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1 increase in pregnancy loss (miscarriages and perinatal mortality).
2 wed up until 7 days after delivery to record perinatal mortality.
3 rm neurological morbidity, and 60% to 80% of perinatal mortality.
4 pid valve malformations associated with high perinatal mortality.
5 aspiration, retinopathy of prematurity, and perinatal mortality.
6 sease, particularly its role in maternal and perinatal mortality.
7 d malaria during pregnancy cause substantial perinatal mortality.
8 tational age, large for gestational age, and perinatal mortality.
9 rtality, while the other showed no change in perinatal mortality.
10 T1 knockout mice exhibited approximately 60% perinatal mortality.
11 gnancy may increase the risk of maternal and perinatal mortality.
12 ormal, neonates showed a higher frequency of perinatal mortality.
13 nces for the offspring in terms of increased perinatal mortality.
14 birth but have impaired growth and increased perinatal mortality.
15 dwarfism, sexual infantilism and significant perinatal mortality.
16 n the central nervous system associated with perinatal mortality.
17 ated with uterine rupture among 145 infants (perinatal mortality 124 per 1,000 total births, 95% CI 7
18 participants; OR, 0.19; 95% CI, 0.07-0.51), perinatal mortality (8 studies, 1140 participants; OR, 0
19 age and birthweight, containing all cases of perinatal mortality [8%]), and higher values of blood pr
21 on length (GL), calving difficulty (CD), and perinatal mortality, also known as stillbirth (SB), in c
22 ion of labour at >/=39 weeks and the risk of perinatal mortality among nulliparous women aged >/=35 y
24 nduction of labour (IOL) around term reduces perinatal mortality and caesarean delivery rates when co
25 ies, albeit with no significant reduction in perinatal mortality and little evidence of neonatal bene
26 than in those that did not, but maternal and perinatal mortality and maternal morbidity did not diffe
27 ng pregnancy results in an increased risk of perinatal mortality and morbidity and is a significant f
29 ongenital heart defects are a major cause of perinatal mortality and morbidity, affecting >1% of all
30 es annually and is associated with increased perinatal mortality and morbidity, as well as poorer lon
36 Slc23a1-/- dams exhibited approximately 45% perinatal mortality, and this was associated with lower
37 of these mice survived, but there was a high perinatal mortality, and those that survived had a decre
38 caesarean delivery was associated with lower perinatal mortality (AOR after planned caesarean at 39 w
39 Atox1(-/-) phenotype, resulting in increased perinatal mortality as well as severe growth retardation
43 between April 2009 and March 2014 to compare perinatal mortality between induction of labour at 39, 4
44 f adverse perinatal outcomes, which included perinatal mortality, bronchopulmonary dysplasia, sepsis,
45 ere was no significant effect on the odds of perinatal mortality but greater odds of neonatal unit ad
46 acenta) is associated with preterm birth and perinatal mortality, but associations with other neonata
48 exhibit a unique phenotype characterized by perinatal mortality, disrupted cerebral cortical layerin
50 sociated with low birth weight and increased perinatal mortality, especially among primigravidae.
51 l inactivation of GATA-6 in VSMCs results in perinatal mortality from a spectrum of cardiovascular de
53 piratory dysfunction is a notorious cause of perinatal mortality in infants and sleep apnoea in adult
55 as moderate to severe neonatal morbidity, or perinatal mortality in the absence of a major congenital
57 e extent to which the effect of abruption on perinatal mortality is mediated through preterm delivery
58 Twin fetuses experience much higher rates of perinatal mortality/morbidity than age- and weight-match
61 A was associated with a nonsignificant lower perinatal mortality of 14 per 1000 births in interventio
63 of link protein could completely prevent the perinatal mortality of link protein-deficient mice and,
64 matopoiesis has been complicated by the high perinatal mortality of SIRT1-deficient mice (SIRT1(-/-))
67 sociation between smoke-free legislation and perinatal mortality, one showed significant reductions i
69 data were analysed with regard to abortion, perinatal mortality, prematurity, toxaemia and congenita
72 f disease activity on spontaneous abortions, perinatal mortality, preterm delivery, and birth weight.
77 gene encoding c-Abl (AIM) display increased perinatal mortality, reduced fertility, foreshortened cr
80 and had gait ataxia, increased frequency of perinatal mortality, scoliosis, resting tremors and ptos
81 mission to a neonatal intensive care unit or perinatal mortality showed adverse trends, these changes
82 efects in the skin, lung, and intestine, and perinatal mortality that are reminiscent of EGF-R knocko
84 ong women who were pregnant, miscarriage and perinatal mortality was 2.7 times higher (95% confidence
87 tion in SGA births, but not preterm birth or perinatal mortality, was observed in the Netherlands aft
88 ion to the neonatal intensive care unit, and perinatal mortality were similar among the infants whose
89 ity but did not report the overall effect on perinatal mortality, while the other showed no change in
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