ライフサイエンスコーパス: peroxide

1 lpropionamidine dihydrochloride and hydrogen peroxide).

2 rimary fibroblasts to a low dose of hydrogen peroxide.

3 erythrocytes to the same extent as hydrogen peroxide.

4 and is involved in controlling resistance to peroxide.

5 phenylenediamine in the presence of hydrogen peroxide.

6 eroxide anion radical to oxygen and hydrogen peroxide.

7 ux and determine the diffusivity of hydrogen peroxide.

8 e presence of the mild oxidant di-tert-butyl peroxide.

9 scribes the reaction of Fe(II) with hydrogen peroxide.

10 d imaging of beta-galactosidase and hydrogen peroxide.

11 se enzymes at rates comparable with hydrogen peroxide.

12 customized creation of nanoclusters of zinc peroxide.

13 nding of the possible mechanisms of hydrogen peroxide.

14 ed by changing the concentration of hydrogen peroxide.

15 peroxiredoxin PrxA, in response to hydrogen peroxide.

16 dioxygen and its congeners such as hydrogen peroxide.

17 dase, superoxide radical anion, and hydrogen peroxide.

18 sed by both urate hydroperoxide and hydrogen peroxide.

19 res could be used as a carrier for carbamide peroxide.

20 ntaining horseradish peroxidase and hydrogen peroxide.

21 presence of exogenous or endogenous hydrogen peroxide.

22 e simultaneously reducing oxygen to hydrogen peroxide.

23 be normalized by the application of hydrogen peroxide.

24 esence of varying concentrations of hydrogen peroxide.

25 nied by increased susceptibility to hydrogen peroxide.

26 radical- and cation-stabilizing groups than peroxides.

27 death induced by the build-up of toxic lipid peroxides.

28 of measuring 8 muL samples of both hydrogen peroxide (0-5 mM, 2.72 x 10(-6) A.mM(-1)) and total chol

29 r exposure of skin to high concentrations of peroxide (0.5-1mM H2O2).

30 luding formaldehyde (0.074g.L(-1)), hydrogen peroxide (21.0g.L(-1)), bicarbonate (4.0g.L(-1)), carbon

31 he Martian surface, iron oxides and hydrogen peroxide, act in synergy with irradiated perchlorates to

32 action concerns the advantageous role of the peroxide acting both as oxidant and reagent for C-O coup

33 6F5)3)2] 4 with isobutene while with benzoyl peroxide afforded [Cp*2Fe][PhC(O)OE(C6F5)3] (E = B 5, Al

34 reactions with sodium borohydride, hydrogen peroxide, alpha-methoxy-alpha-(trifluoromethyl)phenylace

35 oxidease-like catalytic activity in hydrogen peroxide-Amplex red (AR) system (AR is oxidized to resor

36 a consequence of enhanced levels of cellular peroxide and also as a by-product of increased levels of

37 erols accomplished with the oxidative state (peroxide and anisidine value, PV and AV) required for re

38 01) higher omega3 PUFAs % recovery and lower peroxide and anisidine values were observed in nanolipos

39 portant biomarkers such as glucose, hydrogen peroxide and cancer biomarkers and highlight the advanta

40 of multi-valent CNPs in presence of hydrogen peroxide and demonstrate an enzyme-free CNP-based biosen

41 nce reaction occurred with luminol, hydrogen peroxide and HRP enzyme, and the emission of light from

42 H oxidase, as well as the amount of hydrogen peroxide and nitrotyrosine.

43 e resistant cells more sensitive to hydrogen peroxide and nutrient starvation.

44 thesis: oxidative stress induced by hydrogen peroxide and nutrient stresses caused by amino acid or g

45 activity of red blood cells towards hydrogen peroxide and on surface-induced haemolysis.

46 oxide, which acts as a precursor of hydrogen peroxide and other reactive oxygen species that are gene

47 converts superoxide into less toxic hydrogen peroxide and oxygen, functions in the gustatory neuron A

48 nt for Spatzle ligand downstream of hydrogen peroxide and protease function, both of which are known

49 g thiol depletion and generation of hydrogen peroxide and superoxide, but none of them can fully expl

50 interaction is strongly induced by hydrogen peroxide and that DPP3 is required for timely NRF2 induc

51 res could be used as a carrier for carbamide peroxide and that the amount of spheres did not affect t

52 evice capable of the measurement of hydrogen peroxide and total cholesterol.

