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1 s (ORAC, TRAP, HORAC and inhibition of lipid peroxidation).
2  glutathione peroxidase, lipidic and protein peroxidation).
3 c aldehydes produced by metabolism and lipid peroxidation.
4  vitamin E-mediated protection against lipid peroxidation.
5 electivity of the organocatalytic asymmetric peroxidation.
6 resence of nitrotyrosine residues, and lipid peroxidation.
7  could initiate carcinogenesis through lipid peroxidation.
8  droplets, where they are less vulnerable to peroxidation.
9 g a role for myeloperoxidase-dependent lipid peroxidation.
10  (Arabidopsis thaliana) leaves against lipid peroxidation.
11 on observed in tissue inflammation and lipid peroxidation.
12 reactions that are well precedented in lipid peroxidation.
13 ndogenous reactive oxygen species, and lipid peroxidation.
14 diolipin (CL), and resulting in selective CL peroxidation.
15 dienyl-radical intermediates formed in 7-DHC peroxidation.
16 te was the most effective inhibitor of lipid peroxidation.
17 tes hydrogen peroxide accumulation and lipid peroxidation.
18 nificance of PUFA ratios in biological lipid peroxidation.
19  found in the extent of their membrane lipid peroxidation.
20 varieties correlated with tolerance to lipid peroxidation.
21 xide and malondialdehyde, a product of lipid peroxidation.
22 ds that may be caused by activation of lipid peroxidation.
23 M integrity and peripheral measures of lipid peroxidation.
24 graft monitoring membrane fluidity and lipid peroxidation.
25 eatosis score, oil red-O staining, and lipid peroxidation.
26  2.0%, possibly via modifying membrane lipid peroxidation.
27 tract was the most potent inhibitor of lipid peroxidation.
28 vel of unsaturation indicated elevated lipid peroxidation.
29 ion in the hydrophobic region prevents lipid peroxidation.
30 rmed mutagenic DNA adduct derived from lipid peroxidation.
31 so showed strong suppressive effect on lipid peroxidation.
32  of reactive oxygen species (ROS), and lipid peroxidation.
33  showed a strong suppressive effect on lipid peroxidation.
34 o-radicals (ABTS, DPPH) and to inhibit lipid peroxidation.
35 by an increase in oxidative stress and lipid peroxidation.
36 nsfer to the redox partner AhpC required for peroxidation.
37 ha-synuclein generated in vitro by enzymatic peroxidation.
38 PB extract/kg of TTB) displayed higher lipid peroxidation.
39 e oxygen species levels and associated lipid peroxidation.
40 sis by removing fibrogenic products of lipid peroxidation.
41 changing the regio- and stereospecificity of peroxidation.
42 ess and cell membrane damage caused by lipid peroxidation.
43 f nitric oxide, protein nitration, and lipid peroxidation.
44 mulation), oxidative/nitrative stress (lipid peroxidation, 3-nitrotyrosine formation, and expression
45 tly increased ROS content (31-46%) and lipid peroxidation (30-47%), concomitant with decreased operat
46 eutrophil infiltration (MPO activity), lipid peroxidation (4-HNE), and nitric oxide (iNOS) - were sig
47 lpha: 64% +/- 24% increase; P < 0.05), lipid peroxidation (4-hydroxynonenal, measured by ELISA: 0.30
48 ine (8-NO2Gua)) as well as products of lipid peroxidation (8-iso-prostaglandin F2alpha (8-isoPF2alpha
49  nontransferrin bound iron, markers of lipid peroxidation-8alpha-isoprostanes, protein oxidation-adva
50 of trained mice) and oxidative stress (lipid peroxidation, 9.1 +/- 1.4 vs. 5.2 +/- 0.9 mumol mg(-1) ;
51 ecent advances in our understanding of lipid peroxidation, a degenerative process that is believed to
52 s, whereas the Flt3 inhibitor prevents lipid peroxidation, a key mechanism of glutamate-mediated toxi
53                                        Lipid peroxidation, a major consequence of oxidative stress, g
54 y identified the novel end-products of lipid peroxidation, accumulating in circulation in hyperlipide
55 rization, production of free radicals, lipid peroxidation, activation of phospholipase C, IP3 recepto
56  a significant reducing power and anti-lipid peroxidation activities.
