ライフサイエンスコーパス: perivenous

1 ated encephalomyelitis, the co-occurrence of perivenous and confluent demyelination in some individua

2 planted cells was shifted from periportal to perivenous areas by targeted hepatic ablations with carb

3 Transplanted hepatocytes in perivenous areas exhibited inducible cytochrome P450 act

4 eriportal regions of hepatic acinus, whereas perivenous areas were weakly stained or were stain-free.

5 ransplanted cells shifted from periportal to perivenous areas.

6 d thoracic radiologists independently graded perivenous artifact and arterial enhancement.

7 ced iodine concentration appears to diminish perivenous artifact and to result in improved arterial e

8 Perivenous artifacts were statistically significantly re

9 case demonstrated striatal encephalitis with perivenous B- and T-lymphocytic infiltration.

10 In control livers, the distal perivenous cells contained 40% of the total glucokinase

11 f the lobule was taken up by the most distal perivenous cells; this distal perivenous uptake was grea

12 of the total glucokinase of the liver; this perivenous concentration of glucokinase was greatly redu

13 Patients with perivenous demyelination (n = 13; median age 43 years, r

14 s demyelination cohort, 10 patients had only perivenous demyelination and three also had confluent de

15 The perivenous demyelination cohort was more likely than the

16 Among the perivenous demyelination cohort, 10 patients had only pe

17 Perivenous demyelination is associated with meningoencep

18 Perivenous demyelination is the pathological hallmark of

19 Although pathological evidence of perivenous demyelination may be superior to clinical cri

20 All but one patient with perivenous demyelination only had a monophasic course, w

21 d cortical demyelination was found among six perivenous demyelination patients, all of whom had encep

22 We investigated whether perivenous demyelination versus confluent demyelination

23 efined acute disseminated encephalomyelitis (perivenous demyelination), but misdiagnosed acute dissem

24 In contrast with perivenous encephalomyelitis, in which demyelination was

25 of mice and the expression of periportal and perivenous hepatocyte markers was determined by polymera

26 Perivenous hepatocyte-specific expression was confirmed

27 lations, suggesting that both periportal and perivenous hepatocytes are induced.

28 in kidney proximal tubule S3 segments and in perivenous hepatocytes, consistent with the sites of hig

29 e periportal pattern and the last downstream perivenous hepatocytes, respectively.

30 Basolateral RhBG is expressed by perivenous hepatocytes, where it may mediate ammonium up

31 P450 inducibility was originally greater in perivenous hepatocytes; however, periportal cells rapidl

32 KO mice had attenuated perivenous hypoxia, suggesting disruption of the normal

33 that demyelination can occur independent of perivenous inflammatory changes and supports the presenc

34 Surprisingly, in resting liver tissue, perivenous localization of the HIF hydroxylases was obse

35 mice were initially treated with halothane, perivenous necrosis and an infiltration of CD11b(+) Gr-1

36 lidated clinical diagnostic criteria but its perivenous pathological findings distinguish it from mul

37 ibility occurs in the periportal (PP) and/or perivenous (PV) zones in response to hypoxia/reoxygenati

38 s in glucokinase expression in the immediate perivenous region.

39 However, HFD-fed TG mice developed prominent perivenous steatosis with periportal sparing.

40 aging features associated with MS, including perivenous T2-weighted hyperintense lesions and focal le

41 periportal-type (wild-type beta-catenin) and perivenous-type (mutant beta-catenin), which expressed n

42 he most distal perivenous cells; this distal perivenous uptake was greatly diminished in maternal low

43 ointensity, periependymal brainstem lesions, perivenous white matter lesions, Dawson's fingers, curve

44 CCs display mutually exclusive periportal or perivenous zonation programs.

45 of hypoxia and lower gluconeogenesis in the perivenous zone as compared to the rest of the organ.

46 tion of GS in hepatocytes outside the normal perivenous zone was accompanied by a reduction in the ex

47 ntermediate, and the first few layers of the perivenous zone.

48 y fashion in the last 1-2 cell layers of the perivenous zone.

49 ributed downstream to midlobular (zone 2) or perivenous (zone 3) areas.