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1 t diabetic who survived the complications of peripancreatic abscess, enterocutaneous fistula, and art
2 al status of 56 liver lesions and 48 primary peripancreatic adenocarcinomas obtained from 48 patients
3 s (BDPs) from well-differentiated metastatic peripancreatic adenocarcinomas on histological grounds a
4 ng laparoscopy has been used in a variety of peripancreatic and biliary malignancies.
5 volution of disease by reviewing pancreatic, peripancreatic, and ductal changes.
6         Historically, patients with positive peripancreatic aspirate culture have required operation.
7 -Kettering Cancer Center with a diagnosis of peripancreatic cancer, 1363 of whom had adenocarcinoma o
8                                         Most peripancreatic carcinomas harbor activating point mutati
9 as seen in those with diffuse pancreatic and peripancreatic changes.
10 ncer, 250 subjects with other pancreatic and peripancreatic diseases, and 116 controls.
11                            MRC also detected peripancreatic edema and inflammatory changes consistent
12 ntation, acute rejection, and CT findings of peripancreatic edema and/or inflammatory change were sig
13  means of CT and MRI showed small amounts of peripancreatic fluid along with a limited area of intra-
14 rainage of associated or subsequent areas of peripancreatic fluid and/or WOPN.
15 sequently required operation to manage their peripancreatic fluid collection, 37 urgent or emergent.
16 plications or failure of management of their peripancreatic fluid collection.
17 tic calcification, peripancreatic nodes, and peripancreatic fluid collection.
18 ck of vascular encasement, calcification, or peripancreatic fluid collection.
19                                              Peripancreatic fluid collections (PPFC) are a serious co
20 ostoperative complications included: 6 (13%) peripancreatic fluid collections and 2 (4%) pancreatitis
21  been the mainstay of treatment for infected peripancreatic fluid collections and abscesses.
22 en operative and nonoperative management for peripancreatic fluid collections and pseudocysts should
23   No previous study has examined the role of peripancreatic fluid collections and subsequent pseudocy
24 enhancing capsule develops, persistent acute peripancreatic fluid collections are referred to as pseu
25                   Pancreatic pseudocysts and peripancreatic fluid collections associated with acute p
26  the diagnosis of infection in pancreatic or peripancreatic fluid collections in patients with AP.
27 kocytes to detect infection in pancreatic or peripancreatic fluid collections in patients with AP.
28                                              Peripancreatic fluid collections include hematoma/seroma
29 e acute biliary pancreatitis and demonstrate peripancreatic fluid collections or pseudocysts until th
30 3, all patients admitted to our service with peripancreatic fluid collections or pseudocysts were mon
31 mplications included four splenectomies, two peripancreatic fluid collections, one pseudocyst, and on
32 e called acute necrotic collections or acute peripancreatic fluid collections.
33 reatitis survived their acute stage; 151 had peripancreatic fluid collections.
34 tic tail in three fresh cadavers to simulate peripancreatic fluid collections.
35 imary endpoint: development of pancreatic or peripancreatic infection within 42 days following random
36 tween the treatment groups for pancreatic or peripancreatic infection, mortality, or requirement for
37 eatitis and the development of pancreatic or peripancreatic infection; all-cause mortality; requireme
38                                Pancreatic or peripancreatic infections developed in 18% (9 of 50) of
39  of reducing the incidence of pancreatic and peripancreatic infections, although the benefits of doin
40 diffuse pancreatic enlargement, with minimal peripancreatic inflammation and absence of vascular enca
41 ancreatic ducts, and other findings, such as peripancreatic inflammation, encasement of vessels, mass
42 s with potentially resectable pancreatic and peripancreatic lesions.
43 s dependent upon both initial priming in the peripancreatic lymph node and subsequent presentation in
44                                       In the peripancreatic lymph node, division of naive 3A9 cells w
45 d pancreaticoduodenectomy (removing only the peripancreatic lymph nodes en bloc with the specimen) or
46 d pancreaticoduodenectomy (removing only the peripancreatic lymph nodes en bloc with the specimen) or
47 -64) peptide were found spontaneously in the peripancreatic lymph nodes of pre-diabetic NOD mice.
48                                      Several peripancreatic lymph nodes were observed that measured u
49 condary lymphoid structures, most likely the peripancreatic lymph nodes, were essential for the devel
50 -Ig had no axillary, inguinal, popliteal, or peripancreatic lymph nodes.
51                   Nine patients had enlarged peripancreatic lymph nodes.
52 n 10 mm, mural nodules, vascular encasement, peripancreatic lymphadenopathy, or metastases.
53                           Most patients with peripancreatic malignancy are found at exploration to be
54 in many patients with potentially resectable peripancreatic malignancy.
55 is of esophageal (n = 23), gastric (n = 75), peripancreatic (n = 86), or bile duct (n = 11) cancer un
56                                          The peripancreatic necrosis volume of 112.5 ml was a marker
57 sels, mass effect, pancreatic calcification, peripancreatic nodes, and peripancreatic fluid collectio
58          Eighty-five percent of patients had peripancreatic soft tissue invasion microscopically, and
59 l (one of 15 patients) parenchymal swelling, peripancreatic stranding (10 of 15 patients), "halo" (ni
60                                              Peripancreatic stranding was present on 28 scans and was
61                   Seven patients had minimal peripancreatic stranding, with lack of vascular encaseme
62  115 patients with radiologically resectable peripancreatic tumors underwent extended laparoscopy bef
63 ter informed consent, eligible patients with peripancreatic tumors were randomized during surgery eit
64 s useful in evaluating the primary tumor and peripancreatic vascular anatomy.
65 partial regression of tumor contact with any peripancreatic vascular axis was associated with R0 rese
66  provides maximal pancreatic parenchymal and peripancreatic vascular enhancement.
67  improve the enhancement of the pancreas and peripancreatic vasculature, improve tumor conspicuity, a
68  of tumor, bordering pancreas, and all major peripancreatic vessels were obtained for both time inter

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