ライフサイエンスコーパス: periclinal

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1 owed a limited deconstruction of the initial periclinal array followed by a progressive array reorgan

2 atory gene, CYCD6;1, and regulates formative periclinal asymmetric cell divisions in endodermis and c

3 rs are impaired from the "symmetry-breaking" periclinal cell divisions during the transition between

4 ddle cortex (MC), and cortex are produced by periclinal cell divisions that occur at different positi

5 d from microtubules originating on the outer periclinal cell face, pointing to a cell-directed, rathe

6 verse coordinated between the anticlinal and periclinal cell faces.

7 ight lead to chloroplast accumulation on the periclinal cell walls, whereas light intensities over 20

8 seedlings, the cortical array on the outer (periclinal) cell face creates a variety of array pattern

9 oscale and mesoscale structure of the outer (periclinal) cell wall of onion scale epidermis - a model

10 evel of layer-specific expression by using a periclinal chimera that has its L1 layer from Solanum pe

11 We have analyzed periclinal chimeras and mericlinal sectors of jointless

12 insertion in the MADS box region to generate periclinal chimeras expressing alleles with different ac

13 ' lam1 mutant of Nicotiana sylvestris and in periclinal chimeras with lam1 and wild-type (N. glauca)

14 om revertant plants, indicated that all were periclinal chimeras with wild-type fim expression only i

15 autonomy by examining fim expression in flo periclinal chimeras.

16 o development and that none showed the usual periclinal division leading to the formation of the prot

17 liferation and is responsible for triggering periclinal division of subepidermal cells.

18 romotes tapetum differentiation and inhibits periclinal division once a tapetal cell is specified.

19 Moreover, extra periclinal divisions (new wall parallel to surface of th

20 re length of the root tip, but only promotes periclinal divisions at specific sites.

21 scr) and short root (shr) suppress the extra periclinal divisions characteristic of scz mutant roots.

22 nd tissue, and (2) it is required to repress periclinal divisions in the meristem.

23 Disrupting SCR function abolished periclinal divisions in this lateral root primordia cell

24 These protodermal periclinal divisions occur at the expense of normal anti

25 ermis and cortex arise continuously from the periclinal divisions of cells that surround the quiescen

26 The MC arises between days 7 and 14 from periclinal divisions of the endodermis.

27 ferentiate, and, instead, undergo additional periclinal divisions to form extra layers of cells.

28 he parietal layer then undergo two cycles of periclinal divisions to give rise to three wall layers.

29 cell layers1 (Xcl1) mutation causes oblique, periclinal divisions to occur in the protoderm layer.

30 rs are generated properly through successive periclinal divisions, in the ms32 mutant, tapetal precur

31 GRFs increases the meristem size and induces periclinal formative divisions in transit-amplifying cel

32 e rates of light, chloroplasts accumulate in periclinal layers perpendicular to the direction of ligh

33 ues, is abundant in anticlinal and the inner periclinal plasma membrane of 'outside' cells.

34 e-enriched and microtubule-free zones at the periclinal wall in neighboring cells predicted sites of

35 Cortical microtubules adjacent to the periclinal wall were persistently enriched at the convex

36 ght, mesophyll chloroplasts spread along the periclinal walls normal to the light, maximizing absorba

37 creased with increasing cell size, while for periclinal walls, the number of PDs decreased.

38 tubule networks that span the anticlinal and periclinal walls.

39 fferential growth of both the anticlinal and periclinal walls.

40 -1), chloroplasts in pmi2 leaves move to the periclinal walls; 100 micromol m(-2) s(-1) of blue light

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