53 roxidase, cause lethal accumulation of lipid peroxides and induce ferroptotic cell death.

54 PX4), which catalyzes the reduction of lipid peroxides and is a target of ferroptosis inducers, such

55 eine-containing enzyme that dissipates lipid peroxides and thereby prevents the iron-mediated reactio

56 idant enzymes that rapidly detoxify damaging peroxides and/or affect signal transduction and, thus, h

57 es (e.g., hypochlorite, persulfate, hydrogen peroxide) and subsequently with diverse dissolved organi

58 son rotenone, amyloid beta-peptide, hydrogen peroxide, and high levels of glutamate.

59 ctivate H2O2 to form pools of hydroperoxide, peroxide, and superoxide intermediates.

60 a radical couple that recombines either as a peroxide anion 7' to afford the aldehyde-to-carboxylic a

61 se 1 macrophages and that restoring hydrogen peroxide antioxidant defenses suppressed this effect.

62 TAML activators of peroxides are iron(III) complexes.

63 e treated with potassium cyanide or hydrogen peroxide as controls, and epidermal growth factor (EGF)

64 orption and after regeneration with hydrogen peroxide at 20 degrees C.

65 th irreversible reduction of oxygen to bound peroxide at cytochrome c oxidase determining the net flu

66 transients produced by oxidation of hydrogen peroxide at one IrOx NP provided information about the o

67 ica with liquid-phase hydrogen and deuterium peroxides at multiple pH values.

68 id and reversible formation of a pentavalent peroxide ate complex, prior to rate-limiting aryl migrat

69 ed strong evidence pointing towards hydrogen peroxide being the primary current source, confirming th

70 e to Mn during anaerobiosis or when hydrogen peroxide biogenesis is significantly reduced.

71 mbling the conditions of industrial hydrogen peroxide bleaching (P stage).

72 fects associated with hydrogen and carbamide peroxide bleaching.

73 ential generation of superoxide and hydrogen peroxide by AAO4 and the induction of AAO4 expression by

74 aces of oxygen which can generate a reactive peroxide by reaction with [KOt-Bu]4 as indicated by dens

75 odegradation of toxic superoxide to hydrogen peroxide by superoxide dismutase.

76 of pnGFP toward peroxynitrite over hydrogen peroxide by using site-directed mutagenesis, X-ray cryst

77 ssay, we show that within a minute, hydrogen peroxide causes accumulation of Pol II near promoters an

78 ed unadulterated and formaldehyde-, hydrogen peroxide-, citrate-, hydroxide- and starch-adulterated s

79 initial reaction product is a highly labile peroxide complex cis-[Pd(IPr)2(eta(2)-O2)].

80 (ESI-MS) suggested 2 to be a Mn(II) Mn(III) -peroxide complex.

81 ant in the VCs, suggesting active removal of peroxide compounds at DCM.

82 successfully used to determine the hydrogen peroxide concentration in real-time analyses.

83 y, this detector was used to detect hydrogen peroxide concentrations in commercial hair dye products,

84 or over 2 weeks and occurred most rapidly in peroxide-containing "whitening" toothpastes, followed by

85 The hydrogen peroxide content and antimicrobial activity of late harv

86 ontent, antimicrobial activity, and hydrogen peroxide content were selected in current work as the cr

87 peroxide which, after conversion to hydrogen peroxide, contributes to cellular membrane depolarizatio

88 The hydrogen peroxide detection limit of the flexible MSM photodetect

89 ce, coupled with the fact that NNT regulates peroxide detoxification, it was hypothesized that 6J mic

90 Comparisons between current hydrogen peroxide diffusion studies and previously published work

91 but not prostate cancer cells with hydrogen peroxide directly inhibited mmp-3 promoter activity with

92 sily generated from acetic acid and hydrogen peroxide, dramatically increases the effectiveness of di

