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1 ight blindness, and 56% (14/25) with loss of peripheral vision.
2 ld defects specifically affecting central or peripheral vision.
3 re proposed as treatments for PDR that spare peripheral vision.
4 de field of view, involving both central and peripheral vision.
5 n-specific role difficulties and general and peripheral vision.
6 stic explanation for orientation crowding in peripheral vision.
7 primary factor limiting shape perception in peripheral vision.
8 al vision and a dependence on low-resolution peripheral vision.
9 the GVF the measure of choice for changes in peripheral vision.
10 least "some" difficulty with tasks requiring peripheral vision.
11 i may provide an objective measure of VA and peripheral vision.
12 tory targets using a laser pointer guided by peripheral vision.
13 ng, emotional distress, general lighting, or peripheral vision.
14 a selective loss of chromatic sensitivity in peripheral vision.
15 o an increase in reading speed in central or peripheral vision.
16 gested as an explanation for slow reading in peripheral vision.
17 t a different location, within the region of peripheral vision.
18 severely limited in clutter, particularly in peripheral vision.
19 8 for all subscales (P < 0.0001), except for peripheral vision (0.46; P = 0.0003), which also exhibit
22 outcomes: (1) reading and seeing detail, (2) peripheral vision, (3) darkness and glare, (4) household
24 al environments and the fundamental limit on peripheral vision, affecting identification within many
25 motional distress, general dim lighting, and peripheral vision), all with good internal consistency (
26 veal a complex pattern of visual deficits in peripheral vision and indicate a significant role of att
27 Besides the fundus appearance restricted peripheral vision and scotopic electroretinogram confirm
28 rsonal knowledge and deeply held values, use peripheral vision and subsidiary awareness to become awa
29 chromatic and selective S-cone conditions in peripheral vision and whether any association relates to
30 rized by late-onset night blindness, loss of peripheral vision, and diminished or absent electroretin
32 y-of-life measures increased for general and peripheral vision, and near and distance activities, imp
33 ffectively enhances object discrimination in peripheral vision at the goal of the intended saccade.
34 lties (beta = 0.04; R2 = 0.20; P = .01), and peripheral vision (beta = 0.03; R2 = 0.17; P = .03).
35 driving (beta = 0.05; R2 = 0.24; P < .001), peripheral vision (beta = 0.03; R2 = 0.18; P = .02), and
37 hat central retinal damage leads to enhanced peripheral vision by sensitizing the visual system for m
38 le classes of object recognition failures in peripheral vision can be accounted for by a single mecha
40 iously unknown dichotomy between central and peripheral vision could support accurate analysis of att
41 ifference, 16.3; 95% CI, 0.9-31.7; P = .04), peripheral vision (difference, 11.6; 95% CI, 0.8-22.4; P
42 , interphalangeal joint stiffness, decreased peripheral vision, diminished tactile sensation, and hal
44 ll, general vision, distance activities, and peripheral vision domains of theVFQ-25 (partial correlat
46 e measures are needed to estimate changes in peripheral vision during future treatment clinical trial
47 aired individuals often seem to underutilize peripheral vision, even in absence of obvious peripheral
48 ns that afflict the fovea and thus use their peripheral vision exclusively, the signature properties
50 e well-known decline in visual resolution in peripheral vision; however, the main bottleneck for read
51 visual processing speed, visual search, and peripheral vision in driving, especially among patients
52 of focus for resolution acuity measured for peripheral vision indicates that spatial resolution is l
53 tolerance of these place tags in foveal, and peripheral vision is about half the separation of the fe
59 target to protect cone-mediated central and peripheral vision loss in patients with retinitis pigeme
60 ver, PRP can damage the retina, resulting in peripheral vision loss or worsening diabetic macular ede
61 vioral alterations emerging after central or peripheral vision loss suggest that cerebral reorganizat
62 isorder characterized by night blindness and peripheral vision loss, and in many cases leads to blind
63 f people, compensatory recruitment of spared peripheral vision may give rise to cortical thickening.
66 sion," "distance activities," "dependency," "peripheral vision," "self-image," "daily living," and "d
69 onid), providing them with as much, or more, peripheral "vision" than the vespertilionids, but ensoni
71 the association of choroidal thickness and "peripheral vision." The strongest association was the LL
73 our vision that retains chromatic quality in peripheral vision, thus supporting the cone-selective hy
75 nce activities, mental health, and color and peripheral vision, with an overall adjusted mean differe
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