ライフサイエンスコーパス: peritonitis

1 s after a polymicrobial sepsis (experimental peritonitis).

2 ing anastomotic leakage, pelvic abscess, and peritonitis.

3 the inflammatory site in an in vivo model of peritonitis.

4 il transudation during colitis, skin rash or peritonitis.

5 lammatory syndrome and urate crystal-induced peritonitis.

6 P = .01) for patients who did not have fecal peritonitis.

7 ith adrenal insufficiency from polymicrobial peritonitis.

8 t sites of inflammation in a murine model of peritonitis.

9 scites is a variant of spontaneous bacterial peritonitis.

10 l IL-1beta production following Alum-induced peritonitis.

11 gate the role of mast cells in severe septic peritonitis.

12 ects and tissue remodeling during PD-related peritonitis.

13 ammation and rescues mice from polymicrobial peritonitis.

14 ntamoeba histolytica as well as to bacterial peritonitis.

15 n in perforated diverticulitis with purulent peritonitis.

16 nfection, and possibly spontaneous bacterial peritonitis.

17 model of sodium periodate (NaIO(4))-induced peritonitis.

18 and sepsis that are symptomatic of bacterial peritonitis.

19 t lymphangiogenesis in patients with UFF and peritonitis.

20 eritoneal membranes of patients with UFF and peritonitis.

21 yte infiltration in an experimental model of peritonitis.

22 ory exudates during the development of acute peritonitis.

23 ge CD206 expression and efferocytosis during peritonitis.

24 ed anti-inflammatory phenotype in a model of peritonitis.

25 erwent cecal ligation and puncture to induce peritonitis.

26 nscription factor levels during neutrophilic peritonitis.

27 ocyte infiltration in thioglycollate-induced peritonitis.

28 88 in attenuating liver injury during septic peritonitis.

29 IL-1beta, IL-18 and HMGB1 in E. coli-induced peritonitis.

30 al macrophages, and (iii) clearing bacterial peritonitis.

31 row cell cultures, and in vivo in a model of peritonitis.

32 thicillin-resistant S. aureus (MRSA) induced peritonitis.

33 that reduces inflammation in zymosan-induced peritonitis.

34 cts blocked acute inflammation in a model of peritonitis.

35 MN179, isolated from an Iowa laying hen with peritonitis.

36 ment and complement activation during murine peritonitis.

37 beta and IL-18, and increased sensitivity to peritonitis.

38 n in a mouse model of Streptococcus pyogenes peritonitis.

39 tion is indicated to prevent perforation and peritonitis.

40 terations and transport defects during acute peritonitis.

41 ts were observed in a model of acute sterile peritonitis.

42 ity rate comparable to spontaneous bacterial peritonitis.

43 therapeutic strategies for PD-related fungal peritonitis.

44 l response exacerbates septic progression of peritonitis.

45 substantially reduce the risk of PD-related peritonitis.

46 cute perforated diverticulitis with purulent peritonitis.

47 parotomy for indication of acute generalized peritonitis.

48 soon after infection but not during sterile peritonitis.

49 itis (30% versus 49%) and had lower rates of peritonitis (0.30 versus 0.49 episodes per year, P=0.01)

50 ma (2.7%), and intra-abdominal abscess/other peritonitis (1.9%).

51 All patients with fecal peritonitis (15 patients in the laparoscopic peritoneal

52 Deep SSIs included abscesses (58%), peritonitis (28%), deep incisional infections (8%), and

53 33 (1.06-1.65)], abscess [2.18 (1.60-2.97)], peritonitis [3.14 (1.99-4.97)], sepsis [1.88 (1.29-2.73)

54 This group also had fewer patients develop peritonitis (30% versus 49%) and had lower rates of peri

55 proves survival during Klebsiella pneumoniae peritonitis (67 versus 23%, p < 0.005) in mice.

56 PF-562271 reduced monosodium urate-mediated peritonitis, a disease model used for studying the conse

57 patients with proven diagnosis of secondary peritonitis admitted to the ICU were included in the stu

58 In Ag-induced peritonitis (AIP), thioglycolate-induced peritonitis, an

59 up to 50, and impaired zymosan-induced mouse peritonitis along with reduced 5-LO product levels.

60 t were subjected to surgically induced fecal peritonitis and 22 pigs that were subjected to lipopolys

61 differentiating it from a teratoma, meconium peritonitis and abdominal ectopic pregnancy.