93 ochrome P450 OleTJE (CYP152L1) is a hydrogen peroxide-driven oxidase that catalyzes oxidative decarbo

94 ble quantification of intracellular hydrogen peroxide during cancer therapy constitutes an unexplored

95 printed electrochemical sensors for hydrogen peroxide electroreduction integrated with printed electr

96 and oxidative phosphorylation using hydrogen peroxide, employing the phasor approach required fewer i

97 chlorite, 70% isopropyl alcohol, 3% hydrogen peroxide, ethyl alcohol, water immersion, ultraviolet li

98 Given that increased peroxide exposure drives insulin resistance, coupled wit

99 ked down for GIMAP6, treatment with hydrogen peroxide, FasL, or okadaic acid significantly increased

100 ed hydroxyl radical production from hydrogen peroxide (Fenton's reaction) and subsequent aqueous-phas

101 Hydrogen-bearing iron peroxide (FeO2Hx) in the pyrite-type crystal structure w

102 subsequent mediators superoxide and hydrogen peroxide for pulmonary oxygen sensing and signaling.

103 The peroxide formation in the emulsions, containing tailored

104 lectron transfer governed the suppression of peroxide formation instead of plasmon-induced heating th

105 Hydrogen peroxide formation is dramatically suppressed, while the

106 In addition, hydrogen-peroxide formation is identified as a competing process

107 at causes high levels of superoxide and then peroxide formation, which damages DNA and causes hyperac

108 ve cytotoxic effects in cancer cells through peroxide formation.

109 llent yields under transition-metal-free and peroxide-free conditions.

110 light excess (1.5 equiv) of aqueous hydrogen peroxide, from the oxidation of a broad range of olefins

111 y oxygenated molecules that contain multiple peroxide functionalities are readily cationized by the a

112 ence for a feedback mechanism where hydrogen peroxide generated during the oxidation of ascorbic acid

113 ygen consumption and superoxide and hydrogen peroxide generation were measured in 69 infants.

114 compartments resulting in endosomal hydrogen peroxide generation, which suppresses antiviral and humo

115 stresses, subsequently maintaining hydrogen peroxide (H2 O2 ) homeostasis in Arabidopsis.

116 Reducing the levels of hydrogen peroxide (H2 O2 ) in myb36-5 significantly rescues the m

117 h HRP and subsequent treatment with hydrogen peroxide (H2 O2 ) modified the properties of neurotransm

118 2 S until triggered by ROS, such as hydrogen peroxide (H2 O2 ), superoxide (O2(-) ), and peroxynitrit

119 f DNA oxidation [in the presence of hydrogen peroxide (H2O2) and an H2O2/iron (III) chloride (FeCl3)

120 sites were tested for biosensing of hydrogen peroxide (H2O2) and as supercapacitor electrode material

121 h superoxide (O2(-*) or HOO(*)) and hydrogen peroxide (H2O2) and leads to the same set of products (3

122 g intermediate by-products, such as hydrogen peroxide (H2O2) and other reactive oxygen species (ROS),

123 oxidation of styrenes using aqueous hydrogen peroxide (H2O2) and the cationic palladium(II) compound,

124 lso generates superoxide (O2()) and hydrogen peroxide (H2O2) as bona fide products in reactions invol

125 tosan (CH), salicylic acid (SA) and hydrogen peroxide (H2O2) at different concentrations on the antin

126 dge (PS) at the anode and producing hydrogen peroxide (H2O2) at the cathode.

127 Hydrogen peroxide (H2O2) elicits ROS that induces skin aging thro

128 tes of oxygen consumption (VO2) and hydrogen peroxide (H2O2) emission as a function of PCoA concentra

129 he oxidation of cysteine (CSH) with hydrogen peroxide (H2O2) enzymatically generated by alcohol oxida

130 ng of a cathodic cell that produced hydrogen peroxide (H2O2) followed by an ultraviolet (UV) irradiat

131 Electrochemical production of hydrogen peroxide (H2O2) from water oxidation could provide a ver

132 We conceived that extracellular hydrogen peroxide (H2O2) generated by Duox diffuses through the t

133 ethod for noninvasively quantifying hydrogen peroxide (H2O2) in aqueous solutions based on chemical e

