ライフサイエンスコーパス: peritoneum

1 g vs 35.62 mg/kg; P = 0.0003 in the parietal peritoneum).

2 spleen, lymph node, bone marrow, thymus, and peritoneum).

3 . asthma), were particularly elevated in the peritoneum.

4 tiple leiomyomas in the abdominal and pelvic peritoneum.

5 re is effective in the visceral and parietal peritoneum.

6 ype in dialysis membrane chambers in the rat peritoneum.

7 es GRP-induced neutrophil recruitment to the peritoneum.

8 bone marrow and then homed to the spleen and peritoneum.

9 helial cells and probably other cells of the peritoneum.

10 ature splenic NK cells are able to reach the peritoneum.

11 a B1 and follicular B cells persisted in the peritoneum.

12 tion in pro-MMP-9 activation in the inflamed peritoneum.

13 ymorphonuclear leukocytes instilled into the peritoneum.

14 nd chemokine concentrations in the blood and peritoneum.

15 clearance were enhanced within the infected peritoneum.

16 ency results in increased cellularity in the peritoneum.

17 pithelium than ovarian surface epithelium or peritoneum.

18 ogens in many organs, including the lung and peritoneum.

19 microbial translocation from the gut to the peritoneum.

20 eria but not inflammatory cells in lungs and peritoneum.

21 immune responses locally at the level of the peritoneum.

22 V low-grade serous carcinoma of the ovary or peritoneum.

23 ness human peritoneum and, in vivo, to mouse peritoneum.

24 pecific recall eosinophil recruitment to the peritoneum.

25 NK cells and killer dendritic cells into the peritoneum.

26 numerous metastases to lymph node, lung, and peritoneum.

27 sms that up-regulate fibrinolysis within the peritoneum.

28 ectable levels observed on B1 B cells in the peritoneum.

29 t no impairment in neutrophil recruitment to peritoneum.

30 as did neutrophils infiltrating the inflamed peritoneum.

31 een and increased B1a and B1b B cells in the peritoneum.

32 from the bone marrow and accumulation in the peritoneum.

33 apsulated strain, was avirulent in the mouse peritoneum.

34 duced adhesion formation after injury to the peritoneum.

35 earance of apoptotic cells from the inflamed peritoneum.

36 nd no increase of B1a cells in the spleen or peritoneum.

37 2-transfected fibroblasts deposited into the peritoneum.

38 e a potential mechanism for SP action in the peritoneum.

39 rs of the breast, ovary, fallopian tube, and peritoneum.

40 d in neutrophils of the peripheral blood and peritoneum.

41 penetration indices into meninges, bone, and peritoneum.

42 tment and enhancing CXCL10 expression in the peritoneum.

43 colonization and TD Ags translocated to the peritoneum.

44 the low levels of phosphate observed in the peritoneum.

45 ent to make contact with the entire anatomic peritoneum.

46 nd are found in the spleen, lymph nodes, and peritoneum.

47 induced by thioglycolate injection into the peritoneum.

48 cer, metastasis is typically confined to the peritoneum.

49 rrest and were subsequently cleared from the peritoneum.

50 nhanced recovery of viable bacteria from the peritoneum.

51 dent models of inflammation in air pouch and peritoneum.

52 aline) was injected between the mesh and the peritoneum.

53 ) characterized by extensive fibrosis of the peritoneum.

54 ns) outside the uterus, most commonly on the peritoneum.

55 cifically recruited from the spleen into the peritoneum.

56 response to thioglycollate injections in the peritoneum.

57 ytokine and chemokine levels in the infected peritoneum.

58 uction, and triggering their exodus from the peritoneum.

59 n to normal mesothelial cells which line the peritoneum.

60 e serosal surface of the small bowel and the peritoneum.

61 ascular in the vicinity of the lungs and the peritoneum.

62 igh-grade serous carcinomas of the ovary and peritoneum.

63 essels and VEGF-A-positive cells in fibrotic peritoneum.

64 e-switched B cells were only detected in the peritoneum.

65 PH B cells preferentially accumulated in the peritoneum.

66 ssure, carry metastatic cells throughout the peritoneum.

67 t low-grade serous carcinoma of the ovary or peritoneum.

68 phil recruitment into inflamed cremaster and peritoneum.

69 ence of local cytokine production within the peritoneum.

