray) in youths with DBD and low levels o
104 ar and posterior hypothalamus, zona incerta, periaqueductal gray, intermediate layers of the contrala
105 rect evidence supporting the notion that the periaqueductal gray is a site for higher cortical contro
106 ia elicited by morphine in the ventrolateral periaqueductal gray is mediated in part by NMDA and chol
107 f the projections from the ICx to the dorsal periaqueductal gray is sufficient for provoking flight b
108 ral mesencephalic reticular formation (MRF), periaqueductal gray, Kolliker-Fuse nucleus, and pontis c
109 sal lateral hypothalamic area, ventrolateral periaqueductal gray, lateral parabrachial nucleus and ca
110 terminalis, central nucleus of the amygdala, periaqueductal gray, lateral parabrachial nucleus, nucle
111 scle tone such as those in the ventrolateral periaqueductal gray, lateral pontine tegmentum, locus ce
112 ted [(35)S]GTPgammaS binding was observed in periaqueductal gray, locus coeruleus, lateral parabrachi
113 mic nucleus, posterior hypothalamic nucleus, periaqueductal gray, locus coeruleus, nucleus accumbens,
114 ulomotor nucleus, red nucleus, raphe nuclei, periaqueductal gray, locus coeruleus, trigeminal nucleus
115 ned the role of eNOS within the dorsolateral periaqueductal gray mater (dlPAG) on cardiovascular resp
116 bnormal in hippocampus ( approximately 10%), periaqueductal gray matter ( approximately 13%), fimbria
119 al cortex, the lateral hypothalamus, and the periaqueductal gray matter (PAG) are involved in these c
120 ase in the total phosphatase activity in the periaqueductal gray matter (PAG) from morphine-pelleted
121 roinjection of dipyrone (metamizol) into the periaqueductal gray matter (PAG) in rats causes antinoci
123 orsal horn (DH) by brain regions such as the periaqueductal gray matter (PAG) plays a critical role i
126 nervate various targets, including thalamus, periaqueductal gray matter (PAG), and lateral parabrachi
128 r ibotenic acid lesions of the ventrolateral periaqueductal gray matter (vlPAG) attenuated antinocice
129 -immunoreactive (TH-ir) cells in the ventral periaqueductal gray matter (vPAG) expressed Fos protein
130 reased gray matter in the midbrain including periaqueductal gray matter and nucleus cuneiformis, wher
131 e right amygdala and decreased activation in periaqueductal gray matter and the rostral anterior cing
132 rodents, that deep brain stimulation in the periaqueductal gray matter can rapidly and reversibly ma
133 Because chemokine administration into the periaqueductal gray matter inhibits opioid-induced analg
134 , followed by opioid administration into the periaqueductal gray matter of the brain results in an in
135 mygdala, prefrontal cortex, hippocampus, and periaqueductal gray matter to the control of conditioned
137 cingulate cortex, caudate nuclei, brain stem periaqueductal gray matter, cerebellum, and occipital co
138 sterior hypothalamic nuclei), and brainstem (periaqueductal gray matter, dorsal and central superior
139 more modest, but labeling was strong in the periaqueductal gray matter, dorsal raphe nucleus, and la
140 tions to lateral hypothalamus, dorsal raphe, periaqueductal gray matter, pericerulear region, rostrov
141 al scar, these fibers elongated in white and periaqueductal gray matter, reaching the farthest distan
142 rea, Edinger-Westphal nucleus, ventrolateral periaqueductal gray matter, reticular formation, peduncu
146 he hypothalamus; and ventral tegmental area, periaqueductal gray, medial and posterior pretectal nucl
147 the wFMNs: superior colliculus, red nucleus, periaqueductal gray, mesencephalon, pons, and several nu
148 tive axons descended ventromedially into the periaqueductal gray, moving caudally and arborizing exte
149 from identified spino-parabrachial and spino-periaqueductal gray neurons indicated the presence of pa
150 ial longitudinal fasciculus, supraoculomotor periaqueductal gray, nucleus of the optic tract, the inf
151 mine the organization of the medial preoptic-periaqueductal gray-nucleus paragigantocellularis pathwa
153 oidance) dependent on the extended amygdala, periaqueductal gray, or septum, all regions that project
157 ceptors, acts by an unknown mechanism in the periaqueductal gray (PAG) and raphe magnus (RM) to stimu
158 could be mediated in part by actions in the periaqueductal gray (PAG) and the dorsal raphe nucleus (
