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1 nsion was recorded during continuous dynamic perifusion.
2                          RESEARCH DESIGN AND Perifusion analyses of isolated Munc18c- (-/+) or Munc18
3                                        Islet perifusion and calcium imaging studies showed abnormal r
4                                        Islet perifusion and calcium-imaging studies showed abnormal r
5 nsulin secretion ability by in vitro glucose perifusion and explore the expression of insulin pathway
6                                              Perifusion assays using pancreatic islets from transgeni
7                                     Further, perifusion assays with human islets isolated from a dono
8                                   In ex vivo perifusion assays, Smad3-deficient islets exhibit improv
9 e insulin secretion profile in dynamic islet perifusion assays.
10 ucose-stimulated insulin release response in perifusion assays.
11                                        Under perifusion conditions, high glucose concentrations induc
12 n from islets and beta cells under static or perifusion conditions, whereas an inactive structural an
13  glucose-stimulated insulin release in islet perifusion experiments and have significantly reduced pa
14                                              Perifusion experiments indicated that cPLA(2) underexpre
15                                              Perifusion experiments with human islets indicated that
16                                           In perifusion experiments with isolated islets in the absen
17                                           In perifusion experiments, acute insulin responses (AIRs) i
18                                           In perifusion experiments, elevations in extracellular Ca2+
19 e studied using [Ca(2+)] imaging, static and perifusion insulin secretion assays, and gap junction pe
20                                 During islet perifusions, KIC and 2 mM glutamine caused robust dose-d
21                       During local CTX + APA perifusion, L-NNA + INDO abolished SCVD while conducted
22                      Low-Ca2+ or Ca(2+)-free perifusion medium induced oscillatory bursting activity
23 d currents (IPSCs) were usually blocked with perifusion of 10-50 microM bicuculline methiodide (BMI).
24 actional [3H]ACh release was recorded during perifusion of acutely dissociated, [3H]choline-labeled,
25                        However, simultaneous perifusion of explants with ATP (100 micrometer) and PE
26                                              Perifusion of fatty acids restored both responses.
27                    In the second experiment, perifusion of hypothalamic slices with 10(-8) or 10(-7)
28                                              Perifusion of neurexin-1alpha KO mouse islets revealed a
29                                              Perifusion of slices with 7.5-10 mM TEA, a K+ channel bl
30                                              Perifusion of slices with media containing 1-2 microM TT
31 failed to induce an IDAP-like current during perifusion of slices with media containing high [K+]o or
32                                              Perifusion of Syn-1A-betaKO islets showed impaired first
33           Surprisingly, insulin secretion in perifusion or static incubation experiments in response
34  measured every 20 min during a 3-h baseline perifusion period and after depolarization with 56 mM KC
35 functional capacity of islets as assessed by perifusion (r=0.60; P=0.022).
36 s was confirmed in vitro by pancreatic islet perifusion showing an amplified biphasic glucose-stimula
37                               Isolated islet perifusion studies demonstrated that exendin-(9-39) bloc
38                                        Islet perifusion studies failed to demonstrate abnormalities i
39                                              Perifusion studies indicate that the inhibition of [3H]p
40 e to glucose plus isobutyl-methylxanthine in perifusion studies that is clearly larger in magnitude t
41                                              Perifusion studies using pharmacologic inhibitors (genis
42 ycemic clamps, as well as isolated islet and perifusion studies.
43                                            A perifusion study revealed that leptin (50 ng/ml) affecte
44 elease obtained from the same islet lot in a perifusion system (n=12).
45                                      Using a perifusion system to follow secretion over time revealed
46 rent GnRH pulse frequencies using a parallel perifusion system.
47 ch was cut into fine slices and subjected to perifusion to monitor glucagon release.
48 his response was not observed if the insulin perifusion was not switched off when the islets were dep
49            Similarly, in a third experiment, perifusion with 10(-7) M insulin caused a significant de
50                                              Perifusion with 16.7 mmol/l glucose plus 0.1 mmol/l IBMX
51                                              Perifusion with ATP before mechanical stimulation suppre
52                                 During local perifusion with KCa antagonists, iberiotoxin (5 microm)
53 kade of Ca2+ release from internal stores by perifusion with ryanodine or dantrolene, or direct diffu
54         Depletion of internal Ca2+ stores by perifusion with thapsigargin or cyclopiazonic acid also
55 n freshly isolated mouse acinar cells during perifusion with the bile acid taurolithocholic acid 3-su
56 ed pituitary cells and from cells undergoing perifusion with the peptides.

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