ライフサイエンスコーパス: perillyl alcohol

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1 s and vacuolation of basal epithelial cells (perillyl alcohol).

2 eventive agents, difluoromethylornithine and perillyl alcohol.

3 s been achieved from the monoterpene (S)-(-)-perillyl alcohol.

4 t NF-kappaB pathway as a molecular target of perillyl alcohol activity in different cancer cell types

5 te the potential chemopreventive activity of perillyl alcohol against colon carcinogenesis.

6 of incubation, an effect that is blocked by perillyl alcohol, an inhibitor of prenyltransferases, bu

7 vasive adenocarcinomas of the colon, whereas perillyl alcohol at 2 g/kg diet inhibited the incidence

8 Perillyl alcohol at the 1-g/kg level significantly inhib

9 fibroblasts and adipocytes, the presence of perillyl alcohol blocked the ability of insulin to incre

10 Our findings suggest that perillyl alcohol can inhibit NF-kappaB function to modul

11 induced by the monoterpene anticancer agent perillyl alcohol correlates to the increased expression

12 Monoterpenes such as d-limonene and perillyl alcohol derived from orange peels and lavender,

13 ndicate that the colon tumors of animals fed perillyl alcohol exhibited increased apoptosis as compar

14 The chemopreventive activity of perillyl alcohol is mediated through the tumor cell loss

15 (AIN-76A) diet or diets containing 1 and 2 g perillyl alcohol/kg diet, representing 40 and 80% MTD le

16 d 80% maximum tolerated dose (MTD) levels of perillyl alcohol on azoxymethane (AOM)-induced colon car

17 A facile route to perillyl alcohol (POH) differential glycosylation and th

18 The monoterpene perillyl alcohol (POH) has proven efficacious against th

19 We now reveal that a dietary agent perillyl alcohol (POH) in combination with Ad.mda-7 effi

20 Perillyl alcohol (POH) is a monoterpene found in lavende

21 Perillyl alcohol (POH) is a naturally occurring monoterp

22 mechanisms of action of the anticancer agent perillyl alcohol (POH), presently in Phase II clinical t

23 Unique among the monoterpenes, perillyl alcohol short-term treatment induced a persiste

24 .Sle1ab mice with the ras pathway inhibitor, perillyl alcohol, suppressed the increase of ANA.

25 Perillyl alcohol treatment also rapidly elicited reducti

26 influencing constitutive cell survival with perillyl alcohol treatment are not well studied.

27 vation abolished the cell killing effects of perillyl alcohol treatment.

28 Prior to the efficacy study, the MTD of perillyl alcohol was determined in male F344 rats in a 6

29 for the derivatives of carvacrol, thymol and perillyl alcohol were higher (15-30mm) in the case of fi

30 The effects of perillyl alcohol were not limited to these B-lymphoma ce

31 oxy 1-O-glucosides of carvacrol, thymol, and perillyl alcohol were tested against Aspergillus flavus,

32 ckers, the combination of a calcium blocker (perillyl alcohol) with bortezomib suppressed NF-kappaB e

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