ライフサイエンスコーパス: perivascular space

1 ased number of Gaucher cells in the cerebral perivascular space.

2 allowing substances in the CSF to enter the perivascular space.

3 lexity of regulation of hematopoiesis in the perivascular space.

4 s play an important role in the integrity of perivascular space.

5 al stromal cells (MSCs) are important in the perivascular space.

6 l TSP-1 upregulation in the interstitial and perivascular space.

7 nels and elevate [K(+)](o) in the restricted perivascular space.

8 humoral immunity targeting an antigen in the perivascular space.

9 he artery and breaks tissue tolerance in the perivascular space.

10 productively infected MNLs into a potential perivascular space.

11 of CD68+ macrophages in the interstitial and perivascular spaces.

12 t, Tregs were restricted to the meninges and perivascular spaces.

13 ter confinement of CD4(+) lymphocytes to CNS perivascular spaces.

14 9 expression was confined to the vasculature/perivascular spaces.

15 in deposition was found in blood vessels and perivascular spaces.

16 most infiltrating cells remain localized to perivascular spaces.

17 matrix and reduces NP confinement within the perivascular spaces.

18 e immune responses from mucosal surfaces and perivascular spaces.

19 lymphocytes in both the brain parenchyma and perivascular spaces.

20 sular foci at MR imaging are due to enlarged perivascular spaces.

21 oid space and infiltrated the Virchow-Robin (perivascular) space.

22 erivascular macrophages (PVM) located in the perivascular space, a major site of brain Abeta collecti

23 Rac in the Nestin(+) cells would perturb the perivascular space, altering HSC localization and hemato

24 ecruitment prolonged T cell retention in the perivascular space, although no difference in overall CN

25 ity that allowed angiotensin II to enter the perivascular space and activate angiotensin type 1 recep

26 ancer cells were specifically located in the perivascular space and closely associated with blood ves

27 CD40(-/-) mice, T cells were retained in the perivascular space and did not migrate into the parenchy

28 essing mononuclear cells into and out of the perivascular space and leading to progression of disease

29 ouble-negative CD3(+)CD4(-)CD8(-) T cells in perivascular space and reduced vascular oxidative stress

30 rebral ischemia, CD8+ T cells infiltrate the perivascular space and secrete interleukin-16 (IL-16), a

31 roid plexus in early inflammation and in the perivascular space and SIV encephalitis (SIVE) lesions l

32 CD163(+) macrophages accumulated in the perivascular space and SIVE lesions with late inflammati

33 s not required for monocyte migration to the perivascular space and that vascular remodeling followin

34 entitially released drug is cleared from the perivascular space and those by which drug enters the bl

35 acytic inflammation, with a predilection for perivascular spaces and collagenous tissues, was observe

36 p<0.05, and also showed significant rises in perivascular spaces and in cortical parenchyma.

37 lation of mature thymocytes within medullary perivascular spaces and reduced numbers of recent thymic

38 odies exhibited size-dependent access to the perivascular spaces and tunica media basement membranes

39 he deposited drug diffuses directly from the perivascular space, and little arrives from the endovasc

40 ealed white matter hyperintensities, dilated perivascular spaces, and lacunar infarction.

41 cal superficial siderosis, centrum semiovale perivascular spaces, and white matter hyperintensities.

42 cal superficial siderosis, centrum semiovale perivascular spaces, and white matter hyperintensities.

43 caused by a leak of blood and fluid into the perivascular space around the artery, as in several pati

44 hed to VE-cadherin(+) cells, implicating the perivascular space as a near-homogenous location of LT-H

45 We detected NOA-Au into perivascular spaces as early as 30 sec after the beginni

46 the endothelium; (iii) it is detected in the perivascular spaces at early time points (by 1 min) and

47 rocess that requires cells to pass through a perivascular space before entering the parenchyma.

48 ensity - WMH, microbleeds, lacunes, enlarged perivascular spaces, brain atrophy) as seen on structura

49 he meninges, injection site, ventricles, and perivascular spaces but not in the parenchyma.

50 m-dependent activation of the T cells in the perivascular space, but not within the vascular lumen.

51 associated with increasing age and enlarged perivascular spaces, but these did not explain the lacun

52 Drug released into the perivascular space can enter the artery either from the

53 al (thymic epithelial space) and peripheral (perivascular space) components that functions well into

54 ild inflammatory exudates, in endomysial and perivascular spaces, consisted of lymphocytes, histiocyt

55 highly dendritic cells localized within the perivascular space, dermal-epidermal junction, and retic

56 infarcts, microinfarcts, arteriolosclerosis, perivascular space dilation and myelin loss-predicted co

57 orrhage, fibrinoid necrosis, microaneurysms, perivascular space dilation, perivascular haemosiderin l

58 d to characterize how drug released into the perivascular space enters the arterial wall and how it i

59 Enlarged perivascular spaces (EPVS) are a promising neuroimaging

60 ject-based morphologic estimates of enlarged perivascular spaces (ePVSs) in clinical-field-strength (

61 mas are highly invasive tumours that use the perivascular space for invasion and co-opt existing vess

62 pace may provide unique entry sites into the perivascular spaces from the CSF.

