ライフサイエンスコーパス: perifollicular

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1 - and MMP-9-expressing cells were present in perifollicular and dermal inflammatory infiltrates withi

2 nd CD200-deficient C57BL/6 mice, significant perifollicular and intrafollicular inflammation was obse

3 nd interdigitating cells) and present in few perifollicular and intrafollicular lymphoid blasts.

4 s of CD3+, CD4+ T cells were elevated in the perifollicular and papillary dermis although levels were

5 Perifollicular angiogenesis was temporally and spatially

6 d vascular-endothelial-growth-factor-induced perifollicular angiogenesis, whereas the catagen regress

7 issue, by cells associated with sinusoids in perifollicular areas of spleen tissue, and at very high

8 63 expression within the HF infundibulum and perifollicular epidermis.

9 lted in an increased binding of monocytes to perifollicular high endothelial venules (HEVs) of lymph

10 e inflamed skin revealed intrafollicular and perifollicular human CD4(+) cells near F4/80(+) mouse ma

11 Perifollicular IFN-gamma+ CD8 T cells were rare in secon

12 evidence that IL-17+ cells are found in the perifollicular infiltrate of comedones.

13 Nilotinib therapy may induce perifollicular inflammation and widespread persistent al

14 mice developed scarring alopecia and severe perifollicular inflammation.

15 e rs763035 variant, revealed staining in the perifollicular inflammatory infiltrate of rosacea for bo

16 cells and to have FOXP3-positive cells in a perifollicular location.

17 Biopsies revealed perifollicular lymphocytic inflammation and evidence of

18 Thus, perifollicular macrophages contribute to the activation

19 lar B cell areas and the absence of discrete perifollicular marginal and mantle zones; the formation

20 ulted in depigmented hair fiber regrowth and perifollicular neutrophil and eosinophil infiltrates but

21 demonstrated no tendency to localize to the perifollicular region, and were similarly distributed as

22 gregation of plasmacytoid DCs in the splenic perifollicular region, follicular translocation of MZ B

23 In the mutant spleen, the characteristic perifollicular rim marking the marginal zone (MZ), which

24 acterized by focal inflammatory lesions with perifollicular T-cell infiltrates, reflecting the role o

25 CCR6-deficient B(mem) from the marginal and perifollicular to the follicular/germinal center area.

26 tudy aimed to quantify the cyclic changes of perifollicular vascularization and to characterize the b

27 icantly prolonged, associated with increased perifollicular vascularization and vascular proliferatio

28 We found a significant increase in perifollicular vascularization during the growth phase (

29 tinocytes of hair follicles strongly induced perifollicular vascularization, resulting in accelerated

30 heath keratinocytes, associated with reduced perifollicular vascularization.

31 ophages in humans are found primarily in the perifollicular zone, whereas in chimpanzees they also oc

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