ライフサイエンスコーパス: peritoneal cavity

1 eby, promoting neutrophil migration into the peritoneal cavity.

2 production after LPS injection in the murine peritoneal cavity.

3 tress as biologic processes activated in the peritoneal cavity.

4 tation of human endometrial fragments in the peritoneal cavity.

5 on pathophysiological alterations within the peritoneal cavity.

6 fluids reduce immunological defenses in the peritoneal cavity.

7 nude mice had LS174T tumors implanted in the peritoneal cavity.

8 rate and metastasize over the surface of the peritoneal cavity.

9 cal attributes of S. aureus infection in the peritoneal cavity.

10 receptor (M-CSFR) in both the blood and the peritoneal cavity.

11 patients, the disease remains limited to the peritoneal cavity.

12 a integrins to interstitial collagens in the peritoneal cavity.

13 in mice when co-injected with CXCL6 into the peritoneal cavity.

14 paracrine signaling from hepatocytes in the peritoneal cavity.

15 th dozens to hundreds of tumors studding the peritoneal cavity.

16 cells that reside in the normal, uninflamed peritoneal cavity.

17 spleens, peripheral blood, lungs, liver and peritoneal cavity.

18 and neutrophils in cremaster tissue and the peritoneal cavity.

19 IL-17-mediated monocyte recruitment into the peritoneal cavity.

20 ion of labeled donor cells into the mertk-/- peritoneal cavity.

21 robots that are inserted completely into the peritoneal cavity.

22 the fallopian tube, or the mesothelium-lined peritoneal cavity.

23 whole kidney marrow and abundant within the peritoneal cavity.

24 single layer of mesothelial cells lining the peritoneal cavity.

25 , resolving, and chronic inflammation in the peritoneal cavity.

26 f Pseudomonas aeruginosa introduced into the peritoneal cavity.

27 cer, was labeled by virus injection into the peritoneal cavity.

28 host response to bacterial infection of the peritoneal cavity.

29 from lung containing metastases, spleen, and peritoneal cavity.

30 rol modestly, especially when given into the peritoneal cavity.

31 mically via an osmotic pump implanted in the peritoneal cavity.

32 andomized selection of the 9 segments of the peritoneal cavity.

33 lantation of autologous endometrium into the peritoneal cavity.

34 he cecum and placing it into the recipient's peritoneal cavity.

35 enhanced neutrophil recruitment to inflamed peritoneal cavity.

36 ntenance of IgM(+) B cells in the spleen and peritoneal cavity.

37 ion elicited by urate crystals in the murine peritoneal cavity.

38 an increase in the number of bacteria in the peritoneal cavity.

39 mal numbers of B1a, B1b, and B2 cells in the peritoneal cavity.

40 +)IgD(lo) B cells in both the spleen and the peritoneal cavity.

41 neum-induced blood flow decreases within the peritoneal cavity.

42 grows on the omentum when injected into the peritoneal cavity.

43 nation of L. monocytogenes bacteria from the peritoneal cavity.

44 ots" in the liver, the lung airways, and the peritoneal cavity.

45 he formation of macrophage foam cells in the peritoneal cavity.

46 eater LPS-dependent production of TNF in the peritoneal cavity.

47 different sites including liver, kidney, and peritoneal cavity.

48 of Staphylococcus aureus inoculated into the peritoneal cavity.

49 ilar cells also appear somewhat later in the peritoneal cavity.

50 presence of small undetected nodules in the peritoneal cavity.

51 pus, bromodeoxyuridine was injected into the peritoneal cavity.

52 ld amount of free fluid was also seen in the peritoneal cavity.

53 Most of the BM-MSCs formed aggregates in the peritoneal cavity.

54 ltration of neutrophils and T cells into the peritoneal cavity.

55 ents of endometriotic lesion survival in the peritoneal cavity.

56 um along with 150 ml of hemorrhagic fluid in peritoneal cavity.

57 y cytokines and form implants throughout the peritoneal cavity.

58 in microglia and resident macrophages of the peritoneal cavity.

59 neal mucinous carcinomatosis confined to the peritoneal cavity.

60 productive antitumor immune response in the peritoneal cavity.

61 circumvented by T cell administration to the peritoneal cavity.

62 emically, highlighting the uniqueness of the peritoneal cavity.

63 ively activated macrophages (AAM) within the peritoneal cavity.

64 tial step of neutrophil recruitment into the peritoneal cavity.

65 n a few hours of IL-1beta injection into the peritoneal cavity.