62 l responses but resulting in more persistent peritonitis and abscesses.

63 analysis in CD47(-/-) mice by inducing acute peritonitis and aggressive colitis observed consistent r

64 nt to the peritoneum on induction of sterile peritonitis and also reduced inflammation in a model for

65 ression and function in experimental sterile peritonitis and autoimmune arthritis, respectively.

66 s are highly susceptible to Escherichia coli peritonitis and bacteremia.

67 wild-type adults are susceptible to E. coli peritonitis and bacteremia.

68 reduced both C. albicans burden during early peritonitis and C. albicans persistence within abscesses

69 the dissemination of B. fragilis after acute peritonitis and characterized the interactions of the in

70 n in a mouse model of experimentally induced peritonitis and delayed-type hypersensitivity reaction.

71 tients on the day of presentation with acute peritonitis and discriminate between culture-negative, G

72 ality in patients with spontaneous bacterial peritonitis and improves outcome following large volume

73 duction of colitis or thioglycollate-induced peritonitis and in response to tumor necrosis factor-alp

74 recruitment following thioglycollate-induced peritonitis and in vitro chemotaxis were not affected by

75 xperimental models of thioglycollate-induced peritonitis and LPS-triggered lung injury.

76 sis patients presenting with acute bacterial peritonitis and monitoring individuals before and during

77 oneal dialysis, but the relationship between peritonitis and mortality among these patients is not we

78 inary biomarkers in an animal model of fecal peritonitis and recovery.

79 Using a self-resolving peritonitis and resolution indices coupled with lipid me

80 ted defects in solute transport during acute peritonitis and restored ultrafiltration.

81 Peritonitis and sepsis models were created using wild-ty

82 tic accuracy to discriminate the severity of peritonitis and to predict death in the ICU.

83 8), treated with enalapril for 1 week before peritonitis and until study end, received fluids and nor

84 reduced in apo(a)tg and Lp(a)tg mice in both peritonitis and vascular injury inflammatory models, and

85 Feline infectious peritonitis and virulent, systemic calicivirus infection

86 MerTK is linked to the resolution of sterile peritonitis and, after ischemia-reperfusion (I/R) injury

87 AP1189 elicited anti-inflammatory actions in peritonitis and, upon administration at the peak of infl

88 "sigmoidectomy"), AND "Diverticulitis", AND "Peritonitis" AND "therapeutic irrigation" or "lavage" AN

89 mice, and in both in vivo (monosodium urate peritonitis) and in vitro models of inflammation.

90 , long-term (3 d) rat model of sepsis (fecal peritonitis) and recovery was used to understand the tem

91 g nosocomial infections, such as septicemia, peritonitis, and endocarditis.

92 g, serious infections, spontaneous bacterial peritonitis, and hepatorenal syndrome (RR = 0.42, 95% CI

93 ty-acquired pneumonia, spontaneous bacterial peritonitis, and IgE-mediated food allergy.

94 ced peritonitis (AIP), thioglycolate-induced peritonitis, and LPS-induced lung inflammation, CSF-1 ne

95 the cremaster muscle, thioglycollate-induced peritonitis, and LPS-induced lung injury.

96 al recruitment during thioglycollate-induced peritonitis, and they roll slower and adhere more readil

97 ved in the repair process of zymosan-induced peritonitis, and thus, could be the basis of development

98 anscriptomic response to sepsis due to fecal peritonitis, and to compare these with the same paramete

99 In this study, we use IL-1beta-induced peritonitis as a model for an acute immune response, whi

100 Using zymosan-induced peritonitis as an in vivo PMN infiltration model, we sho

101 NA intraperitoneal injection exhibited acute peritonitis as evidenced by marked neutrophil and monocy

102 response signature (SRS) subgroups in fecal peritonitis associated with early mortality (P = 0.01; h

103 a2 whose levels are elevated in experimental peritonitis at times when macrophages are exiting the pe

104 e infiltration during thioglycollate-induced peritonitis between 3 and 24 h that returned to wild typ

105 In septic peritonitis, blood neutrophils and monocytes are rapidly

106 ), we show that, during polymicrobial septic peritonitis, both myeloid and nonmyeloid cells contribut

107 In murine peritonitis, both pre- and posttreatment with CO inhalat

108 ds extend up to 120 days after an episode of peritonitis but the magnitude is greater during the init

109 mice were also more susceptible to abdominal peritonitis, but this was associated with no significant

110 of acute peritoneal dialysis (PD)-associated peritonitis by multicolor flow cytometry and multiplex E

111 16.7% (9/54) for "nonclassical" indications (peritonitis, carcinomatosis, and so on) (P < 0.0001).

