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1  reciprocal accumulation of these Abs in the peripheral circulation).
2 corresponding increase of human cells in the peripheral circulation.
3 cytokines would recruit donor cells into the peripheral circulation.
4 e liver in 16 of the 35 patients, but not in peripheral circulation.
5 r treatment mobilizes OC precursors into the peripheral circulation.
6 al after it can no longer be detected in the peripheral circulation.
7 ins, are also present in soluble form in the peripheral circulation.
8 hormone glucagon-like peptide (GLP)-1 in the peripheral circulation.
9 and exchange interface between the brain and peripheral circulation.
10 l egress from the bone marrow niche into the peripheral circulation.
11 ges at the blood-brain barrier and/or in the peripheral circulation.
12 n was greater in the retina but lower in the peripheral circulation.
13 he local site of infection compared with the peripheral circulation.
14 e of appearance of ingested nutrients in the peripheral circulation.
15 old mobilization in leukemic blasts into the peripheral circulation.
16 rkedly reduced but detectable B cells in the peripheral circulation.
17 he tumor draining lymph nodes but not in the peripheral circulation.
18 ecreases in these NKT cell parameters in the peripheral circulation.
19 ogical Abeta and IgM concentrations found in peripheral circulation.
20 D8(+) T cells, were rapidly decreased in the peripheral circulation.
21 bited profound T cell lymphocytopenia in the peripheral circulation.
22  IGF-1R+CD4+CD3+ cells as those found in the peripheral circulation.
23 mobilization of myeloid lineage cells to the peripheral circulation.
24 ethanol levels in the coronary sinus and the peripheral circulation.
25  endothelium-dependent vasodilation in human peripheral circulation.
26 as selection in the thymus and homing in the peripheral circulation.
27 testinal beta-glucosidases for uptake to the peripheral circulation.
28 cocaine can inhibit NE reuptake in the human peripheral circulation.
29 s the brain from toxic substances within the peripheral circulation.
30  to permit sequestration of parasites in the peripheral circulation.
31 rauma may induce mobilization of CEPs to the peripheral circulation.
32 -forming units-granulocyte-macrophage to the peripheral circulation.
33 parasite stage found in large numbers in the peripheral circulation.
34 f SCID and was found to lack NK cells in his peripheral circulation.
35 t promotes rapid mobilization of CEPs to the peripheral circulation.
36 igher in the pleural compartment than in the peripheral circulation.
37 ascent eosinophils from bone marrow into the peripheral circulation.
38 ors and in breast tumor cells present in the peripheral circulation.
39 e blood flow towards the brain and away from peripheral circulations.
40 ercentage of surface IgM(dim) B cells in the peripheral circulation (0.08% compared with 5-20% in hea
41 rt, and restored endothelial function in the peripheral circulation (89.8 +/- 2.9%).
42 ization or augment EPC mobilization into the peripheral circulation after injury in endothelial nitri
43  increased deformability and reappear in the peripheral circulation, allowing uptake by mosquitoes.
44 rogenitor (MPP) cells in the bone marrow and peripheral circulation, an increase in populations of ea
45 cocaine can inhibit NE reuptake in the human peripheral circulation and (b) whether the NE-mediated p
46 luble receptor, sTNFp55) is increased in the peripheral circulation and amniotic fluid of women with
47 ssing cells mobilize PMNs and HSPCs into the peripheral circulation and are therapeutically useful fo
48 itical role in cholesterol transport in both peripheral circulation and brain.
49 d in the bone marrow can be mobilized to the peripheral circulation and can colonize endothelial flow
50       Sympathetic vasoconstriction regulates peripheral circulation and controls blood pressure, but
51 mparisons are made to recent advances in the peripheral circulation and current gaps in our knowledge
52  (Ang II) is a potent vasoconstrictor in the peripheral circulation and has been implicated in many c
53 Cs are mobilized from the bone marrow to the peripheral circulation and home to the sites of new vess
54 nd integrins unrelated to LFA-1, both in the peripheral circulation and in diseased skin tissue.
55 em or progenitor cells were increased in the peripheral circulation and incorporated into the site of
56 lts demonstrated that BMSCs can recruit from peripheral circulation and participate in wound healing
57  organs that act as an interface between the peripheral circulation and the brain.
58 ered to be the recruitment of cells from the peripheral circulation and the re-entry of mature cells
59 rom the lungs to the tissue but forms in the peripheral circulation, and (iii) SNO-Hb and S-nitrosoth
60  are able to leave the lymph node, enter the peripheral circulation, and migrate to the s.c. immuniza
61 ities of ventilatory control, the lungs, the peripheral circulation, and skeletal muscle appear to co
62 nt a predominant cell source of IL-11 in the peripheral circulation, and the percentage of IL-11(+)CD
63 owever, did not correlate with levels in the peripheral circulation, and there were no T cells or an
64 ent of occlusive disease in the coronary and peripheral circulations, and traditional pharmacotherape
65 elevant, since elevated anandamide levels in peripheral circulation are associated with spontaneous p
66 dulates the balance of T cell subsets in the peripheral circulation as well as multiple inflammatory
67  cells in bone marrow that mobilize into the peripheral circulation at high concentrations.