134 investigate the role of apoplastic hydrogen peroxide (H2O2) in gymnosperm phenolic metabolism, an ex

135 Ps-OA) is reported for detection of hydrogen peroxide (H2O2) in wastewater samples.

136 Hydrogen peroxide (H2O2) is a highly relevant metabolite in many

137 Hydrogen peroxide (H2O2) is an endogenous molecule that plays sev

138 Hydrogen peroxide (H2O2) is an important signaling molecule in ca

139 Nonradical species such as hydrogen peroxide (H2O2) or singlet molecular oxygen, rather than

140 Hydrogen peroxide (H2O2) plays an important role in redox signall

141 One proposed signal is hydrogen peroxide (H2O2) produced by chloroplasts in a light-depe

142 se has been quantified by measuring hydrogen peroxide (H2O2) reduction by chronoamperometry at -0.35V

143 RATIONALE: Hydrogen peroxide (H2O2) regulates vascular tone in the human mic

144 to NO2(-) and NO3(-) in an alkaline hydrogen peroxide (H2O2) solution.

145 was developed for the detection of hydrogen peroxide (H2O2) using a reduced graphene oxide-nafion@si

146 ch catalyzes the reduction of O2 to hydrogen peroxide (H2O2), has been implicated in the cardiac and

147 that CS-generated ROS, particularly hydrogen peroxide (H2O2), impaired adenosine stimulated wound rep

148 photolysis of nitrous acid (HONO), hydrogen peroxide (H2O2), ozone (O3), formaldehyde (HCHO), and ac

149 2) as an oxidant in the presence of hydrogen peroxide (H2O2), we demonstrated that the resulting meth

150 thelial cells produce extracellular hydrogen peroxide (H2O2), which acts as a potent signaling molecu

151 high glucose induced production of hydrogen peroxide (H2O2), which down regulated silent information

152 tly despite an apparent decrease of hydrogen peroxide (H2O2), which was previously shown to be a majo

153 Hydrogen-peroxide (H2O2)-induced growth of small-sized gold nanop

154 intestinal Caco-2 cells exposed to hydrogen peroxide (H2O2)-induced oxidative stress.

155 n 1 (NRX1) targets enzymes of major hydrogen peroxide (H2O2)-scavenging pathways, including catalases

156 ensitive and selective detection of hydrogen peroxide (H2O2).

157 was exacerbated upon treatment with hydrogen peroxide (H2O2).

158 with 3,3-diaminobenzidine (DAB) and hydrogen peroxide (H2O2).

159 ation of a trigger by activation by hydrogen peroxide (H2O2).

160 ins of life, dissipates the oxidant hydrogen peroxide (H2O2).

161 lization processes applying gaseous hydrogen peroxide (H2O2).

162 acterial components, defensin-1 and hydrogen peroxide (H2O2).

163 ROS) [hydroxyl radicals ((*)OH) and hydrogen peroxide (H2O2)] catalyzed by ambient particulate matter

164 However, measuring ROS (hydrogen peroxide, H2O2) content in vivo is now possible using th

165 dissociation energies (BDE) such as dicumyl peroxide, heptafluoroisopropyl iodide and diphenyl disul

166 c substances, such as formaldehyde, hydrogen peroxide, hypochlorite, dichromate, salicylic acid, mela

167 ted to be a significant degrader of hydrogen peroxide in anoxic Escherichia coli Intriguingly, ccp tr

168 d by light (405 nm) as well as with hydrogen peroxide in aqueous phosphate buffer.

169 ed to an increased concentration of hydrogen peroxide in bacterially infected glutathione peroxidase

170 ive quantification of intracellular hydrogen peroxide in Cisplatin-treated human renal HK-2 cells.