70 recovery of highly fluorescent GAS from the peritoneum 1 h after challenge.

71 luded determination of malignancy within the peritoneum (11 sites), lymph nodes (10 sites), and hepat

72 an-elicited neutrophil infiltration into the peritoneum 25-50% and shortened the resolution interval

73 ocyte infiltration and bacterial load in the peritoneum after CLP but dramatically increased the neut

74 Endogenous MDC was detected in the peritoneum after CLP, and neutralization of the MDC with

75 production and neutrophil recruitment to the peritoneum after intraperitoneal administration.

76 these DBP(-/-) mice to recruit cells to the peritoneum after localized thioglycolate injection.

77 We measured KC and MIP-2 in the peritoneum after thioglycollate injection and demonstrat

78 h an increased filtration coefficient of the peritoneum and a decreased reflection coefficient to pro

79 ed significant neutrophil migration into the peritoneum and a significant increase in the levels of C

80 rovides 12-24 h of analgesia to the parietal peritoneum and abdominal wall, and are best used combine

81 crophage and microglial recruitment into the peritoneum and brain, respectively, than in wild-type mi

82 cytokines and higher parasite burden in the peritoneum and brain.

83 urthermore, ATF3-null mice lacked MCs in the peritoneum and dermis, showing that the in vitro results

84 Blood monocytes then enter the inflamed peritoneum and develop into new peritoneal macrophages.

85 n 100% of the animals when injected into the peritoneum and developed vascularized tumors in 85% of t

86 ility of the Deltaspx strain to colonize the peritoneum and disseminate in the bloodstream was signif

87 , these Vgamma4+ T cells are retained in the peritoneum and draining mediastinal lymph nodes for a pr

88 are induced during in vivo growth in the rat peritoneum and during in vitro growth in sputum (mucus)

89 ned evidence that macrophages present in the peritoneum and in menses endometrium can contribute to t

90 MCAs) present in ascites fluid adhere to the peritoneum and induce retraction of the peritoneal mesot

91 1 induces fibrosis and angiogenesis in mouse peritoneum and is associated with increased solute trans

92 tor, in F4/80(hi) macrophages in the spleen, peritoneum and kidney.

93 ous HSCs/HPCs were actively recruited to the peritoneum and liver, respectively, in WT but not Ccr2-/

94 is associated with acute fascioliasis in the peritoneum and liver.

95 in vivo neutrophil recruitment to the mouse peritoneum and lung.

96 acrophage-rich portals of entry, such as the peritoneum and lung.

97 Preferential metastatic sites were lungs, peritoneum and lymph nodes.

98 ese SPM-like cells are not restricted to the peritoneum and may help clear apoptotic cells from tissu

99 acrine manner to promote colonization of the peritoneum and neovascularization of developing tumor de

100 ved in solute and water transport across the peritoneum and of the pathobiology of structural and fun

101 Attachment of OvCa cells to peritoneum and omentum represents the first rate-limitin

102 omentum or peritoneum, and in vivo to mouse peritoneum and omentum.

103 sed the recovery of viable bacteria from the peritoneum and peripheral blood.

104 creased recovery of viable bacteria from the peritoneum and peripheral blood.

105 her concentrations of CXCL1 and CXCL2 in the peritoneum and plasma compared with those predicted to l

106 induced 3- and 4-fold more TNF-alpha in the peritoneum and serum, respectively, of db/db mice as com

107 infiltration ( approximately 44%, 6 h) into peritoneum and shortened resolution interval from 4 to 2

108 equencies of latently infected cells in both peritoneum and spleen by 28 days postinfection.

109 spleen to their long-term residence in adult peritoneum and spleen.

110 ection in mice, g-EAR showed gelation in the peritoneum and sustained, local-regional release of EpoB

111 o increased inflammatory infiltrate into the peritoneum and target organs.

112 y made contact with only 40% of the anatomic peritoneum and that this contact area (A(contact)) could

113 an be activated by antigens drained from the peritoneum and the gastrointestinal tract.

114 from the urinary tract, and 1 each from the peritoneum and the thorax.

115 e parasite migrated from the gut, across the peritoneum and through the liver to mature in the bile d

116 xed to parietal and visceral surfaces of the peritoneum and were filled with either an isotonic or hy

117 mesothelial cells, and full-thickness human peritoneum and, in vivo, to mouse peritoneum.

