ライフサイエンスコーパス: periodontal pathogen

1 FimA, in Porphyromonas gingivalis, a primary periodontal pathogen.

2 luding Porphyromonas gingivalis, a potential periodontal pathogen.

3 hatase and reveal an invasin of an important periodontal pathogen.

4 volved in the production of fimbriae by this periodontal pathogen.

5 from Actinobacillus actinomycetemcomitans, a periodontal pathogen.

6 t not by that of Porphyromonas gingivalis, a periodontal pathogen.

7 valis is a peptide-fermenting asaccharolytic periodontal pathogen.

8 nce the inflammatory response induced by the periodontal pathogen.

9 de antibiotic that is active against several periodontal pathogens.

10 ogenicity and ability to interact with other periodontal pathogens.

11 y lower prevalence and abundance of putative periodontal pathogens.

12 on may be related to an elevated exposure to periodontal pathogens.

13 ctiveness of clarithromycin against invasive periodontal pathogens.

14 cts who harbored two, or all three, of these periodontal pathogens.

15 plex interplay between the immune system and periodontal pathogens.

16 ntal therapy significantly reduced levels of periodontal pathogens.

17 performed to detect the presence of selected periodontal pathogens.

18 haps symptomatic of colonization by residual periodontal pathogens.

19 determined the occurrence of potential major periodontal pathogens.

20 outcomes in addition to previously reported periodontal pathogens.

21 oss, and bone resorption that are induced by periodontal pathogens.

22 chment gain had a high prevalence of these 3 periodontal pathogens.

23 following interaction with several putative periodontal pathogens.

24 were positive for at least one of the target periodontal pathogens.

25 bgingival colonization and multiplication of periodontal pathogens.

26 y at high levels, is primarily restricted to periodontal pathogens.

27 allows more sensitive detection of all three periodontal pathogens.

28 ated with peri-implantitis are recognized as periodontal pathogens.

29 nvironment and a distributional shift of key periodontal pathogens.

30 ymicrobial infection with well-characterized periodontal pathogens.

31 ilable polymerase chain reaction test for 11 periodontal pathogens.

32 s been primarily focused on a small group of periodontal pathogens.

33 d by corrosion may enhance the attachment of periodontal pathogens.

34 ely as a result of increased colonization of periodontal pathogens.

35 tially a good choice for inhibiting invasive periodontal pathogens.

36 altered immune-inflammatory response against periodontal pathogens.

37 overall showed a stronger inhibition of the periodontal pathogens.

38 re- or PCR-positive results for the targeted periodontal pathogens.

39 to 71% increase), bacterial killing of these periodontal pathogens (22 to 38% reduction of bacterial

40 Oral injections of LPS derived from the periodontal pathogen A. actinomycetemcomitans can induce

41 The periodontal pathogen A. actinomycetemcomitans has been k

42 All targeted periodontal pathogens (A. actinomycetemcomitans, P. ging

43 harbored at least one of the following three periodontal pathogens: Aa, Pg, or Bf.

44 bone loss in rats using LPS derived from the periodontal pathogen Actinobacillus actinomycetemcomitan

45 Fresh clinical isolates of the periodontal pathogen Actinobacillus actinomycetemcomitan

46 fresh clinical isolates of the gram-negative periodontal pathogen Actinobacillus actinomycetemcomitan

47 The periodontal pathogen Actinobacillus actinomycetemcomitan

48 Some clinical isolates of the gram-negative periodontal pathogen Actinobacillus actinomycetemcomitan

49 The Gram-negative periodontal pathogen Actinobacillus actinomycetemcomitan

50 Here, we report that the periodontal pathogen Actinobacillus actinomycetemcomitan

51 cts associated with a virulence locus of the periodontal pathogen Actinobacillus actinomycetemcomitan

52 Strains of the periodontal pathogen Actinobacillus actinomycetemcomitan

53 late three catalase-deficient mutants of the periodontal pathogen Actinobacillus actinomycetemcomitan

54 The periodontal pathogen Actinobacillus actinomycetemcomitan

55 zation and confocal microscopy to detect the periodontal pathogens Actinobacillus actinomycetemcomita

56 d with intracellular bacteria, including the periodontal pathogens Actinobacillus actinomycetemcomita

57 Among putative periodontal pathogens, Actinobacillus actinomycetemcomit

58 rbored at least one of the following 3 major periodontal pathogens: Actinobacillus actinomycetemcomit