171 e, CF3CO, MeSO2, CF3SO2) react with hydrogen peroxide in MeOH, THF, MeCN or AcOH to form the correspo

172 indings also demonstrate a role for hydrogen peroxide in the mechanisms tightly regulating NOD-like r

173 that increase levels of endogenous hydrogen peroxide in the yeast Saccharomyces cerevisiae promote s

174 composition, free fatty acid (FFA) content, peroxide index, thermal properties, melting point, consi

175 riming protected cultured DPCs from hydrogen-peroxide-induced cell death and increased the number of

176 viability of 82.90+/-0.78% against hydrogen peroxide-induced oxidative stress on EA.hy926, and was c

177 ogenesis and to protect ECs against hydrogen peroxide-induced oxidative stress.

178 The coregulator hydrogen peroxide-inducible clone 5 (Hic-5) is required for gluco

179 ce of suppressing the formation of undesired peroxide intermediate through plasmonic effects, in whic

180 ature to suppress the formation of undesired peroxide intermediate.

181 O-O bond formation, oxidative activation of peroxide intermediates, and O2 evolution.

182 units; P < 0.05), and released more hydrogen peroxide into the supernatant after hyperoxia exposure (

183 ical methods to accurately quantify hydrogen peroxide is of great interest.

184 ndrial production of superoxide and hydrogen peroxide is potentially important in cell signaling and

185 s at low concentrations-as with the enzymes, peroxide is typically activated with near-quantitative e

186 nstrate that the oxidized sterol, ergosterol peroxide, is necessary and sufficient for Vms1 localizat

187 residues (Cys(53) and Cys(397)) by hydrogen peroxide (k = 17.3 +/- 1.3 m(-1) s(-1) at pH 7.4 and 25

188 ter, calculated as Antioxidant Capacity (AC)/Peroxide Level ratio, assessed here for the first time.

189 ity with enhancing urinary and cardiac lipid peroxide levels, compared to wild type and TRPC3-deficie

190 tering the inputs of irradiation (light) and peroxide (matter) into the concomitant outputs fluoresce

191 and ones involved in detoxification of lipid peroxide-mediated oxidative stress to be different in su

192 ack of NNT activity in Nnt(-/-) mice impairs peroxide metabolism in intact mitochondria.

193 important in coupling intracellular hydrogen peroxide metabolism to glutathione oxidation, and (3) DH

194 and color properties of HA and that hydrogen peroxide might whiten HA-AAAs by oxidizing the benzene r

195 well as the desired reaction product sodium peroxide (Na2O2).

196 study of a single crystal containing uranyl peroxide nanoclusters is reported for pyrophosphate-func

197 on of anilines in the presence of a hydrogen peroxide/O2 system by wavelengths that overlap only with

198 rast, no detectable changes (P>.05) in lipid peroxide occurred within 2h except for samples with 1mg/

199 ar concentrations of superoxide and hydrogen peroxide on irradiation with simulated sunlight in a man

200 The persistent peroxides on the powder further explain previously obser

201 ones (HQ), peroxynitrites (PN), and hydrogen peroxide, on their ability to induce unfolded protein re

202 water) to a 2e(-)/2H(+) process (to hydrogen peroxide) only by increasing the temperature from -50 to

203 .2 eV above lattice oxygen are designated as peroxides (OOH/H2O2) and H2O2 dissolved in water, respec

204 ed from homolytic dissociation of disulfides/peroxides or halide-atom transfer from alkyl halides.

205 T. brucei brucei cells exposed to peroxides or thiol-binding agents were also sensitized t

206 g reactive oxygen species (ROS) from oxygen, peroxides, or ozone.

207 oxin (AhpC) to detoxify ROS such as hydrogen peroxide, organic hydroperoxide, and peroxynitrite.

208 d 52% in the presence of Ca(2+) and hydrogen peroxide over unmodified proteins.

209 In water, they catalyze peroxide oxidation of a broad spectrum of compounds, man

210 mental area under the curve (iAUC) for total peroxide oxidative status after acai consumption relativ

211 nts, including sodium hypochlorite, hydrogen peroxide, ozone and sodium periodate, are described in t

212 gical effects of XO were due to the hydrogen peroxide produced by the enzyme.