112 We report the first case of peritonitis caused by Roseomonas mucosa which led to tec

113 cardiac myocyte level, colon ascendens stent peritonitis cells showed reduced cell shortening, Ca tra

114 age over fibrinogen-deficient mice following peritonitis challenge.

115 thin 6 hours after infection, mice developed peritonitis, characterized by high yeast burdens, neutro

116 ASSO algorithm identified bowel gangrene and peritonitis, coagulopathy, age, the use of stoma, and ch

117 In a murine model of LPS-induced peritonitis, cold exposure potentiated hypothermia and d

118 6 month mortality from sepsis due to CAP or peritonitis combined, 28-day mortality from CAP sepsis,

119 nfection, and possibly spontaneous bacterial peritonitis, community-acquired pneumonia, and infection

120 onset of anuria and reduce the incidence of peritonitis compared with conventional fluid in peritone

121 ng neutrophil influx promoting resolution of peritonitis compared with Ntn1(+/+).

122 codextrin use (R = 0.52; 95% CI, 0.20-0.84), peritonitis count (R = 0.16; 95% CI, 0.03-0.29), and dia

123 th resolvin D1 (RvD1) enhanced resolution of peritonitis, decreased accumulation of apoptotic thymocy

124 inflammatory recipients followed by inducing peritonitis demonstrated that both circulating PMN and t

125 loid cells (IMCs) with thioglycolate-induced peritonitis, DSS-induced colitis, and H. felis-induced g

126 , there were significantly increased odds of peritonitis during the 120 days before death, although t

127 r times to anuria (P=0.009) and to the first peritonitis episode (P=0.01).

128 f BCC and were protective against lethal BCC peritonitis even during coinfection with methicillin-res

129 tored peritoneal function in mouse models of peritonitis, even in mice treated with standard-of-care

130 AIM (-/-) mice subjected to zymosan-induced peritonitis exhibited progressive inflammation and susta

131 Thioglycollate-induced peritonitis experiments with chimeric mice revealed that

132 formation is also detected in vivo in murine peritonitis exudates.

133 anulomatous disease called feline infectious peritonitis (FIP), which is one of the most important fa

134 andomized to receive after 12 hours of fecal peritonitis fluid resuscitation and either norepinephrin

135 evealed impaired monocyte chemotaxis towards peritonitis following high fat diet due to retention of

136 vivo role of YB-1 in murine models of acute peritonitis following LPS injection, in sterile renal in

137 mprised time to anuria, fluid volume status, peritonitis-free survival, technique survival, patient s

138 minated as the causative agent of laying hen peritonitis, Gallibacterium anatis are frequently isolat

139 Perforated diverticulitis with purulent peritonitis has traditionally been treated with open col

140 beta in the peritoneal membrane during acute peritonitis have not been investigated.

141 Colon ascendens stent peritonitis hearts showed a significant increase in oxid

142 Exclusion criteria included patients with peritonitis, hemodynamic instability, unreliable physica

143 Moreover, in animal models of peritonitis, hepatic ischemia-reperfusion injury, Salmon

144 Perforated diverticulitis with purulent peritonitis (Hinchey III) has traditionally been treated

145 During zymosan-induced peritonitis, hPR3Tg displayed an increased accumulation

146 leading to laparotomy was the development of peritonitis in 70%.

147 ild-type mice from established polymicrobial peritonitis in a clinically relevant time frame.

148 Fungal peritonitis in a patient on peritoneal dialysis (PD) is

149 reated case of catheter-related C. difficile peritonitis in a patient undergoing peritoneal dialysis.

150 genously produced during self-limited murine peritonitis in both the initiation and resolution phases

151 recruitment was defective upon induction of peritonitis in HD mice at early disease stages and was n

152 Herein, with the use of microbial-induced peritonitis in mice and liquid chromatography-tandem mas

153 Neutrophilic inflammation in experimental peritonitis in mice decreased iNKT cell T-box transcript

154 The increased peritonitis in mice deficient in IL-4, NKT cells, or IL-

155 Using Staphylococcus aureus peritonitis in mice, we found significant Sod2 gene indu

156 s reduces diabetic host resistance to septic peritonitis in mice.