57 teine, a potent antioxidant, abolished lipid peroxidation activity and ameliorated EAE in IFN-gamma-s
58  Importantly, "free" myelin debris and lipid peroxidation activity at CNS lesions was increased in mi
59  (OSE), which are products of enhanced lipid peroxidation and a major target of innate natural antibo
60 eral, AntiOxCINs derivatives prevented lipid peroxidation and acted as inhibitors of the mitochondria
61 o tropical fruit juices (FA and FB) on lipid peroxidation and antioxidant enzymes in rats.
62 ical of neuronal damage with increased lipid peroxidation and cellular oxidant levels but no alterati
63 cyte adhesion in culture and increases lipid peroxidation and cyclooxygenase-2 (COX-2) levels in cult
64         FA-AKI in mice associates with lipid peroxidation and downregulation of glutathione metabolis
65 ability and reduced levels of membrane lipid peroxidation and electrolyte leakage under drought or sa
66 nCeO2 L(-1), the roots showed enhanced lipid peroxidation and electrolyte leakage, while at 500 mg L(
67 , as indicated by reduced ROS, lowered lipid peroxidation and enhanced photosynthesis.
68  aldehyde, is produced endogenously by lipid peroxidation and exogenously by combustion of organic ma
69 utathione peroxidase (GPx), vitamin E, lipid peroxidation and liver enzymes in hyperlipidaemia rabbit
70 dialdehyde (MDA) epitopes, products of lipid peroxidation and markers for enhanced oxidative stress,
71          Our observations suggest that lipid peroxidation and mitochondrial biogenesis are the key in
72          Our observations suggest that lipid peroxidation and mitochondrial biogenesis are the key in
73                                  Also, lipid peroxidation and mitochondrial dysfunction appeared to b
74  seen including elevated mitochondrial lipid peroxidation and mitochondrial membrane defects, as well
75           MJ33 increased the levels of lipid peroxidation and mitochondrial O2(* horizontal line ) pr
76 hemical link between antioxidant load, lipid peroxidation and mitochondrial physiology.
77                              Levels of lipid peroxidation and of superoxide anion (O2(* horizontal li
78 identify 5-HT as a potent inhibitor of lipid peroxidation and offer a different perspective on the ro
79                                        Lipid peroxidation and polar compounds formation in sunflower
80 ase [ECSOD] mimetics), porphyrins, and lipid peroxidation and protein carbonylation blockers/inhibito
81   Rice seedlings also exhibited severe lipid peroxidation and protein carbonylation, for oxidative st
82 ytoprotective activity on lymphocytes, lipid peroxidation and protein degradation.
83 ars, sucrose, ethylene, ascorbic acid, lipid peroxidation and reactive oxygen species.
84 xidase activity, which catalyzes cardiolipin peroxidation and results in mitochondrial damage during
85  activation of SAT1 expression induces lipid peroxidation and sensitizes cells to undergo ferroptosis
86 adduct (where the Trp is further modified by peroxidation) and its associated mobile arginine.
87 stress parameters such as glutathione, lipid peroxidation, and calcium levels along with the glutathi
88 odification and oxidation of proteins, lipid peroxidation, and DNA fragmentation.
89 ions in pathogenic eicosanoid species, lipid peroxidation, and extracellular receptor kinase 1/2 acti
90 sible symptoms (i.e. cell death), less lipid peroxidation, and lower NADPH oxidase activity, indicati
91 d oxidative phosphorylation, increased lipid peroxidation, and neuroinflammmation.
92 th altered antioxidant enzyme content, lipid peroxidation, and oxidative DNA adducts.
93 ch can quench singlet oxygen, suppress lipid peroxidation, and prevent oxidative damage.