59 ne whether Prevotella intermedia, a putative periodontal pathogen, activates populations of specific

60 The periodontal pathogen Aggregatibacter (Actinobacillus) ac

61 cytolethal distending toxin (Cdt), from the periodontal pathogen Aggregatibacter actinomycetemcomita

62 crease in antibacterial activity against the periodontal pathogen Aggregatibacter actinomycetemcomita

63 hal distending toxin (Cdt), expressed by the periodontal pathogen Aggregatibacter actinomycetemcomita

64 Biofilm formation by the periodontal pathogen Aggregatibacter actinomycetemcomita

65 e major clonal lineages of the Gram-negative periodontal pathogen Aggregatibacter actinomycetemcomita

66 The periodontal pathogen Aggregatibacter actinomycetemcomita

67 The periodontal pathogen Aggregatibacter actinomycetemcomita

68 sed a dual-species biofilm consisting of the periodontal pathogen Aggregatibacter actinomycetemcomita

69 e cytolethal distending toxin (Cdt) from the periodontal pathogen Aggregatibacter actinomycetemcomita

70 The aim of this investigation is to quantify periodontal pathogens (Aggregatibacter actinomycetemcomi

71 sent investigation was to identify potential periodontal pathogens among these newly identified speci

72 Porphyromonas gingivalis is a keystone periodontal pathogen and its lipopolysaccharide (PgLPS)

73 hat patterns of high and low levels of eight periodontal pathogens and antibody levels against those

74 After the 2-week study period, putative periodontal pathogens and cariogenic bacteria were overa

75 maxillas and spleens from mice infected with periodontal pathogens and compared to those in the maxil

76 his may suggest possible association between periodontal pathogens and DG.

77 y assessed the associations between putative periodontal pathogens and early-onset periodontitis (EOP

78 ave the capacity to respond to components of periodontal pathogens and express both pro- and anti-inf

79 antibacterial activity against most putative periodontal pathogens and has been shown to kill bacteri

80 rivative possessing antimicrobial effects on periodontal pathogens and inhibitory properties on matri

81 Clarithromycin inhibits several periodontal pathogens and is concentrated inside gingiva

82 High levels of periodontal pathogens and low maternal IgG antibody resp

83 o 19 species, including established/putative periodontal pathogens and non-pathogenic bacteria, in 5,

84 It increases the acquisition of periodontal pathogens and periodontal disease, colonizat

85 for the presence of familial aggregation of periodontal pathogens and periodontitis and have alluded

86 ciated with the subgingival presence of some periodontal pathogens and periodontitis.

87 hol consumption on the levels of subgingival periodontal pathogens and proinflammatory cytokines (int

88 In addition, putative periodontal pathogens and resistance of subgingival bact

89 MC-2 was correlated with traditional periodontal pathogens and several newly identified putat

90 ediates of microbial burden (levels of eight periodontal pathogens) and local inflammatory response (

91 e we report that Porphyromonas gingivalis, a periodontal pathogen, and Escherichia coli LPS induce os

92 ship of gingivitis with salivary biomarkers, periodontal pathogens, and interleukin (IL)-1 polymorphi

93 king, age, IgG levels against other selected periodontal pathogens, and race.

94 of host cells by a limited number of 'model' periodontal pathogens, and therefore may not adequately

95 The wide variability found in periodontal pathogen antibiotic-resistance patterns shou

96 the hypothesis that oral bacteria other than periodontal pathogens are also associated with pregnancy

97 ntal disease, high levels of colonization of periodontal pathogens are associated with an increased r

98 concluded, based upon current evidence, that periodontal pathogens are communicable; however, they ar

99 Periodontal pathogens are detrimental to periodontal hea

100 While traditional putative periodontal pathogens are implicated, research involving

101 Periodontal pathogens are present in atherosclerotic pla

102 e by impairing the innate immune response to periodontal pathogens as well as by increasing free radi

103 Furthermore, in response to these periodontal pathogens (as mono- and polymicrobial heat-k

104 Persistence of putative and novel periodontal pathogens, as well as low prevalence of bene

105 The oral spirochete Treponema denticola, a periodontal pathogen associated with human periodontitis

106 f this study was to test the hypothesis that periodontal pathogens associated with aggressive periodo

107 nema denticola, and Tannerella forsythia are periodontal pathogens associated with the etiology of ad