157 s and for protective efficacy against lethal peritonitis in mice.

158 ned (75%) during the resolution of zymosan A peritonitis in mice.

159 Finally, models of peritonitis in p53(-/-) and pifithrin-alpha-treated mice

160 ammasome is activated during acute bacterial peritonitis in patients on PD, and this activation assoc

161 ell-characterized model of sterile resolving peritonitis in the mouse.

162 /liposome-induced interleukin-1beta-mediated peritonitis in vivo.

163 cites, encephalopathy, spontaneous bacterial peritonitis) increased with severity of AKI.

164 The Mannheim Peritonitis Index and Acute Physiology and Chronic Healt

165 A mouse model of peritonitis indicated that monocyte/macrophage recruitme

166 molol on myocardial and vascular function in peritonitis-induced septic rats and to explore the infla

167 de (LPS); underwent induction E. coli 018:K1 peritonitis induction, followed by treatment with AB103;

168 during endotoxemia and severe polymicrobial peritonitis, integrin VLA-3 (CD49c/CD29) is specifically

169 bleeding, female sex, spontaneous bacterial peritonitis, intermediate increases in serum creatinine

170 ere pulmonary embolism, pneumonia, secondary peritonitis, invasive aspergillosis, endocarditis and my

171 Peritonitis is a common and serious complication in indi

172 Acute peritonitis is a frequent medical condition that can tri

173 Peritonitis is a major complication of peritoneal dialys

174 Spontaneous bacterial peritonitis is an acute bacterial infection of ascitic f

175 Here, we report that resolution of acute peritonitis is delayed in obese diabetic (db/db) mice.

176 t was previously thought, the risk of septic peritonitis is reduced and not increased with each recur

177 Peritonitis is the major disease problem of laying hens

178 uals with a history of spontaneous bacterial peritonitis, known to have defective cellular immunity.

179 ed during polymicrobial versus monomicrobial peritonitis, leading to increased inflammatory infiltrat

180 opolysaccharide- or Escherichia coli-induced peritonitis led to IL-1beta release in the peritoneal me

181 acterium anatis are frequently isolated from peritonitis lesions.

182 a role for G. anatis in the pathogenesis of peritonitis, little is known about the organism's virule

183 by macrophages derived from zymosan-induced peritonitis, M1- and M2a-like bone marrow derived macrop

184 cell infiltration in a zymosan-induced mouse peritonitis model accompanied by impaired levels of cyst

185 fects of hydrogen sulfide in vivo, we used a peritonitis model by way of intraperitoneal injection of

186 Using a sterile peritonitis model in mice, we identified interleukin (IL

187 In a mouse peritonitis model of gout, using monosodium urate crysta

188 y, pharmacokinetics, and efficacy in a mouse peritonitis model of infection, which led to the discove

189 In this study, we used a murine peritonitis model to characterize the inflammatory mecha

190 A controlled murine S. aureus peritonitis model with and without inhaled CO was examin

191 d in inflamed cremaster muscle venules, in a peritonitis model, and in an in vitro chemotaxis assay.

192 In the peritonitis model, EP2(-/-) mice exhibited a higher perc

193 In a murine peritonitis model, HlgAB contributes to S. aureus bacter

194 In the mouse peritonitis model, LNZ (at doses that mimic human pharma

195 Employing a mouse peritonitis model, we also determined that cells collect

196 Using an in vivo peritonitis model, we have validated that the IL-1beta i

197 Using a self-limited zymosan-induced peritonitis model, we show that the Ac2-26 NPs (100 ng p

198 a significant effect on virulence in a mouse peritonitis model, whereas a double-deletion mutant was

199 +) and 3-Cl-Tyr were detected readily in the peritonitis model, whereas in the arterial inflammation

200 reduce RIPK2-mediated effects in an in vivo peritonitis model.

201 lular recruitment is inhibited in an in vivo peritonitis model.

202 P) monocytes in vivo using a sterile zymosan peritonitis model.

203 el and the cecal ligation and puncture (CLP) peritonitis model.

204 monosodium urate crystal inflammatory murine peritonitis model.

205 nt of the inflammatory response in the mouse peritonitis model.

206 r to be concordant with those from the mouse peritonitis model.

207 also reduced mortality in a murine S. aureus peritonitis model.