66 was observed in CD8(+) memory T cells in the peritoneal cavity.

67 and tumor metastasis within the pleural and peritoneal cavities.

68 mbrane chambers that were implanted into rat peritoneal cavities.

69 dialysis membrane chambers implanted in rat peritoneal cavities.

70 In mast cells harvested from peritoneal cavities, 2 cytokine signals are required for

71 es (4/5), spleens (4/10), livers (6/10), and peritoneal cavities (8/10).

72 ynecological malignancies disseminate in the peritoneal cavity - a condition known as peritoneal carc

73 In the peritoneal cavity, a further loss was demonstrated early

74 r CLP with a concomitant increase within the peritoneal cavity, a pattern that was ablated in CXCR3-d

75 rotein 10, IP-10) increase in plasma and the peritoneal cavity after CLP, peak at 8 hours after infec

76 Migration of inflammatory monocytes into the peritoneal cavity after CLP, which is dependent on VLA-4

77 ished the accumulation of macrophages in the peritoneal cavity after thioglycolate injection.

78 However, the peritoneal cavity also contains relatively high frequenc

79 crophage population within bacteria-infected peritoneal cavities and increased the systemic level of

80 septic shock, in part, by migration into the peritoneal cavity and amplification of the proinflammato

81 ancer cells primarily disseminate within the peritoneal cavity and are only superficially invasive.

82 esiding in nonlymphoid organs, including the peritoneal cavity and fat pads, are more resistant to ap

83 Mesothelial cells (MCs) line the peritoneal cavity and help define peritoneal response to

84 In the peritoneal cavity and liver, C1q enhancement of type 2 m

85 eated transfused neutrophils to the inflamed peritoneal cavity and lungs was significantly elevated.

86 re, neutrophil recruitment into the inflamed peritoneal cavity and lymphocyte homing to secondary lym

87 veloped increased neutrophil influx into the peritoneal cavity and more efficient bacterial clearance

88 cells (IgM>>IgG) that migrate rapidly to the peritoneal cavity and persist there indefinitely, ready

89 ary, they disseminate into the organs of the peritoneal cavity and produce ascites, typical of ovaria

90 ch2(+/-) mice have reduced B1 B cells of the peritoneal cavity and show a severe reduction in MZ B ce

91 luding the accumulation of B-1a cells in the peritoneal cavity and spleen.

92 mentum collected antigens and cells from the peritoneal cavity and supported T cell-dependent B cell

93 red cytokine and chemokine production in the peritoneal cavity and suppressed neutrophil recruitment.

94 inhibits infiltration of macrophages to the peritoneal cavity and suppresses the activity of TNF-alp

95 d on B1 cells outside of the bone marrow and peritoneal cavity and that pathogenic natural IgM titers

96 B-1a cells reside in both the peritoneal cavity and the spleen.

97 C5a-mediated leukocyte recruitment into the peritoneal cavity and tumor regression were suppressed i

98 /kg body weight of Escherichia coli into the peritoneal cavity and within 1 hr were immediately place

99 tigens, particulates, and pathogens from the peritoneal cavity and, depending on the stimuli, promoti

100 sent <1% of total B cells, accumulate in the peritoneal cavity, and account for near-normal levels of

101 om the colon, blood, thymus, spleen, uterus, peritoneal cavity, and allergen-challenged lung.

102 tion of IFN-regulated gene expression in the peritoneal cavity, and an increased production of myeloi

103 CSF-1-dependent M-NFS-60 tumor cells in the peritoneal cavity, and diminished the accumulation of ma

104 bdominal aorta) and catheters (jugular vein, peritoneal cavity, and distal abdominal aorta).

105 e bone marrow, peripheral blood, spleen, and peritoneal cavity, and improved responses to T-independe

106 y cell populations was delayed in the brain, peritoneal cavity, and intestinal lamina propria, indica

107 iver, adrenal gland, kidney medulla, spleen, peritoneal cavity, and intestine.

108 se model, augmented cfB levels in the serum, peritoneal cavity, and major organs including the kidney

109 GM3 BLT mice and populate the immune system, peritoneal cavity, and peripheral tissues.

110 spontaneous metastasis to the lungs, to the peritoneal cavity, and resulted in 90% lethality within

111 , the persistence of kappa+ B-1 cells in the peritoneal cavity, and significant levels of serum IgM,k

112 lorohydrins were also instilled in the rats' peritoneal cavity, and their effects on peritoneal macro

113 surprising observation that B-1 cells in the peritoneal cavity are activated as early as 1 h after na

114 contact skin immunization, B-1 cells in the peritoneal cavity are activated to migrate to the lympho

115 opped gallstones in the peritoneal and extra-peritoneal cavity are usually asymptomatic.