108 ophaga spp. have been implicated as putative periodontal pathogens associated with various periodonta

109 ibody, dichotomized at the median, for three periodontal pathogens (Bacteroides forsythus [IgG Bf], P

110 Periodontal pathogens belonging to the genera Fusobacter

111 ission is not limited to the well-recognized periodontal pathogens, but instead may also involve the

112 highest proportional recoveries of putative periodontal pathogens, but the recoveries by the various

113 Periodontal pathogens/byproducts may reach the placenta

114 Strains of the periodontal pathogen Campylobacter rectus express a 150-

115 tainment proofs 1 to 6 provides support that periodontal pathogens can contribute to atherosclerosis.

116 Porphyromonas gingivalis, a periodontal pathogen, can efficiently invade human gingi

117 Porphyromonas gingivalis is a periodontal pathogen capable of invading primary culture

118 sent an overreaction of the host response to periodontal pathogens caused by excessive production of

119 We used GWAS data of CP (n = 4,504) and periodontal pathogen colonization (n = 1,020) from a coh

120 6); AP3B2, p = 2.2 x 10(-6)) and 2 with high periodontal pathogen colonization (red complex-KCNK1, p

121 ce on host genetic risk loci associated with periodontal pathogen colonization.

122 P. gingivalis and the other periodontal pathogens colonize and interact with gingiva

123 Periodontal pathogens colonized implants symptomatic thr

124 P. gingivalis and B. forsythus, red complex periodontal pathogens, colonized several implants, altho

125 Periodontal pathogens commonly associated with chronic a

126 est positive for antiCl, likely because some periodontal pathogens contain antigens homologous to the

127 periodontitis patients contain aCl, and some periodontal pathogens contain antigens with peptide sequ

128 ew findings provide mechanistic support that periodontal pathogens create a unique microenvironment i

129 Risk for harboring 2 or more periodontal pathogens decreased with the years the paren

130 Rats infected with the periodontal pathogens displayed a five-fold increase in

131 ne the role of saliva-derived biomarkers and periodontal pathogens during periodontal disease progres

132 ues, we tested the ability of three putative periodontal pathogens-Eikenella corrodens, Porphyromonas

133 It is known that periodontal pathogens elicit host-derived immune respons

134 bjects and to be the most prevalent putative periodontal pathogens examined.

135 has a lasting suppressive effect on selected periodontal pathogens for the majority of sites in patie

136 hogens and several newly identified putative periodontal pathogens: Fretibacterium fastidiosum, Freti

137 d with increased odds of detection of common periodontal pathogens from individuals with aggressive p

138 culated that periodontal probes can transmit periodontal pathogens from site to site.

139 support the hypothesis of a translocation of periodontal pathogens from subgingival microbiota to the

140 patients with and without DM, and 2) isolate periodontal pathogens from these patients' blood.

141 A defining characteristic of the suspected periodontal pathogen Fusobacterium nucleatum is its abil

142 s among the presence of three orange-complex periodontal pathogens (Fusobacterium nucleatum, Prevotel

143 that growth of the PFOR-containing anaerobic periodontal pathogens, grown in both monospecies as well

144 Since a few periodontal pathogens have been reported to invade oral

145 However, some periodontal pathogens have developed strategies to evade

146 ampylobacter rectus has been implicated as a periodontal pathogen; however, association with periodon

147 Filifactor alocis is a recently recognized periodontal pathogen; however, little is known regarding

148 ent reports focusing on virulence factors of periodontal pathogens implicated proteinases as major de

149 ion was conducted to confirm the presence of periodontal pathogens in bone particles harvested intrao

150 gingivalis is recognized as one of the major periodontal pathogens in chronic periodontitis, a common

151 Importantly, the levels of IL-17 induced by periodontal pathogens in CII-specific T cells directly c

152 Detection of periodontal pathogens in early periodontitis suggests an

153 ion frequency and levels of recovery of some periodontal pathogens in failing implants are significan

154 ot an adequate method for identifying DNA of periodontal pathogens in low quantities because of the h

155 biotic resistance among selected subgingival periodontal pathogens in patients with CP.

156 a key role in the innate immune responses to periodontal pathogens in periodontal disease.

157 e types and relative proportions of putative periodontal pathogens in plaque associated with ligature

158 nested multiplex PCR method to detect three periodontal pathogens in subgingival plaque collected be

159 and their association with the levels of key periodontal pathogens in subgingival plaque.