118 ne levels both within the local environment (peritoneum) and systemically (plasma), which in turn cor

119 g vs 5.48 mg/kg; P = 0.00001 in the visceral peritoneum, and 66.16 mg/kg vs 35.62 mg/kg; P = 0.0003 i

120 in a 25-75% reduction of cells invading the peritoneum, and a 7-fold increase in LXA(4) identified b

121 lial cells lining the lungs, pericardium and peritoneum, and are often associated with occupational a

122 with primary macrophages derived from lung, peritoneum, and blood but not spleen.

123 responses in the spleen, lungs, bone marrow, peritoneum, and blood using a mouse model of endotoxemia

124 el of metastatic OvCa, full human omentum or peritoneum, and in vivo to mouse peritoneum and omentum.

125 ominal and involved the original tumor site, peritoneum, and liver.

126 ulation, neutrophil extravasation into lung, peritoneum, and skin wounds was reduced in Tph1(-/-) mic

127 infection, mononuclear cells from the liver, peritoneum, and spleen were isolated.

128 on in specific microenvironments such as the peritoneum, and we would propose other privileged sites,

129 lial cancer of the ovary, fallopian tube, or peritoneum (any stage, grade 2 to 3 if stage I) and meas

130 expression and possibly localization to the peritoneum appear to provide some protection to autoreac

131 nally in some mesoderm cells of the visceral peritoneum arranged in an approximately midventral row r

132 We observed NK migration into the infected peritoneum as early as 6 h after vaccinia inoculation.

133 mulation in the interstitium (edema) and the peritoneum (ascites) of nephrotic patients is classicall

134 ncer metastasized to lymph nodes, liver, and peritoneum at a significantly higher rate compared with

135 ntable temperature probe was placed into the peritoneum before asphyxia.

136 activation-involvement of the liver, spleen, peritoneum, bones, and marrow-are frequent.

137 Compartments such as the bladder, peritoneum, brain, eye and skin are often sites of disea

138 poradic EOC commonly remains confined to the peritoneum, BRCA1/2-deficient ovarian cancer frequently

139 t between wild-type MIP-2 and mutants in the peritoneum, but all activity of the mutants is lost in t

140 C chemokine ligand 12 injection sites in the peritoneum, but not to CXC chemokine ligand 12.

141 ing cells showed increased adhesion to mouse peritoneum, but the overall number of metastatic nodules

142 11beta-HSD activity in cells elicited in the peritoneum by a single thioglycolate injection in mice.

143 inhibited recruitment of leukocytes into the peritoneum by specifically activating the A(3)AR.

144 whether host responses originating from the peritoneum can influence PspC expressed by pneumococci i

145 suggest that statins administered within the peritoneum can up-regulate local fibrinolysis, while the

146 e used in humans, injection of NBPs into the peritoneum caused recruitment of neutrophils, followed b

147 ragilis and colonic contents escape into the peritoneum, causing abscesses and bacteremia.

148 ease in neutrophil and monocyte entry to the peritoneum compared to scramble mice.

149 y more neutrophils and fewer bacteria in the peritoneum compared with C57BL/6 mice 24 h after CLP.

150 10) concentrations increase in the blood and peritoneum concordant with the peritoneal recruitment of

151 n their ability to migrate into the inflamed peritoneum, confirming that CD13 phosphorylation is rele

152 macrophages and mesothelial cells lining the peritoneum contain MCP-1, which is released following th

153 cell subsets within lymphoid tissues and the peritoneum could be depleted efficiently in vivo through

154 gly, although NK trafficking to the infected peritoneum depended on G alpha(i) protein-coupled recept

155 Cs), the main source of IL-6 and VEGF in the peritoneum, do not bear the cognate IL-6 receptor and ar

156 giant cells are formed in the inflamed mouse peritoneum during the resolution phase of inflammation,

157 ompared with long-term PD control and uremic peritoneum, EPS peritoneum showed thicker submesothelial

158 ells residing in the bone marrow, blood, and peritoneum, even though cells from the last site had hig

159 -resistant papillary serous carcinoma of the peritoneum experienced a partial response lasting 22 mon

160 Within the peritoneum, F. hepatica antibodies coincided with an int

161 ed to both lymphoid and non-lymphoid organs (peritoneum, fat pads, and lung).

