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1 r hemorrhage grade of 3 or greater or cystic periventricular leukomalacia.
2 ing diseases, such as multiple sclerosis and periventricular leukomalacia.
3 prematurity, necrotizing enterocolitis, and periventricular leukomalacia.
4 to have hypoxic brain injury in the form of periventricular leukomalacia.
5 time to surgery (P=0.028) than those without periventricular leukomalacia.
6 severe intracranial hemorrhage and/or cystic periventricular leukomalacia.
7 e rates of severe intracranial hemorrhage or periventricular leukomalacia.
8 se and severe intraventricular hemorrhage or periventricular leukomalacia.
9 ll as severe intraventricular hemorrhage and periventricular leukomalacia.
10 ks gestation, the period of highest risk for periventricular leukomalacia.
11 amate receptor blockade in a rodent model of periventricular leukomalacia.
12 supported by the simultaneous occurrence of periventricular leukomalacia.
13 ce of severe intraventricular hemorrhage and periventricular leukomalacia (12.4 percent vs. 23.5 perc
14 incidence of intraventricular hemorrhage and periventricular leukomalacia (33.3 percent and 38.2 perc
15 7.5 percent vs. 23.9 percent, P=0.03) and of periventricular leukomalacia alone (5.2 percent vs. 9.0
16 ic white matter injury is thought to mediate periventricular leukomalacia, an age-dependent white mat
17 n lesions of white matter disorders, such as periventricular leukomalacia and multiple sclerosis.
18 hogenesis of white matter disorders, such as periventricular leukomalacia and multiple sclerosis.
20 tial failure of recovery in insults, such as periventricular leukomalacia and multiple sclerosis.
21 or translation as a therapeutic strategy for periventricular leukomalacia and that the mechanism of p
22 ncluding multiple sclerosis, cerebral palsy (periventricular leukomalacia), and spinal cord injury.
23 lasia, sepsis, intraventricular haemorrhage, periventricular leukomalacia, and necrotising enterocoli
25 asia, severe intracranial hemorrhage, cystic periventricular leukomalacia, and/or severe retinopathy
26 eath; death, intraventricular hemorrhage, or periventricular leukomalacia; and death or necrotizing e
28 on, length of stay, intracranial hemorrhage, periventricular leukomalacia, chronic lung disease, pate
30 alence of structural brain abnormalities and periventricular leukomalacia, fetal and postnatal cerebr
31 epsis, intraventricular hemorrhage >grade 2, periventricular leukomalacia >grade 1, or necrotizing en
32 associated with BAS; however, neonates with periventricular leukomalacia had lower preoperative oxyg
33 plications with respect to the mechanisms of periventricular leukomalacia in infants and of persisten
34 se and severe intraventricular hemorrhage or periventricular leukomalacia in premature infants are as
35 severe intracranial hemorrhage and/or cystic periventricular leukomalacia in the neonatal period.
36 ith clinical potential for disorders such as periventricular leukomalacia in the preterm and neonatal
40 mes of myelination or remyelination, such as periventricular leukomalacia leading to cerebral palsy,
41 elination is also noted in children with the periventricular leukomalacia of cerebral palsy, another
42 the risk of intracranial hemorrhage, cystic periventricular leukomalacia, or other complications.
43 mbined end point of intracranial hemorrhage, periventricular leukomalacia, or ventriculomegaly (17.5
44 riventricular/intraventricular hemorrhage or periventricular leukomalacia (PIVH/PVL), retinopathy of
45 tributes to mental or physical impairment in periventricular leukomalacia (pre- or perinatal white ma
46 splasia, severe intraventricular hemorrhage, periventricular leukomalacia, proven sepsis, necrotizing
47 brain injuries, including the development of periventricular leukomalacia (PVL) and cerebral palsy (C
48 causes of perinatal brain injury leading to periventricular leukomalacia (PVL) and cerebral palsy.
51 al developing white matter, which results in periventricular leukomalacia (PVL), a condition associat
52 te of cerebral palsy in premature infants is periventricular leukomalacia (PVL), a disorder of the im
53 is generally thought to consist primarily of periventricular leukomalacia (PVL), a distinctive form o
54 lying cerebral palsy in premature infants is periventricular leukomalacia (PVL), a lesion of the imma
55 ellular models of Huntington's disease (HD), periventricular leukomalacia (PVL), and kidney dysfuncti
56 eriventricular white matter injury, that is, periventricular leukomalacia (PVL), the dominant form of
57 e infant leads to white matter injury termed periventricular leukomalacia (PVL), the leading cause of
58 d and one coinciding with the peak period of periventricular leukomalacia (PVL), the major disorder u
59 e matter is important in the pathogenesis of periventricular leukomalacia (PVL), the major pathologic
60 white-matter damage of immaturity, including periventricular leukomalacia (PVL), was the most common
61 ctor for multiple brain pathologies, notably periventricular leukomalacia (PVL), which is distinguish
65 schemic damage to the cerebral white matter [periventricular leukomalacia (PVL)] is a common and lead
66 o the periventricular cerebral white matter [periventricular leukomalacia (PVL)] results in cerebral
67 II or IV intraventricular hemorrhage, cystic periventricular leukomalacia, severe bronchopulmonary dy
68 lar hemorrhage grade of 3 or greater, cystic periventricular leukomalacia, surgical necrotizing enter
71 associated with premature birth and typical periventricular leukomalacia, who had a dramatic improve
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