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1 ctivity regulates blood pressure by altering peripheral vascular resistance.
2 sure, there is no decrease in either MSNA or peripheral vascular resistance.
3 em may be due, in part, through reduction of peripheral vascular resistance.
4 een sympathetic activity and the increase in peripheral vascular resistance.
5 The effect of water is mediated by increased peripheral vascular resistance.
6 nsiveness, and vascular volume and increased peripheral vascular resistance.
7 a and a sympathetically mediated increase in peripheral vascular resistance.
8 output (36%), stroke volume (30%), and total peripheral vascular resistance (45%), along with increas
9 in peripheral mean arterial pressure (PMAP), peripheral vascular resistance, AIx, aortic stiffness an
11 -Hb is a composite effect created by reduced peripheral vascular resistance and direct inhibition of
12 were improved by drug treatments that reduce peripheral vascular resistance and hypertension, consist
13 siologic parameters (e.g., decrease in total peripheral vascular resistance and increase in cardiac s
14 be a compensatory mechanism for the blunted peripheral vascular resistance and lower central volume.
16 s been traditionally ascribed to decrease in peripheral vascular resistance and to an antiatherogenic
17 ETA/B receptor antagonist TAK-044 decreases peripheral vascular resistance and, to a lesser extent,
18 greater effects on arterial pressure, total peripheral vascular resistance, and cardiac index, compa
19 re, cardiac output, stroke volume, and total peripheral vascular resistance by diaspirin crosslinked
20 in constant BP in salt resistance, renal and peripheral vascular resistance falls and is associated w
21 index and lowered mean arterial pressure and peripheral vascular resistance in patients and healthy c
22 harmacologic strategies designed to maintain peripheral vascular resistance in patients with insuffic
25 this study investigated how changes in fetal peripheral vascular resistance, in plasma concentrations
26 Cooling resulted in a greater increase in peripheral vascular resistance index (+515+/-160 versus
27 whereas reducing mean arterial pressure and peripheral vascular resistance index (P<0.001 for all) i
28 ed fall in cardiac output with no changes in peripheral vascular resistance may contribute to (pre)sy
30 s a reduced ability of astronauts to augment peripheral vascular resistance, often resulting in ortho
31 ts of a sympathetically mediated increase in peripheral vascular resistance, parasympathetically medi
33 ine in CHF rats may be due to a reduction in peripheral vascular resistance since epicardial lidocain
34 7% +/- 3% of baseline and increases in total peripheral vascular resistance to 146% +/- 5% of baselin
35 nd (3) assess the contribution of changes in peripheral vascular resistance to the glucocorticoid-ind
36 en, high SNA is associated with higher total peripheral vascular resistance (TPR), and appears to be
37 ine-induced hypertension consist of elevated peripheral vascular resistance, ventricular vascular unc
38 rates between groups were not observed, and peripheral vascular resistance was significantly greater
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