94 egion of the cells and were related to lipid peroxidation, and structural changes of nucleic acids.
95 poxy alcohols are major products formed from peroxidation, and the basic mechanisms of product format
96 positively with EVOO to inhibit phospholipid peroxidation, and thus, McPC-EEVOO could be a potential
97 e spectrometry, anti-low-density lipoprotein peroxidation, anti-AAPH-induced hemolysis, and oxygen ra
98 ths, and that elevated serum levels of lipid peroxidation are reported in BD, these serum measures ma
99 mation of hydroxyl radicals leading to lipid peroxidation as the primary mechanism of bacterial inact
100 e roots showed delayed PPD and reduced lipid peroxidation as well as decreased H2O2 accumulation.
101 yl radicals), copper-induced LDL-cholesterol peroxidation, as well as alpha-glucosidase and lipase ac
102 sruption of the antioxidant system and lipid peroxidation, as well as alterations in lysosomal membra
103  and in L-alpha-phosphatidylcholine liposome peroxidation assay measured following formation of conju
104                             Endogenous lipid peroxidation (assay A) and induced lipid peroxidation (a
105 pid peroxidation (assay A) and induced lipid peroxidation (assay B) were evaluated in liver homogenat
106 radical absorbance capacity (ORAC) and lipid peroxidation assayed as thiobarbituric acid reactive sub
107 eomics data, enzymatic activities, and lipid peroxidation assays, we identified glutathione peroxidas
108 of phenolic compounds in non-enzymatic lipid peroxidation assays.
109  beta-carotene bleaching system and liposome peroxidation assays.
110 nging, beta-carotene-linoleic acid and lipid peroxidation assays; the antibacterial activity was eval
111 insights into asthma mechanisms once lipidic peroxidation assessed by urinary metabolomics is related
112 cury were not associated with elevated lipid peroxidation biomarkers.
113 between cadmium, lead, and mercury and lipid peroxidation biomarkers.
114 altered antioxidant enzyme content and lipid peroxidation, but better withstood insults.
115 evated H(2)O(2) flux, and increases in lipid peroxidation, but no effect on longevity.
116 nd kidney dysfunction; Fer-1 inhibited lipid peroxidation, but not mitochondrial reactive oxygen spec
117 5-HT contributes to the termination of lipid peroxidation by direct interaction with active groups of
118    These lipids are the substrates for lipid peroxidation by lipoxygenase enzymes.
119             XBP1 activation, fueled by lipid peroxidation byproducts, induced a triglyceride biosynth
120  scale of inhibition properties of the lipid peroxidation can be devised.
121                                              Peroxidation can be explained by a mechanism in which th
122                           Increases in lipid peroxidation can cause ferroptosis, a form of cell death
123 s (protocatechuates) inhibited linoleic acid peroxidation catalyzed by soybean lipoxygenase-1 (EC 1.1
124 plets protect glia and also neuroblasts from peroxidation chain reactions that can damage many types
125      OA cartilage had higher levels of lipid peroxidation compared to control cartilage, and lipid pe
126 e control, and decreased the degree of lipid peroxidation compared with the HF diet.
127 he pex11a line showed higher levels of lipid peroxidation content and lower expression of genes invol
128 lve ROS generation, carnosol inhibited lipid peroxidation, contrary to carnosic acid.
129 ochondrial function by measuring H2O2, lipid peroxidation, cytochrome c oxidase activity and mitochon
130  that protects RPE cells in vitro from lipid peroxidation cytotoxicity mediated by 4-hydroxynonenal (
131 vy hydrogen isotope deuterium at the site of peroxidation (D-PUFA) prevented PUFA oxidation and block
132  levels of reactive oxygen species and lipid peroxidation, depleting and oxidizing glutathione and ni
133 del of assessment (HOMA), and systemic lipid peroxidation determined by plasma F2-isoprostane levels.
