1 While the in vitro
peroral absorption seemed to be less predictable, it ten
2 s at the 4N-position to optimize potency and
peroral absorption.
3 ty and overcome complications attendant with
peroral administration by developing a new nanovesicular
4 ic bioavailability of the gels compared with
peroral administration ranged from 54% to 69%.
5 have excellent pharmacokinetics in mice, and
peroral administration showed the compound to dose-depen
6 identity of the lung cell types targeted by
peroral arsenic and the associated immune mechanisms rem
7 Peroral arsenic has little effect on local airway immune
8 We aimed to determine the impact of
peroral arsenic on pulmonary antibacterial host defense.
9 nd higher polymers and is protective against
peroral challenges with T1L either when the MAb is passi
10 cyclosporine A (CsA), a model peptide, upon
peroral dosing to rodents led to maximum plasma concentr
11 e proposed approach offers new prospects for
peroral drug delivery and beyond.
12 Peroral endoscopic myotomy (POEM) and submucosal tunneli
13 s of a large series of patients treated with
peroral endoscopic myotomy (POEM) in a single European c
14 Pilot studies have indicated that
peroral endoscopic myotomy (POEM) might be a safe and ef
15 ne disease not amenable to surgical therapy,
peroral endoscopic removal, or simple percutaneous retri
16 After
peroral entry into mice, reovirus replicates within the
17 with vehicle or pazopanib (2.5 mg per mouse
peroral every other day) was initiated.
18 almonella enterica serovar Typhimurium SR-11
peroral infection of BALB/c mice.
19 al sections taken from Peyer's patches after
peroral infection of mice showed that, unlike its wild-t
20 athogenesis of acute toxoplasmosis following
peroral infection was examined in both genetically susce
21 Following
peroral infection with 10 cysts of the ME49 strain, all
22 orter time than did wild-type controls after
peroral infection with ME49 cysts.
23 ies the genetic susceptibility of B6 mice to
peroral infection with T. gondii, whereas the same cytok
24 in Peyer's patches in C57BL/6 mice following
peroral infection with T. gondii.
25 After
peroral infection with the ME49 strain of T. gondii, C57
26 a remarkable difference in susceptibility to
peroral infection with Toxoplasma gondii among inbred st
27 n in the small intestine following sublethal
peroral infection with Toxoplasma gondii and that this d
28 e small intestines in C57BL/6 mice following
peroral infection with Toxoplasma gondii, we performed s
29 ble of systemic spread in newborn mice after
peroral inoculation and produces lethal encephalitis.
30 rus that infects Peyer's patches (PPs) after
peroral inoculation of mice.
31 K poliovirus was unstable in feces following
peroral inoculation of mice.
32 Following
peroral inoculation of newborn mice, both viruses replic
33 After
peroral inoculation, reovirus strain type 1 Lang replica
34 ne was equivalent for both strains following
peroral inoculation.
35 a chronic carrier state in BALB/c mice after
peroral inoculation.
36 to confirm that non-endoscopic
peroral manometric placement of WC is as effective and b
37 es that cause systemic disease in mice after
peroral (
p.o.) inoculation and primary replication in th
38 were randomly assigned to WC with unsedated
peroral placement or SC after esophageal manometry (ESM)
39 Eighteen (8%) of 219 groups performed
peroral pneumocolon examinations and 80 (37%) performed
40 The authors conclude that noninvasive
peroral portal venous phase CT enterography with use of
41 r Typhimurium SR-11 wild-type strain via the
peroral route and is highly attenuated via the intraperi
42 treated BALB/c mice infected via the natural
peroral route died within 8 days of infection.
43 nity following infection through the natural
peroral route.
44 This study investigates the effect of
peroral Salmonella enterica serovar Typhimurium infectio
45 o be superior to other imaging tests such as
peroral small-bowel examinations, conventional CT, and b
46 Monotherapy with
peroral stavudine capsules or peroral zidovudine capsule
47 ases the susceptibility of mice to a primary
peroral T. gondii infection with cysts and impairs their
48 f 490 mg/dl and consumed equivalent doses of
peroral Te.
49 A murine model of
peroral Toxoplasma gondii infection was used to determin
50 Cx. pipiens had poor
peroral vector competence and a higher VT rate as compar
51 ced and supports previous data demonstrating
peroral virulence attenuation of pmrH mutants.
52 The noninvasive
peroral water CT enterography protocol had similar accur
53 Unsedated
peroral WC insertion is better tolerated than SC pH-metr
54 notherapy with peroral stavudine capsules or
peroral zidovudine capsules.