208 ow a failure of leukocyte extravasation in a peritonitis model.

209 psis in mice using the colon ascendens stent peritonitis model.

210 ued by prior treatment with 12(S)-HETE, in a peritonitis model.

211 to adoptive transfer into an on-going murine peritonitis model.

212 inflammatory activity in a SEB-induced mouse peritonitis model.

213 showed impaired neutrophil recruitment in a peritonitis model.

214 dies using small animals (p < 0.0001) and in peritonitis models (p < 0.0001).

215 zymosan air pouch and thioglycolate-induced peritonitis models, the i.m. treatment with xylazine or

216 f AIM in prevention of progression of fungal peritonitis models.

217 Moreover, in a thioglycollate-induced peritonitis mouse model, Sema4A was detected in circulat

218 peptide (AC3-I) and in colon ascendens stent peritonitis myocytes isolated from mutant mice that have

219 was also preserved in colon ascendens stent peritonitis myocytes isolated from transgenic mice expre

220 eticulum Ca content in colon ascendens stent peritonitis myocytes.

221 The sheep were randomized to fecal peritonitis (n = 10) or a sham procedure (n = 5), and cr

222 d to intensive care with sepsis due to fecal peritonitis (n = 117) or community-acquired pneumonia (n

223 = 2), infected joint prosthesis (n = 2), and peritonitis (n = 2) being the most common, thus expandin

224 In zymosan-initiated peritonitis, neutrophil polymorphonuclear leukocyte infi

225 lear neutrophil (PMN) infiltration in murine peritonitis (ng/mouse range) as well as enhancing human

226 ed leukocyte infiltration in zymosan-induced peritonitis nor regulated miR-208a and IL-10 in these mi

227 associated with necrosis in zymosan-induced peritonitis of AIM (-/-) mice.

228 ions produced in living macrophage cells and peritonitis of living mice with high contrast.

229 ammation, induced by thioglychollate-induced peritonitis or following infection withSalmonella enteri

230 e presence of clinical symptoms and signs of peritonitis or intra-abdominal abscess and isolation of

231 O-deficient (Mpo(-/-)) mice with established peritonitis or localized arterial inflammation, and tiss

232 diagnoses of ascites, spontaneous bacterial peritonitis, or esophageal variceal hemorrhage.

233 diagnoses for ascites, spontaneous bacterial peritonitis, or esophageal variceal hemorrhage.

234 asome-dependent inflammation in the model of peritonitis (P<0.05) in wild-type but not in miR-21 knoc

235 group or one of three groups in which fecal peritonitis (peritoneal instillation of 2 g/kg autologou

236 l replacement therapy, spontaneous bacterial peritonitis, positive blood culture, and infection by fu

237 imen of Ertapenem in patients with localized peritonitis ranging from mild to moderate severity.

238 Animals with zymosan peritonitis reached a clinical and biochemical nadir on

239 Peritonitis remains a major cause of morbidity and morta

240 nt therapies, fungal-bacterial polymicrobial peritonitis remains a serious complication for surgery p

241 d colonic perforation with purulent or fecal peritonitis remains controversial.

242 Bacterial peritonitis remains the main cause of technique failure

243 date macrophages was impaired during sterile peritonitis, resulting in enhanced and prolonged inflamm

244 Furthermore, colon ascendens stent peritonitis S2814A mice showed preserved ejection fracti

245 ion (UTI) (28.5%), and spontaneous bacterial peritonitis (SBP) (22.5%) were the most prevalent infect

246 fectious diseases were spontaneous bacterial peritonitis (SBP) (645, 49.8%), urinary tract infection

247 ver 20 points and with spontaneous bacterial peritonitis (SBP) as a precipitating event was almost si

248 Spontaneous bacterial peritonitis (SBP) can be a severe complication occurring

249 owever, development of spontaneous bacterial peritonitis (SBP) in these patients could preclude treat

250 Spontaneous bacterial peritonitis (SBP) is a common, life-threatening complica

251 tract infection (UTI), spontaneous bacterial peritonitis (SBP), and sepsis without specific focus (SW

252 e in case of suspected spontaneous bacterial peritonitis (SBP).

253 which can also lead to spontaneous bacterial peritonitis (SBP).

254 ract infections (52%), spontaneous bacterial peritonitis (SBP, 23%) and spontaneous bacteremia (21%)

255 oteinase-8 is a critical component of septic peritonitis secondary to intestinal compromise.

