1                                              Pericholecystic abscess, intraluminal membranes, and wal

2 wall appearance, distention, bile color, and pericholecystic fluid were not individually significant

3 udge and/or stones, (c) wall appearance, (d) pericholecystic fluid, and (e) common bile duct size and

4   Sensitivities of increased wall thickness, pericholecystic fluid, and adjacent fat signal intensity

5  thickening, echotexture, and intramural and pericholecystic fluid.

6 enhancement (P<.001) and increased transient pericholecystic hepatic enhancement (P=.003) were the mo

7 der wall enhancement and increased transient pericholecystic hepatic enhancement had the highest comb

8 der wall enhancement and increased transient pericholecystic hepatic enhancement were 74% (14 of 19 p

9 the development of intramural (P = .015) and pericholecystic (P = .006) fluid.

10 nstrated gallbladder nonvisualization with a pericholecystic rim sign at 1 hr, a cholescintigraphic p

11  from 72% (gallbladder nonvisualization with pericholecystic rim sign before morphine) to 86% (gallbl

12 tion at 1 hr (before morphine); those with a pericholecystic rim sign were further evaluated for pers

13 ntigrams were analyzed for the presence of a pericholecystic rim sign, marked or mild, associated wit

14  1 hr of gallbladder nonvisualization with a pericholecystic rim sign, regardless of its intensity.

15  cholecystitis, however, 10 (32%) had a mild pericholecystic rim sign.

16 nstrated gallbladder nonvisualization with a pericholecystic rim sign.

17                   Of 24 patients with marked pericholecystic rim signs, 21 had acute cholecystitis.

18 ered present if there was enlargement of the pericholecystic space (i.e., gallbladder fossa) and the

19                                              Pericholecystic stranding was seen in 19, biliary dilata

20 tudied - wall thickness, intramural nodules, pericholecystic stranding, wall thickness, THAD, fat in