162 data collection process or had done a formal peritoneum-focused review of individual pre-treatment sc

163 110gamma(-/-)) CD8 effector T cells into the peritoneum following i.p. challenge with VV.

164 hibited impaired migration into the inflamed peritoneum following secondary transfer into wild-type r

165 recruitment at a primary site of infection (peritoneum) following lethal murine ehrlichial infection

166 aintained the polymeric nanoparticles in the peritoneum for 1 week, which we had previously shown wou

167 ID1 was increased in peritoneum from women with endometriosis and TGF-beta1 i

168 ple anatomic sites, including the spleen and peritoneum; however, the contribution of distinct cell t

169 Metastases were found in the peritoneum in 70 (59%), nodes in 20 (17%), and liver in

170 infusion reduced leukocyte migration to the peritoneum in a murine model of thioglycolate-induced pe

171 caused by deposition of AIM at the necrotic peritoneum in AIM (+/+) mice.

172 toneal dialysis are dependent on the area of peritoneum in contact with the dialysis solution, rats w

173 s a result, neutrophils entered the inflamed peritoneum in greater numbers or for a longer duration.

174 id not affect invasion of PDAC into bowel or peritoneum in mice.

175 okines in vitro and increased migration from peritoneum in response to lipopolysaccharide in vivo.

176 Neutrophilic influx into the peritoneum in response to necrotic cells in vivo was als

177 lay no acceleration of infiltration into the peritoneum in spite of elevated L-selectin surface level

178 display the linear dimension of the anatomic peritoneum in the sampling plane.

179 2, is enough to keep the cells viable in the peritoneum in vivo and under normal culture conditions.

180 nies by colorectal cancer cells in the mouse peritoneum in vivo.

181 isplay a profound defect in migration to the peritoneum in vivo.

182 nds in vitro, and to an aseptically inflamed peritoneum in vivo.

183 s, particularly eosinophils, to the inflamed peritoneum in vivo.

184 nstrate increased numbers of B1 cells in the peritoneum, increased serum IgM, IgG, and IgG 2a and sho

185 enovirus to overexpress gremlin in the mouse peritoneum, induced submesothelial thickening, fibrosis,

186 We also found that the peritoneum induces different behavior in mature and imma

187 cavity, usually in the sub-hepatic or retro-peritoneum inferior to the sub-hepatic space.

188 ead of exfoliated tumor cells throughout the peritoneum, initially via the peritoneal fluid, and late

189 eferential recruitment of Th1 cells into the peritoneum is also seen when cytokine response gene 2 (C

190 ltered immune cell composition of the female peritoneum is controlled by elevated tissue chemokine ex

191 eosinophil recruitment to both the lung and peritoneum is impaired by the lack of CD28, suggesting a

192 s concluded that less than half of the mouse peritoneum is in contact with a large volume of solution

193 These data suggest that the peritoneum is not only home to a new subset of tissue-re

194 Given that adhesion to the abdominal peritoneum is the first step in ovarian cancer dissemina

195 eptic abscesses to the lungs, joints, liver, peritoneum, kidneys, and brain.

196 ade serous carcinoma of the ovary (LGSOC) or peritoneum (LGSPC) is a rare subtype of ovarian or perit

197 n WT and TFE3(-/-) mice were observed in the peritoneum, lung, and skin.

198 tly in subsets of macrophages located in the peritoneum, marginal zone of the spleen, and the medulla

199 ls are rapidly released and recruited to the peritoneum membrane lumen vasculature where they reside

200 or and was particularly advantageous for the peritoneum, mesentery, and bowel.

201 tumor invades locally into dermal layers and peritoneum, metastasizes to the lung, and induces a nons

202 In the peritoneum, MR imaging and CT (A(z) = 0.96 for both) wer

203 s of cancer of the ovary, fallopian tube, or peritoneum (N = 404).

204 tion tubing in the subcutaneous layer (n=3), peritoneum (n=3), aorta (n=3), or carotid artery (n=3).

205 cogen-induced neutrophil emigration into the peritoneum, neutrophil emigration across either the pulm

206 lysis membrane chambers implanted within the peritoneum of a rat.

207 these peptides were co-administered into the peritoneum of baboons with endometriosis, cells in lesio

208 09, decreased neutrophil accumulation in the peritoneum of db/db mice, as well as the accumulation of

209 ed the islets and increased in number at the peritoneum of diabetic but not prediabetic mice.