116 imarily produced by B1a cells outside of the peritoneal cavity, are integrally involved.

117 and B-2 cell numbers increased in lungs and peritoneal cavity as early as day 1 postinfection, but l

118 ring nude mice, PTX was best retained in the peritoneal cavity as PTX-gel (microparticulate PTX entra

119 the parasites grew exponentially within the peritoneal cavity (as measured by light emitted from luc

120 ion of polymorphonuclear leukocytes into the peritoneal cavity, as well as abdominal writhings.

121 ave fewer cells than controls present in the peritoneal cavity, as well as fewer leukocytes leaving t

122 ger RNA for 145 C. albicans genes within the peritoneal cavity at 48 hours.

123 tion-elicited effector cells did not promote peritoneal cavity B cell depletion in FcR-deficient mice

124 By contrast, peritoneal cavity B cells expressed normal CD20 densitie

125 Importantly, IL-10 production by peritoneal cavity B cells significantly reduced disease

126 Bone marrow and spleen mature B cell and peritoneal cavity B-1 cell numbers were also halved, whe

127 Peritoneal cavity B-1 cells are believed to produce IgM

128 Peritoneal cavity B-1a B cells exhibited the most acapsu

129 detected was significantly lower in B6.Sle2 peritoneal cavity B-1a cells than in B6, with or without

130 In addition, a significant number of peritoneal cavity B-1a cells were recovered in lethally

131 pe Galalpha1,3Gal (Gal), to demonstrate that peritoneal cavity B-1b cells with anti-Gal receptors pro

132 Innate-like splenic marginal zone (MZ) and peritoneal cavity B1 B lymphocytes share critical respon

133 In this study, peritoneal cavity B10 cells shared similar cell surface

134 The BCR repertoire of peritoneal cavity B10 cells was diverse, as occurs in th

135 However, peritoneal cavity B10 cells were 10-fold more frequent a

136 Cdkn2c expression in the splenic B cells and peritoneal cavity B1a cells from Sle2c1-carrying mice, w

137 lenic B cells and increased proliferation of peritoneal cavity B1a cells.

138 ing rate and cumulative dose released in the peritoneal cavity based on the in vitro release data.

139 d in the spleen but remained elevated in the peritoneal cavity beyond 40 d postimmunization.

140 strointestinal system as well as pleural and peritoneal cavities but not the brain.

141 e expression in macrophages infiltrating the peritoneal cavity but was not observed in the resident p

142 blished MM-AN tumor nodules in the flank and peritoneal cavity by 85 to 90% without detectable toxici

143 o AVF and sham mice by protein transfer into peritoneal cavity by minipump for 4 weeks.

144 n be used to visualize ovarian tumors in the peritoneal cavity by multimodal MR and near infra-red im

145 al pathogens and affect the cells lining the peritoneal cavity by triggering local inflammation and i

146 ctomized controls after adoptive transfer of peritoneal cavity cells from B6 GalT(-/-) mice.

147 Cytofluorimetry of splenocytes and peritoneal cavity cells of MDA-LDL- or heat shock protei

148 rticles, which distribute more evenly in the peritoneal cavity compared to the large microparticles,

149 ited invasion of inflammatory cells into the peritoneal cavity compared with mice deficient in CD40 a

150 eased accumulation of neutrophils within the peritoneal cavity compared with wild-type control mice,

151 phaMbeta2 suppressed Mvarphi egress from the peritoneal cavity, decreased the production of anti-infl

152 igen receptor stimulation that is blunted in peritoneal cavity-derived B1 B cells.

