1 at P(0) protein was mistargeted to abaxonal, periaxonal, and mesaxon membranes in P(0)(tg) Schwann ce

2 absent myelin sheaths, vacuolation, enlarged periaxonal collars, oligodendrocyte cell death, and axon

3 t treatment, viral spread and replication in periaxonal glial cells confound study of axonal transpor

4 ssociated glycoprotein (MAG) is expressed in periaxonal membranes of myelinating glia where it is bel

5 orted in the Golgi apparatus and targeted to periaxonal membranes.

6 m current Kdr as a result of accumulation of periaxonal potassium in concert with a reduced influx of

7 s, and MAG and MAP1B also colocalized in the periaxonal region of myelinated axons.

8   We show that in response to axonal injury, periaxonal Schwann cells release erythropoietin (EPO), w

9      Concurrently, the fluid in the expanded periaxonal space accumulated into droplets that appeared

10 (+)-K(+)-ATPase extrudes these ions into the periaxonal space beneath the myelin rather than into the

11 yelin lamellae of the sheath and a distended periaxonal space between the axon and myelin sheaths.

12 esence of a small working volume such as the periaxonal space between the myelin sheath and the axon

13 rs in the myelinated axon and the restricted periaxonal space between the myelin sheath and the axon.

14  and to indicate accumulation of ions in the periaxonal space followed by their release at the parano

15        All 3 had numerous macrophages in the periaxonal space of myelinated internodes, and rare intr

16 vere cases IgG and C3d were found within the periaxonal space of the myelinated internodes, bound to

17 ther in the inner myelin intralaminar and/or periaxonal space that separates axons from myelinating S

18 egrity and plasticity and maintenance of the periaxonal space.

19  In particular, the development of extensive periaxonal spaces in the large-diameter axons was reduce