ライフサイエンスコーパス: periodontal pocket

1 rochete that inhabits the gingival sulcus or periodontal pocket.

2 hance the maintenance of PMN function in the periodontal pocket.

3 hysiologic relevance, i.e., reachable in the periodontal pocket.

4 omposition of the bacterial community in the periodontal pocket.

5 omposition of the bacterial community in the periodontal pocket.

6 ses or other oral bacteria to survive in the periodontal pocket.

7 se bone formation on local delivery into the periodontal pocket.

8 1.55 to 10.76 in women who had at >/=1 5-mm periodontal pocket.

9 plays in tissue destruction occurring in the periodontal pocket.

10 lls live and die in the gingival crevice and periodontal pocket.

11 of evading the host response in the inflamed periodontal pocket.

12 profile for the delivery of doxycycline into periodontal pockets.

13 olled-release delivery system (DH) placed in periodontal pockets.

14 rs and also with a higher percentage of deep periodontal pockets.

15 s of repair, a common status within inflamed periodontal pockets.

16 therapeutic agents against these species in periodontal pockets.

17 air polishing (SubGPAP) in moderate-to-deep periodontal pockets.

18 has been shown to remove biofilms in shallow periodontal pockets.

19 ith periodontitis because of the presence of periodontal pockets.

20 re produced from inflammatory tissues within periodontal pockets.

21 hypothetical data to model the treatment of periodontal pockets.

22 portant thiol source for H(2)S production in periodontal pockets.

23 tself as one of the predominant pathogens in periodontal pockets.

24 ive periodontal disease (> 10% of sites with periodontal pockets 4+ mm) had an increase of approximat

25 SRP showed PD improvements only in moderate periodontal pockets (4 to 6 mm).

26 increase in availability of thin inserts for periodontal pocket access.

27 In deep periodontal pocket analysis (probing depth [PD] >/= 7 mm

28 ce its ability to survive and persist in the periodontal pocket and may play an important role in inf

29 presence of Porphyromonas gingivalis in the periodontal pocket and the high levels of gingipain acti

30 ilized by oral spirochetes to survive in the periodontal pocket and transition from a minor to a domi

31 elated to periodontal condition, measured as periodontal pocketing and gingival bleeding in this low-

32 r of the human oral microbiome that inhabits periodontal pockets and contributes to chronic periodont

33 However, change in periodontal pockets and DMFT over time varied according

34 d as the number of teeth with deep (>/=4 mm) periodontal pockets and the number of bleeding sextants

35 se their viability within gingival crevices, periodontal pockets and the oral cavity die by necrosis

36 etween probing attachment changes in treated periodontal pockets and the prevalence of selected perio

37 span of neutrophils in gingival crevices and periodontal pockets and therefore into the pathogenesis

38 tudents (36 periodontally healthy and 2 with periodontal pockets) and 58 healthy A. actinomycetemcomi

39 dontitis (defined as at least two sites with periodontal pockets), and 82.6% sensitive for at least t

40 retained root fragments, root-surface decay, periodontal pockets, and problem-motivated dental visits

41 d from clinically healthy gingival crevices, periodontal pockets, and the oral cavity (saliva) were e

42 /g in thermosetting agents) for non-invasive periodontal pocket anesthesia was evaluated.

43 In this large cohort study, the presence of periodontal pockets as measured by CPITN was positively

44 helial cells may promote colonization of the periodontal pocket, as well as retention of treponeme co

45 nd two or more sites with >/= 6 mm or deeper periodontal pocket, associated with elevated salivary MM

46 least two sites with BOP and two sites with periodontal pockets but a lower relationship for single-

47 g did not affect the rate of repopulation of periodontal pockets by the tested pathogens; 2) thorough

48 subgingival cultivable microflora in shallow periodontal pockets compared to curets and is safe when

49 e likelihood of demonstrating a reduction in periodontal pockets compared to erratic compliers under

50 ge amounts of H2S have been reported in deep periodontal pockets, cystalysin may also function in viv

51 Gingival and periodontal pocket depth and attachment levels were reco

52 There was no association between periodontal pocket depth and the detection of H. pylori.

53 gingival bleeding, gingival recession level, periodontal pocket depth, and calculus were made by dent

54 A one-time use of the 3-CO(2) laser in periodontal pockets did not sterilize or substantially r

55 s) are being used to treat residual inflamed periodontal pockets during periodontal maintenance thera

56 Periodontal pockets experience a higher temperature duri

57 alis (Pg), and Prevotella intermedia (Pi) in periodontal pockets following scaling and root planing (

58 l (either placebo or test gel) placed in the periodontal pockets for 30 seconds.

59 ical migration of the junctional epithelium, periodontal pocket formation, alveolar bone resorption,

60 t periodontitis, measured by the presence of periodontal pockets > or = 4 mm, was found in about 30%

61 Fifty-two patients with 4 periodontal pockets > or = 5 mm and bleeding on probing

62 ted had two quadrants with a minimum of four periodontal pockets > or = 5 mm in depth with two sites

63 ely to have a reduction in the percentage of periodontal pockets >3 mm compared to erratic compliers,

64 and approximately 20% of the population had periodontal pocketing (>3 mm).

65 no reduction, reduction in the percentage of periodontal pockets>3 mm versus no reduction, no increas

66 abolic pathway leading to H(2)S formation in periodontal pockets has not been determined.