153 8(+) splenocytes from dnTGF-betaRII mice and peritoneal cavity-derived, but not spleen-derived, CD19(

154 ts with PCI, the presence of free air in the peritoneal cavity does not confirm an intestinal perfora

155 the accumulation of lipid-rich lymph in the peritoneal cavity due to disruption of the lymphatic sys

156 The efficacy of irrigating the peritoneal cavity during appendectomy for perforated app

157 hermore, addition of exogenous PGE2 into the peritoneal cavity during infection overrode the protecti

158 t natural killer (NK) cells migrate into the peritoneal cavity during intraabdominal sepsis, but the

159 ion of hepatocytes with microcarriers in the peritoneal cavity efficiently rescued animals with liver

160 -reduction by gaining direct access into the peritoneal cavity, enabling elevated drug levels versus

161 ere transplanted into the liver, kidney, and peritoneal cavity (encapsulated or nonencapsulated) of r

162 and antigen presentation occurred within the peritoneal cavity, even in the presence of highly activa

163 in both normal and V(H)11t mouse spleen and peritoneal cavity express the highest levels of CXCR5, w

164 Similarly, iNKT cells from the human peritoneal cavity expressed lower transcription factor l

165 In the peritoneal cavity, FcRH3 was found on B1 cells, and not

166 fect in inflammatory cell recruitment to the peritoneal cavity following B. pahangi infection.

167 provide effective drug concentrations in the peritoneal cavity for an extended period of time.

168 nesis and proinflammatory environment in the peritoneal cavity for the establishment and maintenance

169 When injected into the peritoneal cavity, free and liposomal alendronic acid we

170 ice with inflammation of the airways, or the peritoneal cavity had poor priming ability resulting in

171 ollectively, these data demonstrate that the peritoneal cavity has a unique environment capable of el

172 numerically small B10 cell subset within the peritoneal cavity has regulatory function and is importa

173 s in large microcapsules transplanted in the peritoneal cavity have failed to reverse diabetes in hum

174 owed decreased macrophage migration into the peritoneal cavity; however, MP inhibited this effect.

175 multiple tissues, including the pleural and peritoneal cavities in mice.

176 distributed solid tumors and ascites in the peritoneal cavity in 100% of animals.

177 , Ly6C(+) monocytes constitutively enter the peritoneal cavity in a CCR2-dependent manner, where they

178 promote the migration of neutrophils to the peritoneal cavity in a CXCR2-dependent manner.

179 eased the levels of several cytokines in the peritoneal cavity in Cot/tpl2 KO mice compared with Wt c

180 howed a diminished ability to migrate to the peritoneal cavity in cox-2(-/-) mice.

181 ion of elicited monocytes/macrophages in the peritoneal cavity in response to inflammatory thioglycol

182 nocytes were preferentially recruited to the peritoneal cavity in response to pristane.

183 gammadelta T cells accumulated in the peritoneal cavity in response to tumor challenge and cou

184 ginal zone and primary follicles, and in the peritoneal cavity in the mutant animals, as were T-indep

185 rcentage of Dectin-1-expressing cells in the peritoneal cavity increased by 774% with cecal ligation

186 W/W(v) mice had neutrophil migration to the peritoneal cavity, increased CXCR2 expression, and reduc

187 ired for survival and abscess formation in a peritoneal cavity infection model.

188 lation of the footpad, or inoculation of the peritoneal cavity (intraperitoneal [i.p.] inoculation).

189 eatment of malignant disease confined to the peritoneal cavity is based on the theoretical potential

190 retention of surgical foreign bodies in the peritoneal cavity is estimated to occur once in every 10

191 Tumor cell metastasis to the peritoneal cavity is observed in patients with tumors of

192 The peritoneal cavity is recognized as an important site for

193 though colonization of Peyer patches and the peritoneal cavity is significantly impaired.

194 When injected into the mouse peritoneal cavity, lactoferrin also caused a marked recr

195 t/detect and kill metastasized tumors in the peritoneal cavity, leading to significant suppression of

196 flammation in the mouse cremaster muscle and peritoneal cavity led to ICAM-1 expression on intravascu

197 The simulated drug release in the peritoneal cavity linearly correlated with treatment eff

198 These findings demonstrate that peritoneal cavity Mac-1(+) B-1 cells are precursors of M

199 In contrast, peritoneal cavity macrophages basally express NAIP1 and

200 These findings suggest that spread to the peritoneal cavity may require no or very little further

201 m (n = 4), portal venous system (n = 1), and peritoneal cavity (n = 1).

202 mosan-induced neutrophils were isolated from peritoneal cavities of betaarr2-deficient (betaarr2(-/-)

203 this subset, macrophages were collected from peritoneal cavities of mice injected with saline, thiogl

204 on macrophage foam cell formation within the peritoneal cavities of mice.

205 ng freshly isolated human PEL cells into the peritoneal cavities of NOD/SCID mice without in vitro ce

206 L cells were detected in both the spleen and peritoneal cavity of animals for up to 75 days.

207 s persisted in the intestinal tissue and the peritoneal cavity of drug-cured mice for weeks.