256 Peritonitis secondary to perforated diverticulitis has c

257 opean community-acquired pneumonia (CAP) and peritonitis sepsis cases, and 477 controls from the Unit

258 combined, 28-day mortality from CAP sepsis, peritonitis sepsis, pneumococcal sepsis or sepsis in you

259 erapeutic efficacies, using either the mouse peritonitis-sepsis model or the thigh infection model.

260 rization, major bleeding, sepsis, pneumonia, peritonitis, severe arrhythmia, or renal failure.

261 In conclusion, peritonitis significantly associates with mortality in p

262 n alphaCD40-induced acute colitis and during peritonitis, suggesting an altered monocyte migration.

263 wenty-four hours after colon ascendens stent peritonitis surgery, we observed that wild type mice had

264 o caecal ligation and puncture (CLP)-induced peritonitis than wild-type (WT) mice.

265 ar recruitment site for neutrophils in acute peritonitis that is indispensable for host defence again

266 a of BG2 (1 x 10(8) or 1 x 10(7) CFU) caused peritonitis that progressed to abscesses.

267 In murine experimental peritonitis, the application of SLC1 drastically reduced

268 o study resolution of thioglycollate-induced peritonitis, the model in which earlier work indicated t

269 During peritonitis, the peritoneal membrane undergoes structura

270 o patients presented with signs of bacterial peritonitis; the third had pyomyositis of the thigh.

271 aride or cecal ligation and puncture-induced peritonitis, these marginated cells are rapidly released

272 f P. aeruginosa was used in a mouse model of peritonitis to show the requirement of NLRC4.

273 condary prophylaxis of spontaneous bacterial peritonitis, to 90% for assays for cell number and type

274 A final diagnosis of CNS, complicated with peritonitis tracking into the scrotal sacs was arrived a

275 111 patients with acute peritonitis underwent emergency laparotomy: number of pe

276 tion: prior episode of spontaneous bacterial peritonitis, upper gastrointestinal bleeding, and low-pr

277 0001), particularly in spontaneous bacterial peritonitis, urinary tract infection, and pneumonia (26%

278 Results were compared to those in mouse peritonitis, using LNZ via oral gavage at 80 and 120 mg/

279 Feline infectious peritonitis virus (FIPV) belongs to the genus Alphacoron

280 Feline infectious peritonitis virus (FIPV) is the leading cause of death i

281 ain protease (M(pro)) from feline infectious peritonitis virus (FIPV), which leads to lethal systemic

282 Thioglycollate-stimulated peritonitis was also attenuated.

283 ere pre-injected (ip.) with sCD48, and then, peritonitis was induced by SEB injection; peritoneal lav

284 INTERVENTIONS AND MEASUREMENTS: Fecal peritonitis was induced in conscious, fluid-resuscitated

285 Fecal peritonitis was induced, and the sheep were randomized t

286 ivo, neutrophil infiltration in self-limited peritonitis was reduced in human ALX/FPR2-overexpressing

287 10,14-tetramethylpentadecane (TMPD)-mediated peritonitis was similarly aggravated in STING-deficient

288 y window 6 months before death, the odds for peritonitis was six-fold higher during the 30 days immed

289 Using a murine model of peritonitis, we also demonstrate that these agents are h

290 Using a mouse model of peritonitis, we demonstrate that neutrophils elicited in

291 ng a murine model of foreign body-associated peritonitis, we demonstrated that the ability of the Del

292 Using a murine model of peritonitis, we have demonstrated that monomicrobial inf

293 tant S. aureus (CA MRSA) in a mouse model of peritonitis, we observed in vivo expression of Nuc activ

294 variceal bleeding, and spontaneous bacterial peritonitis were associated with a higher mortality rate

295 protected 20% of mice (1 of 5) from E. coli peritonitis, whereas 100% (15 of 15) survived when AB103

296 T-cell activation in a model of PMA-elicited peritonitis, whereas topical application of MCV1 markedl

297 in the pathogenesis of spontaneous bacterial peritonitis, which is a common complication of liver cir

298 ment and IL-1beta generation in alum-induced peritonitis, which is a typical IL-1 signaling-dependent

299 total, there were 1446 reported episodes of peritonitis with 27% of patients having >/= 2 episodes.

300 ns of cecal contents to induce polymicrobial peritonitis without tissue compromise in the recipient.