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1 proven sepsis, necrotizing enterocolitis, or perinatal death).
2 aged >/=35 years may reduce overall rates of perinatal death.
3 nt should include discussion of the risks of perinatal death.
4 rm of ryanodine receptor; Ry1R) resulting in perinatal death.
5 on in rates of preterm birth, stillbirth, or perinatal death.
6 the risks of stillbirth, neonatal death, and perinatal death.
7 rm parturition, to prevent preterm birth and perinatal death.
8 The primary outcome was perinatal death.
9 gnancies, is a leading cause of maternal and perinatal death.
10 l intensive care/special care baby unit, and perinatal death.
11 architecture, causing severe proteinuria and perinatal death.
12 sive care, macrosomia, low Apgar scores, and perinatal death.
13 Whole-body deletion of Derlin-2 results in perinatal death.
14 interstitium, reduced airway branching, and perinatal death.
15 mphangiogenesis defects in mouse embryos and perinatal death.
16 th in the 7 days after birth, stillbirth, or perinatal death.
17 preterm birth, fetal-growth restriction, or perinatal death.
18 f routine induction of labour on the risk of perinatal death.
19 a 33% reduction in litter size and frequent perinatal death.
20 h caused congestive heart failure leading to perinatal death.
21 ice leads to cystic kidneys and embryonic or perinatal death.
22 r at 40 weeks would be required to prevent 1 perinatal death.
23 Prenatal induction resulted in perinatal death.
24 160 pregnancies and accounts for 50% of all perinatal deaths.
25 ed Scottish national databases of births and perinatal deaths.
26 There were no perinatal deaths.
27 associated with a lower risk of in-hospital perinatal death (0.08% versus 0.26%; adjusted risk ratio
28 I, 1.18-1.30; I2 = 80%; n = 18 studies); for perinatal death, 1.16 (95% CI, 1.00-1.35; I2 = 93.7%; n
29 more than 16,274 stillbirths, more than 4311 perinatal deaths, 11,294 neonatal deaths, and 4983 infan
30 5 singleton births, there were 13,027 (0.6%) perinatal deaths, 116,043 (5.6%) preterm live-births and
31 eated infertility also increased the risk of perinatal death (2.7 [1.5-4.7]); the risks associated wi
32 tio [OR] 3.3, 95% CI 1.2-9.0, I(2)=58%), and perinatal deaths (2.3, 1.2-4.1, I(2)=73%) compared with
33 was 7.0 per 1000 births, of which 3.6% were perinatal deaths, 20% prenatally diagnosed, and 5.6% TOP
34 e no significant differences in the rates of perinatal death (3.2% in the pessary group and 2.4% in t
36 d in 2.0 per 1000 births, of which 8.1% were perinatal deaths, 40% were prenatally diagnosed, and 14%
37 (95% CI, 46-51), and 59 (95% CI, 55-63); for perinatal death, 66, 73 (95% CI, 67-81), and 86 (95% CI,
40 or anxiety have higher risks of miscarriage, perinatal death and decisions to terminate a pregnancy i
41 y abruption result in increased frequency of perinatal death and decreased fetal size and gestational
43 ary outcome was a composite of stillbirth or perinatal death and neonatal complications, including hy
44 Whether supplementation reduces the risk of perinatal death and neonatal convulsions requires furthe
45 omposite outcome that included stillbirth or perinatal death and several neonatal complications, it d
49 ulsions in the control group compared with 3 perinatal deaths and no neonatal convulsions in the DHA
50 studies (in which cases were stillbirths or perinatal deaths), and randomised controlled trials of m
51 estern Scotland with databases of maternity, perinatal death, and birth and death certifications to a
52 ffect with protection from preterm birth and perinatal death, and partial correction of reduced birth
53 , premature births, small full-term infants, perinatal deaths, and births of live healthy infants.
55 riction and early delivery, the high risk of perinatal death associated with abruption persisted.
57 f this analysis was to estimate the risks of perinatal death associated with treated and untreated in
62 vessel formation resulting in hemorrhage and perinatal death, but the mechanism of brain hemorrhage i
63 s a composite of adverse perinatal outcomes (perinatal death, chronic lung disease, neonatal sepsis,
64 Wnt1-expressing neural crest cells leads to perinatal death, cleft palate and other cranial bone def
66 cterize the relationship between smoking and perinatal death, defined as the combination of stillbirt
67 enetic disruption of myomaker in mice causes perinatal death due to an absence of multi-nucleated mus
74 d its homozygous deletion in mice results in perinatal death from respiratory failure due to the lack
76 he effect of a trial of labor on the risk of perinatal death in otherwise uncomplicated term pregnanc
78 ecropsy is done in less than 60% of cases of perinatal death in the UK, despite the value of the proc
79 r coagulation (both in the Foley group); ten perinatal deaths, including two stillbirths (both in the
80 The risks of stillbirth, neonatal death, and perinatal death increased with the amount smoked by the
81 The risk of cerebral palsy, like the risk of perinatal death, is lowest in babies who are of above av
82 ldbirth Checklist, on a composite outcome of perinatal death, maternal death, or maternal severe comp
83 l pregnancies and their outcome (live birth, perinatal death, miscarriage or termination) among women
84 ing policy would be lower than the number of perinatal deaths (n = 189) caused by lack of prenatal ca
87 ational diabetes, fetal death or stillbirth, perinatal death, or admission to neonatal intensive care
88 g risk of a mother experiencing at least one perinatal death over the study was 9.0 per 1000 women.
89 women have a significantly increased risk of perinatal death, particularly associated with prematurit
90 ted with a reduction of 3.7 (95% CI 2.2-5.2) perinatal deaths per 1000 pregnancies and 2.3 (0.9-3.7)
91 omparison, the reductions were 4.1 (2.5-5.7) perinatal deaths per 1000 pregnancies and 2.4 (0.7-4.1)
93 how how the gestational age-specific risk of perinatal death (PND) can be decomposed as the product o
94 dorsal midline brain structure formation and perinatal death, presumably by interfering with expressi
96 ries) and morbidities (acute and long-term), perinatal deaths, preterm birth, and intrauterine growth
99 he population attributable risk fraction for perinatal death related to infertility was 6.2% (3.4-9.0
100 RR = 1.22, 95% CI: 1.14, 1.30 (n = 28)), and perinatal death (sRR = 1.33, 95% CI: 1.25, 1.41 (n = 46)
101 Qualitative reports have suggested that perinatal death takes a significant emotional toll on st
102 l birth was associated with a higher rate of perinatal death than was planned in-hospital birth (3.9
103 terval [CI], 0.81 to 1.22) or in the rate of perinatal death; there was a significant reduction in th
104 5 515), the overall rate of delivery-related perinatal death was 12.9 (95% confidence interval [CI],
105 A population-based case-control study of perinatal deaths was carried out in Leicestershire Healt
106 ations between active or passive smoking and perinatal death were included in the meta-analyses.
108 Mice deficient in MARCKS exhibited universal perinatal death with defects in neurulation, fusion of t
109 ion with preterm delivery; 55% of the excess perinatal deaths with abruption were due to early delive
110 ce were born small and weak and succumbed to perinatal death within 12 h because of neuromuscular res
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