ライフサイエンスコーパス: peripheral resistance

1 despite reduced diastolic pressure and total peripheral resistance.

2 ystem, and increased vascular tone and total peripheral resistance.

3 rterial enlargement in response to increased peripheral resistance.

4 lity, systemic compliance, stroke volume and peripheral resistance.

5 ormal, where it is quickly reflected off the peripheral resistance.

6 cant increases in SS RBC adhesion and in the peripheral resistance.

7 sure, occurs as the consequence of increased peripheral resistance.

8 ke volume, possibly as a result of decreased peripheral resistance.

9 ut (5.5 vs. 4.9 l/min; P < 0.001), and lower peripheral resistance (1,487 vs. 1,666; P = 0.01), paral

10 15% vs. -5 +/- 10%), and a lower decrease in peripheral resistance (-17 +/- 12% vs. -26 +/- 12%) (p <

11 n increase in blood pressure (43%) and total peripheral resistance (65%) without any change in heart

12 cts peripheral resistance to blood flow, and peripheral resistance affects DBP.

13 d by elevated heart rate and decreased total peripheral resistance and arterial blood pressure.

14 on were hypotensive, with decreases in total peripheral resistance and filtration fraction on day 1 i

15 rolled for, hypertensive subjects had higher peripheral resistance and lower arterial compliance than

16 late vasoconstriction, which increases total peripheral resistance and mean arterial pressure.

17 Peripheral resistance and peak filling rate were unchang

18 Total peripheral resistance and perivascular fibrosis in the h

19 during EFP than MLP (P = 0.030), while total peripheral resistance and plasma noradrenaline were not

20 nts present with low cardiac output and high peripheral resistance and that they respond poorly to fl

21 pressure and is a fundamental determinant of peripheral resistance and, hence, organ perfusion and sy

22 high rate of LV hypertrophy, in spite of low peripheral resistances and low-to-normal blood pressure,

23 Cardiac index, total peripheral resistance, and blood volume were not differe

24 stemic oxygen delivery, stroke volume, total peripheral resistance, and organ blood flow in the liver

25 ate, preload, and cardiac output; decreasing peripheral resistance; and increasing ventricular compli

26 ecause endothelial dysfunction and increased peripheral resistance are hallmarks of hypertension, det

27 between central control of vascular flow and peripheral resistance are unclear.

28 ar volumes and end-systolic elastance (Ees), peripheral resistance, arterial elastance (Ea), arterial

29 Autonomic sympathetic nerves innervate peripheral resistance arteries, thereby regulating vascu

30 ationship between large artery stiffness and peripheral resistance artery function.

31 f altered arterial stiffness versus impaired peripheral resistance but is not superior to SBP+DBP in

32 ut and decreases in left atrial pressure and peripheral resistance but without eliciting a supplement

33 sure, portal pressure, cardiac output, total peripheral resistance, central blood volume, and extrace

34 er (P = 0.09), heart rate was similar, total peripheral resistance decreased (2172 +/- 364 vs. 2543 +

35 nfusion before any symptoms developed, total peripheral resistance decreased 24% +/- 20% in group II

36 to tilt before any symptoms developed, total peripheral resistance decreased 9% +/- 14% in group I fr

37 Cardiac index and total peripheral resistance decreased in MSA patients by 33.4+

38 Total peripheral resistance decreased with rosuvastatin in bot

39 s include increased cardiac work with normal peripheral resistance, diffuse slowing on electroencepha

40 a means for maintenance of vascular tone and peripheral resistance during hibernation.

41 Total peripheral resistance during tilt post flight was higher

42 tput, and calculated stroke volume and total peripheral resistance, during supine rest and 10 min of

43 ined by LV end-diastolic dimension and total peripheral resistance estimated by thoracic impedance.

44 to 86.9 +/- 21.7 ml (P = 0.06) and increased peripheral resistance from 1106 +/- 246 to 1246 +/- 222

45 EC size, occlusion of capillaries, elevated peripheral resistance, heart failure, and death.

46 subjects, accompanied by a decrease in total peripheral resistance in 16 of them (64%, group A).

