ライフサイエンスコーパス: permissivity

1 changes at positions 288 and 330 can confer permissivity.

2 transcriptional states of either silence or permissivity.

3 nd which mediate their effects on lymphocyte permissivity.

4 ructure likely contributes to the CalS8 base permissivity.

5 isomerase inhibitors regulate SAMHD1 and HIV permissivity at a post-RT step, revealing a mechanism by

6 that the specificity of ligand response and permissivity coevolved to enable signal discrimination.

7 The host mechanisms regulated by such permissivity factors are potential targets for anti-HIV-

8 ified four mouse carcinoma lines of variable permissivity for adenoviral gene expression, cytopathic

9 ECM attachment probably influences substrate permissivity for dendrite growth and contributes to the

10 human tumor cell lines were correlated with permissivity for EBOV glycoprotein (GP)-mediated entry.

11 ion was observed between RhoC expression and permissivity for EBOV vector transduction.

12 egration sites differ substantially in their permissivity for expression, but even the largest LCR co

13 glycosylation also acts as a determinant of permissivity for ferret, hamster, and guinea pig DPP4.

14 , such as the mitotic spindle checkpoint and permissivity for gene amplification, are also normal in

15 and this correlates with a sharp increase in permissivity for HIV infection in these stimulated cells

16 dependent on TCF-1 and GATA-3, a stochastic-permissivity function dependent on Notch signaling, and

17 als maintaining Il4 and Il13 transcriptional permissivity in naive cells.

18 ANTXR1 is necessary for permissivity in vitro and in vivo.

19 al determinants mediating MERS-CoV host cell permissivity, including virus spike interactions with th

20 Additionally, target cell permissivity increased after loading with heterologous V

21 a third parameter of substrate recognition, permissivity, is equally important.

22 t the amount of virus encountered and/or the permissivity of a dendritic cell to infection can alter

23 Due to the extensive permissivity of CD36(+) EPCs, we significantly improved

24 gn1, that is responsible for determining the permissivity of ex vivo macrophages to Legionella pneumo

25 methods for quickly and easily assessing the permissivity of existing UaaRSs and for generating permi

26 cells with GFP reporter virus confirmed the permissivity of follicular cells ex vivo.

27 tant to infection, HepG2 and HH29, conferred permissivity of HCV pseudotype infection.

28 not limited to HCV; TNF-alpha increased the permissivity of hepatoma cells to infection by Lassa, me

29 IFN-alpha induction correlated with the permissivity of pDCs to CD46- but not CAR-utilizing Ad s

30 g T-cell line, MT2 was used to determine the permissivity of RPE to HTLV-1 infection in vitro.

31 ty of the natural product assembly lines and permissivity of tailoring enzymes offer prospects for re

32 The permissivity of TFR cells to HIV-1 has not previously be

33 The permissivity of TFR cells to HIV-1 is unknown.

34 We then increased the permissivity of the initial UaaRSs to allow for incorpor

35 ple experiments, we determined the degree of permissivity of two UaaRSs for a family of structurally

36 ntiviral factor APOBEC3G (A3G) in regulating permissivity to HIV infection.

37 In this study, we investigated TFH permissivity to HIV-1 ex vivo by spinoculating and cultu

38 ines are comparable to human T-cell lines in permissivity to HIV-1 transduction, infection of murine

39 ule 3 grabbing nonintegrin) failed to confer permissivity to HIV-HCV pseudotype infection.

40 Activation of T cells increased their permissivity to HSV infection.

41 to be a novel target of infection and their permissivity to infection increases with development.

42 Regardless of the permissivity to infection, both BALB/c and C57BL/6 BMDC

43 lines were systematically compared for their permissivity to primary infection, replication, and spre

44 m distinct brain regions showed differential permissivity to replication of several positive-stranded

45 prevalence is due to differences in mucosal permissivity to SIV (i.e., whether the genetic bottlenec

46 s, providing a mechanism for their increased permissivity to support HCV entry.

47 d virus-induced cell killing correlated with permissivity to viral replication.

48 HIV-1 permissivity was conferred to the minus clone in a manne

49 Our crystal structure explains this permissivity, where an extended coil unique to MVt is un