ライフサイエンスコーパス: periarteriolar

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1 ting a fern-like pattern with sparing of the periarteriolar area in all cases.

2 ncreasing vascular compliance, regression of periarteriolar collagen area, improvement of coronary re

3 o suggest a homology between this vasoactive periarteriolar fat and both periarterial and visceral fa

4 association with VEGFR2 blockade resulted in periarteriolar inflammation with macrophages, and B cell

5 ly arrays of splenic macrophages surrounding periarteriolar lymphatic sheaths and a red pulp depletio

6 ver, BDC-Idd9 T cells accumulated in splenic periarteriolar lymphatic sheaths, whereas BDC T cells we

7 cur in the marginal zone and surrounding the periarteriolar lymphocyte sheaths.

8 X40 is maximally expressed by T cells in the periarteriolar lymphoid sheath (PALS) 3 d after primary

9 20+ B cells clustered within the T cell-rich periarteriolar lymphoid sheath (PALS) and surrounding fo

10 were observed in or near the CD4 T cell-rich periarteriolar lymphoid sheath (PALS) during the entire

11 ion of Lm from the marginal zone (MZ) to the periarteriolar lymphoid sheath (PALS) was inhibited by p

12 n the splenic marginal zone, migrated to the periarteriolar lymphoid sheath (PALS) where they grew ex

13 pheral lymphoid organs and accumulate in the periarteriolar lymphoid sheath but are unable to generat

14 ls (FRCs) form a network in the T cell zone (periarteriolar lymphoid sheath, PALS) of the WP on which

15 the chemokine receptor CCR7, are home to the periarteriolar lymphoid sheath, whereas activated TH2 ce

16 s that were exclusively found in the splenic periarteriolar lymphoid sheath.

17 but C4H3+ DC rapidly accumulate in the outer periarteriolar lymphoid sheaths (PALS) and in follicular

18 on, activated splenic T cells proliferate in periarteriolar lymphoid sheaths (PALS) and subsequently

19 old increase in the T cell-rich areas of the periarteriolar lymphoid sheaths (PALs).

20 , foci of KJ1-26+ cells were observed in the periarteriolar lymphoid sheaths at day 3; these were not

21 ion at the expense of HSC-maintaining NG2(+) periarteriolar niche cells.

22 alters the distribution of HSCs from NG2(+) periarteriolar niches to LEPR(+) perisinusoidal niches.

23 ail to migrate from the marginal zone to the periarteriolar region of the spleen.

24 n the spleen, where they were located in the periarteriolar sheath, marginal zone, and interfollicula

25 al reveal a novel function for podoplanin on periarteriolar stromal cells in the bone marrow: promoti

26 ssive lengthening stimulates the activity of periarteriolar sympathetic nerves; this activity propaga

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