134 a-tocopherol, the oxysterol profile of 7-DHC peroxidation differed distinctly from the profile observ
135 W: 8.5 +/- 4.4 days, P=0.1357), nor in lipid peroxidation during 16-hr cold ischemia (P=0.672), or re
136        We report here the mechanism of lipid peroxidation during ferroptosis, which involves phosphor
137  report that PPARgamma is activated by lipid peroxidation end products, such as 4-hydroxynonenal, who
138 ased antioxidant defense, and elevated lipid peroxidation end-products in spite of comparable nitrosa
139  inhibited in vitro microsomal lipid and LDL peroxidation, exhibiting potent free-radical scavenging
140                     Antioxidant 11 inhibited peroxidation for 7-fold longer than that recorded with a
141 elluro)phenols and resorcinols could inhibit peroxidation for only 89-228 min, some of the bis(alkylt
142 vative 20c was regenerable and could inhibit peroxidation for substantially longer time (>410 min) th
143 olefinic band elicited seizure-induced lipid peroxidation further confirmed by the thiobarbituric aci
144                                        Lipid peroxidation have not changed significantly however, the
145 ted increased reactive oxygen species, lipid peroxidation, histological evidence of balloon degenerat
146 6.51 +/- 3.6 mug/mL) and inhibition of lipid peroxidation (IC50=12.34 +/- 2.3 mug/mL) as compared to
147  ability of these compounds to inhibit lipid peroxidation in a liposome membrane system was examined
148 reases in hepatic weight, lipid content, and peroxidation in C57BL/6J mice.
149 es promise to help clarify the role of lipid peroxidation in cell death and human disease.
150 utathione concentrations and decreased lipid peroxidation in cultured hepatocytes.
151 itochondrial biogenesis, and increased lipid peroxidation in female mouse offspring exposed to an obe
152 rmore their effectiveness in retarding lipid peroxidation in fish oil was evaluated by an accelerated
153 tration was negatively correlated with lipid peroxidation in foliar tissue under ozone stress and zin
154  prevents anemia, ROS accumulation and lipid peroxidation in Gpx4-deficient cells remain high.
155 ntional red grape juice consumption on lipid peroxidation in healthy individuals.
156  and correlated with the inhibition of lipid peroxidation in human erythrocytes (LP) and total conten
157 ty and vitamin E level and on reducing lipid peroxidation in hypercholesterolaemia rabbit, thereby pr
158 on of this protocol, tissue injury and lipid peroxidation in jejunum and ileum were analyzed by histo
159  flavonoids were also found to prevent lipid peroxidation in L6 myoblast.
160 ng the mechanisms of inhibition of the lipid peroxidation in micelles, in view of bibliographic data,
161 n, mitochondrial hyperpolarization and lipid peroxidation in neuronal cells, but they do so by distin
162 ction (FRAP) assays and by the effect on oil peroxidation in Oxipres apparatus.
163 E-42 has an enhanced capacity to cause lipid peroxidation in primary cortical mouse neurons compared
164  We used it to probe the importance of lipid peroxidation in progression of NASH beyond simple steato
165                   MERTK expression and lipid peroxidation in synaptoneurosomes also increased to a si
166                 Basil addition inhibited fat peroxidation in the cakes, measured as the malondialdehy
167 y, to co-localize amyloid deposits and lipid peroxidation in tissue slides from patients affected by
168  a main endogenous product of cellular lipid peroxidation in tissues and is reported to play pathogen
169 ydroxy-2,6-alkadienals), biomarkers of lipid peroxidation, in exhaled breath condensate of three heal
170 ne-to-glutathione disulfide ratio, and lipid peroxidation indicated that HFD-induced oxidative stress
171 cies production and hence the level of lipid peroxidation, indicating a role of TAG in protection aga
172 We found no significant differences in lipid peroxidation, indicating that oxidative stress may not b
173 d fractions of sea buckthorn inhibited lipid peroxidation induced by H2O2, however, the non-polar fra
174  formation of ethylene as a product of lipid peroxidation induced by the respiratory burst.