160 his study is to detect and quantify the main periodontal pathogens in the oral microbiota of postmeno

161 ion is to compare the presence and number of periodontal pathogens in the subgingival microbiota of s

162 ted with decreased levels of IgG antibody to periodontal pathogens in women with periodontitis when a

163 logical testing showed the presence of known periodontal pathogens including Actinobacillus actinomyc

164 responses to carbohydrate antigens from non-periodontal pathogens including Haemophilus influenzae b

165 At 6 weeks, all membranes showed periodontal pathogens, including Porphyromonas gingivali

166 y 40% compared to that of HC, and killing of periodontal pathogens, including Porphyromonas gingivali

167 loading time increased, but colonization by periodontal pathogens, including red complex species, wa

168 their precise roles are not well understood, periodontal pathogens, including Treponema denticola, ar

169 Porphyromonas gingivalis, an important periodontal pathogen, infects primary gingival epithelia

170 One of the features of this periodontal pathogen is its ability to attach to a varie

171 The simultaneous detection of three periodontal pathogens is an advantage of this technique

172 ibroblasts that occurs during infection with periodontal pathogens is, in part, mediated by TNF.

173 Porphyromonas gingivalis, an important periodontal pathogen, is an effective colonizer of oral

174 Porphyromonas gingivalis, a key periodontal pathogen, is capable of invading a variety o

175 Treponema denticola, a periodontal pathogen, is relatively resistant to human b

176 If P. gingivalis is a periodontal pathogen, it would be expected to be present

177 the rate of preterm delivery, a decrease in periodontal pathogen load, and a decrease in both GCF IL

178 tudy was devised to test the hypothesis that periodontal pathogens may be present and affect human pl

179 This study indicates that periodontal pathogens may play important roles in the sh

180 in the field and addressed the link between periodontal pathogens measured with the benzoyl-DL-argin

181 The major fimbriae of this periodontal pathogen mediate binding to host gingival ep

182 that proteinases expressed by the infecting periodontal pathogens might activate latent host MMPs to

183 In the surgical specimens positive for periodontal pathogens, more than 1 species was most ofte

184 Thus, in order to colonize, successful periodontal pathogens must devise means to interfere wit

185 t group also demonstrated significantly less periodontal pathogens of red and orange complexes and a

186 th Porphyromonas gingivalis (Pg), a putative periodontal pathogen, on the progression of atherosclero

187 Likewise, periodontal pathogens, opportunistic pathogens, or norma

188 Baseline levels of selected periodontal pathogens or changes in these bacteria resul

189 lis (P gingivalis) (10(7) CFU), an important periodontal pathogen, or vehicle once per week for 14 or

190 The presence of periodontal pathogens P.g., P.i., C.r., and B.f. in subg

191 serum levels of IgG antibody to the panel of periodontal pathogens (P = 0.0018), to P. gingivalis (P

192 Thus, S. mutans and the periodontal pathogens, P. gingivalis and B. forsythus, w

193 The periodontal pathogens Pg, Pi, Tf, and Fn are associated

194 t highly virulent strains or clonal types of periodontal pathogens play a major role in the initiatio

195 te genome representing a novel strain of the periodontal pathogen Porphyromonas gingivalis (P. gingiv

196 tide (VIP) modulates immune responses to the periodontal pathogen Porphyromonas gingivalis (Pg).

197 The periodontal pathogen Porphyromonas gingivalis adheres to

198 ibute to osteolytic lesions, we injected the periodontal pathogen Porphyromonas gingivalis adjacent t

199 mine the antibacterial effects of EMD on the periodontal pathogen Porphyromonas gingivalis and 2) to

200 The periodontal pathogen Porphyromonas gingivalis and the en

201 Two types of fimbriae, FimA and Mfa1, of the periodontal pathogen Porphyromonas gingivalis are respon

202 However, the periodontal pathogen Porphyromonas gingivalis can contro

203 The periodontal pathogen Porphyromonas gingivalis employs a

204 Lipopolysaccharide (LPS) derived from the periodontal pathogen Porphyromonas gingivalis has been r

205 The capsular polysaccharide (CPS) of the periodontal pathogen Porphyromonas gingivalis is an impo

206 Previously, we have established that the periodontal pathogen Porphyromonas gingivalis is capable

207 The periodontal pathogen Porphyromonas gingivalis is implica

208 One of the features of the periodontal pathogen Porphyromonas gingivalis is the pre

209 f lipopolysaccharide (LPS) purified from the periodontal pathogen Porphyromonas gingivalis on human m