210 ment blocked neutrophil recruitment into the peritoneum of E-selectin(-/-) mice, but had only a parti

211 trual endometrial tissue introduced into the peritoneum of immunocompetent mice.

212 he heritable disease cystic fibrosis and the peritoneum of individuals undergoing continuous ambulato

213 ly more PspC than did D39 collected from the peritoneum of intraperitoneally (i.p.)-infected mice.

214 vely transferred cells that entered into the peritoneum of mertk-/- mice.

215 ioglycollate, neutrophils recruited into the peritoneum of mice were shown to bind more plasminogen t

216 icle-induced pro-inflammatory cascade in the peritoneum of mice.

217 and spleen compared with the bone marrow and peritoneum of normal mice as well as younger mice with l

218 bridoma cells were then transferred into the peritoneum of nude mice to study chronic thyroid stimula

219 resident population of memory T cells in the peritoneum of PD patients and speculate that these cells

220 The peritoneum of puromycin aminonucleoside rats displayed a

221 sion formation was surgically induced in the peritoneum of rats receiving daily doses of the NK-1R an

222 tumor cells were injected directly into the peritoneum of severe combined immunodeficient mice, and

223 aine (according to weight) atomized onto the peritoneum of the right iliac fossa and pelvis, or 20 mL

224 rable neutrophil migration into the inflamed peritoneum of transgenic and wild-type (WT) mice.

225 and FcepsilonRI double-positive cells in the peritoneum of wild-type (WT) and TFE3 knockout (TFE3(-/-

226 xhibit reduced neutrophil recruitment to the peritoneum on induction of sterile peritonitis and also

227 u expression of endostatin either inside the peritoneum or by the ovarian tumor cells inhibited perit

228 lines as compared to benign tissues (pleura, peritoneum or cysts).

229 a was increased in lesions compared with the peritoneum or eutopic endometrium.

230 nonuclear phagocytes derived from either the peritoneum or from bone marrow in vitro into syngeneic r

231 uced leukocyte transendothelial migration to peritoneum or lungs was significantly lower in Eng(+/-)

232 not observed following inoculation into the peritoneum or lymph nodes, or after oral ingestion.

233 mice had the phenotype of B-1 B cells in the peritoneum or marginal zone B cells in the spleen, where

234 s and attach preferentially on the abdominal peritoneum or omentum, where the cancer cells revert to

235 Thus, the injection of pMCKhLPL into the peritoneum or quadriceps muscle results in plasma trigly

236 her, pluripotent Mullerian stem cells in the peritoneum or subjacent mesenchyme undergo glial metapla

237 adrenal glands, the leukocyte recruitment to peritoneum or the bacterial clearance in liver.

238 re) recruitment of inflammatory cells to the peritoneum, or improve phagocytic cell killing of pathog

239 he spleen, type 2 cytokine production in the peritoneum, or mucus hypersecretion in the gastrointesti

240 , suggesting that monocytes recruited to the peritoneum originate in the spleen.

241 that metastasized to the liver, lung, skin, peritoneum, ovary, and lymph nodes were established.

242 f the drug in both the visceral and parietal peritoneum (P = 0.0058 and P = 0.0044, respectively).

243 3 years) with laparoscopic M1 disease in the peritoneum (P1, adjacent to stomach, 9%; P2, few distant

244 We addressed this hypothesis by studying the peritoneum permeability of rats with puromycin aminonucl

245 ants in GP often arise from cells within the peritoneum, presumably pluripotent Mullerian stem cells,

246 ian tube cancers or primary carcinoma of the peritoneum; prior initial therapy with platinum/paclitax

247 The presence of antigen in the peritoneum promotes local proliferation of recruited T c

248 Papillary serous carcinoma of the peritoneum (PSCP) is believed to develop de novo from th

249 man GW39 colon cancer cells into the hamster peritoneum represents a model of tumor spillage that may

250 y osmotic pump (LPS pump) implanted into the peritoneum resulted in sustained, widespread NF-kappaB a

251 ng the Rluc were imaged while located in the peritoneum, s.c. layer, as well as in the liver and lung

252 g-term PD control and uremic peritoneum, EPS peritoneum showed thicker submesothelial fibrosis, with

253 CCR4(-/-) cells home normally to the peritoneum, showing that they do not have a general defe

254 ng local anesthetic between the mesh and the peritoneum significantly reduces postoperative pain and

255 sessed the E. chaffeensis clearance from the peritoneum, spleen, and liver by C57BL/6J mice using a T

256 senger RNA in macrophages recovered from the peritoneum, spleen, liver and lung, and lowered serum Tn

257 HSCs/HPCs recovered from the peritoneum successfully engrafted into the BM of irradia

258 is at times when macrophages are exiting the peritoneum, suggesting that its proteolytic shedding may

259 stratified by tumors that perforate visceral peritoneum (T4a) versus tumors that invade or are adhere

260 imes more efficient in migrating to inflamed peritoneum than CD8(IL-15) cells.

