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1 derate to severe collagen deposition seen as perisinusoidal and bridging fibrosis using H&E, Trichome
2 chronic ASH with macrophage inflammation and perisinusoidal and pericellular fibrosis to AH in 57% (1
5 orm to sinusoidal endothelial cells or other perisinusoidal cells, such as hepatic stellate cells, wo
7 lls and Tcf21(+) stromal cells thus create a perisinusoidal EMH niche in the spleen, which is necessa
8 tic steatosis, substantial inflammation, and perisinusoidal fibrosis (i.e., steatohepatitis), associa
9 r and portal inflammation scores (P < 0.01), perisinusoidal fibrosis (P < 0.001), and NAS > or =5 (P
10 ic hepatosclerosis in which there is diffuse perisinusoidal fibrosis (type IV collagen) without zonal
13 he mean steatosis (1.6 vs. 2.16, P <.04) and perisinusoidal fibrosis scores (0.35 vs. 0.9, P <.049) w
17 d by steatosis, ballooning degeneration, and perisinusoidal fibrosis; type 2 was characterized by ste
18 d, it is now apparent that the activation of perisinusoidal hepatic stellate cells, which is a key ev
20 IgD(hi) B cells can occupy an extravascular perisinusoidal niche in the bone marrow in addition to t
22 and non-dividing HSCs thus reside mainly in perisinusoidal niches with Lepr(+)Cxcl12(high) cells thr
24 thin the metaphysis of long bones not in the perisinusoidal space and are needed for bone development
26 onectin in normal liver was localized to the perisinusoidal space surrounding the periportal and the
27 brinogen in normal liver is localized to the perisinusoidal space surrounding the periportal region.
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