208 , respectively, n = 9) was injected into the peritoneal cavity of female Lewis rats 30 min before int

209 when the cell lines were implanted into the peritoneal cavity of female nude mice, every line secret

210 lation was found in spleen, bone marrow, and peritoneal cavity of humanized mice and included distinc

211 e bone marrow, spleen, peripheral blood, and peritoneal cavity of JAK2V617F transgenic mice than from

212 lasmacytoid DCs, T cells, and B cells in the peritoneal cavity of mertk-/- mice when compared with wi

213 6(-)PD-L2(-)MHCII(-)UCP1(+) phenotype in the peritoneal cavity of mice and during the formation of li

214 directly to s.c. xenograft tumors and to the peritoneal cavity of mice bearing primary and metastatic

215 capable of imaging hydrogen peroxide in the peritoneal cavity of mice during a lipopolysaccharide-in

216 ure from a flow stream in vitro and from the peritoneal cavity of mice in vivo.

217 The abundance of B-1a cells found in the peritoneal cavity of mice is under genetic control.

218 nclude fabrication and implantation into the peritoneal cavity of mice, incubation, retrieval via per

219 yi nematodes are implanted surgically in the peritoneal cavity of mice, we identified differential ex

220 measure the amount of ovarian tumors in the peritoneal cavity of mice.

221 ith these tachyzoites once inoculated in the peritoneal cavity of mice.

222 oliferation was drastically decreased in the peritoneal cavity of Nbs1(B/B) mice.

223 rowing subcutaneously or multifocally in the peritoneal cavity of nonobese diabetic/severe combined i

224 capsulated canine pancreatic islets into the peritoneal cavity of pancreatectomized canines.

225 unting long-term recall responses enrich the peritoneal cavity of PD patients.

226 Before reperfusion, the peritoneal cavity of recipients was irrigated with delta

227 The IL-6-rich peritoneal cavity of SHIP(-/-) mice shows more than 700-

228 ters were subsequently transplanted into the peritoneal cavity of streptozotocin-induced diabetic sev

229 CD8+ T cells in the peritoneal cavity of TCF-1-deficient mice had decreased

230 trophils compared with WT neutrophils in the peritoneal cavity of TG-exposed WT mice.

231 attenuated recruitment of neutrophils in the peritoneal cavity of TRPC6(-/-) mice.

232 Fewer DCs also accumulated in the peritoneal cavity of UV-chimeric mice (ie, mice transpla

233 both in a Transwell chamber assay and in the peritoneal cavity of wild-type (WT) mice.

234 l as the recruitment of neutrophils into the peritoneal cavity of wild-type mice, but not RICK-defici

235 Within the peritoneal cavity, omental tissue is a common site for m

236 effect, myeloid cells were isolated from the peritoneal cavity or bone marrow.

237 ad extravasation of contrast medium into the peritoneal cavity or the presacral space on a postoperat

238 nd in rats with islets transplanted into the peritoneal cavity or under the kidney capsule.

239 clusters into the mesothelial cell lining of peritoneal cavity organs; however, the tumor-specific fa

240 There is no advantage to irrigation of the peritoneal cavity over suction alone during laparoscopic

241 ansplanted into the hepatic sinus (H group), peritoneal cavity (P group), omentum (O group), and kidn

242 d that the presence of a foreign body in the peritoneal cavity (PC) might alter the inflammatory resp

243 CDR-3 of the Ig H chain (CDR-H3) content in peritoneal cavity (PerC) B cells, we analyzed the compos

244 Peritoneal cavity (PerC) B-1 cells have long been known

245 The peritoneal cavity (PerC) is a unique compartment within

246 In vivo, the introduction of sHz into the peritoneal cavity produces an inflammatory response char

247 vehicle for cancer cell dissemination in the peritoneal cavity, protecting cells from environmental s

248 Fibrosis of the peritoneal cavity remains a serious, life-threatening pr

249 ion of memory CD8 T cells to lymph nodes and peritoneal cavity required G(i)-coupled receptor signali

250 hese Ab-secreting cells (ASC) originate from peritoneal cavity-resident cells, because transfer of pe

251 (MZ) B cells and B1 B cells enriched in the peritoneal cavity respond preferentially to T cell-indep

252 l-free viral transmission of HHV-6A into the peritoneal cavity resulted in detectable viral DNA in at

253 oss of vascular fluid into the intestine and peritoneal cavity, resulting in rapid (less than 30 min)

254 responses at the site of initial infection (peritoneal cavity) revealed that CLP TRPV1KO mice exhibi

255 ently clear Klebsiella pneumoniae from their peritoneal cavities reveals an essential role for this s

256 II, CD80, and CD86) become detectable in the peritoneal cavity, skin, lung, and lymph nodes under inf

257 y imaged in most mouse organs, including the peritoneal cavity, stomach, small intestine, and colon.