261 -producing efferocytosing macrophages in the peritoneum that activate invariant natural killer T (iNK

262 When transferred directly into the peritoneum, the mature NK cells survive but do not divid

263 are not hyporesponsive; however, within the peritoneum, these B-1 cells are induced to express Lck a

264 0(4) cells in 0.5 ml) were injected into the peritoneum through a midline incision.

265 g fibrinolytic activity in the postoperative peritoneum, thus enabling fibrinous adhesions to persist

266 nd accelerates macrophage clearance from the peritoneum, thus promoting resolution of inflammation an

267 ed a phage display library with female mouse peritoneum tissue and selected phage clones by binding t

268 We find the peritoneum to be rich in glutamate, a glutaminolysis-fue

269 uring the transition of pneumococci from the peritoneum to the blood.

270 ivated beta1 integrin and relocated from the peritoneum to the inflamed skin and intestine upon innat

271 ormal mesothelial cells lining the pleura or peritoneum to the tumor-associated cancer antigen 125 (C

272 amma) and IP-10 production within the septic peritoneum together with local and systemic increases of

273 e faster in vitro, which, in the case of the peritoneum, translates to more rapid in vivo monocyte/ma

274 of Ly6C(hi) inflammatory monocytes into the peritoneum was abolished in type I interferon (IFN-I) re

275 timulated neutrophil transmigration into the peritoneum was also similar in both the heterozygous con

276 er of inflammatory monocytes in the inflamed peritoneum was associated with a lack of differentiation

277 h VWF-mediated neutrophil recruitment to the peritoneum was described to require the VWF receptor on

278 The absence of PMN in the peritoneum was due in part to lysis of intraperitoneal P

279 hesized that only a fraction of the anatomic peritoneum was in contact with the therapeutic solution

280 petitively inhibit tumor cell seeding of the peritoneum was investigated.

281 nd that macrophage recruitment into inflamed peritoneum was markedly reduced in the MCP-1-deficient a

282 late-induced neutrophil recruitment into the peritoneum was more severely impaired in P-Rex1(-/-) Vav

283 density of blood vessels per unit length of peritoneum was significantly higher for patients with me

284 Neutrophil trafficking in the peritoneum was similar in all strains after injection of

285 etriosis occurs on organ surfaces facing the peritoneum, we subtracted a phage display library with f

286 colate-induced macrophage recruitment to the peritoneum were indistinguishable between MPhi Sphk dKO

287 differences in monocyte recruitment into the peritoneum were observed after thioglycollate injection.

288 stimulate inflammation at the surface of the peritoneum when injected into the abdominal cavity of mi

289 intestine but instead form aggregates in the peritoneum where they produce immunoregulatory molecules

290 atment of large areas such as the pleura and peritoneum, where curative radiation doses cannot be tol

291 ions and agonist-induced neutrophilia in the peritoneum, whereas Galpha(i3) plays a less critical but

292 erthermia enhances diffusion in the visceral peritoneum, whereas high pressure is effective in the vi

293 reatment with neutrophil infiltration to the peritoneum, whereas other investigated cell types remain

294 chaffeensis was cleared in 2 weeks from the peritoneum, whereas the pathogen from tick cells persist

295 eloid-derived suppressor cells (MDSC) in the peritoneum, which expressed functional arginase 1, and p

296 of structural and functional changes in the peritoneum with long-term PD has provided new targets fo

297 the camera to virtually any location in the peritoneum with no working envelope restrictions and the

298 g a radiolabeled protein that adhered to the peritoneum with which it came in contact.

299 ve markedly decreased PMN infiltrates to the peritoneum with zymosan and altered the dynamics of this

300 IL-6 was also detected in the peritoneum within an hour of infection, prior to the app