160 CeL neurons directly project to the midbrain periaqueductal gray (PAG) and the paraventricular nucleu
161 se VMH efferents travel caudally through the periaqueductal gray (PAG) and then ventrally through the
163 The different subdivisions of the midbrain periaqueductal gray (PAG) are intricately (and different
164 attention or expectancy have identified the periaqueductal gray (PAG) as a key brainstem structure i
165 the endogenous cannabinoid anandamide in the periaqueductal gray (PAG) by in vivo microdialysis in th
166 he generation of DRRs and stimulation of the periaqueductal gray (PAG) can induce the release of GABA
167 binds to mu-opioid receptors (MORs), and the periaqueductal gray (PAG) contains a dense population of
170 onnectivity fluctuations between the DMN and periaqueductal gray (PAG) dynamically tracked spontaneou
172 pothalamic nucleus (VMH) that project to the periaqueductal gray (PAG) form a crucial segment of the
173 NK(1)-Substance P receptors in the midbrain periaqueductal gray (PAG) in defensive rage behavior in
174 imulation in different parts of the midbrain periaqueductal gray (PAG) in the cat generates four diff
175 l evidence that neurons in the ventrolateral periaqueductal gray (PAG) innervate the medial pericoeru
183 t.SIGNIFICANCE STATEMENT We demonstrate that periaqueductal gray (PAG) microglia contribute to the se
184 expression is increased in the ventrolateral periaqueductal gray (PAG) neurons following precipitated
185 and spontaneous firings of rat ventrolateral periaqueductal gray (PAG) neurons, either mechanically d
186 tracellular Ca(2+) levels in dissociated rat periaqueductal gray (PAG) neurons, which express GPR55 m
188 ala by recording neurons in the amygdala and periaqueductal gray (PAG) of rats during fear conditioni
191 M), but not following co-injections into the periaqueductal gray (PAG) or into the spinal subarachnoi
192 by lesions of brainstem regions such as the periaqueductal gray (PAG) or the rostral ventromedial me
195 nal motor system, in which the mesencephalic periaqueductal gray (PAG) plays a central role, as demon
199 beta-endorphin in the rVLM as well as in the periaqueductal gray (PAG) that are involved in EA-mediat
200 ng emotions and projects to the amygdala and periaqueductal gray (PAG) to modulate emotional response
202 on of prefrontal cortical projections to the periaqueductal gray (PAG) were defined with retrograde a
203 lamus (CH) have been shown to project to the periaqueductal gray (PAG) which, in turn, sends descendi
204 ult of increased microglia activation in the periaqueductal gray (PAG), a central locus mediating the
205 state functional connectivity (rs-fc) of the periaqueductal gray (PAG), a key region in the descendin
206 yngeal nerve (ISLN) activates neurons of the periaqueductal gray (PAG), a midbrain region implicated
207 We found that pain PEs were encoded in the periaqueductal gray (PAG), a structure important for pai
208 havior in the cat elicited from the midbrain periaqueductal gray (PAG), and that such effects are blo
209 halamus, the amygdala, the hypothalamus, the periaqueductal gray (PAG), and the brainstem reticular f
210 ciation between the brainstem areas, such as periaqueductal gray (PAG), and the headache phase of mig
211 rticular, the medullary reticular formation, periaqueductal gray (PAG), and ventromedial nucleus of t
214 d 81, 96, 106, and 82 tolerance genes in the periaqueductal gray (PAG), prefrontal cortex, temporal l
215 ministered systemically or directly into the periaqueductal gray (PAG), produces a significantly grea
216 stent with the location of areas such as the periaqueductal gray (PAG), rostral ventral medulla, and
217 We show that BDNF-containing neurons in the periaqueductal gray (PAG), the central structure for pai
218 ctions to the rostral midbrain including the periaqueductal gray (PAG), the deep layers of the superi
219 lowing administration into the ventrolateral periaqueductal gray (PAG), the dorsal PAG, and the rostr
220 or-expressing neurons (LepRb neurons) in the periaqueductal gray (PAG), the largest population of Lep
221 D), the lateral hypothalamic area (LHA), the periaqueductal gray (PAG), the parabrachial nucleus (Pb)
222 lateral and the upper lateral portion of the periaqueductal gray (PAG), the Su3 and PV2 nuclei of the
223 ds and to determine the role of the midbrain periaqueductal gray (PAG), which mediates stress-induced