258 al and regional malignancies confined in the peritoneal cavity such as ovarian cancer.

259 ized to nearby and distant organs within the peritoneal cavity, such as abdominal lymph nodes, mesent

260 on of cells and cellular aggregates into the peritoneal cavity, survival of matrix-detached cells in

261 hours after infection, and are higher in the peritoneal cavity than in plasma.

262 ontrast to recent studies in the pleural and peritoneal cavities, the proliferation of resident and a

263 ributed to the retention of B-1 cells in the peritoneal cavity through high expression of the chemoki

264 itive cells were preferentially noted in the peritoneal cavity tissue site; spleen cells displayed a

265 d redistribution of radioactivity out of the peritoneal cavity to circulating blood, which cleared vi

266 and monocytes are rapidly recruited into the peritoneal cavity to control infection, but the role of

267 migration of cox-2(-/-) macrophages from the peritoneal cavity to lymph nodes, as well as cell adhesi

268 fluids continuously circulating through the peritoneal cavity to rapidly induce and maintain hypothe

269 pairs dendritic cell (DC) migration from the peritoneal cavity to the draining lymph node in mice imm

270 that microbes disseminated rapidly from the peritoneal cavity to the lung and spleen, where they rep

271 that CD11c(+)Gr-1(+) cells migrate from the peritoneal cavity to the spleen.

272 Following release into the peritoneal cavity, tumor cells rapidly attach to the ome

273 Leukocyte recruitment into the peritoneal cavity upon induction of inflammation was not

274 induce the recruitment of neutrophils in the peritoneal cavity via CXCR2.

275 tingly, the total number of Th2 cells in the peritoneal cavity was comparable to those found in the d

276 The detection of tumors in the peritoneal cavity was exclusively possible with sequenti

277 Access to the peritoneal cavity was gained using a modified blunt dila

278 107 colony-forming units); 24 hrs later, the peritoneal cavity was lavaged and the major organs were

279 ment or localization of B10 cells within the peritoneal cavity was not dependent on the presence of c

280 oglycollate, neutrophil recruitment into the peritoneal cavity was reduced, and the delayed macrophag

281 utrophil and monocyte extravasation into the peritoneal cavity was severely reduced in St3Gal4(-/-) m

282 ent neutrophil recruitment into the inflamed peritoneal cavity was sharply suppressed in Tyrobp(-/-)

283 o recruitment of these cells to the inflamed peritoneal cavity was significantly enhanced.

284 However, using the peritoneal cavity, we found that although innate immune-

285 rophages in the spleen, lymph nodes, and the peritoneal cavity were reduced.

286 leen B cells adoptively transferred into the peritoneal cavity were similarly resistant to mAb-induce

287 ells from blood and spleen into the inflamed peritoneal cavity where they appear to facilitate the ac

288 m circulating monocytes and recruited to the peritoneal cavity where they differentiate into macropha

289 s the main site of neutrophil entry into the peritoneal cavity, where MMP-2 and MMP-9 act synergistic

290 t arise in spleen but rapidly migrate to the peritoneal cavity, where they persist indefinitely but d

291 rmed metastasis in mice when injected in the peritoneal cavity, whereas aggressive ovarian cancer cel

292 e content of the resident macrophages in the peritoneal cavity, whereas it reduced the content of the

293 RF2 normally populated the marginal zone and peritoneal cavity, whereas mice using inverted RF1 had i

294 CCR3 and CCR6, but not CXCR3, to home to the peritoneal cavity, whereas Th9 homing to the CNS during

295 ortion of the autoislets placed within their peritoneal cavities, which appeared to be functioning no

296 viral vectors are essentially limited to the peritoneal cavity, which has advantages for safety and d

297 red for Ly6C(hi) monocyte trafficking to the peritoneal cavity, which is thought to be one of the ini

298 cancer cells disseminate readily within the peritoneal cavity, which promotes metastasis, and are of

299 multicellular aggregates or spheroids in the peritoneal cavity, which seed abdominal surfaces and org

300 on of SpA, showed neutrophil influx into the peritoneal cavity with peak numbers appearing at 8 h.