63 have pleomorphic lymphoid infiltrates in the perivascular spaces, had significantly increased numbers

64 lthough T cells could reach the meninges and perivascular space in the absence of TNFR1, recruitment

65 grade), whereas the severity of MRI-visible perivascular spaces in the basal ganglia was associated

66 justed analyses, the severity of MRI-visible perivascular spaces in the centrum semi-ovale was indepe

67 's disease, we hypothesized that MRI-visible perivascular spaces in the centrum semi-ovale would be a

68 We also hypothesized that MRI-visible perivascular spaces in the centrum semi-ovale would be a

69 nocytes into the choroid plexus stromata and perivascular spaces in the cerebra of rhesus macaques ac

70 iods when fluid and solutes are driven along perivascular spaces in the reverse direction to the flow

71 e attachment of solutes to the lining of the perivascular space, in order to produce a valve-like eff

72 though almost all the drug released into the perivascular space is cleared through the extravascular

73 n through the endothelial cell wall into the perivascular space is well characterized; however, mecha

74 However, whether large perivascular spaces (L-PVSs) (>3 mm in diameter) visible

75 ucuronidase reached brain parenchyma via the perivascular spaces lining blood vessels.

76 Using MRI-visible perivascular space location and severity together with o

77 t the anatomical distribution of MRI-visible perivascular spaces may reflect the underlying cerebral

78 cts, lacunes, white matter hyperintensities, perivascular spaces, microbleeds, and brain atrophy.

79 within Gelfoam matrices and implanted in the perivascular space of 42 injured arteries.

80 epileptiform discharges, WBCs populated the perivascular space of a leaky BBB.

81 lium controls repair even when placed in the perivascular space of injured vessels.

82 and inflammatory cells that appeared in the perivascular space of intramural coronary arteries and a

83 find that glioma cells, as they populate the perivascular space of preexisting vessels, displace astr

84 exclusively target the meninges and vascular/perivascular space of the gray and white matter of the b

85 ion or fibrosis in the media, adventitia, or perivascular space of vessels treated with the beta-part

86 distribution to deep brain regions along the perivascular spaces of all vessel types, with sdAb acces

87 to inflammatory changes in the meninges and perivascular spaces of both wild-type and complement-def

88 fluid, although convective transport in the perivascular spaces of cerebral blood vessels was also e

89 he brain surface and convective transport in perivascular spaces of cerebral blood vessels.

90 rne macrophages repopulated the meninges and perivascular spaces of chimeric animals.

91 microscopic examination, these cavities were perivascular spaces of mostly arteriolar origin.

92 sed numbers in both the brain parenchyma and perivascular spaces of pre-AIDS brains.

93 sing exchangeable Zn2+ concentrations in the perivascular spaces of the brain.

94 tions to localize infiltrating leukocytes to perivascular spaces, preventing CNS parenchymal infiltra

95 The perivascular space (PVS) of human thymus increases in vo

96 Perivascular space (PVS) within the brain is an importan

97 rain and increased their presence within the perivascular space (PVS).

98 ite matter lesion load, frequency of dilated perivascular spaces (PVS) and abnormalities in cerebral

99 MRI-visible perivascular spaces (PVS) are potential neuroimaging mar

100 Perivascular spaces (PVSs) in brain have a close relatio

101 th advancing age, an increased visibility of perivascular spaces (PVSs) on magnetic resonance imaging

102 Infiltration of splenic blood vessel perivascular spaces resulted in vascular compression and

103 MRI-visible perivascular space severity in either location did not p

104 genesis, the migration of monocytes into the perivascular space surrounding collateral arteries and t

105 can, are not abundant in brain except in the perivascular space that is contacted by astrocytes but n

106 Perivascular spaces that are visible on magnetic resonan

107 ze CXCR4-expressing mononuclear cells to the perivascular space, thereby limiting the parenchymal inf

108 channels, which locally release K+ into the perivascular space to activate SMC Kir channels and caus

109 ation away from their vitronectin-containing perivascular space toward fibronectin-rich tumor tissue.

110 -carboxybenzoylsulphonamide (TFL-Zn)] in the perivascular space was significantly decreased in the ne

111 MRI-visible perivascular spaces were rated using a validated 4-point

112 cytes were detected only in the vascular and perivascular spaces while albumin and IgG extravasates w

113 d with inflammation of the vascular wall and perivascular space with cells of monocyte/microglia line

114 method for multimodal autoidentification of perivascular spaces yields individual whole-brain morpho