67 L-6(+) cells) were elevated adjacent to deep periodontal pockets; however, there was no significant e

68 t that local delivery of antimicrobials into periodontal pockets improve periodontal health.

69 Local delivery of antimicrobials into periodontal pockets improves periodontal health.

70 nd subgingival plaque taken from the deepest periodontal pocket in each sextant may yield the most re

71 ent study evaluated the clinical response of periodontal pockets in beagle dogs after treatment with

72 s increase in tissue cells and especially in periodontal pockets in patients with CP, and the periodo

73 mmunologic benefits in the treatment of deep periodontal pockets in single-rooted teeth in patients w

74 , for the beagle dogs with severely infected periodontal pockets in this study, treatment with subgin

75 to the proliferation of P. gingivalis within periodontal pockets in which erythrocytes are abundant.

76 n destruction and bone resorption locally in periodontal pockets, in a double-blind placebo-controlle

77 , promotes the growth of the pathogen in the periodontal pocket, initially by enhancing its survivabi

78 athione, a readily available thiol source in periodontal pockets, is a suitable substrate for H(2)S p

79 The hypothesis of this study is that periodontal pockets may be associated with elevated bloo

80 P. gingivalis colonization of the periodontal pockets may increase visfatin secretion.

81 GCF was collected from two periodontal pockets (mean +/- SD: 5.1 +/- 1.0 mm) at bas

82 The microorganism was isolated from a periodontal pocket of a patient with periodontal disease

83 ted with pain in the upper left quadrant and periodontal pocketing of at least 6 mm in each of the fo

84 Twenty patients exhibited no periodontal pockets of > or = 5 mm, other than the study

85 est gel was administered by syringe into the periodontal pockets of 18 systemically healthy adult vol

86 Twenty-two patients revealed multiple periodontal pockets of 5 mm or more and numerous pathoge

87 re capable of establishing themselves in the periodontal pockets of nonimmunocompromised individuals

88 The periodontal pockets of one quadrant in each patient were

89 the most abundant bacteria identified in the periodontal pockets of periodontitis patients.

90 Oral H. pylori was detected from periodontal pockets of various depths and the dorsum of

91 gn endodontic-periodontic lesion with a 7-mm periodontal pocket on tooth #15 in a 40-year-old, non-sm

92 Diode laser therapy was applied to periodontal pockets on days 1, 3, and 7 after SRP.

93 25(OH)D level and teeth with deep (>/=4 mm) periodontal pockets or bleeding sextants.

94 ithin diseased gingiva adjacent to 4 to 6 mm periodontal pockets (P <0.001) and were not correlated w

95 n higher proportions in deep than in shallow periodontal pockets (P = 0.02).

96 neutrophils were harvested from prespecified periodontal pockets, purified, stained, and examined by

97 temic azithromycin (AZM) in combination with periodontal pocket reduction surgery in the treatment of

98 valis in the inflammatory environment of the periodontal pocket requires an ability to overcome oxida

99 in the inflammatory microenvironment of the periodontal pocket requires an ability to overcome oxida

100 e 2 DM and CP, local delivery of 1% ALN into periodontal pockets resulted in a significant increase i

101 ment loss, probing depths, and percentage of periodontal pocket sites > or =5 mm were measured.

102 Blood vessel features in periodontal pocket soft tissues may be significant in th

103 s that the local delivery of 1% ALN into the periodontal pocket stimulated a significant increase in

104 tudy show that local delivery of MF into the periodontal pocket stimulated significant increase in th

105 t that permits irradiation directly into the periodontal pocket/sulcus.

106 cquire systemic access through the ulcerated periodontal pocket surface; conclusive evidence supporti

107 ving subgingival biofilm in moderate-to-deep periodontal pockets than SRP.

108 major metabolic end product detected in deep periodontal pockets that is produced by resident periodo

109 serts may have advantages in negotiating the periodontal pocket, the relatively narrow structure may

110 However, in diseased periodontal pockets, treponemes thrive and become a domi

111 a matching placebo gel was applied into the periodontal pocket using a blunt applicator.

112 somewhat lower proportion of teeth with deep periodontal pockets was found in higher serum 25(OH)D qu

113 The presence of periodontal pockets was positively associated with highe

114 The periodontal pockets were exposed to a KTP laser with the

115 ingival biofilm specimens from inflamed deep periodontal pockets were removed before treatment from 4

116 emorrhagic gingiva adjacent to a > or = 3 mm periodontal pocket) were studied.

117 hemorrhagic gingiva adjacent to a > or =3 mm periodontal pocket) were studied.

118 djacent to a < or =3 mm, 4 to 6 mm, or >6 mm periodontal pocket) were studied.

119 plex with respect to the gingival sulcus and periodontal pockets (where the very different defensive

120 ophils are recruited in large numbers to the periodontal pocket, where they play a crucial role in th

121 ationale that indicates that the presence of periodontal pockets which can harbor pathogenic microorg

122 Periodontal pockets with a mean probing depth of 5.5 +/-

123 removing most of the subgingival biofilm in periodontal pockets with an APD < or =3 mm.

124 In periodontal pockets with PD of >/=5.4 mm, a single subgi

125 were randomly assigned to receive SubGPAP in periodontal pockets with probing depths of 4 to 9 mm, Su

126 subgingival debridement efficacy of GPAP in periodontal pockets with various depths.