213 icient, flat-plate, dual-chambered microbial peroxide producing cell (MPPC) as an anaerobic energy-co

214 The reason is that the protonated peroxide product is always very high in energy.

215 ally and experimentally investigate hydrogen peroxide production activity trends for a range of metal

216 reased residence times and an absence of the peroxide production seen with previous substrate site in

217 ging clean energy reactions such as hydrogen peroxide production, carbon dioxide reduction, and nitro

218 the mitochondria, where it promotes hydrogen peroxide production.

219 ctivity is compromised and the production of peroxide radicals is increased.

220 Hydrogen peroxide reduced thrombospondin 2 (an MMP-3 suppressor)

221 nium-binding proteins putatively involved in peroxide reduction from gammaproteobacteria were abundan

222 oxygen reduction reaction (ORR) or hydrogen peroxide reduction on platinum, has been investigated.

223 ticles, which subsequently catalyze hydrogen peroxide reduction to detect PSA and PSMA.

224 rtant role in the detoxification of hydrogen peroxide released either during photorespiration or as a

225 plex genetic and molecular basis of hydrogen peroxide resistance, we show two examples where multiple

226 our-fold more hydroxyl radicals and hydrogen peroxide, respectively, than TiO2 at 160 mmHg.

227 on and susceptibility to killing by hydrogen peroxide, respectively.

228 d HSC are also important sources of hydrogen peroxide resulting from the activation of NADPH oxidase

229 occurs by a three-step mechanism, where the peroxide reversibly associates with the enzyme; then it

230 The acid, peroxide, saponification and iodine values and unsaponif

231 O2 scavenger, mitochondria-targeted hydrogen peroxide scavenger ebselen, reduced Sirt3 S-glutathionyl

232 increase in DPPH (48.32-59.62%) and hydrogen peroxide scavenging activities (35.44-63.07mM-Trolox/g)

233 Peroxide sensing is essential for bacterial survival dur

234 ystem through chemiluminescence for hydrogen peroxide sensing.

235 Here the authors use a fluorescent hydrogen peroxide sensor to provide evidence that H2O2 is transfe

236 cting duct cells in the presence of hydrogen peroxide showed increased activity of p53 at Serine 15.

237 the induction of AAO4 expression by hydrogen peroxide shown here suggest a self-amplification mechani

238 the CYP121-catalyzed reaction, we tested the peroxide shunt pathway by using rapid kinetic techniques

239 ere carried out at -0.20V by adding hydrogen peroxide solution onto the electrode surface in the pres

240 res (lambdamax =460, 610 nm) typical of a Mn-peroxide species and a 29-line EPR signal typical of a M

241 wed that the leptospiral PerR belongs to the peroxide stimulon in pathogenic species and is involved

242 ski7 hbs1 mutants are sensitive to hydrogen peroxide stress and accumulate an NSD substrate.

243 individual inactivation of newly identified peroxide stress genes had modest or no obvious consequen

244 Peroxide stress regulators (PerRs) are homodimeric trans

245 ely, our findings unraveled the scope of the peroxide stress regulon and expand the repertoire of oxi

246 1 coordinates transcriptional changes during peroxide stress that modify global metabolism and facili

247 ster females but not males adapt to hydrogen peroxide stress, whereas males but not females adapt to

248 ted conditions as well as following hydrogen peroxide stress.

249 for PerR dimeric assembly and for regulating peroxide stress.

250 metabolic pathways controlled by Spx during peroxide stress.

251 sequencing to uncover the scope of the H2O2 (peroxide)-stress regulon and to further explore the sign

252 on of oxidation in the Fc region of hydrogen peroxide-stressed Rituximab, using a single, commerciall

253 wing treatment with antibiotics and hydrogen peroxide, supporting the importance of the protein-prote

254 nd produces proinflammatory lipids and lipid peroxides that exacerbate beta-cell dysfunction and macr

255 reby prevents the iron-mediated reactions of peroxides that induce ferroptotic cell death.

256 The flux of hydrogen peroxide through 1-mm discs of bovine enamel was measure

257 easing superoxide radical anion and hydrogen peroxide through a mechanism that promotes activation of

258 of spheres did not affect the diffusivity of peroxide through the enamel discs.

