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1          Recent developments have shown that peroxidative activation of isoniazid by the mycobacteria
2 he sole catalase and is also responsible for peroxidative activation of isoniazid, an anti-tuberculos
3                                          The peroxidative activity of LPO increases in proportion to
4  H(2)O(2) causes tyrosine-dependent in vitro peroxidative aggregation of proteins, including alpha-sy
5          To examine the role of Arg38 in the peroxidative and peroxygenative activity of horseradish
6 trast, an F41A mutation has little effect on peroxidative catalysis.
7 d relate this function to protection against peroxidative cell injury.
8           A novel approach for assessing the peroxidative chain initiation potency of lipid hydropero
9  with EtOOH in PGH2 synthase II suggest that peroxidative cleavage is not the initiating event in dio
10 ld be inconsistent with a mechanism in which peroxidative cleavage precedes AA dioxygenation.
11 ngs are consistent with a mechanism in which peroxidative cleavage precedes AA dioxygenation.
12                                        Thus, peroxidative coupling of extensin monomers and resistanc
13 geting of ovoperoxidase may lead to spurious peroxidative cross-linking activity and contribute to th
14 te that SA acts as an electron donor for the peroxidative cycle of catalase.
15 iological conditions mitochondria can repair peroxidative damage in part through a remodeling mechani
16 evere steatosis, fibrosis, inflammation, and peroxidative damage than wild-type C57BL/6 mice.
17 embrane and biological systems by preventing peroxidative damage to lipids.
18 sfer LOOH-enriched LDL to Cu2+-induced chain peroxidative damage was assessed by monitoring the accum
19 thereby potentially enhance dissemination of peroxidative damage was examined in this study.
20 dialdehyde (MDA) was measured as a marker of peroxidative damage, and mRNA expression of CuZn-SOD, Mn
21 ile on an MCD diet had reduced steatosis and peroxidative damage, compared with mice not receiving FG
22 hly sensitive in situ monitoring of cellular peroxidative damage.
23  play a vital role in cytoprotection against peroxidative damage.
24 t to a eukaryotic-like oscillatory oxidative-peroxidative enzymatic cycle by these AHPs that leads to
25  ring A-D or B-C, a known soluble product of peroxidative heme degradation, as a modifying species.
26 not contribute to peroxynitrite formation or peroxidative injury of C. parvum-infected mucosa and had
27 sensitivity of lipid-metabolizing enzymes to peroxidative injury, as well as the reported effects of
28                   However, CPR did stimulate peroxidative metabolism by the simple system containing
29                                              Peroxidative metabolism was not affected by the interact
30                         We conclude that the peroxidative modification of ANT is conformation-depende
31 that the CeDUOX1 protein supports controlled peroxidative polymerization of tyrosine residues and ind
32      4-Hydroxyalkenal species are a class of peroxidative products of polyunsaturated fatty acids, wh
33              The F41H/H42A double mutant has peroxidative properties intermediate between those of th
34 T, implicating a role for a Trp-32-dependent peroxidative reaction in the covalent aggregation of hSO
35 ably due to siphoning catalase into the slow peroxidative reaction.
36                       This paper reviews the peroxidative reactions of Diversozymes, including peroxi
37 oxidase, in addition to catalyzing classical peroxidative reactions, also acts as a P450 cytochrome a
38 onvenient and sensitive method for measuring peroxidative status in TBI.
39     This is the first reported evidence that peroxidative stress damage can be selectively targeted a
40 tivity exhibited in these mice suggests that peroxidative stress plays an important role in ileal and
41 ame proteins that become carbonylated during peroxidative stress.
42                 The pH profiles of different peroxidative substrates showed optimal activities at var
43                                              Peroxidative treatment of rat heart mitochondria results
44 bstitutes, albeit imperfectly, for His-42 in peroxidative turnover of the enzyme.

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