175 equence of sequence-specific repair of lipid peroxidation-induced DNA adduct, 1, N(6)-ethenoadenine (
176 ntration- and time-dependent effect on lipid peroxidation, inducing both pro-oxidant actions at low (
177 adicals, inhibit non-enzymatic linoleic acid peroxidation, inhibit human serum oxidation in the prese
178 ces produced in Southern Brazil showed lipid peroxidation inhibition abilities in healthy subjects, r
179 e antiradical activity and significant lipid peroxidation inhibition activities, with their IC50 resu
180 free radical scavenging activities and lipid peroxidation inhibition activities.
181 correlated with nitrite scavenging and lipid peroxidation inhibition activities.
182         Superoxide radical scavenging, lipid peroxidation inhibition and cupric ion reducing activiti
183  dicaffeoylquinic acid; whereas higher lipid peroxidation inhibition was attributed to the presence o
184 cavenging activity, reducing power and lipid peroxidation inhibition) and antitumour potential (again
185 cavenging activity, reducing power and lipid peroxidation inhibition) and individual phenolic profile
186 cavenging activity, reducing power and lipid peroxidation inhibition) of dried powder formulations of
187  properties (mainly reducing power and lipid peroxidation inhibition), antibacterial activity against
188 drazyl (DPPH) free radical scavenging, lipid peroxidation inhibition, nitrite scavenging and super ox
189                                        Lipid peroxidation is a major consequence of oxidative stress
190                                        Lipid peroxidation is connected to increases in mitochondrial
191                                    Substrate peroxidation is measured by microamperage-level current
192                         How BD affects lipid peroxidation is not known.
193                       The reduction of lipid peroxidation levels and activity of hepatic enzymes (ala
194  and is photoprotective, as it reduces lipid peroxidation levels.
195  is strongly associated with increased lipid peroxidation levels.
196 liar reactive oxygen species (ROS) and lipid peroxidation levels.
197  by sensing a wide range of endogenous lipid peroxidation ligands and activating innate immune signal
198 /or RD) in BD, and also examined serum lipid peroxidation (lipid hydroperoxides, LPH and 4-hydroxy-2-
199                                        Lipid peroxidation (LPO) is induced by a variety of abiotic an
200 oduced endogenously via reactions with lipid peroxidation (LPO) products.
201 DA), which is a significant product of lipid peroxidation (LPO), total oxidant status (TOS), total an
202 t maxilla was used for the analysis of lipid peroxidation (malondialdehyde [MDA]) and antioxidant enz
203                          Resistance to lipid peroxidation maps genetically to transmembrane and membr
204 50 +/- 360%, respectively, and urinary lipid peroxidation marker malondialdehyde was decreased by 32
205  levels of malondialdehyde (MDA), as a lipid peroxidation marker, and 8-hydroxydeoxyguanosine (8-OHdG
206  of several SPs, we found increases in lipid peroxidation markers in Trsp-deficient epithelial cells.
207               Brain death (BD)-related lipid peroxidation, measured as serum malondialdehyde (MDA) le
208 , indicating the occurrence of cell-membrane peroxidation mediated by ROS.
209 peutic uses for ferrostatins, and that lipid peroxidation mediates diverse disease phenotypes.
210 f normal PLA2G6 gene activity leads to lipid peroxidation, mitochondrial dysfunction and subsequent m
211 rmine the effects of SOD2 depletion on lipid peroxidation, mtDNA damage, and mitochondrial respiratio
212 NPs exposed snails did not undergo any lipid peroxidation nor change in the antioxidant contents, whi
213                Confocal microscopy and lipid peroxidation observations show that Cu and citric acid i
214                                    No sulfur peroxidation occurs, and the system remains stable.
215 e suggests that this process, known as lipid peroxidation, occurs in vivo under a variety of conditio
216 ivo from free-radical-catalyzed nonenzymatic peroxidation of alpha-linolenic acid (1).
217               At the onset of apoptosis, the peroxidation of cardiolipin at the inner mitochondrial m
218 recedented highly enantioselective catalytic peroxidation of enals.