210 re, we investigate the direct effects of the periodontal pathogen Porphyromonas gingivalis on osteocl

211 el to study the effect of infection with the periodontal pathogen Porphyromonas gingivalis on pregnan

212 Finally, the abundance of the periodontal pathogen Porphyromonas gingivalis trended wi

213 lutinin B (rHagB), a virulence factor of the periodontal pathogen Porphyromonas gingivalis, has been

214 In the periodontal pathogen Porphyromonas gingivalis, the CTD i

215 allows invading microorganisms, such as the periodontal pathogen Porphyromonas gingivalis, to readil

216 eated diabetic mice, infected with the human periodontal pathogen Porphyromonas gingivalis, with solu

217 LR4(-/-)) mice were orally infected with the periodontal pathogen Porphyromonas gingivalis.

218 The presence of periodontal pathogens Porphyromonas gingivalis (P.g.), P

219 activity of major virulence factors from the periodontal pathogens Porphyromonas gingivalis and Actin

220 Repeated oral inoculations of periodontal pathogens Porphyromonas gingivalis and Prevo

221 For example, the periodontal pathogens Porphyromonas gingivalis and Tanne

222 Growth inhibition of the periodontal pathogens Porphyromonas gingivalis, Prevotel

223 , and Campylobacter rectus), two red-complex periodontal pathogens (Porphyromonas gingivalis and Tann

224 The presence of periodontal pathogens (Porphyromonas gingivalis, Aggrega

225 We postulated that the periodontal pathogen, Porphyromonas gingivalis may suppr

226 colonization with a well-characterized human periodontal pathogen, Porphyromonas gingivalis We found

227 es and subsequent proteomic responses to the periodontal pathogen, Porphyromonas gingivalis, donor-ma

228 ar premolars followed by an application of a periodontal pathogen, Porphyromonas gingivalis, to induc

229 the present study, LPS derived from the oral periodontal pathogen, Porphyromonas gingivalis, was comp

230 Deoxyribonucleic acids (DNA) of periodontal pathogens, Porphyromonas gingivalis (Pg) and

231 hich the most extensively studied "putative" periodontal pathogens, Porphyromonas gingivalis and Acti

232 species in a newly described red complex of periodontal pathogens, Porphyromonas gingivalis and Bact

233 o characterize host responses of interest to periodontal pathogens, Porphyromonas gingivalis was intr

234 To determine whether three putative periodontal pathogens, Porphyromonas gingivalis, Campylo

235 antibacterial activity against two anaerobic periodontal pathogens: Porphyromonas gingivalis and Acti

236 Thirteen (59%) of the 22 periodontal pathogen-positive surgical specimens were po

237 Recent evidence suggests that certain periodontal pathogens preferentially stimulate T cells e

238 as well as a cysteine proteinase of another periodontal pathogen, Prevotella intermedia, resulted in

239 ormerly Bacteroides forsythus) is one of the periodontal pathogens recently implicated in the develop

240 is report was to compare the distribution of periodontal pathogens recovered from failing implants an

241 hat activation of the acquired immunity by a periodontal pathogen reduces the coupling of bone format

242 United States frequently yielded subgingival periodontal pathogens resistant in vitro to therapeutic

243 of the patients with CP revealed subgingival periodontal pathogens resistant to at least one of the t

244 een percent of patients harbored subgingival periodontal pathogens resistant to both amoxicillin and

245 parental and feoB1-deficient strains of the periodontal pathogen revealed that the feoB1-deficient m

246 Porphyromonas gingivalis, an invasive periodontal pathogen, secretes the HAD family phosphoser

247 pants assembled into three clusters based on periodontal pathogens, serum and salivary biomarkers.

248 In response to endotoxins derived from periodontal pathogens, several osteoclast-related mediat

249 selected controls from the above cohort, the periodontal pathogen-specific maternal serum IgG levels

250 provide mechanistic support that SCFAs from periodontal pathogens stimulate KSHV replication and inf

251 We hypothesized that virulence factors of periodontal pathogens such as lipopolysaccharide stimula

252 onizing the colonization and accumulation of periodontal pathogens such as P. gingivalis.