47 In vivo, human U-II markedly increases total peripheral resistance in anaesthetized non-human primate

48 aptation may contribute to the regulation of peripheral resistance in eNOS-KO mice.

49 t dilatation may contribute to the increased peripheral resistance in hypertension.

50 es to the elevation of wall shear stress and peripheral resistance in hypertension.

51 rdiac output (CO) but minor changes in total peripheral resistance in saline-treated rats.

52 ntry communication) and physiologic factors (peripheral resistance in the branch vessels, pump output

53 ge of factors: one key element of control is peripheral resistance in tissue capillary beds.

54 he true-lumen outflow caused by lowering the peripheral resistance in true-lumen branch vessels.

55 (P<0.05) compared with baseline, while total peripheral resistance increased (P<0.05).

56 Na+ intake, while baseline CO decreased and peripheral resistance increased in this group.

57 nd 1.15 versus 1.02 mm Hg/mL x m2) and total peripheral resistance index (3027 and 2805 versus 2566 d

58 n cardiac index, a 28% increase in the total peripheral resistance index (p < .01), and a 33% decreas

59 The total peripheral resistance index and stroke volume index tend

60 ed with increases in both cardiac output and peripheral resistance index.

61 This increased peripheral resistance is the result of an increased acti

62 Although total peripheral resistance markedly increased, mean arterial

63 mean arterial pressure of 18 mm Hg and total peripheral resistance of 665 AU and increases in heart r

64 roximately 20 mm Hg and an increase in total peripheral resistance of approximately 30%.

65 01) while accentuating the increase in total peripheral resistance (P=0.012).

66 decreases in left atrial pressure (P<0.001), peripheral resistance (P=0.014), and hematocrit (P<0.001

67 women relative wall thickness (r = 0.11) and peripheral resistance (r = -0.17).

68 Although total peripheral resistance remains decreased, Na+ retention c

69 epsis), cardiac output, stroke volume, total peripheral resistance, systemic oxygen delivery, and org

70 oth muscle cells of a primary determinant of peripheral resistance - the small mesenteric artery.

71 Viscosity affects peripheral resistance to blood flow, and peripheral resi

72 cially the visceral type, is associated with peripheral resistance to insulin actions and hyperinsuli

73 modalities, such as exercise, may overcome a peripheral resistance to insulin, thus preventing GDM or

74 Peripheral resistance to leptin due to its impaired brai

75 ng leptin and have the potential to induce a peripheral resistance to leptin, similar to the central

76 roke volume (SV), cardiac output (CO), total peripheral resistance (TPR) and arterial compliance to a

77 tivity (MSNA) is positively related to total peripheral resistance (TPR) and inversely related to car

78 asures of preejection period (PEP) and total peripheral resistance (TPR) in healthy black (n=76) and

79 ed, whereas heart rate (HR), MSNA, and total peripheral resistance (TPR) increased during HUT (all P<

80 Baseline total peripheral resistance (TPR) was not different between th

81 decrease in cardiac contractility and total peripheral resistance (TPR) were similar in TRPV(1)(+/+)

82 a sympathetically-mediated increase in total peripheral resistance (TPR).

83 Microvascular blockage resulted in increased peripheral resistance units (PRU).

84 d action of nitric oxide and endothelin-1 in peripheral resistance vessels of patients with syndrome

85 Total peripheral resistance was calculated from mean arterial

86 2 +/- 0.40 l min(-1), P = 0.01), while total peripheral resistance was greater (1327 +/- 117 vs. 903

87 Supine leg peripheral resistance was greater than control resistanc

88 ght with CFD than in those with PFD, whereas peripheral resistance was not significantly different.

89 volume (r=0.88+/-0.13, P<0.05), while total peripheral resistance was related to MSNA during 45 min

90 with control rats, but the increase in total peripheral resistance was significantly attenuated.

91 In the other 9 subjects, total peripheral resistance was well maintained even at presyn

92 eart rate, mean arterial pressure, and total peripheral resistance were greater, whereas cardiac outp

93 Upright heart rate and total peripheral resistance were greater, whereas stroke volum

94 Blood pressure and total peripheral resistance were higher during rest and tiltin

95 decrease in diastolic pressure and estimated peripheral resistance were observed in troglitazone-trea