219 )/Ascorbic acid (Fe(+3)/AsA) system mediated peroxidation of l-alpha-phosphatidylcholine aqueous disp
220 ibitory effect of HLP on oxidation and lipid peroxidation of LDL was defined in vitro.
221 ompounds were found to inhibit azo-initiated peroxidation of linoleic acid an order of magnitude more
222 f a defined pathway for O2 in regulating the peroxidation of linoleic acid by soybean lipoxygenase 1.
223 ompounds were found to inhibit azo-initiated peroxidation of linoleic acid much more efficiently than
224 d the inhibition of the metmyoglobin-induced peroxidation of linoleic acid.
225 -determining step in the tocopherol-mediated peroxidation of lipids in human low-density lipoproteins
226 hotosensitizer; irradiation at 730 nm led to peroxidation of liposomal lipids, allowing drug release.
227 process of intrinsic apoptosis relies on the peroxidation of mitochondrial lipids as a critical molec
228                Oxidative stress triggers the peroxidation of omega-6-polyunsaturated fatty acids to r
229 mes, which in turn drive ferroptosis through peroxidation of polyunsaturated fatty acids (PUFAs) at t
230 rated during oxidative stress and subsequent peroxidation of polyunsaturated fatty acids.
231  radicals by mitochondria thus causing lipid peroxidation, oxidative and acidic stress, which can lea
232 001), blood pressure (P < 0.0001), and lipid peroxidation (P = 0.001) were also observed for the HF a
233 l in that, despite the diversity of possible peroxidation pathways giving cyclic compounds and oligom
234 al electrical resistance, and markers of the peroxidation process of membrane lipids, MDA, fatty acid
235 e stress (peroxiredoxin-3/5) and for a lipid peroxidation product (hydroxynonenal).
236                      In GDM cells, the lipid peroxidation product 4-hydroxynonenal (HNE) failed to in
237   CaARP-expressing plants showed lower lipid peroxidation product content in presence or absence of s
238 and lower plasma concentrations of the lipid peroxidation product F2-isoprostanes (18.5 pg/mL, interq
239 activated by 4-hydroxynonenal (HNE), a lipid peroxidation product generated naturally during oxidativ
240 notoxic N-nitroso compounds (NOCs) and lipid peroxidation products (LPOs) in the gut.
241 lly diverse group of products, such as lipid peroxidation products (LPP).
242 ctional role of aS in sequestering the early peroxidation products of fatty acids, thereby reducing t
243                These data suggest that lipid peroxidation products play a role in progression of live
244 ies and adipose tissue, we demonstrated that peroxidation products produced in the vascular wall (ie,
245                      Tissue content of lipid peroxidation products was increased in G6PDX mice in res
246 eventing the damage to biomolecules by lipid peroxidation products, a novel concept in vision researc
247 iet and traditional medicines and from lipid peroxidation products, in human prostate and renal speci
248                         These could be lipid peroxidation products, including isolevuglandins (isoLGs
249 iscriminators of SLE included elevated lipid peroxidation products, MDA, gamma-glutamyl peptides, GGT
250                                        Lipid peroxidation products, such as 4-hydroxy-trans-2-nonenal
251  from 2,3-epoxyaldehydes of endogenous lipid peroxidation products, were present in all subjects (16.
252 lable for the generation of neurotoxic lipid peroxidation products.
253 antibodies against protein adducted by lipid peroxidation products.
254 of oxidative stress signals related to lipid peroxidation, protein carbonylation, and nitration in WA
255 oxidant enzymes were elevated, as were lipid peroxidation, protein nitrosylation, and ROS.
256 HCV replicase is uniquely regulated by lipid peroxidation, providing a mechanism for attenuating repl
257                                        Lipid peroxidation, regulated in part through sphingosine kina
258 ayed an increased membrane leakage and lipid peroxidation relative to Cu-GGH and OV-3 alone.
