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1 are one cause of a severe, recessive form of persistent hyperinsulinemic hypoglycemia of infancy.
2 l, has been demonstrated to be causative for persistent hyperinsulinemic hypoglycemia of infancy.
3 urea type 1 receptor (Sur1) or Kir6.2, cause persistent hyperinsulinemic hypoglycemia of infancy.
4 ly rectifying potassium channel Kir6.2 cause persistent hyperinsulinemic hypoglycemia of infancy.
5 ine that we have derived from a patient with persistent hyperinsulinemic hypoglycemia of infancy.
7 ls equivalent to those seen in patients with persistent hyperinsulinemic hypoglycemia of infancy, but
8 ceptor (SUR) of an individual diagnosed with persistent hyperinsulinemic hypoglycemia of infancy gene
9 ndent SUR1 mutations involved in the disease persistent hyperinsulinemic hypoglycemia of infancy have
11 e human glk gene associated with the disease persistent hyperinsulinemic hypoglycemia of infancy (PHH
12 including adult-onset (type 2) diabetes and persistent hyperinsulinemic hypoglycemia of infancy (PHH
13 he Kir6.2 and SUR1 genes are associated with persistent hyperinsulinemic hypoglycemia of infancy (PHH
14 Persistent hyperinsulinemic hypoglycemia of infancy (PHH
15 er the SUR1 or Kir6.2 channel subunit causes persistent hyperinsulinemic hypoglycemia of infancy (PHH
16 NES2Y cells were derived from a patient with persistent hyperinsulinemic hypoglycemia of infancy; the
17 Whereas persistent hyperinsulinemic hypoglycemia of infancy unde
18 A116P and V187D, previously correlated with persistent hyperinsulinemic hypoglycemia of infancy, wer
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