253 Periodontal pathogens such as Porphyromonas gingivalis a

254 Known putative periodontal pathogens such as Porphyromonas gingivalis,

255 tegerin) combined with red-complex anaerobic periodontal pathogens (such as Porphyromonas gingivalis

256 cells of the periodontium to encounter known periodontal pathogens, such as Actinobacillus actinomyce

257 was associated with elevated levels of known periodontal pathogens, such as P. nigrescens, T. forsyth

258 Virulence factors from periodontal pathogens, such as Porphyromonas gingivalis

259 crobial co-occurrence analysis revealed that periodontal pathogens, such as Porphyromonas gingivalis,

260 The virulence of periodontal pathogens, such as Porphyromonas gingivalis,

261 ssociation of miR-146a expression with these periodontal pathogens, suggesting that miR-146a may dire

262 Aggregatibacter actinomycetemcomitans, a periodontal pathogen, synthesizes leukotoxin (LtxA), a p

263 athogens by reconstructing the genome of the periodontal pathogen Tannerella forsythia and also ident

264 We found the periodontal pathogen Tannerella forsythia to be associat

265 e genes, (iv) a genome reconstruction of the periodontal pathogen Tannerella forsythia, (v) 239 bacte

266 Antepartum, the levels of periodontal pathogens tended to be higher in the preterm

267 tis have higher levels of GCF biomarkers and periodontal pathogens than clinically healthy sites from

268 ctinobacillus actinomycetemcomitans is a key periodontal pathogen that adheres to and invades oral ep

269 Porphyromonas gingivalis is a periodontal pathogen that also localizes to atherosclero

270 Porphyromonas gingivalis (Pg) is a major periodontal pathogen that contains immunostimulatory com

271 Porphyromonas gingivalis is a consensus periodontal pathogen that has been implicated in adult f

272 Treponema denticola is a consensus periodontal pathogen that has recently been associated w

273 obacillus actinomycetemcomitans are putative periodontal pathogens that may be harbored in subgingiva

274 addition, in comparison with other anaerobic periodontal pathogens, the removal of 8-oxoG was unique

275 gingival epithelium forms a barrier against periodontal pathogens, this study was undertaken to dete

276 response to systemic inflammation induced by periodontal pathogens through mechanisms involving downr

277 parameters and the salivary presence of six periodontal pathogens to age-related macular degeneratio

278 The periodontal pathogen Treponema denticola produces dentil

279 The metabolism of glutathione by the periodontal pathogen Treponema denticola produces hydrog

280 ue samples were assessed for the presence of periodontal pathogens using indirect immunofluorescence.

281 nd assessed their response to infection with periodontal pathogens using microarray analysis, quantit

282 ontal pockets and the prevalence of selected periodontal pathogens was assessed in 10 patients with a

283 Subgingival infection with target periodontal pathogens was determined by indirect immunof

284 The subgingival presence of suspected periodontal pathogens was determined by non-selective an

285 d" DNA-DNA hybridization quantification of 8 periodontal pathogens was performed.

286 Detection of known periodontal pathogens was rare.

287 Putative periodontal pathogens were detected as a major component

288 minutes after membrane placement, suspected periodontal pathogens were detected in several ePTFE mem

289 istically significant reductions in selected periodontal pathogens were evident at day 7 but not at d

290 cocci, was measured in 78.2% of thrombi, and periodontal pathogens were measured in 34.7%.

291 , whereas the carriage rates of the examined periodontal pathogens were not.

292 Microbiologic assessments of eight putative periodontal pathogens were performed using the checkerbo

293 determine whether Gram-negative, facultative periodontal pathogens were sensitive to the protegrins.

294 The samples were cultured, and selected periodontal pathogens were tested in vitro for susceptib

295 ar lipids of Porphyromas gingivalis, a known periodontal pathogen, were previously shown to promote d

296 nas gingivalis are virulence factors of this periodontal pathogen which likely act by interrupting ho

297 Porphyromonas gingivalis is a periodontal pathogen whose primary niche is the anaerobi

298 y worsen oral health, favoring the growth of periodontal pathogens, whose detection could improve the

299 Campylobacter rectus is a periodontal pathogen with a 150-kDa protein on its cell

300 ence intervals (CIs) for the associations of periodontal pathogens with total cancer and site-specifi