259 s showed no increase in ROS content or lipid peroxidation relative to well-watered controls, despite
260 ric acid (TBA) number, an indicator of lipid peroxidation responsible for off-flavour generation.
261  that photoexcited SWCNTs can catalyze lipid peroxidation similarly to lipoxygenases.
262 operties of a number of antioxidants against peroxidation, started by a 2,2'-azobis[2-(2-imidazolin-2
263 e male germ line, with the products of lipid peroxidation stimulating free radical generation by the
264 ciently than alpha-tocopherol in a two-phase peroxidation system containing excess N-acetylcysteine a
265 e predominantly associated with higher lipid peroxidation (TBARS) [exp(beta) = 1.09-1.78, p < 0.01-0.
266 dant activity (FRAP, ABTS), as well as lipid peroxidation (TBARS) were determined at the end of the e
267 electrophilic aldehyde produced during lipid peroxidation that forms covalent adducts on proteins, al
268 a propagation rate constant for free radical peroxidation that is 200 times that of cholesterol.
269 scular glycogen, and oxidative stress (lipid peroxidation) that occurred following approximately 30 m
270 vity, reducing power and inhibition of lipid peroxidation, the antitumour potential was tested in hum
271 vity, reducing power and inhibition of lipid peroxidation; the antitumour potential was tested in hum
272         PSEE exhibited a protection of lipid peroxidation threefold higher than a positive control.
273 s, which paralleled with reductions in lipid peroxidation, thus suggesting plants from the highest al
274 d in TTB as a protective agent against lipid peroxidation to extend its shelf-life up to two months.
275 e fused bicyclic core and a cobalt-catalyzed peroxidation to install the peroxide functional group.
276                                        Lipid peroxidation took place after 3 weeks of storage in dark
277  levels in both endogenous and induced lipid peroxidation up to 35% and 70%, respectively.
278                              Increased lipid peroxidation via oxidative stress was also detected by t
279   Reducing LD accumulation in glia and lipid peroxidation via targeted lipase overexpression and/or l
280                                Hepatic lipid peroxidation was also elevated in the nose-only group.
281 ant status (SOD, CAT, GPX and GSH) and lipid peroxidation was also studied.
282    Quality of olive oils was improved, since peroxidation was inhibited.
283  acids illustrated that, in the light, lipid peroxidation was predominantly due to the production of
284  sham laparotomy, but this increase in lipid peroxidation was prevented by preconditioning with remif
285  as measured by glutathione levels and lipid peroxidation was significantly reduced in rapamycin-trea
286 ion compared to control cartilage, and lipid peroxidation was similarly elevated in SOD2-depleted cho
287  the antioxidant role of Vitamin E, as lipid peroxidation was suppressed in HeLa cells both under bas
288 ary malondialdehyde (MDA), a marker of lipid peroxidation, was measured in 24 hour urine collections.
289 ver, MDA adduct formation, a marker of lipid peroxidation, was not affected by any of the four HBQs t
290 (ROS) and their downstream products of lipid peroxidation, we investigated the effect of nerve termin
291 etermine possible underlying causes of lipid peroxidation, we investigated the renal redox balance by
292 ccumulation, apoptosis, and changes in lipid peroxidation were attenuated.
293       Enhanced electrolyte leakage and lipid peroxidation were found in the shoots of seedlings grown
294 ion, amount of glutathione stores, and lipid peroxidation were similar irrespective of the insult to
295 unsaturated fatty acids, which inhibit lipid peroxidation, were able to partially rescue the locomoto
296 sity lipoprotein, serum amyloid A, and lipid peroxidation, were significantly altered by polybacteria
297 scorbic acid degradation, and membrane lipid peroxidation, which enhanced total phenolics content and
298 werful effect in non-enzymatic linoleic acid peroxidation with IC50 values 2.4 mM +/- 0.21 and 0.055
299 arly and integral component of in vivo lipid peroxidation with important clinical implications as a b
300  anion (and indirectly, a causative of lipid peroxidation) within the mitochondria matrix.

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