259 re produced from the ORR byproduct, hydrogen peroxide, through a 1 electron reduction pathway (H2O2 +

260 d its derived intermediates such as hydrogen peroxide to afford a variety of metal-oxygen intermediat

261 atments N-acetylcysteine, EDTA, and hydrogen peroxide to disrupt in vitro biofilms and kill equine re

262 catalyze reaction of o-phenylenediamine with peroxide to form 2,3-diaminophenazone was evaluated.

263 icles in catalytic decomposition of hydrogen peroxide to oxygen and water.

264 tone diboration, react rapidly with hydrogen peroxide to release alcohols, aldehydes, and ketones thr

265 ikely competitive pathway is the cleavage of peroxide to the alpha-oxy radical (likely catalyzed by C

266 ransformation but also as a reductant of the peroxide to the corresponding alpha-hydroxy ketone.

267 Consistent with this mode of action, peroxide treatment of parental HeLa cells elevated phosp

268 sented study, DHNQ was treated with hydrogen peroxide under alkaline conditions at pH 10, resembling

269 saponification value (183.8mg KOH/g of oil), peroxide value (8.2meq/kg of oil) and refractive index (

270 ork explored, for the first time, monitoring peroxide value (PV) of omega-3 rich algae oil using ATR-

271 spectra, density, refractive index, acidity, peroxide value (PV), p-anisidine index (p-An), oxidation

272 water-in-oil emulsion was evaluated through peroxide value (PV), p-anisidine, TBARs inhibition and o

273 ith voluntary industry-set maximum limits on Peroxide Value (PV), para-Anisidine Value (p-AV), and TO

274 te their physicochemical properties, such as peroxide value (PV), volatile compounds (VOCs), particle

275 of these emulsions, as shown by their lowest peroxide value and concentration of volatiles such as 1-

276 acidity, syneresis, fatty acid composition, peroxide value as well as sensory tests were investigate

277 ned (e.g. values of r=0.320 and r=0.793 with peroxide value for butter and back-fat, respectively, an

278 e control oil, the conjugated dienes and the peroxide value observed were respectively of 41.8+/-0.8

279 Iodine and peroxide value were also affected by processing.

280 Lipid oxidation was tracked by measuring the peroxide value, acidity, conjugated dienes and trienes.

281 nificant reduction in acidity, syneresis and peroxide value.

282 observed (e.g. TBA value correlated with the peroxide value: r=0.466 for butter and r=0.898 for back-

283 exture of the samples, coupled with the high peroxide values (13-539meqO2/kgfat) measured at the end

284 ndary oxidation in emulsions as evidenced by peroxide values (PVs) and secondary volatile oxidation p

285 of TPB extract/kg of TTB revealed the lowest peroxide values at all the determination intervals.

286 ity to capture free radicals, and the lowest peroxide values, conjugated dienes and conjugated triene

287 thing, decolonization for MRSA, and hydrogen peroxide vaporizer for MDR-AB) were used less commonly.

288 t 98% for H2O2 production.Producing hydrogen peroxide via electrochemical oxidation of water is an at

289 n source of increased production of hydrogen peroxide was a block in respiratory chain and diversion

290 Hydrogen peroxide was also formed in 10% yield consistent with a

291 , a primary scavenger of endogenous hydrogen peroxide was also identified.

292 Furthermore, hydrogen peroxide was characterized as the central modulator for

293 We observed that hydrogen peroxide was generated during thermal treatment (up to 7

294 electrochemical water oxidation to hydrogen peroxide was observed, albeit with low (15%) Faradaic yi

295 acid, maximum levels of measurable hydrogen peroxide were achieved with an initial concentration of

296 lling depended on the production of hydrogen peroxide, which required increased supply of NADH for re

297 theoretical calculations, we find that iron peroxide with a varying amount of hydrogen has a high de

298 We replaced hydrogen peroxide with t-butyl hydroperoxide and found that, alth

299 d cause a negative effect (i.e., increase of peroxide yield), in which the hot electron transfer of A

300 Analogous reactions with di-tert-butyl peroxide yielded [Cp*2Fe][(mu-HO)(B(C6F5)3)2] 4 with iso