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1 t diabetic who survived the complications of peripancreatic abscess, enterocutaneous fistula, and art
2 al status of 56 liver lesions and 48 primary peripancreatic adenocarcinomas obtained from 48 patients
3 s (BDPs) from well-differentiated metastatic peripancreatic adenocarcinomas on histological grounds a
7 -Kettering Cancer Center with a diagnosis of peripancreatic cancer, 1363 of whom had adenocarcinoma o
12 ntation, acute rejection, and CT findings of peripancreatic edema and/or inflammatory change were sig
13 means of CT and MRI showed small amounts of peripancreatic fluid along with a limited area of intra-
15 sequently required operation to manage their peripancreatic fluid collection, 37 urgent or emergent.
20 ostoperative complications included: 6 (13%) peripancreatic fluid collections and 2 (4%) pancreatitis
22 en operative and nonoperative management for peripancreatic fluid collections and pseudocysts should
23 No previous study has examined the role of peripancreatic fluid collections and subsequent pseudocy
24 enhancing capsule develops, persistent acute peripancreatic fluid collections are referred to as pseu
26 the diagnosis of infection in pancreatic or peripancreatic fluid collections in patients with AP.
27 kocytes to detect infection in pancreatic or peripancreatic fluid collections in patients with AP.
29 e acute biliary pancreatitis and demonstrate peripancreatic fluid collections or pseudocysts until th
30 3, all patients admitted to our service with peripancreatic fluid collections or pseudocysts were mon
31 mplications included four splenectomies, two peripancreatic fluid collections, one pseudocyst, and on
35 imary endpoint: development of pancreatic or peripancreatic infection within 42 days following random
36 tween the treatment groups for pancreatic or peripancreatic infection, mortality, or requirement for
37 eatitis and the development of pancreatic or peripancreatic infection; all-cause mortality; requireme
39 of reducing the incidence of pancreatic and peripancreatic infections, although the benefits of doin
40 diffuse pancreatic enlargement, with minimal peripancreatic inflammation and absence of vascular enca
41 ancreatic ducts, and other findings, such as peripancreatic inflammation, encasement of vessels, mass
43 s dependent upon both initial priming in the peripancreatic lymph node and subsequent presentation in
45 d pancreaticoduodenectomy (removing only the peripancreatic lymph nodes en bloc with the specimen) or
46 d pancreaticoduodenectomy (removing only the peripancreatic lymph nodes en bloc with the specimen) or
47 -64) peptide were found spontaneously in the peripancreatic lymph nodes of pre-diabetic NOD mice.
49 condary lymphoid structures, most likely the peripancreatic lymph nodes, were essential for the devel
55 is of esophageal (n = 23), gastric (n = 75), peripancreatic (n = 86), or bile duct (n = 11) cancer un
57 sels, mass effect, pancreatic calcification, peripancreatic nodes, and peripancreatic fluid collectio
59 l (one of 15 patients) parenchymal swelling, peripancreatic stranding (10 of 15 patients), "halo" (ni
62 115 patients with radiologically resectable peripancreatic tumors underwent extended laparoscopy bef
63 ter informed consent, eligible patients with peripancreatic tumors were randomized during surgery eit
65 partial regression of tumor contact with any peripancreatic vascular axis was associated with R0 rese
67 improve the enhancement of the pancreas and peripancreatic vasculature, improve tumor conspicuity, a
68 of tumor, bordering pancreas, and all major peripancreatic vessels were obtained for both time inter
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