238 tic (MPO) produced dense labeling within the periaqueductal gray (PAG); anterogradely labeled fibers
240 he substantia nigra, ventral tegmental area, periaqueductal gray, parabrachial nucleus, and dorsal va
241 were confined to the ventral tegmental area, periaqueductal gray, parabrachial nucleus, and locus coe
242 sal lateral hypothalamic area, ventrolateral periaqueductal gray, parabrachial nucleus, and nucleus o
243 ei, hippocampus, amygdala, substantia nigra, periaqueductal gray, paratrochlear nucleus, paralemnisca
244 the two fiber types was also observed in the periaqueductal gray, particularly in the vicinity of the
245 ypothalamic areas, Edinger-Westphal nucleus, periaqueductal gray, pedunculopontine tegmental nucleus,
246 d nucleus of the stria terminalis, amygdala, periaqueductal gray, raphe and parabrachial nuclei) and
247 gdala, hypothalamus, zona incerta, thalamus, periaqueductal gray, raphe nuclei, lateral parabrachial
248 These findings indicate that neurons in the periaqueductal gray region of the brain have an inherent
250 xide (NO) donating compounds into the dorsal periaqueductal gray region of the midbrain (PAG) decreas
252 crease in the levels of mu receptor in their periaqueductal gray region, while AS-MOR MM rats showed
254 al cortex, lateral orbitofrontal cortex, and periaqueductal gray relative to controls but showed grea
256 pramammillary nuclei; ventrolateral midbrain periaqueductal gray; rostral and midlevel ventrolateral
257 sis that neurons in the rostral ventromedial periaqueductal gray (rvmPAG) are a source of inhibitory
258 olaminergic and cholinergic cell groups, the periaqueductal gray, several brainstem reticular nuclei,
259 formation, cerebellum, parabrachial nucleus, periaqueductal gray, thalamus, hypothalamus, amygdala, b
260 und no evidence that neurons of the midbrain periaqueductal gray that project to the RVM are postsyna
261 measured in the dorsal central gray matter (periaqueductal gray), the locus coeruleus, the ventromed
262 inalis, the medial and central amygdala, the periaqueductal gray, the dorsal raphe, and the locus coe
263 amic nuclei, the medial geniculate body, the periaqueductal gray, the ventral tegmental area, the sup
264 ual anterior cingulate cortex (sgACC) to the periaqueductal gray to the rostral ventromedial medulla
265 orphine microinjected into the ventrolateral periaqueductal gray using both the foot-withdrawal and t
266 and lateral nuclei of the hypothalamus; and periaqueductal gray, ventral tegmental area, substantia
268 electrical stimulation of the ventrolateral periaqueductal gray (vl PAG) versus the dorsolateral per
269 resynaptic GABA release in the ventrolateral periaqueductal gray (vlPAG) activates the descending ant
270 ulatory regions, including the ventrolateral periaqueductal gray (vlPAG) and locus ceruleus (LC).
271 een shown that the ventrolateral part of the periaqueductal gray (VLPAG) and the adjacent dorsal deep
275 een central amygdala (CeA) and ventrolateral periaqueductal gray (vlPAG) is implicated in several dis
277 n (PG) E2 signaling within the ventrolateral periaqueductal gray (vlPAG) is pronociceptive in naive a
279 inociception elicited from the ventrolateral periaqueductal gray (vlPAG) on two nociceptive measures.
281 hat the ventrolateral column of the midbrain periaqueductal gray (vlPAG) region mediates the hypotens
282 ed IS induces DeltaFosB in the ventrolateral periaqueductal gray (vlPAG), and levels of the protein a
286 la (17%), midline raphe (40%), ventrolateral periaqueductal gray (VLPAG, 15%), lateral hypothalamic a
287 croinjecting morphine into the ventrolateral periaqueductal gray (vPAG) develops with repeated admini
289 istration of morphine into the ventrolateral periaqueductal gray (vPAG), a key structure contributing
290 of neurons of the sympathoinhibitory ventral periaqueductal gray (VPAG), or sympathoexcitatory dorsal
292 l forebrain (SLEA) and the ventral tegmentum/periaqueductal gray (VT/PAG), while foci of increased si
294 eta-endorphin analgesia in the ventrolateral periaqueductal gray was confirmed by its sensitivity to
296 lus retroflexus, interpeduncular nuclei, and periaqueductal gray were also stained with this antibody
300 CG and OFC, mPFC, LHA, VMN, hippocampus, and periaqueductal gray, with largest effect sizes in mPFC a
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