ライフサイエンスコーパス: periodontal infection

1 control of patients with DMt2 by eliminating periodontal infection.

2 , such as those in chronic P.gingivalis (PG) periodontal infection.

3 meters as healthy or mild or moderate/severe periodontal infection.

4 d clinical measures and serologic markers of periodontal infection.

5 nal gingival swelling that mimicked an acute periodontal infection.

6 increase the host susceptibility to oral and periodontal infection.

7 for understanding the role of these cells in periodontal infection.

8 ate the mechanisms of NKT cell activation in periodontal infections.

9 ory responses in atheromatous lesions due to periodontal infections.

10 c bacteremia, which occurs frequently during periodontal infections.

11 ip of inflammation to obesity, diabetes, and periodontal infections.

12 genic factor linking obesity to diabetes and periodontal infections.

13 al epithelial cells are essential aspects of periodontal infections.

14 understanding of the role of F. nucleatum in periodontal infections.

15 eful antimicrobial agents in therapy against periodontal infections.

16 ease in attachment loss may represent active periodontal infection accelerated by pregnancy.

17 nisms underlying the host immune response in periodontal infection and alveolar bone resorption.

18 does not prove, a causal association between periodontal infection and atherosclerotic cardiovascular

19 does not prove a causal association between periodontal infection and cardiovascular disease and sug

20 ly explore and describe familial patterns of periodontal infection and other aspects of periodontal d

21 has the potential for the early detection of periodontal infection and progression to identify incipi

22 Effective treatment of periodontal infection and reduction of periodontal infla

23 Elimination of periodontal infection and reduction of periodontal infla

24 ty at Buffalo, Buffalo, New York, i.e., the "Periodontal Infection and Risk for Myocardial Infarction

25 ntial causative role of chronic T. denticola periodontal infection and vascular atherosclerosis in vi

26 tablished the plausibility of a link between periodontal infections and atherogenesis, and have ident

27 th of time of loading; history of implant or periodontal infections; and whether implants replaced si

28 Initiation and progression of periodontal infections are clearly modified by local and

29 nce from epidemiologic studies suggests that periodontal infections are independently associated with

30 erlying host defense impairment coupled with periodontal infection by HCMV and A. actinomycetemcomita

31 otentially be developed as a vaccine against periodontal infection by P. gingivalis.

32 lammatory burden was measured as 1) clinical periodontal infection categorized as no periodontal dise

33 Sera from patients with periodontal infections contain elevated levels of C-reac

34 Recent data suggest periodontal infection contributes to chronic kidney dise

35 determine whether the prevalence of maternal periodontal infection could be associated with preterm l

36 ving periodontal disease, or may result from periodontal infection-genetic interactions.

37 Maternal periodontal infection has been associated with adverse m

38 Maternal periodontal infection has been associated with increased

39 al peptides (AMPs) as therapeutic agents for periodontal infections has great advantages, such as bro

40 Periodontal infections have a microbial etiology.

41 Periodontal infections have been associated with a state

42 at the increased oxidative stress induced by periodontal infection in rats can be ameliorated by bone

43 Periodontal infections in individuals with pre-existing

44 It shows that periodontal infections in patients with depressed PMN fu

45 and may be clinically useful in treatment of periodontal infections involving enterococci.

46 ration of host defence cells are symptoms of periodontal infection, iron products released from blood

47 Periodontal infection is also associated with increased

48 Maternal periodontal infection is associated with an increased ri

49 controls to establish an association between periodontal infection markers and rheumatic activity.

50 c kidney disease (CKD), we hypothesized that periodontal infection may affect the systemic inflammato

51 Transient bacteremia caused by periodontal infection may facilitate bacterial transmiss

52 These findings suggest that periodontal infection may influence the development of N

53 Chronic infections, including periodontal infections, may predispose to cardiovascular

54 iodontal pathogen; however, association with periodontal infections of other Campylobacter species, e

55 uction by reversing the inhibitory effect of periodontal infection on collagen synthesis.

56 available regarding the effects of long-term periodontal infection on diabetes mellitus (DM) control.

57 The effect of periodontal infection on systemic diseases and condition

58 moderate or reduce the inhibitory effects of periodontal infection on the expression of type I and ty

59 It was hypothesized that if periodontal infections predispose low birth weights and

60 Control of periodontal infections should thus be an important part

61 Individuals with periodontal infection showed, approximately, five times

62 e subjects might represent a contribution of periodontal infections to systemic inflammation in relat

63 A periodontal infection was categorized by clinical parame

64 To investigate the lymphatic function after periodontal infection, we used K14-VEGF receptor 3-Ig (K

65 form of early-onset periodontitis linked to periodontal infection with uncontrolled inflammation and