68 ific CD8(+) T cells can be maintained in the peripheral circulation at high frequency in the absence
69 CD8+ cells and decreased NK1.1+ cells in the peripheral circulation at the time of transplantation.
70  LSP and RAG-1+ ELP were both present in the peripheral circulation, but RAG-1+ ELP had no exact coun
71 , injected labeled MSCs had cleared from the peripheral circulation by 15 minutes after injection.
72 ngent selection either in the bone marrow or peripheral circulation by deletion, induction of anergy,
73 E degeneration, BMCs were mobilized into the peripheral circulation by granulocyte-colony stimulating
74 t appears that BMCs, when mobilized into the peripheral circulation, can home to focal areas of RPE d
75 tastatic disease characterized by EVs in the peripheral circulation containing mtDNA.
76 ted Lewis rats, but could be measured in the peripheral circulation during small bowel allograft reje
77 expressing granulocytes were obtained in the peripheral circulation during the early posttransplant p
78                 I-FABP was released into the peripheral circulation early in the evolution of acute r
79 NGFR+/TK+ donor lymphocytes persisted in the peripheral circulation for 6 months in both syngeneic an
80 nts had persistent donor class II DNA in the peripheral circulation for at least 1 year after transpl
81 ema, and elevated levels of TNF-alpha in the peripheral circulation from cardiac spillover.
82 erotic occlusive disease in the coronary and peripheral circulations; however, long-term results are
83  and oxytocin (OT) levels in the IPS and the peripheral circulation in nine normal volunteers, before
84 n be mobilized from the bone marrow into the peripheral circulation in response to a number of stimul
85 er over time and failed to mobilize into the peripheral circulation in response to cytokine stimulati
86 ashion to attenuate its own release into the peripheral circulation in response to increased plasma o
87                       SCS is used to improve peripheral circulation in selected patients with ischemi
88 dy suggest that fetal cells migrate from the peripheral circulation into multiple organs in women wit
89 n of the thymus, a low CD3 percentage in the peripheral circulation is also associated with a CD8(low
90 he constitutive number of eosinophils in the peripheral circulation is also demonstrated.
91                      Vascular disease in the peripheral circulation is associated with significant mo
92 clusion, an adequate insulin delivery in the peripheral circulation, obtained by islet transplantatio
93 es more CD4(+) T cells than basophils in the peripheral circulation of filaria-infected patients, the
94                       When injected into the peripheral circulation of mice with either, orthotopic p
95 e, the presence of normal blood cells in the peripheral circulation of neonatal and adult sph/sph mic
96  that KLK4-specific CD4 T cells exist in the peripheral circulation of normal male donors and the ide
97 to muscarinic stimulation is impaired in the peripheral circulation of patients with congestive heart
98 ulating tumor cells (CTCs) isolated from the peripheral circulation of patients with metastatic color
99 specific CD8+ T cells were determined in the peripheral circulation of patients with squamous cell ca
100 y, IL-10 must either access the CNS from the peripheral circulation or be delivered directly to it by
101 ar proteins not previously identified in the peripheral circulation or have functional roles relevant
102 ic, with the capacity to enter skin from the peripheral circulation, patrol within tissue, and migrat
103  of high-avidity autoreactive T cells in the peripheral circulation, perhaps due to this capability o
104 to the delivery of viable platelets into the peripheral circulation (peripheral platelet mass turnove
105 t displace the parasite from the deep to the peripheral circulation, promoting its elimination at the
106  (patrolling) monocytes in the heart and the peripheral circulation provides evidence that the massiv
107 monstrate that overexpression of SDF1 in the peripheral circulation results in the mobilization of he
108 tor cells (EPCs) from the bone marrow to the peripheral circulation (see the related article beginnin
109 s that the MUC1 alpha chain is shed into the peripheral circulation, sequesters circulating antitande
110 termine the proportion of each subset in the peripheral circulation, spleen, and bronchotracheal lymp
111  is found more commonly in the liver than in peripheral circulation, suggesting a tissue-specific loc
112 otective interface between the brain and the peripheral circulation that maintains the extracellular
113 As thymocytes leave the thymus and enter the peripheral circulation, the expression of CXCR4 is again
114 r necrosis factor-alpha (TNF-alpha) in their peripheral circulation, the structural and functional ef
115        Tregs are also small in number in the peripheral circulation, thus they require ex vivo expans
116  to the recruitment of mature cells from the peripheral circulation to the development of airway eosi
117 ing marked redistribution of blood flow from peripheral circulations to the brain.
118 ves a redistribution of blood flow away from peripheral circulations towards essential vascular beds,
119 rbed from the intestine intact and reach the peripheral circulation unchanged.
120 xtracellular osmotic conditions found in the peripheral circulation vs. the bone marrow could dictate
121 hocytes subsequently traffic to the lungs or peripheral circulation, we compared the immune responses
122 ve of neutrophils that are released into the peripheral circulation where they traverse to sites of i
123 CR4 interaction and releases BM PCs into the peripheral circulation, where the mobilized cells are re
124 one of the major antigen presenting cells in peripheral circulation, which are chronically exposed to
125 ecially relevant to pain caused by issues of peripheral circulation, which is commonly observed in di
126 s (OCs), CD34+ cells were mobilized into the peripheral circulation with granulocyte colony-stimulati
127 thesized that elevation of SDF1 level in the peripheral circulation would result in mobilization of p

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